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Gaceta médica de México, ISSN 0016-3813, 03/1970, Volume 100, Issue 3, p. 279
Journal Article
PLoS pathogens, ISSN 1553-7374, 2013, Volume 9, Issue 11, p. e1003773
Interferons (IFNs) are a group of cytokines with a well-established antiviral function. They can be induced by viral infection, are secreted and bind to... 
CELLS | DEFENSE | VIRUS-INFECTION | DISTINCT | RIG-I | MICROBIOLOGY | IRF-7 | ANTIVIRAL RESPONSE | VIROLOGY | GENE | SIGNALING PATHWAY | ADAPTER PROTEIN | PARASITOLOGY | Epithelial Cells - metabolism | Influenza A virus - genetics | Respiratory Mucosa - virology | Interferon Regulatory Factor-7 - genetics | Interferon Type I - immunology | Interferon Regulatory Factor-3 - genetics | Interleukins - metabolism | Orthomyxoviridae Infections - genetics | Respiratory Mucosa - pathology | Interleukins - genetics | Interleukins - immunology | Respiratory Mucosa - immunology | Adaptor Proteins, Signal Transducing - immunology | Interferon Type I - metabolism | Influenza A virus - immunology | Membrane Proteins - metabolism | Interferon Regulatory Factor-3 - immunology | Nerve Tissue Proteins - immunology | Membrane Proteins - genetics | Orthomyxoviridae Infections - metabolism | Epithelial Cells - pathology | Membrane Proteins - immunology | Interferon Regulatory Factor-7 - immunology | Interferon Regulatory Factor-7 - metabolism | Nerve Tissue Proteins - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Influenza A virus - metabolism | Epithelial Cells - immunology | Epithelial Cells - virology | Adaptor Proteins, Signal Transducing - genetics | Interferon Regulatory Factor-3 - metabolism | Interferon Type I - genetics | Mice | Respiratory Mucosa - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Orthomyxoviridae Infections - immunology | Epithelial cells | Influenza | Physiological aspects | Host-parasite relationships | Interferon | Genetic aspects | Genetic transcription | Research | Health aspects | Airway (Medicine) | Interleukins/metabolism | Nerve Tissue Proteins/immunology | Orthomyxoviridae Infections/genetics | Orthomyxoviridae Infections/metabolism | Membrane Proteins/genetics | Adaptor Proteins, Signal Transducing/genetics | Interferon Regulatory Factor-7/genetics | Interleukins/immunology | Membrane Proteins/immunology | Life Sciences | Orthomyxoviridae Infections/immunology | Influenza A virus/genetics | Nerve Tissue Proteins/metabolism | Respiratory Mucosa/immunology | Immunology | Interferon Type I/immunology | Epithelial Cells/immunology | Epithelial Cells/virology | Interferon Regulatory Factor-7/immunology | Epithelial Cells/metabolism | Interferon Regulatory Factor-3/immunology | Respiratory Mucosa/pathology | Influenza A virus/immunology | Interleukins/genetics | Interferon Regulatory Factor-3/genetics | Influenza A virus/metabolism | Adaptor Proteins, Signal Transducing/metabolism | Epithelial Cells/pathology | Adaptor Proteins, Signal Transducing/immunology | Interferon Type I/genetics | Interferon Regulatory Factor-3/metabolism | Membrane Proteins/metabolism | Nerve Tissue Proteins/genetics | Interferon Regulatory Factor-7/metabolism | Interferon Type I/metabolism | Respiratory Mucosa/metabolism | Respiratory Mucosa/virology | Cytokines | Genes | Rodents | Genomics | Genomes | Kinases | Experiments | Viral infections
Journal Article
Nature cell biology, ISSN 1476-4679, 2017, Volume 19, Issue 6, pp. 698 - 710
textabstractThe IRE1-XBP1 signalling pathway is part of a cellular programme that protects against endoplasmic reticulum (ER) stress, but also controls... 
