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1. Full Text Precision medicine in patients with allergic diseases: Airway diseases and atopic dermatitis—PRACTALL document of the European Academy of Allergy and Clinical Immunology and the American Academy of Allergy, Asthma & Immunology
by Muraro, Antonella, MD and Lemanske, Robert F., MD and Hellings, Peter W., MD and Akdis, Cezmi A., MD and Bieber, Thomas, MD and Casale, Thomas B., MD and Jutel, Marek, MD and Ong, Peck Y., MD and Poulsen, Lars K., PhD and Schmid-Grendelmeier, Peter, MD and Simon, Hans-Uwe, MD and Seys, Sven F., PhD and Agache, Ioana, MD
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 137, Issue 5, pp. 1347 - 1358
In this consensus document we summarize the current knowledge on major asthma, rhinitis, and atopic dermatitis endotypes under the auspices of the PRACTALL... 
Allergy and Immunology | phenotype | allergic skin disease | Precision medicine | allergic asthma | endotype | personalized care | allergic rhinitis | biomarker | Key words Precision medicine | CORTICOSTEROID RESPONSIVENESS | IL-13 EXPRESSION | SURROGATE END-POINTS | SEVERE EOSINOPHILIC ASTHMA | BARRIER FUNCTION | IMMUNOLOGY | CHRONIC RHINOSINUSITIS | ALLERGY | INTERNATIONAL CONSENSUS | DOUBLE-BLIND | GENE-EXPRESSION | EVALUATE SAFETY | Rhinitis - immunology | Dermatitis, Atopic - drug therapy | Europe | Humans | Asthma - immunology | Dermatitis, Atopic - immunology | Societies, Scientific | Asthma - drug therapy | Respiratory System - immunology | Rhinitis - drug therapy | Precision Medicine | Skin - immunology | Food allergy | Care and treatment | Atopic dermatitis | Asthma | Food allergies | Disease | Cytokines | Inflammation | Metabolism | Patients | Blood | Quality of life | Medicine | Genotype & phenotype | Immunology | Biomarkers | Volatile organic compounds--VOCs | Index Medicus | Abridged Index Medicus
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2. Full Text Phenotyping the Immune Response to Trauma: A Multiparametric Systems Immunology Approach
by Seshadri, Anupamaa and Brat, Gabriel A and Yorkgitis, Brian K and Keegan, Joshua and Dolan, James and Salim, Ali and Askari, Reza and Lederer, James A
Critical Care Medicine, ISSN 0090-3493, 09/2017, Volume 45, Issue 9, pp. 1523 - 1530
OBJECTIVE:Trauma induces a complex immune response that requires a systems biology research approach. Here, we used a novel technology, mass cytometry by... 
trauma | mass cytometry | immune phenotype | immunology | cytometry by time-of-flight | MORTALITY | CELLS | BIOLOGY | EXPRESSION | SEVERITY | BURN INJURY | CRITICAL CARE MEDICINE | Intensive Care Units | Humans | Middle Aged | Phagocytosis - immunology | Immunophenotyping | Male | Interleukin-17 - biosynthesis | Respiratory Burst - immunology | Wounds and Injuries - immunology | Injury Severity Score | CD4-Positive T-Lymphocytes - immunology | Flow Cytometry | Time Factors | Leukocytes, Mononuclear - immunology | Chemokine CCL2 - biosynthesis | Adult | Female | Interleukins - biosynthesis | Cytokines - biosynthesis | Interferon-gamma - biosynthesis | Index Medicus | Abridged Index Medicus
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3. Full Text Distinct TLR-mediated pathways regulate house dust mite–induced allergic disease in the upper and lower airways
by Ryu, Ji-Hwan, PhD and Yoo, Jung-Yeon, BS and Kim, Min-Ji, BS and Hwang, Sang-Gyu, MS and Ahn, Kwang Chul, MS and Ryu, Jae-Chan, BS and Choi, Mi-Kyung, PhD and Joo, Jung Hee, PhD and Kim, Chang-Hoon, MD, PhD and Lee, Sang-Nam, PhD and Lee, Won-Jae, PhD and Kim, Jaesang, PhD and Shin, Dong Min, DDS, PhD and Kweon, Mi-Na, PhD and Bae, Yun Soo, PhD and Yoon, Joo-Heon, MD, PhD
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2012, Volume 131, Issue 2, pp. 549 - 561
Background Allergic rhinitis (AR) and asthma are 2 entities of allergic airway diseases that frequently occur together, which is referred to as united airways... 
