Articles

Search Articles

Advanced search
Help

Catalogue

Articles

Databases

X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (1961) 1961
index medicus (1266) 1266
animals (1254) 1254
resting phase, cell cycle (847) 847
mice (731) 731
resting phase, cell cycle - drug effects (694) 694
cell cycle (641) 641
female (617) 617
apoptosis (533) 533
male (526) 526
cell line, tumor (516) 516
g1 phase (504) 504
g1 phase - drug effects (481) 481
cell biology (451) 451
cells, cultured (439) 439
oncology (415) 415
biochemistry & molecular biology (401) 401
expression (384) 384
apoptosis - drug effects (357) 357
resting phase, cell cycle - genetics (318) 318
cell proliferation (307) 307
flow cytometry (303) 303
resting phase, cell cycle - physiology (289) 289
cancer (284) 284
adult (282) 282
analysis (280) 280
proliferation (277) 277
cell line (276) 276
immunology (274) 274
cell proliferation - drug effects (273) 273
time factors (253) 253
neurosciences (249) 249
rats (242) 242
phosphorylation (239) 239
cell cycle - drug effects (236) 236
activation (228) 228
resting phase, cell cycle - immunology (228) 228
research (211) 211
growth (209) 209
proteins (204) 204
article (199) 199
gene expression (187) 187
antineoplastic agents - pharmacology (186) 186
signal transduction (186) 186
dose-response relationship, drug (184) 184
g1 phase - physiology (180) 180
middle aged (177) 177
cell division (174) 174
mice, inbred c57bl (169) 169
differentiation (167) 167
blotting, western (165) 165
tumor cells, cultured (164) 164
research article (163) 163
cell division - drug effects (162) 162
in-vitro (162) 162
in-vivo (161) 161
s phase (161) 161
pharmacology & pharmacy (159) 159
biology (153) 153
physiological aspects (153) 153
cell cycle - physiology (150) 150
transfection (150) 150
cells (147) 147
physiology (141) 141
mice, inbred balb c (137) 137
protein (137) 137
cell survival - drug effects (136) 136
life sciences (136) 136
marine & freshwater biology (133) 133
molecular sequence data (133) 133
g1 phase - genetics (132) 132
gene-expression (131) 131
health aspects (131) 131
inhibition (129) 129
kinetics (123) 123
down-regulation (122) 122
multidisciplinary sciences (121) 121
dna (119) 119
mice, transgenic (119) 119
rna, messenger - metabolism (118) 118
genetics & heredity (113) 113
signal transduction - drug effects (113) 113
gene (110) 110
induction (110) 110
resting energy-expenditure (110) 110
biotechnology & applied microbiology (108) 108
medicine (107) 107
aged (105) 105
endocrinology & metabolism (105) 105
studies (105) 105
hematology (104) 104
cell growth (102) 102
microbiology (102) 102
mice, knockout (99) 99
brain (97) 97
genes (97) 97
metabolism (97) 97
cycle (96) 96
dna damage (96) 96
fibroblasts - cytology (96) 96
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (3260) 3260
Chinese (20) 20
Russian (12) 12
Japanese (5) 5
French (3) 3
Italian (2) 2
German (1) 1
Korean (1) 1
Spanish (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


1. Full Text Metformin induced AMPK activation, G0/G1 phase cell cycle arrest and the inhibition of growth of Esophageal squamous cell carcinomas in vitro and in vivo
by Cai, Xianbin and Hu, Xi and Tan, Xiaojun and Cheng, Weijie and Wang, Qinjia and Chen, Xiaofeng and Guan, Yinghong and Chen, Chong and Jing, Xubin
PLoS ONE, ISSN 1932-6203, 07/2015, Volume 10, Issue 7, p. e0133349
Esophageal squamous cell carcinomas (ESCC) have become a severe threat to health and the current treatments for ESCC are frequently not effective. Recent... 
