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PLoS pathogens, ISSN 1553-7374, 2013, Volume 9, Issue 11, p. e1003773
Interferons (IFNs) are a group of cytokines with a well-established antiviral function. They can be induced by viral infection, are secreted and bind to... 
CELLS | DEFENSE | VIRUS-INFECTION | DISTINCT | RIG-I | MICROBIOLOGY | IRF-7 | ANTIVIRAL RESPONSE | VIROLOGY | GENE | SIGNALING PATHWAY | ADAPTER PROTEIN | PARASITOLOGY | Epithelial Cells - metabolism | Influenza A virus - genetics | Respiratory Mucosa - virology | Interferon Regulatory Factor-7 - genetics | Interferon Type I - immunology | Interferon Regulatory Factor-3 - genetics | Interleukins - metabolism | Orthomyxoviridae Infections - genetics | Respiratory Mucosa - pathology | Interleukins - genetics | Interleukins - immunology | Respiratory Mucosa - immunology | Adaptor Proteins, Signal Transducing - immunology | Interferon Type I - metabolism | Influenza A virus - immunology | Membrane Proteins - metabolism | Interferon Regulatory Factor-3 - immunology | Nerve Tissue Proteins - immunology | Membrane Proteins - genetics | Orthomyxoviridae Infections - metabolism | Epithelial Cells - pathology | Membrane Proteins - immunology | Interferon Regulatory Factor-7 - immunology | Interferon Regulatory Factor-7 - metabolism | Nerve Tissue Proteins - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Influenza A virus - metabolism | Epithelial Cells - immunology | Epithelial Cells - virology | Adaptor Proteins, Signal Transducing - genetics | Interferon Regulatory Factor-3 - metabolism | Interferon Type I - genetics | Mice | Respiratory Mucosa - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Orthomyxoviridae Infections - immunology | Epithelial cells | Influenza | Physiological aspects | Host-parasite relationships | Interferon | Genetic aspects | Genetic transcription | Research | Health aspects | Airway (Medicine) | Interleukins/metabolism | Nerve Tissue Proteins/immunology | Orthomyxoviridae Infections/genetics | Orthomyxoviridae Infections/metabolism | Membrane Proteins/genetics | Adaptor Proteins, Signal Transducing/genetics | Interferon Regulatory Factor-7/genetics | Interleukins/immunology | Membrane Proteins/immunology | Life Sciences | Orthomyxoviridae Infections/immunology | Influenza A virus/genetics | Nerve Tissue Proteins/metabolism | Respiratory Mucosa/immunology | Immunology | Interferon Type I/immunology | Epithelial Cells/immunology | Epithelial Cells/virology | Interferon Regulatory Factor-7/immunology | Epithelial Cells/metabolism | Interferon Regulatory Factor-3/immunology | Respiratory Mucosa/pathology | Influenza A virus/immunology | Interleukins/genetics | Interferon Regulatory Factor-3/genetics | Influenza A virus/metabolism | Adaptor Proteins, Signal Transducing/metabolism | Epithelial Cells/pathology | Adaptor Proteins, Signal Transducing/immunology | Interferon Type I/genetics | Interferon Regulatory Factor-3/metabolism | Membrane Proteins/metabolism | Nerve Tissue Proteins/genetics | Interferon Regulatory Factor-7/metabolism | Interferon Type I/metabolism | Respiratory Mucosa/metabolism | Respiratory Mucosa/virology | Cytokines | Genes | Rodents | Genomics | Genomes | Kinases | Experiments | Viral infections
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 08/2009, Volume 157, Issue 2, pp. 316 - 324
Summary There are increased numbers of activated T lymphocytes in the bronchial mucosa of stable chronic obstructive pulmonary disease (COPD) patients. T helper type 17 (Th17... 
