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Journal Article
Autophagy, ISSN 1554-8627, 08/2016, Volume 12, Issue 8, pp. 1272 - 1291
Journal Article
Circulation Research, ISSN 0009-7330, 04/2015, Volume 116, Issue 8, pp. 1312 - 1323
Rationale: Matrix vesicles (MVs), secreted by vascular smooth muscle cells (VSMCs), form the first nidus for mineralization and fetuin-A, a potent circulating... 
exosomes | extracellular matrix | vascular calcification | CALCIPROTEIN PARTICLES | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTEIN | RETICULOCYTE MATURATION | EXTRACELLULAR CALCIUM | MATRIX VESICLES | HUMAN ATHEROSCLEROTIC PLAQUES | MULTIVESICULAR ENDOSOMES | ARTERIAL CALCIFICATION | PERIPHERAL VASCULAR DISEASE | CHRONIC KIDNEY-DISEASE | HEMATOLOGY | FETUIN-A | Exosomes - metabolism | Sphingomyelin Phosphodiesterase - genetics | Muscle, Smooth, Vascular - metabolism | Calcium - metabolism | Humans | Middle Aged | Vascular Calcification - genetics | Myocytes, Smooth Muscle - pathology | Secretory Vesicles - metabolism | Exosomes - pathology | Male | Exocytosis | Case-Control Studies | Young Adult | Intercellular Signaling Peptides and Proteins - metabolism | Vascular Calcification - metabolism | Transfection | RNA Interference | Time Factors | Sphingomyelin Phosphodiesterase - metabolism | Vascular Calcification - physiopathology | Adult | Female | Proteomics - methods | Tetraspanins - metabolism | Myocytes, Smooth Muscle - metabolism | Exosomes - secretion | Cytokines - metabolism | Cells, Cultured | Vascular Calcification - pathology | Myocytes, Smooth Muscle - secretion | Protein Transport | Secretory Vesicles - secretion | Muscle, Smooth, Vascular - pathology | Adolescent | alpha-2-HS-Glycoprotein - metabolism | Secretory Vesicles - pathology | Muscle, Smooth, Vascular - secretion | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2004, Volume 101, Issue 26, pp. 9683 - 9688
Journal Article
EMBO reports, ISSN 1469-221X, 01/2010, Volume 11, Issue 1, pp. 45 - 51
Autophagy is the cellular homeostatic pathway that delivers large cytosolic materials for degradation in the lysosome. Recent evidence indicates that autophagy... 
mitophagy | Nix | LC3 | GABARAP | selective autophagy | Selective autophagy | Mitophagy | APOPTOSIS | PROTEIN | RETICULOCYTE MATURATION | UBIQUITIN | BNIP3 | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL-DEATH | CELL BIOLOGY | STRUCTURAL BASIS | DEGRADATION | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Humans | Cercopithecus aethiops | Molecular Sequence Data | Substrate Specificity | Autophagy - physiology | Mitochondrial Proteins - genetics | Proto-Oncogene Proteins - chemistry | Mitochondrial Proteins - metabolism | Tumor Suppressor Proteins - chemistry | Tumor Suppressor Proteins - genetics | Membrane Proteins - metabolism | Binding Sites | Proto-Oncogene Proteins - metabolism | Amino Acid Sequence | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Cells, Cultured | Ubiquitin-Protein Ligases - metabolism | Proto-Oncogene Proteins - genetics | Mitochondria - metabolism | Saccharomyces cerevisiae Proteins - genetics | Blotting, Western | Amino Acid Motifs | Autophagy-Related Protein 8 Family | Animals | Membrane Proteins - chemistry | Reticulocytes - cytology | Mitochondrial Proteins - chemistry | Saccharomyces cerevisiae Proteins - metabolism | Protein Binding | Mice | Receptors, GABA-A - metabolism | COS Cells | Proteins | Mitochondria | Cellular biology | Cytoplasm | Index Medicus | Scientific Report
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 03/2005, Volume 280, Issue 11, pp. 10491 - 10500
Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) induces programmed cell death through the caspase activation cascade and translocation of... 