CANCER-CELLS | PROSTATE | APOPTOSIS | CISPLATIN | ANGIOGENESIS | SIGNALING PATHWAY | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | RESISTANCE | PROLIFERATION | INDUCTION | Cyclin D1 - metabolism | AMP-Activated Protein Kinases - metabolism | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Male | Esophageal Neoplasms - pathology | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Esophageal Neoplasms - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Cell Survival - drug effects | Metformin - pharmacology | Tumor Suppressor Protein p53 - metabolism | Enzyme Activation - drug effects | Blotting, Western | Hypoglycemic Agents - pharmacology | Xenograft Model Antitumor Assays | Animals | Carcinoma, Squamous Cell - drug therapy | Tumor Burden - drug effects | Mice, Nude | Cell Line, Tumor | Resting Phase, Cell Cycle - drug effects | Cell Proliferation - drug effects | G1 Phase Cell Cycle Checkpoints - drug effects | Esophageal Neoplasms - drug therapy | Diabetics | Squamous cell carcinoma | Growth | Cell cycle | Metformin | Tumor proteins | Health aspects | Esophageal cancer | Flow cytometry | Regulations | p53 Protein | Colorectal cancer | Kinases | Epidemiology | Anticancer properties | Angiogenesis | Cell activation | Cell growth | Gastroenterology | Xenografts | Tumorigenesis | Inhibition | Growth factors | G1 phase | Esophageal carcinoma | Enzymes | Adenosine | Diabetes mellitus | Cyclin-dependent kinases | Health risks | Risk reduction | Cell division | Cultures | Metabolism | Esophagus | Cyclin-dependent kinase inhibitor p21 | Hospitals | Cell lines | Cyclin-dependent kinase inhibitor p27 | Antitumor activity | Hypoxia | Cancer
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
2. Full Text Synthesis of novel guttiferone E and xanthochymol derivatives with cytotoxicities by inducing cell apoptosis and arresting the cell cycle phase
by Lin, Xin and Tian, Dongsong and Fu, Yanhui and Li, Yanan and Li, Yanmei and Huang, Liejun and Gu, Wei and Song, Jialei and Ben-David, Yaacov and Wen, Min and Yuan, Chunmao and Hao, Xiaojiang
European Journal of Medicinal Chemistry, ISSN 0223-5234, 01/2019, Volume 162, pp. 765 - 780
The mixture of (guttiferone E and xanthochymol), an inseparable polycyclic polyprenylated acylphloroglucinol (PPAP), showed moderate cytotoxic activities. The... 
Cell cycle arrest | Cytotoxicity | Polycyclic polyprenylated acylphloroglucinol | Chemical transformation | Cell apoptosis | CHEMISTRY, MEDICINAL | ACYLPHLOROGLUCINOLS | NATURAL-PRODUCTS | DRUGS | BENZOPHENONES | CANCER | Cyclin D1 - drug effects | Cyclin D1 - metabolism | Benzophenones - chemistry | Magnetic Resonance Spectroscopy | Humans | bcl-2-Associated X Protein - metabolism | Benzophenones - pharmacology | Leukemia - drug therapy | bcl-2-Associated X Protein - drug effects | Polycyclic Compounds - pharmacology | Cytotoxins | Proto-Oncogene Proteins c-bcl-2 - metabolism | Cell Cycle Checkpoints | Spectrometry, Mass, Electrospray Ionization | Proto-Oncogene Proteins c-bcl-2 - drug effects | Cell Line, Tumor | Resting Phase, Cell Cycle - drug effects | Molecular Structure | Apoptosis
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
3. Full Text Honokiol causes G0-G1 phase cell cycle arrest in human prostate cancer cells in association with suppression of retinoblastoma protein level/phosphorylation and inhibition of E2F1 transcriptional activity
by Eun-Ryeong Hahm and Shivendra V. Singh
Molecular Cancer Therapeutics, ISSN 1535-7163, 10/2007, Volume 6, Issue 10, pp. 2686 - 2695
The present study was undertaken to gain insights into the mechanism of cell cycle arrest caused by honokiol, a constituent of oriental herb Magnolia... 
