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Proceedings of the National Academy of Sciences, ISSN 0027-8424, 08/2011, Volume 108, Issue 32, pp. E440 - E449
Astrocytes regulate synaptic connectivity in the CNS through secreted signals. Here we identified two astrocyte-secreted proteins, hevin and SPARC, as... 
Extracellular matrix | Synaptic cleft 1 | Matricellular protein | Osteonectin | SPARC-Like | osteonectin | SC1 | synaptic cleft 1 | RAT | EXTRACELLULAR-MATRIX GLYCOPROTEIN | INTEGRIN | MULTIDISCIPLINARY SCIENCES | RECEPTOR | matricellular protein | CELL-SURVIVAL | SUPERIOR COLLICULUS | SPARC-Like 1 | EXPRESSION | extracellular matrix | SUPERFICIAL LAYERS | BRAIN | Central Nervous System - ultrastructure | Central Nervous System - metabolism | Extracellular Matrix Proteins - deficiency | Humans | Superior Colliculi - ultrastructure | Calcium-Binding Proteins - antagonists & inhibitors | Culture Media, Conditioned - pharmacology | Retinal Ganglion Cells - metabolism | Retinal Ganglion Cells - cytology | Osteonectin - deficiency | Synapses - metabolism | HEK293 Cells | Neurogenesis - drug effects | Extracellular Matrix Proteins - metabolism | Astrocytes - cytology | Calcium-Binding Proteins - chemistry | Calcium-Binding Proteins - metabolism | Protein Structure, Tertiary | Astrocytes - drug effects | Retinal Ganglion Cells - ultrastructure | Synapses - drug effects | Extracellular Matrix Proteins - chemistry | Superior Colliculi - metabolism | Extracellular Matrix Proteins - antagonists & inhibitors | Superior Colliculi - drug effects | Osteonectin - metabolism | Rats | Calcium-Binding Proteins - deficiency | Synapses - ultrastructure | Osteonectin - chemistry | Rats, Sprague-Dawley | Astrocytes - ultrastructure | Animals | Central Nervous System - cytology | Superior Colliculi - cytology | Mice | Retinal Ganglion Cells - drug effects | Astrocytes - secretion | Proteins | Brain | Rodents | Cells | Retinal ganglion cells | Synaptogenesis | Astrocytes | Neural networks | Central nervous system | Superior colliculus | Synapses | Index Medicus | Biological Sciences | PNAS Plus
Journal Article
Journal Article
Current Biology, ISSN 0960-9822, 02/2014, Volume 24, Issue 3, pp. 310 - 315
Journal Article
Neuron, ISSN 0896-6273, 12/2016, Volume 92, Issue 6, pp. 1294 - 1307
Journal Article
Glia, ISSN 0894-1491, 11/2018, Volume 66, Issue 11, pp. 2366 - 2384
Excitotoxicity is well known in the neuronal death in the brain and is also linked to neuronal damages in the retina. Recent accumulating evidence show that... 
cytokines | retinal ganglion cells | tumor necrosis factor | excitotoxicity | microglia | SMALL-MOLECULE | D-ASPARTATE RECEPTORS | AQUEOUS-HUMOR | FACTOR-KAPPA-B | EXPERIMENTAL GLAUCOMA | NEUROSCIENCES | FACTOR-ALPHA | TUMOR-NECROSIS-FACTOR | NEURONAL NMDA RECEPTORS | MOUSE MODEL | GLUTAMATE LEVELS | Tumor Necrosis Factor-alpha - metabolism | Optic Nerve Injuries - chemically induced | Male | Nerve Degeneration - chemically induced | N-Methylaspartate - pharmacology | Microglia - physiology | Organic Chemicals - pharmacology | Microfilament Proteins - metabolism | Cell Death - drug effects | Cytokines - genetics | Microfilament Proteins - genetics | Animals, Newborn | Calcium-Binding Proteins - metabolism | Retinal Ganglion Cells - ultrastructure | Retinal Ganglion Cells - physiology | Cytokines - metabolism | Microglia - drug effects | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Excitatory Amino Acid Agonists - pharmacology | Transcription Factor Brn-3A - metabolism | Gene Expression Regulation - drug effects | Pyrroles - pharmacology | Animals | Cell Death - physiology | Signal Transduction - drug effects | Transcription Factor Brn-3A - genetics | Microglia - ultrastructure | Aminopyridines - pharmacology | Mice | Retinal Ganglion Cells - drug effects | Calcium-Binding Proteins - genetics | Medical research | Neurons | Cell death | Medicine, Experimental | Eye diseases | Biochemistry | Ganglion | Tumor necrosis factor receptors | Brain | Gene regulation | Retina | Interleukin 6 | Neurotoxicity | Neurodegeneration | Interleukin 1 | Minocycline | Tumor necrosis factor-TNF | Colony-stimulating factor | Degeneration | Inhibition | Damage accumulation | Pharmacology | Inflammation | siRNA | Tumor necrosis factor-α | Gene expression | Ablation | Microglia | Retinal ganglion cells | Excitotoxicity | Brain damage | Cell migration | Apoptosis | Index Medicus
Journal Article