Allergy and Immunology | pathogen associated molecular pattern | allergic asthma | innate immunity | house dust mite | Allergic rhinitis | reactive oxygen species | epithelium | β-glucans | dual oxidase 2 | Toll-like receptor | ACTIVATION | beta-glucans | DENDRITIC CELLS | IMMUNOLOGY | DECTIN-1 | RHINITIS | TOLL-LIKE RECEPTOR-2 | RESPONSES | EPITHELIAL-CELLS | ALLERGY | INFLAMMATION | LIPID RAFTS | ASTHMA | Asthma - metabolism | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Rhinitis, Allergic | NADPH Oxidases - metabolism | Hypersensitivity - immunology | NADPH Oxidases - immunology | Lipopolysaccharides - immunology | Rhinitis, Allergic, Perennial - metabolism | Hypersensitivity - metabolism | Reactive Oxygen Species - immunology | Respiratory Mucosa - immunology | Asthma - immunology | Lung - metabolism | Nasal Mucosa - metabolism | beta-Glucans - immunology | Respiratory System - immunology | Rhinitis, Allergic, Perennial - immunology | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Immunity, Innate - immunology | beta-Glucans - metabolism | Animals | Nasal Mucosa - immunology | Respiratory System - metabolism | Epithelial Cells - immunology | Toll-Like Receptor 2 - immunology | Mice | Respiratory Mucosa - metabolism | Dual Oxidases | Lung - immunology | Pyroglyphidae - immunology | Medical colleges | Asthma | Lipids | Gene expression | Allergies | Immune system | Index Medicus | Abridged Index Medicus
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4. Full Text The airway microbiome in patients with severe asthma: Associations with disease features and severity
by Huang, Yvonne J., MD and Nariya, Snehal, BS and Harris, Jeffrey M., MD, PhD and Lynch, Susan V., PhD and Choy, David F., BS and Arron, Joseph R., MD, PhD and Boushey, Homer, MD
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 136, Issue 4, pp. 874 - 884
Background Asthma is heterogeneous, and airway dysbiosis is associated with clinical features in patients with mild-to-moderate asthma. Whether similar... 
Allergy and Immunology | lung | Microbiota | inflammation | 16S ribosomal RNA | body mass index | asthma control | steroids | TH2 | 2 | T | CELLS | PHENOTYPES | COMMUNITIES | INDUCTION | IMMUNOLOGY | T(H)2 | MESSENGER-RNA | ALLERGY | DYNAMICS | CHEMOKINE | EXPRESSION | Dysbiosis - drug therapy | Humans | Middle Aged | Male | Respiratory System - microbiology | Tacrolimus Binding Proteins - metabolism | Adrenal Cortex Hormones - therapeutic use | Dysbiosis - microbiology | Eosinophils - immunology | Leukocytes - immunology | Young Adult | Dysbiosis - immunology | Respiratory Mucosa - immunology | Asthma - immunology | Adult | Female | Respiratory System - immunology | Severity of Illness Index | Asthma - microbiology | Administration, Inhalation | Asthma - drug therapy | Disease Progression | Tacrolimus Binding Proteins - genetics | Th17 Cells - immunology | Klebsiella - immunology | Care and treatment | Asthma | Studies | Bacterial infections | Phylogenetics | Genomes | Inflammation | Gene expression | Methods | Index Medicus | Abridged Index Medicus
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5. Full Text ICOS:ICOS-Ligand Interaction Is Required for Type 2 Innate Lymphoid Cell Function, Homeostasis, and Induction of Airway Hyperreactivity
by Maazi, Hadi and Patel, Nisheel and Sankaranarayanan, Ishwarya and Suzuki, Yuzo and Rigas, Diamanda and Soroosh, Pejman and Freeman, Gordon J and Sharpe, Arlene H and Akbari, Omid
Immunity, ISSN 1074-7613, 03/2015, Volume 42, Issue 3, pp. 538 - 551
Allergic asthma is caused by Th2-cell-type cytokines in response to allergen exposure. Type 2 innate lymphoid cells (ILC2s) are a newly identified subset of... 