Rb | Honokiol | E2F1 | chemoprevention | cell cycle | Prostatic Neoplasms - metabolism | Transcription, Genetic - drug effects | Phosphorylation | Immunoprecipitation | Luciferases - metabolism | Reactive Oxygen Species - metabolism | Humans | Male | G1 Phase - drug effects | Lignans - pharmacology | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Biphenyl Compounds - pharmacology | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Prostatic Neoplasms - drug therapy | Prostatic Neoplasms - pathology | Drugs, Chinese Herbal | RNA, Small Interfering - pharmacology | Retinoblastoma Protein - metabolism | Tumor Cells, Cultured - drug effects | Cell Cycle Proteins - metabolism | Tumor Suppressor Protein p53 - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - antagonists & inhibitors | Antioxidants - pharmacology | E2F1 Transcription Factor - metabolism | Acetylcysteine - pharmacology | Resting Phase, Cell Cycle - drug effects | Cell Proliferation - drug effects | Antineoplastic Agents, Phytogenic - pharmacology | Cell Cycle - drug effects | E2F1 Transcription Factor - genetics
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
4. Full Text Low doses of cisplatin or gemcitabine plus Photofrin/photodynamic therapy: Disjointed cell cycle phase-related activity accounts for synergistic outcome in metastatic non–small cell lung cancer cells (H1299)
by Elvira Crescenzi and Angela Chiaviello and Gianfranco Canti and Elena Reddi and Bianca Maria Veneziani and Giuseppe Palumbo
Molecular Cancer Therapeutics, ISSN 1535-7163, 03/2006, Volume 5, Issue 3, pp. 776 - 785
We compared the effects of monotherapy (photodynamic therapy or chemotherapy) versus combination therapy (photodynamic therapy plus a specific drug) on the... 
CDDP | Photodynamic therapy | Gemcitabine | Photofrin | Combined therapy | APOPTOSIS | ARREST | PHTHALOCYANINE PHOTOSENSITIZER PC-4 | PHOTODYNAMIC THERAPY | POTENTIATION | ONCOLOGY | DNA-DAMAGE | TUMOR-CELLS | ACCUMULATION | MURINE TUMORS | CHEMOTHERAPY | Lung Neoplasms - drug therapy | Photosensitizing Agents - therapeutic use | Humans | Lung Neoplasms - pathology | Antineoplastic Agents - therapeutic use | G1 Phase - drug effects | Photochemotherapy | Dose-Response Relationship, Drug | Deoxycytidine - therapeutic use | Carcinoma, Non-Small-Cell Lung - secondary | Cisplatin - therapeutic use | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Dihematoporphyrin Ether - therapeutic use | Resting Phase, Cell Cycle - drug effects | Carcinoma, Non-Small-Cell Lung - drug therapy | Deoxycytidine - analogs & derivatives
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
5. Full Text A novel cell-cycle-indicator, mVenus-p27K -, identifies quiescent cells and visualizes G0-G1 transition
by Oki, Toshihiko and Nishimura, Koutarou and Kitaura, Jiro and Togami, Katsuhiro and Maehara, Akie and Izawa, Kumi and Sakaue-Sawano, Asako and Niida, Atsushi and Miyano, Satoru and Aburatani, Hiroyuki and Kiyonari, Hiroshi and Miyawaki, Atsushi and Kitamura, Toshio
Scientific Reports, ISSN 2045-2322, 02/2014, Volume 4, Issue 1, p. 4012
The quiescent (G0) phase of the cell cycle is the reversible phase from which the cells exit from the cell cycle. Due to the difficulty of defining the G0... 
PROGENITOR CELLS | SKELETAL-MUSCLE | MUSCLE REGENERATION | CDK INHIBITORS | S-PHASE ENTRY | MULTIDISCIPLINARY SCIENCES | HEMATOPOIETIC STEM-CELLS | NICHE | SATELLITE CELLS | P27(KIP1) | G-G TRANSITION | RNA-Binding Proteins - genetics | Selenium-Binding Proteins - genetics | Tumor Suppressor Protein p53 - biosynthesis | Stem Cells - cytology | Retinoblastoma Protein - biosynthesis | RNA-Binding Proteins - biosynthesis | Selenium-Binding Proteins - biosynthesis | Apoptosis Regulatory Proteins - genetics | Cyclin-Dependent Kinase Inhibitor p16 - biosynthesis | G1 Phase - genetics | Apoptosis Regulatory Proteins - biosynthesis | Recombinant Fusion Proteins - biosynthesis | Cell Line | Cyclin-Dependent Kinase Inhibitor p21 - biosynthesis | Gene Expression | Carrier Proteins - biosynthesis | Bacterial Proteins - genetics | Mice, Transgenic | MicroRNAs - biosynthesis | Carrier Proteins - genetics | Cyclin-Dependent Kinase Inhibitor p57 - biosynthesis | Animals | Resting Phase, Cell Cycle - genetics | Recombinant Fusion Proteins - genetics | Luminescent Proteins - genetics | Mice | 3T3 Cells | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Cyclin-dependent kinase | Rodents | Stem cells | Cell cycle | Defective mutant | Transgenic mice | Stem cell transplantation | Cyclin-dependent kinase inhibitor p27 | Fusion protein
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
6. Full Text The mechanism of the G0/G1 cell cycle phase arrest induced by activation of PXR in human cells
by Zhuang, Weiliang and Jia, Zhenhua and Feng, Huahua and Chen, Jun and Wang, Hang and Guo, Yanghao and Meng, Chun
Biomedicine & Pharmacotherapy, ISSN 0753-3322, 2011, Volume 65, Issue 7, pp. 467 - 473
Abstract Context Pregnane X receptor (PXR) is an important transcriptional regulator that plays important roles in the cell metabolism and cell growth by... 