bronchial biopsies | pathology | autoimmunity | neutrophils | emphysema | Autoimmunity | Pathology | Bronchial biopsies | Emphysema | Neutrophils | DISTINCT | IMMUNOLOGY | IL-23 | INTERLEUKIN-17 | INFLAMMATION | IL-17 | GENE-EXPRESSION | HOST-DEFENSE | SMOOTH-MUSCLE-CELLS | DIFFERENTIATION | TH17 CELLS | Immunohistochemistry | Smoking - adverse effects | Humans | Middle Aged | Interleukin-17 - immunology | RNA, Messenger - analysis | Male | DNA Primers - genetics | Bronchi - immunology | Case-Control Studies | Receptors, Retinoic Acid - genetics | Interleukin-23 - genetics | Interleukins - genetics | Receptors, Thyroid Hormone - genetics | T-Lymphocytes, Helper-Inducer - immunology | Interleukins - immunology | Receptors, Retinoic Acid - immunology | Polymerase Chain Reaction | Statistics, Nonparametric | Female | Receptors, Thyroid Hormone - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 | Interleukin-23 - immunology | Analysis of Variance | Mucous Membrane - immunology | Aged | Respiratory Function Tests | Pulmonary Disease, Chronic Obstructive - immunology | Lung diseases, Obstructive | Messenger RNA | Cytokines | Interleukins | Automobile driving | T cells | Motor vehicle driving | CD8 antigen | Mucosa | Helper cells | Interleukin 22 | Interleukin 23 | Inflammation | Lymphocytes T | Epithelium | Gene expression | Endothelial cells | Orphan receptors | CD4 antigen | Polymerase chain reaction | Biopsy | Interleukin 21 | Chronic obstructive pulmonary disease | Translational Studies
Journal Article
PLoS pathogens, ISSN 1553-7374, 2010, Volume 6, Issue 5, p. e1000902
Despite recent advances in our understanding of the pathogenesis of attaching and effacing (A/E) Escherichia coli infections, the mechanisms by which the host... 
DISRUPTION IN-VIVO | YERSINIA-ENTEROCOLITICA | EFFACING PATHOGEN | PURIFIED RABBIT | MICROBIOLOGY | ENTAMOEBA-HISTOLYTICA | ENTERICA SEROTYPE TYPHIMURIUM | VIROLOGY | ENTEROPATHOGENIC ESCHERICHIA-COLI | HUMAN INTESTINAL MUCIN | HOST-DEFENSE | CITROBACTER-RODENTIUM INFECTION | PARASITOLOGY | Mucin-2 - genetics | Mucin-2 - metabolism | Intestinal Mucosa - metabolism | Epithelial Cells - metabolism | Bacterial Adhesion - immunology | Enterobacteriaceae Infections - metabolism | Mice, Inbred C57BL | Bacterial Translocation - immunology | Intestinal Mucosa - microbiology | Enterobacteriaceae Infections - immunology | Animals | Colitis - microbiology | Mucin-2 - immunology | Intestinal Mucosa - immunology | Mice, Mutant Strains | Epithelial Cells - immunology | Epithelial Cells - microbiology | Colitis - metabolism | Mice | Citrobacter rodentium | Colitis - immunology | Escherichia coli | Mucins | Physiological aspects | Causes of | Development and progression | Genetic aspects | Research | Colitis | Translocation | Pathogens | Commensals | Secretion | Mortality | Mucosa | Oral administration | Diarrhea | Mucus | Epithelium | Infection | Large intestine | mucin | Fluorescence in situ hybridization | Colon | Feces | Intestinal microflora | Digestive tract | Colonization | Inflammatory bowel disease | Medical research | Bacterial infections | Microbiology | E coli | Rodents | Bacteria | Experiments | Bars
Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 2013, Volume 131, Issue 2, pp. 549 - 561
... . In contrast to this general concept, we hypothesized that innate immunity of the upper and lower airways is respectively distinctive, because the immunologic conditions of the nasal and lung mucosa... 
Allergy and Immunology | pathogen associated molecular pattern | allergic asthma | innate immunity | house dust mite | Allergic rhinitis | reactive oxygen species | epithelium | β-glucans | dual oxidase 2 | Toll-like receptor | ACTIVATION | beta-glucans | DENDRITIC CELLS | IMMUNOLOGY | DECTIN-1 | RHINITIS | TOLL-LIKE RECEPTOR-2 | RESPONSES | EPITHELIAL-CELLS | ALLERGY | INFLAMMATION | LIPID RAFTS | ASTHMA | Asthma - metabolism | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Rhinitis, Allergic | NADPH Oxidases - metabolism | Hypersensitivity - immunology | NADPH Oxidases - immunology | Lipopolysaccharides - immunology | Rhinitis, Allergic, Perennial - metabolism | Hypersensitivity - metabolism | Reactive Oxygen Species - immunology | Respiratory Mucosa - immunology | Asthma - immunology | Lung - metabolism | Nasal Mucosa - metabolism | beta-Glucans - immunology | Respiratory System - immunology | Rhinitis, Allergic, Perennial - immunology | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Immunity, Innate - immunology | beta-Glucans - metabolism | Animals | Nasal Mucosa - immunology | Respiratory System - metabolism | Epithelial Cells - immunology | Toll-Like Receptor 2 - immunology | Mice | Respiratory Mucosa - metabolism | Dual Oxidases | Lung - immunology | Pyroglyphidae - immunology | Medical colleges | Asthma | Lipids | Gene expression | Allergies | Immune system
Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 2014, Volume 134, Issue 3, pp. 613 - 621.e7
Journal Article
Trends in Immunology, ISSN 1471-4906, 2011, Volume 32, Issue 9, pp. 402 - 411
House dust mites (HDM; Dermatophagoides sp.) are one of the commonest aeroallergens worldwide and up to 85% of asthmatics are typically HDM allergic.... 