HEMATOPOIETIC-CELLS | DEATH RECEPTORS | IMMUNE-SYSTEM | BIOCHEMISTRY & MOLECULAR BIOLOGY | MITOCHONDRIA | MEDIATED APOPTOSIS | DEFICIENT MICE | CYTOCHROME-C RELEASE | BCL-2 FAMILY | TNF | BH3-ONLY PROTEINS | Caspase 8 | Leukemia - pathology | Membrane Glycoproteins - metabolism | Humans | RNA, Messenger - metabolism | TNF-Related Apoptosis-Inducing Ligand | Caspase 3 | Protein Synthesis Inhibitors - pharmacology | Binding Sites | Amino Acid Sequence | bcl-X Protein | Jurkat Cells | Down-Regulation | Rats | Neoplasm Proteins - chemistry | Reticulocytes - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Plasmids - metabolism | Tumor Necrosis Factor-alpha - chemistry | Cell Line, Tumor | Aspartic Acid - chemistry | HeLa Cells | Mutation | Proteasome Endopeptidase Complex - metabolism | RNA, Small Interfering - metabolism | Tumor Necrosis Factor-alpha - metabolism | Immunoprecipitation | Membrane Glycoproteins - chemistry | BH3 Interacting Domain Death Agonist Protein | Molecular Sequence Data | Neoplasm Proteins - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Caspases - metabolism | Cell Death | Proto-Oncogene Proteins c-bcl-2 - chemistry | Protein Structure, Tertiary | Mutagenesis, Site-Directed | Cell Survival | Cytochromes c - metabolism | Glutathione Transferase - metabolism | Mitochondria - metabolism | Cycloheximide - pharmacology | Point Mutation | Animals | Apoptosis Regulatory Proteins | Carrier Proteins - metabolism | Myeloid Cell Leukemia Sequence 1 Protein | Microscopy, Fluorescence | Apoptosis | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2012, Volume 109, Issue 50, pp. 20590 - 20595
There is no effective treatment for the cardiomyopathy of the most common autosomal recessive ataxia, Friedreich ataxia (FA). This disease is due to decreased... 
Heart | Protein metabolism | Energy metabolism | Diet | Liver | Cardiomyopathies | Mice | Down regulation | Ferritins | Skeletal muscle | Ferroportin 1 | Hemojuvelin | Transferrin receptor 1 | Heme oxygenase | heme oxygenase | MECHANISM | HEME-SYNTHESIS | MULTIDISCIPLINARY SCIENCES | CARDIOMYOPATHY | TRAFFICKING | hemojuvelin | RETICULOCYTES | ferroportin 1 | SUCCINYLACETONE | METABOLISM | transferrin receptor 1 | TISSUES | FERRITIN | PROTEINS | Mitochondria, Heart - metabolism | Creatine Kinase, MM Form - metabolism | Friedreich Ataxia - pathology | Humans | Cardiomegaly - pathology | Iron, Dietary - administration & dosage | Iron-Binding Proteins - metabolism | Friedreich Ataxia - genetics | Mice, Mutant Strains | Myocardium - metabolism | Disease Models, Animal | Iron - blood | Myocardium - ultrastructure | Microscopy, Electron, Transmission | Friedreich Ataxia - metabolism | Creatine Kinase, MM Form - genetics | Signal Transduction | Spectroscopy, Mossbauer | Liver - metabolism | Iron - metabolism | Mice, Knockout | Animals | Cardiomegaly - prevention & control | Iron-Binding Proteins - antagonists & inhibitors | Iron-Binding Proteins - genetics | Iron Regulatory Protein 2 - metabolism | Cardiomegaly - metabolism | Physiological aspects | Ataxia | Development and progression | Iron in the body | Metabolism | Health aspects | Phosphotransferases | Oxidative stress | Mitochondria | Transmission electron microscopy | Rodents | Iron | Mutation | Gene expression | Index Medicus | Biological Sciences
Journal Article
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2012, Volume 143, Issue 3, pp. 698 - 707.e4
Journal Article
Journal Article