Internal Medicine | Medical Education | Retinoid X receptor alpha | G0/G1 cell cycle phase arrest | Pregnane X receptor | Competitive bind | MEDICINE, RESEARCH & EXPERIMENTAL | APOPTOSIS | PROLIFERATION | OVARIAN-CANCER | XENOBIOTIC RECEPTOR | IDENTIFICATION | PREGNANE-X-RECEPTOR | TARGET GENES | INHIBITION | GENE-EXPRESSION | PHARMACOLOGY & PHARMACY | NF-KAPPA-B | Binding, Competitive | Transcription, Genetic - drug effects | Retinoid X Receptor alpha - metabolism | Humans | RNA, Small Interfering - pharmacology | Recombinant Fusion Proteins - physiology | Receptors, Steroid - drug effects | Cell Line, Tumor - drug effects | G1 Phase - drug effects | Cell Division - drug effects | Hep G2 Cells - drug effects | Ultraviolet Rays - adverse effects | RNA Interference | Hep G2 Cells - metabolism | Retinoid X Receptor alpha - genetics | Hep G2 Cells - radiation effects | Resting Phase, Cell Cycle - drug effects | DNA Damage | Rifampin - pharmacology | G1 Phase Cell Cycle Checkpoints - drug effects | Cell Line, Tumor - metabolism | Real-Time Polymerase Chain Reaction | Neuroblastoma - pathology | Physiological aspects | Enzymes | Cell cycle
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
7. Full Text Metformin induces apoptosis and cell cycle arrest mediated by oxidative stress, AMPK and FOXO3a in MCF-7 breast cancer cells
by Queiroz, Eveline A. I. F and Puukila, Stephanie and Eichler, Rosangela and Sampaio, Sandra C and Forsyth, Heidi L and Lees, Simon J and Barbosa, Aneli M and Dekker, Robert F. H and Fortes, Zuleica B and Khaper, Neelam
PLoS ONE, ISSN 1932-6203, 05/2014, Volume 9, Issue 5, p. e98207
Recent studies have demonstrated that the anti-diabetic drug, metformin, can exhibit direct antitumoral effects, or can indirectly decrease tumor proliferation... 
PROSTATE | OBESITY | ACTIVATED PROTEIN-KINASE | INHIBITION | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | IN-VIVO | GROWTH | AKT | AUTOPHAGY | TUMOR SUPPRESSORS | AMP-Activated Protein Kinases - metabolism | Oxidants - pharmacology | Hydrogen - pharmacology | Apoptosis - drug effects | Humans | Metformin - pharmacology | Enzyme Activation - drug effects | Neoplasm Proteins - metabolism | Breast Neoplasms - metabolism | Hypoglycemic Agents - pharmacology | Breast Neoplasms - pathology | Forkhead Transcription Factors - metabolism | Cell Line, Tumor | Female | Resting Phase, Cell Cycle - drug effects | Oxidative Stress - drug effects | Forkhead Box Protein O3 | G1 Phase Cell Cycle Checkpoints - drug effects | AMP-Activated Protein Kinases - genetics | Oxidative stress | Cancer cells | Cell cycle | Breast cancer | Metformin | Diabetes therapy | Apoptosis | Flow cytometry | Cell culture | Phosphorylation | Hydrogen peroxide | Bax protein | Bcl-2 protein | AKT protein | Superoxide dismutase | Biology | Activation | Kinases | Caspase-3 | Cancer therapies | Western blotting | Necrosis | Proteins | Signal transduction | Cell growth | Catalase | Penicillin | FOXO3 protein | G1 phase | Diabetes mellitus | Extracellular signal-regulated kinase | Caspase | MAP kinase | Pharmacology | Gene expression | Insulin | Zinc | Polymerase chain reaction | Cytometry | Molecular modelling | Cell number | Diabetes | Viability | Prostate | Cancer
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
8. Full Text A Hyperfused Mitochondrial State Achieved at G₁-S Regulates Cyclin E Buildup and Entry into S Phase
by Kasturi Mitra and Christian Wunder and Badrinath Roysam and Gang Lin and Jennifer Lippincott-Schwartz
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2009, Volume 106, Issue 29, pp. 11960 - 11965
Mitochondria undergo fission-fusion events that render these organelles highly dynamic in cells. We report a relationship between mitochondrial form and cell... 