HUMAN INTERLEUKIN-5 | ADAPTIVE IMMUNITY | RESPONSES | TH2 DIFFERENTIATION | TRANSCRIPTION FACTOR GATA3 | REGULATORY T-CELLS | ALLERGIC LUNG INFLAMMATION | RECEPTOR | IMMUNOLOGY | MOLECULE ICOS | EXPRESSION | Respiratory System - pathology | Humans | STAT5 Transcription Factor - immunology | Homeostasis | Interleukin-13 - immunology | Interleukin-5 - genetics | Interleukins - genetics | STAT5 Transcription Factor - genetics | Interleukin-13 - genetics | Interleukin-2 - immunology | Lymphocytes - immunology | Interleukins - immunology | Asthma - immunology | Inducible T-Cell Co-Stimulator Protein - genetics | Female | Inducible T-Cell Co-Stimulator Protein - immunology | Respiratory System - immunology | Inducible T-Cell Co-Stimulator Ligand - immunology | Signal Transduction | Interleukin-33 | Gene Expression Regulation | Mice, Transgenic | Immunity, Innate | Asthma - genetics | Interleukin-5 - immunology | Interleukin-2 - genetics | Lymphocytes - pathology | Animals | Inducible T-Cell Co-Stimulator Ligand - genetics | Asthma - pathology | Allergens | T cells | Asthma | Immunology | Compliance | Pathogenesis | Lymphocytes | Rodents | Cloning | Ligands | Software | Histology | Gene expression | Experiments | Index Medicus
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6. Full Text Resident viruses and their interactions with the immune system
by Duerkop, Breck A and Hooper, Lora V
Nature Immunology, ISSN 1529-2908, 07/2013, Volume 14, Issue 7, pp. 654 - 659
The human body is colonized with a diverse resident microflora that includes viruses. Recent studies of metagenomes have begun to characterize the composition... 
CYCLIC GMP-AMP | BACTERIOPHAGE PARTICLES | DENDRITIC CELLS | CYTOSOLIC DNA | HOST | HUMAN GUT VIROME | MICROBIOTA | INFECTION | DIVERSITY | IMMUNOLOGY | HUMAN FECES | Gastrointestinal Tract - virology | Viruses - genetics | Humans | Mouth - immunology | Viruses - immunology | Host-Pathogen Interactions - immunology | Gastrointestinal Tract - immunology | Respiratory System - virology | Mouth - virology | Nasal Cavity - virology | Skin - virology | Nasal Cavity - immunology | Respiratory System - immunology | Skin - immunology | Viruses | Genetic aspects | Research | Microbiota (Symbiotic organisms) | Analysis | Immune system | Index Medicus
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7. Full Text The Th17-ELR+ CXC chemokine pathway is essential for the development of central nervous system autoimmune disease
by Carlson, Thaddeus and Kroenke, Mark and Rao, Praveen and Lane, Thomas E and Segal, Benjamin
Journal of Experimental Medicine, ISSN 0022-1007, 04/2008, Volume 205, Issue 4, pp. 811 - 823
The ELR+ CXC chemokines CXCL1 and CXCL2 are up-regulated in the central nervous system (CNS) during multiple sclerosis ( MS) and its animal model, experimental... 
MEDIATED HOST-DEFENSE | MEDICINE, RESEARCH & EXPERIMENTAL | NEUTROPHIL | MULTIPLE-SCLEROSIS | DENDRITIC CELLS | RECEPTOR-2 LIGANDS | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | MONONUCLEAR-CELLS | IMMUNE INVASION | IMMUNOLOGY | BLOOD-BRAIN-BARRIER | MICROARRAY ANALYSIS | Autoimmune Diseases of the Nervous System - prevention & control | Recurrence | Blood-Brain Barrier - immunology | Myelin Sheath - immunology | Central Nervous System - pathology | Inflammation | Adoptive Transfer | Receptors, Interleukin-8B - deficiency | Autoimmune Diseases of the Nervous System - pathology | Central Nervous System - immunology | Autoimmune Diseases of the Nervous System - immunology | Blood-Brain Barrier - pathology | Chemokine CXCL1 - immunology | Chemokine CXCL2 - immunology | Animals | T-Lymphocytes, Helper-Inducer - immunology | Bone Marrow Cells - immunology | Leukocytes, Mononuclear - immunology | Mice | Mice, Inbred BALB C | Index Medicus
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8. Full Text Proteinase activated receptor‐2‐mediated dual oxidase‐2 up‐regulation is involved in enhanced airway reactivity and inflammation in a mouse model of allergic asthma
by Nadeem, Ahmed and Alharbi, Naif O and Vliagoftis, Harissios and Tyagi, Manoj and Ahmad, Sheikh F and Sayed‐Ahmed, Mohamed M
Immunology, ISSN 0019-2805, 07/2015, Volume 145, Issue 3, pp. 391 - 403
Airway epithelial cells ( AEC s) express a variety of receptors, which sense danger signals from various aeroallergens/pathogens being inhaled constantly.... 
proteinase‐activated receptor‐2 | cockroach extract allergens | allergic asthma | airway epithelium | dual oxidase‐2 | reactive oxygen species | Allergic asthma | Reactive oxygen species | Proteinase-activated receptor-2 | Airway epithelium | Cockroach extract allergens | Dual oxidase-2 | PATHWAYS | LUNG | OXIDATIVE STRESS | dual oxidase-2 | REDOX CONTROL | BREATH CONDENSATE | IMMUNOLOGY | EPITHELIAL-CELLS | NITRIC-OXIDE | proteinase-activated receptor-2 | HYDROGEN-PEROXIDE | MICE | NITROTYROSINE | Respiratory System - pathology | Asthma - metabolism | Epithelial Cells - metabolism | Onium Compounds - immunology | Reactive Oxygen Species - metabolism | Asthma - prevention & control | Apoptosis - drug effects | Anti-Infective Agents - pharmacology | Epithelial Cells - drug effects | Receptor, PAR-2 - metabolism | NADPH Oxidases - metabolism | Oxidative Stress - immunology | NADPH Oxidases - immunology | Male | Anti-Infective Agents - immunology | Signal Transduction - immunology | Biphenyl Compounds - immunology | Inflammation - metabolism | Receptor, PAR-2 - antagonists & inhibitors | Receptor, PAR-2 - immunology | Allergens - immunology | Biphenyl Compounds - pharmacology | Reactive Oxygen Species - immunology | Asthma - immunology | Respiratory Hypersensitivity - prevention & control | Cockroaches | Respiratory System - immunology | Disease Models, Animal | NADPH Oxidases - antagonists & inhibitors | Respiratory Hypersensitivity - metabolism | Inflammation - immunology | Onium Compounds - pharmacology | Respiratory Hypersensitivity - immunology | Piperazines - pharmacology | Blotting, Western | Animals | Apoptosis - immunology | Up-Regulation - immunology | Respiratory System - metabolism | Epithelial Cells - immunology | Piperazines - immunology | Inflammation - prevention & control | Mice, Inbred BALB C | Oxidative Stress - drug effects | Dual Oxidases | Oxidases | Allergens | Inflammation | Asthma | Oxidative stress | Rodents | Apoptosis | Index Medicus | Original
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9. Full Text S100A8, S100A9 and S100A12 activate airway epithelial cells to produce MUC5AC via extracellular signal‐regulated kinase and nuclear factor‐κB pathways
by Kang, Jin Hyun and Hwang, Sae Mi and Chung, Il Yup
Immunology, ISSN 0019-2805, 01/2015, Volume 144, Issue 1, pp. 79 - 90
Airway mucus hyperproduction is a common feature of chronic airway diseases such as severe asthma, chronic obstructive pulmonary disease and cystic fibrosis,... 
S100A12 | MUC | S100A9 | S100A8 | airway epithelial cells | Airway epithelial cells | MUC5AC | SEVERE ASTHMA | MUCIN GENE-EXPRESSION | PROTEIN-KINASE | ALPHA | NEUTROPHIL ELASTASE | IMMUNOLOGY | TOLL-LIKE RECEPTOR-4 | MESSENGER-RNA | GROWTH | DEPENDENT MECHANISM | GLYCATION END-PRODUCTS | Cell Line | Inflammation - pathology | Gene Expression Regulation - immunology | Humans | NF-kappa B - immunology | Epithelial Cells - pathology | Inflammation - immunology | Toll-Like Receptor 4 - immunology | Bronchi - immunology | S100 Proteins - immunology | MAP Kinase Signaling System - immunology | Extracellular Signal-Regulated MAP Kinases - immunology | S100A12 Protein | Mucin 5AC - immunology | Epithelial Cells - immunology | Calgranulin B - immunology | I-kappa B Kinase - immunology | Calgranulin A - immunology | Bronchi - pathology | Index Medicus | Original
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