HOMEOSTASIS | ER-STRESS | IN-VIVO | IRE1 | TRANSCRIPTION FACTOR XBP-1 | ENDOPLASMIC-RETICULUM STRESS | DIFFERENTIATION | MAMMALIAN-CELLS | UNFOLDED-PROTEIN-RESPONSE | TRANSLATIONAL CONTROL | CELL BIOLOGY | Transcription Factor CHOP - genetics | Protein Biosynthesis | Dendritic Cells - pathology | JNK Mitogen-Activated Protein Kinases - metabolism | RNA, Messenger - metabolism | Respiratory Mucosa - pathology | Time Factors | Intestinal Mucosa - enzymology | Membrane Proteins - metabolism | Protein-Serine-Threonine Kinases - metabolism | Signal Transduction | Cell Survival | Membrane Proteins - genetics | RNA, Messenger - genetics | Protein-Serine-Threonine Kinases - genetics | Respiratory Mucosa - enzymology | Activating Transcription Factor 4 - genetics | Genotype | Mice, Transgenic | RNA Stability | Unfolded Protein Response | Phenotype | Animals | Activating Transcription Factor 4 - metabolism | Dendritic Cells - enzymology | X-Box Binding Protein 1 - metabolism | Endoplasmic Reticulum Stress | Transcription Factor CHOP - metabolism | X-Box Binding Protein 1 - genetics | Apoptosis | Intestinal Mucosa - pathology | Regulators | Mucosa | Signal transduction | Alterations | Cell activation | Intestine | Immune system | Stresses | Spleen | Cell survival | Dendritic cells | mRNA turnover | Protein biosynthesis | JNK protein | Ribonucleic acid--RNA | Survival | Stress | Signaling | Lungs | Protein synthesis | Cell death | Decay | Shutdowns | Endonuclease | Endoplasmic reticulum
Journal Article
The American surgeon, ISSN 0003-1348, 1/2016, Volume 82, Issue 1, pp. 1 - 5
Mucosal melanoma represents a distinct minority of disease sites and portends a worse outcome. The ideal treatment and role of adjuvant therapy remains unknown... 
SURGERY | ANORECTAL MELANOMA | OUTCOMES | KIT | Chemotherapy | Lymphatic system | Databases | Surgery | Immunotherapy | Melanoma | Mutation | Vaccines | Radiation therapy | Trends | Skin cancer
Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 2016, Volume 138, Issue 1, pp. 16 - 27
Chronic obstructive pulmonary disease (COPD) is associated with chronic inflammation affecting predominantly the lung parenchyma and peripheral airways that... 
Allergy and Immunology | cytokine | chemokine | autoantibody | nuclear factor κB | macrophage | Inflammation | neutrophil | oxidative stress | PROGENITOR CELLS | EXHALED BREATH CONDENSATE | nuclear factor kappa B | BRONCHIAL-MUCOSA | INNATE LYMPHOID-CELLS | IMMUNOLOGY | COPD OVERLAP SYNDROME | PERIPHERAL AIRWAYS | ALLERGY | ALVEOLAR MACROPHAGES | GROWTH-FACTOR | SMALL-AIRWAY OBSTRUCTION | Leukocytes - pathology | Pulmonary Disease, Chronic Obstructive - diagnosis | Inflammasomes - metabolism | Cytokines - metabolism | Oxidative Stress | Humans | Risk Factors | Pulmonary Disease, Chronic Obstructive - etiology | Inflammation - etiology | Leukocytes - immunology | Respiratory Mucosa - pathology | Inflammation - metabolism | Phenotype | Animals | Respiratory Mucosa - immunology | Inflammation Mediators - metabolism | Inflammation - diagnosis | Pulmonary Disease, Chronic Obstructive - metabolism | Respiratory Mucosa - metabolism | Leukocytes - metabolism | Autoimmunity | Osteoporosis | Lung diseases, Obstructive | Care and treatment | Autoantibodies | Corticosteroids | Comorbidity | T cells | Medical colleges | Endothelial growth factors | Respiratory agents | Superoxide | Macrophages | Endothelium | Disease | Cytokines | Antimicrobial agents | Lymphocytes | DNA methylation | Biomarkers | Air pollution | Chronic obstructive pulmonary disease | Volatile organic compounds--VOCs | Health risk assessment | Nitrogen dioxide | Metabolic disorders
Journal Article
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