Mitochondria | Cell growth | HCT116 cells | Interphase | Cell nucleus | Cell cycle | Cell lines | Cellular immunity | Gene expression regulation | Cyclins | Mitochondrial morphology | Dynamin-related protein 1 | Microtubule-Associated Proteins - metabolism | HCT116 Cells | Humans | Tumor Suppressor Protein p53 - metabolism | Mitochondria - metabolism | Resting Phase, Cell Cycle | G1 Phase | GTP Phosphohydrolases - metabolism | Mitochondrial Proteins - metabolism | S Phase | Membrane Potential, Mitochondrial | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Cyclin E - metabolism | dynamin-related protein 1 | Biological Sciences | cell cycle | mitochondrial morphology
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
9. Full Text Antiproliferative activity of the isoindigo 5′-Br in HL-60 cells is mediated by apoptosis, dysregulation of mitochondrial functions and arresting cell cycle at G0/G1 phase
by Saleh, Ayman M and El-Abadelah, Mustafa M and Aziz, Mohammad Azhar and Taha, Mutasem O and Nasr, Amre and Rizvi, Syed A.A
Cancer Letters, ISSN 0304-3835, 2015, Volume 361, Issue 2, pp. 251 - 261
Highlights • 5'-Br is a novel isoindigo showing strong, selective antiproliferative activity against various hematological and solid tumor cell lines. • The... 
Hematology, Oncology and Palliative Medicine | CDK | Anticancer | Mitochondria dysfunction | Cell cycle | Isoindigo | Apoptosis | CHRONIC MYELOGENOUS LEUKEMIA | WILD-TYPE P53 | INDIRUBIN DERIVATIVES | TUMOR SUPPRESSION | DNA-DAMAGE | DOWN-REGULATION | ACUTE MYELOID-LEUKEMIA | IN-VITRO | ONCOLOGY | BIOLOGICAL EVALUATION | GENE AMPLIFICATION | Apoptosis - drug effects | Leukemia, Myeloid, Acute - pathology | Humans | Mitochondria - metabolism | G1 Phase - drug effects | Mitochondria - drug effects | Cell Division - drug effects | HL-60 Cells | Leukemia, Myeloid, Acute - drug therapy | Indoles - pharmacology | Resting Phase, Cell Cycle - drug effects | Cell Proliferation - drug effects | Cell Cycle - drug effects | Leukemia | Kinases | Gene expression | Cancer therapies | Proteins | Mitochondria | Chemotherapy | Drug dosages | Binding sites | Cancer | Crystal structure | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
10. Full Text Local injection of lentivirus-delivered livinshrna suppresses lung adenocarcinoma growth by inducing a G0/G1 phase cell cycle arrest
by Chen, Yu-Sheng and Li, Hong-Ru and Miao, Yan and Chen, Wen-Ying and Li, You-Tang and Wang, Gui-Qing and Wu, Zheng-Cai
International Journal of Clinical and Experimental Pathology, ISSN 1936-2625, 2012, Volume 5, Issue 8, pp. 796 - 805
The inhibitor of apoptosis protein (IAP) plays an important role in tumorigenesis and may be a potential target for cancer therapy. Livin, which belongs to... 