Allergy and Immunology | GROWTH-FACTOR RECEPTOR | STRUCTURAL BIOLOGY | DENDRITIC CELLS | EPITHELIAL-CELLS | INNATE IMMUNE-RESPONSES | IMMUNOTHERAPY | AIRWAY INFLAMMATION | T-CELL | NLRP3 INFLAMMASOME | IMMUNOLOGY | THYMIC STROMAL LYMPHOPOIETIN | Allergens - adverse effects | Immunotherapy - methods | Epithelial Cells - metabolism | Bronchial Hyperreactivity - immunology | Dendritic Cells - immunology | Humans | Adrenal Cortex Hormones - therapeutic use | Bronchi - immunology | Antigens, Dermatophagoides - immunology | Allergens - immunology | Respiratory Mucosa - immunology | Asthma - immunology | Toll-Like Receptors - metabolism | beta-Glucans - immunology | Bronchi - pathology | Dendritic Cells - metabolism | Disease Models, Animal | Spores, Fungal - immunology | Bronchial Hyperreactivity - therapy | Respiratory Mucosa - cytology | Toll-Like Receptors - immunology | Bronchial Hyperreactivity - pathology | Animals | Epithelial Cells - immunology | Asthma - therapy | Mice | Respiratory Mucosa - metabolism | Chitin - immunology | Asthma - pathology | Cytokines - biosynthesis | Adrenal Cortex Hormones - administration & dosage | Pyroglyphidae - immunology | Allergens | Analysis | Asthma | Chitinase | Antigens | Enzymes | Disease | Pathogenesis | Lipids | Smooth muscle | Allergies | Molecular weight | Proteins | Airway management | Proteases | Collagen | Ligands | Chemokines | Immune system | Chronic illnesses
Journal Article
PLoS pathogens, ISSN 1553-7366, 06/2013, Volume 9, Issue 6, p. e1003407
Although vitamin D deficiency is a common feature among patients presenting with active tuberculosis, the full scope of vitamin D action during Mycobacterium... 
AIM2 INFLAMMASOME | INFLAMMASOME ACTIVATION | D DEFICIENCY | INNATE | MICROBIOLOGY | BETA-DEFENSINS | CUTTING EDGE | VIROLOGY | HOST-DEFENSE | 1,25-DIHYDROXYVITAMIN D-3 | IL-1-BETA | D-RECEPTOR | PARASITOLOGY | Receptors, Interleukin-1 Type I - immunology | NLR Family, Pyrin Domain-Containing 3 Protein | Coculture Techniques | Humans | Mycobacterium tuberculosis - immunology | Caspase 1 - metabolism | Male | Interleukin-1beta - genetics | Paracrine Communication - immunology | Vitamins - pharmacology | Respiratory Mucosa - microbiology | Respiratory Mucosa - immunology | Carrier Proteins - immunology | Tuberculosis - pathology | Paracrine Communication - genetics | Macrophages, Peritoneal - immunology | Calcitriol - pharmacology | Epithelial Cells - microbiology | Mice | Macrophages, Peritoneal - metabolism | beta-Defensins - immunology | Epithelial Cells - metabolism | Caspase 1 - immunology | Respiratory Mucosa - pathology | Receptors, Interleukin-1 Type I - metabolism | Female | Interleukin-1beta - biosynthesis | beta-Defensins - metabolism | Mycobacterium tuberculosis - metabolism | Tuberculosis - metabolism | Cell Line | beta-Defensins - genetics | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Interleukin-1beta - immunology | Immunity, Innate | Gene Expression Regulation - drug effects | Tuberculosis - immunology | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Macrophages, Peritoneal - microbiology | Tuberculosis - genetics | Caspase 1 - genetics | Epithelial Cells - immunology | Receptors, Interleukin-1 Type I - genetics | Respiratory Mucosa - metabolism | beta-Defensins - biosynthesis | Mycobacterium tuberculosis - genetics
Journal Article