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1. Full Text A novel cell-cycle-indicator, mVenus-p27K -, identifies quiescent cells and visualizes G0-G1 transition
by Oki, Toshihiko and Nishimura, Koutarou and Kitaura, Jiro and Togami, Katsuhiro and Maehara, Akie and Izawa, Kumi and Sakaue-Sawano, Asako and Niida, Atsushi and Miyano, Satoru and Aburatani, Hiroyuki and Kiyonari, Hiroshi and Miyawaki, Atsushi and Kitamura, Toshio
Scientific reports, ISSN 2045-2322, 02/2014, Volume 4, Issue 1, pp. 4012 - 4012
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | RNA-Binding Proteins - genetics | Selenium-Binding Proteins - genetics | Tumor Suppressor Protein p53 - biosynthesis | Stem Cells - cytology | Retinoblastoma Protein - biosynthesis | RNA-Binding Proteins - biosynthesis | Selenium-Binding Proteins - biosynthesis | Apoptosis Regulatory Proteins - genetics | Cyclin-Dependent Kinase Inhibitor p16 - biosynthesis | G1 Phase - genetics | Apoptosis Regulatory Proteins - biosynthesis | Recombinant Fusion Proteins - biosynthesis | Cell Line | Cyclin-Dependent Kinase Inhibitor p21 - biosynthesis | Gene Expression | Carrier Proteins - biosynthesis | Bacterial Proteins - genetics | Mice, Transgenic | MicroRNAs - biosynthesis | Carrier Proteins - genetics | Cyclin-Dependent Kinase Inhibitor p57 - biosynthesis | Animals | Resting Phase, Cell Cycle - genetics | Recombinant Fusion Proteins - genetics | Luminescent Proteins - genetics | Mice | 3T3 Cells | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Cyclin-dependent kinase | Rodents | Stem cells | Cell cycle | Defective mutant | Transgenic mice | Stem cell transplantation | Cyclin-dependent kinase inhibitor p27 | Fusion protein | Index Medicus
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2. Full Text Antiproliferative activity of the isoindigo 5′-Br in HL-60 cells is mediated by apoptosis, dysregulation of mitochondrial functions and arresting cell cycle at G0/G1 phase
by Saleh, Ayman M and El-Abadelah, Mustafa M and Aziz, Mohammad Azhar and Taha, Mutasem O and Nasr, Amre and Rizvi, Syed A.A
Cancer letters, ISSN 0304-3835, 2015, Volume 361, Issue 2, pp. 251 - 261
Hematology, Oncology and Palliative Medicine | CDK | Anticancer | Mitochondria dysfunction | Cell cycle | Isoindigo | Apoptosis | Life Sciences & Biomedicine | Oncology | Science & Technology | Apoptosis - drug effects | Leukemia, Myeloid, Acute - pathology | Humans | Mitochondria - metabolism | G1 Phase - drug effects | Mitochondria - drug effects | Cell Division - drug effects | HL-60 Cells | Leukemia, Myeloid, Acute - drug therapy | Indoles - pharmacology | Resting Phase, Cell Cycle - drug effects | Cell Proliferation - drug effects | Cell Cycle - drug effects | Index Medicus
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3. Full Text miR-206 functions as a novel cell cycle regulator and tumor suppressor in clear-cell renal cell carcinoma
by Xiao, Haibing and Xiao, Wei and Cao, Jing and Li, Heng and Guan, Wei and Guo, Xiaolin and Chen, Ke and Zheng, Tao and Ye, Zhangqun and Wang, Ji and Xu, Hua
Cancer letters, ISSN 0304-3835, 2016, Volume 374, Issue 1, pp. 107 - 116
Hematology, Oncology and Palliative Medicine | tumor suppressor | ccRCC | cell cycle | miR-206 | MiR-206 | CcRCC | Tumor suppressor | Cell cycle | Life Sciences & Biomedicine | Oncology | Science & Technology | Cyclin D1 - metabolism | Kidney Neoplasms - genetics | Cyclin-Dependent Kinase 4 - genetics | Humans | Carcinoma, Renal Cell - genetics | Male | MicroRNAs - metabolism | Kidney Neoplasms - metabolism | Cell Cycle Proteins - genetics | Cell Growth Processes - genetics | Carcinoma, Renal Cell - pathology | Cyclin-Dependent Kinase 9 - genetics | Cell Cycle Proteins - metabolism | Cyclin-Dependent Kinase 4 - metabolism | G1 Phase Cell Cycle Checkpoints - genetics | Carcinoma, Renal Cell - metabolism | Animals | Resting Phase, Cell Cycle - genetics | Cyclin D1 - genetics | Cell Line, Tumor | Kidney Neoplasms - pathology | Cyclin-Dependent Kinase 9 - metabolism | Mice | Mice, Inbred BALB C | MicroRNAs - genetics | Cancer | Genetic research | MicroRNA | Analysis | Index Medicus
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4. Full Text Bcl‐xL/Bcl‐2 coordinately regulates apoptosis, cell cycle arrest and cell cycle entry
by Janumyan, Yelena M and Sansam, Courtney G and Chattopadhyay, Anuja and Cheng, Ningli and Soucie, Erinn L and Penn, Linda Z and Andrews, David and Knudson, C.Michael and Yang, Elizabeth
The EMBO journal, ISSN 0261-4189, 10/2003, Volume 22, Issue 20, pp. 5459 - 5470
apoptosis | Bcl‐2 | BAD | Bcl‐xL | cell cycle | Bcl-2 | Bcl-X | Cell cycle | Apoptosis | Recombinant Proteins - metabolism | Cell Line | bcl-X Protein | Rats | Resting Phase, Cell Cycle | Proto-Oncogene Proteins c-bcl-2 - metabolism | Contact Inhibition | G1 Phase | Animals | Carrier Proteins - metabolism | Mutagenesis | Cell Division | Alleles | Cell Cycle - physiology | Apoptosis - physiology | Kinetics | Proto-Oncogene Proteins c-bcl-2 - genetics | bcl-Associated Death Protein | Index Medicus | Bcl-xL
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5. Full Text Honokiol causes G0-G1 phase cell cycle arrest in human prostate cancer cells in association with suppression of retinoblastoma protein level/phosphorylation and inhibition of E2F1 transcriptional activity
by Eun-Ryeong Hahm and Shivendra V. Singh
Molecular cancer therapeutics, ISSN 1535-7163, 10/2007, Volume 6, Issue 10, pp. 2686 - 2695
Honokiol | E2F1 | chemoprevention | cell cycle | Prostatic Neoplasms - metabolism | Transcription, Genetic - drug effects | Phosphorylation | Immunoprecipitation | Luciferases - metabolism | Reactive Oxygen Species - metabolism | Humans | Male | G1 Phase - drug effects | Lignans - pharmacology | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Biphenyl Compounds - pharmacology | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Prostatic Neoplasms - drug therapy | Prostatic Neoplasms - pathology | Drugs, Chinese Herbal | RNA, Small Interfering - pharmacology | Retinoblastoma Protein - metabolism | Tumor Cells, Cultured - drug effects | Cell Cycle Proteins - metabolism | Tumor Suppressor Protein p53 - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - antagonists & inhibitors | Antioxidants - pharmacology | E2F1 Transcription Factor - metabolism | Acetylcysteine - pharmacology | Resting Phase, Cell Cycle - drug effects | Cell Proliferation - drug effects | Antineoplastic Agents, Phytogenic - pharmacology | Cell Cycle - drug effects | E2F1 Transcription Factor - genetics | Index Medicus
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6. Full Text Porcine epidemic diarrhea virus through p53-dependent pathway causes cell cycle arrest in the G0/G1 phase
by Sun, Pei and Wu, Haoyang and Huang, Jiali and Xu, Ying and Yang, Feng and Zhang, Qi and Xu, Xingang
Virus research, ISSN 0168-1702, 07/2018, Volume 253, pp. 1 - 11
Cell cycle arrest | PEDV | Virus replication | p53 pathway | Life Sciences & Biomedicine | Science & Technology | Virology | Cyclin-Dependent Kinase 2 - metabolism | Porcine epidemic diarrhea virus | Swine Diseases - genetics | Chlorocebus aethiops | Swine Diseases - virology | Swine Diseases - metabolism | Tumor Suppressor Protein p53 - metabolism | Coronavirus Infections - physiopathology | Cyclin-Dependent Kinase 2 - genetics | Resting Phase, Cell Cycle | Tumor Suppressor Protein p53 - genetics | G1 Phase | Animals | Coronavirus Infections - metabolism | Coronavirus Infections - veterinary | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Coronavirus Infections - virology | Swine | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | G1 Phase Cell Cycle Checkpoints | Swine Diseases - physiopathology | Vero Cells | Virus diseases | Epidemics | Medical research | Cell cycle | Diarrhea | Medicine, Experimental | Tumor proteins | Health aspects | Cells | Index Medicus
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7. Full Text PSMD2 regulates breast cancer cell proliferation and cell cycle progression by modulating p21 and p27 proteasomal degradation
by Li, Yunhai and Huang, Jing and Zeng, Beilei and Yang, Dejuan and Sun, Jiazheng and Yin, Xuedong and Lu, Mengqi and Qiu, Zhu and Peng, Weiyan and Xiang, Tingxiu and Li, Hongzhong and Ren, Guosheng
Cancer letters, ISSN 0304-3835, 08/2018, Volume 430, pp. 109 - 122
Breast cancer | p27 | Ubiquitin-proteasome system | PSMD2 | p21 | Cell cycle | Life Sciences & Biomedicine | Oncology | Science & Technology | RNA, Small Interfering - genetics | Up-Regulation | Cell Proliferation | Humans | Middle Aged | TNF Receptor-Associated Factor 2 - genetics | Gene Expression Profiling | Resting Phase, Cell Cycle | Gene Knockdown Techniques | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Injections, Intralesional | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | G1 Phase Cell Cycle Checkpoints | Ubiquitin Thiolesterase - metabolism | Female | Kaplan-Meier Estimate | TNF Receptor-Associated Factor 2 - metabolism | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - pathology | Mice, Nude | Cell Line, Tumor | Breast Neoplasms - mortality | Mice | Proteasome Endopeptidase Complex - metabolism | RNA, Small Interfering - administration & dosage | RNA, Small Interfering - metabolism | Ubiquitin | Immunohistochemistry | Phosphates | Epigenetic inheritance | RNA | Genes | Genomics | Development and progression | Gene expression | Ligases | Glutathione transferase | Analysis
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8. Full Text Isolation of Quiescent and Nonquiescent Cells from Yeast Stationary-Phase Cultures
by Chris Allen and Sabrina Büttner and Anthony D. Aragon and Jason A. Thomas and Osorio Meirelles and Jason E. Jaetao and Don Benn and Stephanie W. Ruby and Marten Veenhuis and Frank Madeo and Margaret Werner-Washburne
The Journal of cell biology, ISSN 0021-9525, 7/2006, Volume 174, Issue 1, pp. 89 - 100
Yeasts | Cell culture techniques | Scars | Mother cells | Cell cycle | Stem cells | Cultured cells | Cells | Daughter cells | Apoptosis | APOPTOSIS | REGULATOR | COLONIES | SUPEROXIDE | DNA | GENE-EXPRESSION | STRESS | IDENTIFICATION | SACCHAROMYCES-CEREVISIAE | EXIT | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Microscopy - methods | Reactive Oxygen Species - metabolism | Mitosis | Cells, Cultured | Cell Separation - methods | Saccharomyces cerevisiae - cytology | Flow Cytometry | Resting Phase, Cell Cycle - physiology | Sensitivity and Specificity | Glucose - chemistry | Cell Cycle - physiology | Centrifugation, Density Gradient - methods | Apoptosis - physiology | Saccharomyces cerevisiae - isolation & purification | Cell culture | Research | Yeast fungi | Yeast | Analysis | Index Medicus | s
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9. Anti-Cancerous Effect of Rutin Against HPV C33A Cervical Cancer Cells via G0/G1 Cell Cycle Arrest and Apoptotic Induction
by Khan, Fahad and Pandey, Pratibha and Upadhyay, Tarun K and Jafri, Asif and Jha, Niraj K and Mishra, Rashmi and Singh, Vineeta
Endocrine, metabolic & immune disorders drug targets, ISSN 1871-5303, 01/2020, Volume 20, Issue 3, pp. 409 - 418
Pharmacology & Pharmacy | Endocrinology & Metabolism | Immunology | Life Sciences & Biomedicine | Science & Technology | Cell Survival - drug effects | G1 Phase Cell Cycle Checkpoints - physiology | Apoptosis - drug effects | Humans | Rutin - pharmacology | Cell Cycle Checkpoints - physiology | Uterine Cervical Neoplasms - drug therapy | Uterine Cervical Neoplasms - physiopathology | Alphapapillomavirus | Dose-Response Relationship, Drug | Cell Cycle Checkpoints - drug effects | Resting Phase, Cell Cycle - physiology | HEK293 Cells | Cell Line, Tumor | Female | Resting Phase, Cell Cycle - drug effects | Apoptosis - physiology | Antineoplastic Agents, Phytogenic - pharmacology | Antineoplastic Agents, Phytogenic - therapeutic use | G1 Phase Cell Cycle Checkpoints - drug effects | Rutin - therapeutic use | Cell Survival - physiology
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10. Full Text Glioma-specific cation conductance regulates migration and cell cycle progression
by Rooj, Arun K and McNicholas, Carmel M and Bartoszewski, Rafal and Bebok, Zsuzsanna and Benos, Dale J and Fuller, Catherine M
The Journal of biological chemistry, ISSN 0021-9258, 02/2012, Volume 287, Issue 6, pp. 4053 - 4065
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Peptides | Humans | Amiloride - analogs & derivatives | Neoplasm Proteins - antagonists & inhibitors | Spider Venoms - pharmacology | Epithelial Sodium Channels - metabolism | G1 Phase - drug effects | Neoplasm Proteins - metabolism | Glioma - metabolism | Glioma - genetics | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Epithelial Sodium Channels - genetics | Sodium Channels - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | G1 Phase - genetics | Neoplasm Proteins - genetics | Nerve Tissue Proteins - antagonists & inhibitors | Acid Sensing Ion Channels | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Cell Cycle Checkpoints | Resting Phase, Cell Cycle - genetics | Amiloride - pharmacology | Cell Line, Tumor | Resting Phase, Cell Cycle - drug effects | Epithelial Sodium Channel Blockers | Sodium Channels - genetics | Cell Movement | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Index Medicus | Membrane Biology | Sodium Channels | Psalmotoxin-1 | ENaC | Acid-sensing Ion Channels (ASIC) | Cancer | MAPKs
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11. Full Text HSP90 inhibitor, celastrol, arrests human monocytic leukemia cell U937 at G0/G1 in thiol-containing agents reversible way
by Peng, Bin and Xu, Limin and Cao, Fanfan and Wei, Tingxuan and Yang, Chunxin and Uzan, Georges and Zhang, Denghai
Molecular cancer, ISSN 1476-4598, 04/2010, Volume 9, Issue 1, pp. 79 - 79
Biochemistry & Molecular Biology | Oncology | Life Sciences & Biomedicine | Science & Technology | Cell Cycle Proteins - drug effects | Immunoprecipitation | Triterpenes - pharmacology | Sulfhydryl Compounds - pharmacology | Apoptosis - drug effects | Cell Separation | Humans | G1 Phase - drug effects | Blotting, Western | Dose-Response Relationship, Drug | Flow Cytometry | HSP90 Heat-Shock Proteins - antagonists & inhibitors | U937 Cells | Resting Phase, Cell Cycle - drug effects | Antineoplastic Agents - pharmacology | Cell Proliferation - drug effects | Antimitotic agents | Leukemia | Patient outcomes | Heat shock proteins | Physiological aspects | Genetic aspects | Dosage and administration | Research | Drug therapy | Antineoplastic agents | Index Medicus | Triterpenes | Cell Proliferation | HSP90 Heat-Shock Proteins | Biochemistry, Molecular Biology | Genomics | Antineoplastic Agents | Life Sciences | G1 Phase | G0 Phase | Cell Cycle Proteins | Apoptosis | Sulfhydryl Compounds | Cancer
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