Xenograft tumor model | RNA interference | Lung adenocarcinoma | Cell cycle | Livin gene | CANCER CELLS | cell cycle | SURVIVIN | lung adenocarcinoma | PATHOLOGY | COLON-CANCER | APOPTOSIS PROTEIN FAMILY | ONCOLOGY | LIVIN/ML-IAP | ACUTE LYMPHOBLASTIC-LEUKEMIA | SPLICING VARIANTS | PROGNOSTIC-SIGNIFICANCE | xenograft tumor model | INHIBITOR | EXPRESSION | Adenocarcinoma - pathology | Cell Proliferation | Adenocarcinoma of Lung | Inhibitor of Apoptosis Proteins - genetics | Humans | Lung Neoplasms - pathology | Male | RNA Interference | Female | Lentivirus - genetics | Adenocarcinoma - genetics | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | Lung Neoplasms - genetics | Gene Transfer Techniques | Down-Regulation | RNA, Small Interfering - pharmacology | Gene Silencing | Xenograft Model Antitumor Assays | Animals | Mice, Nude | Adaptor Proteins, Signal Transducing - genetics | Cell Line, Tumor | Resting Phase, Cell Cycle - drug effects | Mice | Mice, Inbred BALB C | G1 Phase Cell Cycle Checkpoints - drug effects | Apoptosis | Genetic Therapy - methods
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
11. Full Text RNA-binding proteins ZFP36L1 and ZFP36L2 promote cell quiescence
by Galloway, Alison and Saveliev, Alexander and Łukasiak, Sebastian and Hodson, Daniel J and Bolland, Daniel and Balmanno, Kathryn and Ahlfors, Helena and Monzón-Casanova, Elisa and Mannurita, Sara Ciullini and Bell, Lewis S and Andrews, Simon and Díaz-Muñoz, Manuel D and Cook, Simon J and Corcoran, Anne and Turner, Martin
Science, ISSN 0036-8075, 04/2016, Volume 352, Issue 6284, pp. 453 - 459
Progression through the stages of lymphocyte development requires coordination of the cell cycle. Such coordination ensures genomic integrity while cells... 
TRANSITION | RECOMBINATION | CYCLIN-E | MESSENGER-RNA | TRISTETRAPROLIN | MULTIDISCIPLINARY SCIENCES | S PHASE | COORDINATION | PROLIFERATION | TUMORIGENESIS | PROGRESSION | RNA-Binding Proteins - genetics | RNA, Messenger - metabolism | Cyclins - metabolism | Tristetraprolin - physiology | Conserved Sequence | Transcription, Genetic | G1 Phase - genetics | Nuclear Proteins - genetics | B-Lymphocytes - cytology | Pre-B Cell Receptors | RNA-Binding Proteins - physiology | Mice, Inbred C57BL | Gene Expression Regulation | Selection, Genetic | V(D)J Recombination | G1 Phase - physiology | S Phase - genetics | Mice, Knockout | Immunoglobulin mu-Chains - genetics | Animals | Resting Phase, Cell Cycle - genetics | Tristetraprolin - genetics | Resting Phase, Cell Cycle - physiology | S Phase - physiology | Mice | Nuclear Proteins - physiology | B cells | RNA | Binding proteins | Protein binding | Cell interaction | Observations | Lymphocytes | Health aspects | Proteins | Antigens | Ribonucleic acid--RNA | Genes
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
12. Full Text Isolation of Quiescent and Nonquiescent Cells from Yeast Stationary-Phase Cultures
by Chris Allen and Sabrina Büttner and Anthony D. Aragon and Jason A. Thomas and Osorio Meirelles and Jason E. Jaetao and Don Benn and Stephanie W. Ruby and Marten Veenhuis and Frank Madeo and Margaret Werner-Washburne
The Journal of Cell Biology, ISSN 0021-9525, 7/2006, Volume 174, Issue 1, pp. 89 - 100
Quiescence is the most common and, arguably, most poorly understood cell cycle state. This is in part because pure populations of quiescent cells are typically... 
Yeasts | Cell culture techniques | Scars | Mother cells | Cell cycle | Stem cells | Cultured cells | Cells | Daughter cells | Apoptosis | APOPTOSIS | REGULATOR | COLONIES | SUPEROXIDE | DNA | GENE-EXPRESSION | STRESS | IDENTIFICATION | SACCHAROMYCES-CEREVISIAE | EXIT | CELL BIOLOGY | Microscopy - methods | Reactive Oxygen Species - metabolism | Mitosis | Cells, Cultured | Cell Separation - methods | Saccharomyces cerevisiae - cytology | Flow Cytometry | Resting Phase, Cell Cycle - physiology | Sensitivity and Specificity | Glucose - chemistry | Cell Cycle - physiology | Centrifugation, Density Gradient - methods | Apoptosis - physiology | Saccharomyces cerevisiae - isolation & purification | Cell culture | Research | Yeast fungi
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
13. Full Text Global analysis of proliferation and cell cycle gene expression in the regulation of hematopoietic stem and progenitor cell fates
by Passegué, Emmanuelle and Wagers, Amy J and Giuriato, Sylvie and Anderson, Wade C and Weissman, Irving L
Journal of Experimental Medicine, ISSN 0022-1007, 12/2005, Volume 202, Issue 11, pp. 1599 - 1611
Knowledge of the molecular networks controlling the proliferation and fate of hematopoietic stem cells (HSC) is essential to understand their function in... 
MEDICINE, RESEARCH & EXPERIMENTAL | COLONY-STIMULATING FACTOR | MOBILIZATION | IN-VIVO | BONE-MARROW | MICE | DIFFERENTIATION | IMMUNOLOGY | BLOOD | Leukemia - pathology | Cell Separation | Humans | Gene Expression Regulation - physiology | Hematopoietic Stem Cell Transplantation | Leukemia - therapy | Gene Expression Profiling | Leukemia - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Cell Transformation, Neoplastic - metabolism | Animals | Hematopoietic Stem Cells - cytology | Resting Phase, Cell Cycle - physiology | Graft Survival - physiology | Hematopoietic Stem Cells - physiology | Hematopoiesis - physiology | Mice | Hematopoietic Stem Cell Mobilization | Cell Transformation, Neoplastic - pathology | Cell Differentiation - physiology
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
14. Full Text MicroRNA-149 Inhibits Proliferation and Cell Cycle Progression through the Targeting of ZBTB2 in Human Gastric Cancer
by Wang, Ying and Zheng, Xiushan and Zhang, Zhiyong and Zhou, Jinfeng and Zhao, Guohong and Yang, Jianjun and Xia, Limin and Wang, Rui and Cai, Xiqiang and Hu, Hao and Zhu, Cailin and Nie, Yongzhan and Wu, Kaichun and Zhang, Dexin and Fan, Daiming
PLoS ONE, ISSN 1932-6203, 10/2012, Volume 7, Issue 10, p. e41693
An increasing body of evidence indicates that miR-149 can both suppress and promote tumor growth depending on the tumor type. However, the role of miR-149 in... 
SUPPRESSOR | OVEREXPRESSION | REGULATOR | PATHWAY | MULTIDISCIPLINARY SCIENCES | EXPRESSION PROFILES | P53 | Immunohistochemistry | Stomach Neoplasms - genetics | Cell Line | Cell Proliferation | Down-Regulation | Humans | Gene Expression Regulation, Neoplastic | 3' Untranslated Regions - genetics | Repressor Proteins - genetics | Stomach Neoplasms - metabolism | MicroRNAs - metabolism | Stomach Neoplasms - pathology | Binding Sites - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Resting Phase, Cell Cycle - genetics | Base Sequence | Cell Line, Tumor | Cell Cycle Checkpoints - genetics | MicroRNAs - genetics | G1 Phase - genetics | Mutation | Repressor Proteins - metabolism | MicroRNA | Tumor proteins | Stomach cancer | Cell cycle | Cancer | Cell proliferation | Thoracic surgery | Disease | Laboratories | Epithelial cells | p53 Protein | Biology | mRNA | Retailing | Tissues | Hepatitis | Cell growth | Transfection | Physiology | Gastric cancer | G1 phase | MiRNA | Melanoma | Gene expression | Ribonucleic acid--RNA | Cyclin-dependent kinase inhibitor p21 | Hospitals | 3' Untranslated regions | MicroRNAs | Cell lines | Tumor suppressor genes | Prostate | Binding sites | Apoptosis | Tumors | RNA | Ribonucleic acid
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights