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Science, ISSN 0036-8075, 10/2018, Volume 362, Issue 6410, pp. 91 - 95
Journal Article
Nature, ISSN 0028-0836, 03/2013, Volume 495, Issue 7440, pp. 241 - 245
Journal Article
Modern Pathology, ISSN 0893-3952, 2011, Volume 24, Issue 1, pp. 82 - 89
Cellular angiofibroma represents a rare benign mesenchymal tumor, occurring mainly in the superficial soft tissue of the genital region. The involvement of... 
genital mesenchymal tumors | soft tissue | spindle cell lipoma | mammary-type myofibroblastoma | cellular angiofibroma | MESENCHYMAL TUMOR | ANGIOMYOFIBROBLASTOMA | FEMALE GENITAL-TRACT | SARCOMATOUS TRANSFORMATION | PATHOLOGY | SOFT-TISSUE | BENIGN NEOPLASM DISTINCT | SOLITARY FIBROUS TUMOR | AGGRESSIVE ANGIOMYXOMA | CLINICOPATHOLOGICAL ANALYSIS | SUPERFICIAL ANGIOMYXOMA | Antigens, CD34 - metabolism | Humans | Middle Aged | Lipoma - genetics | Angiofibroma - metabolism | Male | Lipoma - pathology | Breast Neoplasms - metabolism | Angiofibroma - genetics | Urogenital Neoplasms - metabolism | Vulvar Neoplasms - pathology | Neoplasms, Muscle Tissue - metabolism | Gene Deletion | Vulvar Neoplasms - metabolism | Aged, 80 and over | Biomarkers, Tumor - metabolism | Adult | Female | Lipoma - metabolism | DNA, Neoplasm - analysis | Soft Tissue Neoplasms - genetics | Neoplasms, Muscle Tissue - pathology | Retinoblastoma Protein - metabolism | Soft Tissue Neoplasms - metabolism | Urogenital Neoplasms - pathology | In Situ Hybridization, Fluorescence | Neoplasms, Muscle Tissue - genetics | Breast Neoplasms - genetics | Vulvar Neoplasms - genetics | Breast Neoplasms - pathology | Retinoblastoma Protein - genetics | Urogenital Neoplasms - genetics | Angiofibroma - pathology | Soft Tissue Neoplasms - pathology | Aged | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 2013, Volume 497, Issue 7447, pp. 108 - 112
Cancers acquire resistance to systemic treatment as a result of clonal evolution and selection(1,2). Repeat biopsies to study genomic evolution as a result of... 
HETEROGENEITY | LUNG-CANCER | ACTIVATION | EVOLUTION | VARIANTS | AXL | MULTIDISCIPLINARY SCIENCES | TAMOXIFEN RESISTANCE | TUMOR-CELLS | MUTATIONS | Lung Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - genetics | Genomics | Humans | Ovarian Neoplasms - pathology | Lung Neoplasms - pathology | Antineoplastic Agents - therapeutic use | Ovarian Neoplasms - genetics | Neoplasms - genetics | DNA Mutational Analysis | Female | Antineoplastic Agents - pharmacology | DNA, Neoplasm - analysis | Ovarian Neoplasms - drug therapy | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Carcinoma, Non-Small-Cell Lung - genetics | Intercellular Signaling Peptides and Proteins - genetics | Breast Neoplasms - drug therapy | Genome, Human - genetics | Neoplasms - drug therapy | Phosphatidylinositol 3-Kinases - genetics | Drug Resistance, Neoplasm - genetics | Exome - genetics | Breast Neoplasms - genetics | Class I Phosphatidylinositol 3-Kinases | Breast Neoplasms - pathology | Retinoblastoma Protein - genetics | Plasma - chemistry | Alleles | Carcinoma, Non-Small-Cell Lung - drug therapy | DNA, Neoplasm - genetics | Neoplasms - pathology | Mediator Complex Subunit 1 - genetics | Drug Resistance, Neoplasm - drug effects | Evolution, Molecular | Antimitotic agents | Care and treatment | Physiological aspects | Dosage and administration | Genetic aspects | Research | Nucleotide sequencing | Drug resistance | Antineoplastic agents | DNA sequencing | Blood plasma | Cancer | Plasma | Biopsy | Breast cancer | Genomes | Mutation | Patients | Deoxyribonucleic acid--DNA | Tumors | Index Medicus
Journal Article
The EMBO Journal, ISSN 0261-4189, 01/2010, Volume 29, Issue 1, pp. 222 - 235
Journal Article
Nature Communications, ISSN 2041-1723, 2015, Volume 6, Issue 1, pp. 6377 - 6377
Tyrosine kinase inhibitors are effective treatments for non-small-cell lung cancers (NSCLCs) with epidermal growth factor receptor (EGFR) mutations. However,... 
GROWTH-FACTOR RECEPTOR | DRUG-SENSITIVITY | GEFITINIB | AMPLIFICATION | MODELS | GENE | MULTIDISCIPLINARY SCIENCES | MUTATION | ACQUIRED-RESISTANCE | KINASE INHIBITORS | CHEMOTHERAPY | Lung Neoplasms - drug therapy | Recurrence | Adenocarcinoma - pathology | Adenocarcinoma of Lung | Humans | Lung Neoplasms - metabolism | ErbB Receptors - genetics | Gene Expression Regulation, Neoplastic | bcl-X Protein - genetics | Lung Neoplasms - pathology | Small Cell Lung Carcinoma - drug therapy | Small Cell Lung Carcinoma - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Adenocarcinoma - metabolism | Adenocarcinoma - genetics | Antineoplastic Agents - pharmacology | Lung - metabolism | Gefitinib | ErbB Receptors - deficiency | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Lung - pathology | Aniline Compounds - pharmacology | ErbB Receptors - antagonists & inhibitors | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Retinoblastoma Protein - deficiency | Small Cell Lung Carcinoma - genetics | Sulfonamides - pharmacology | Adenocarcinoma - drug therapy | Disease Progression | Drug Resistance, Neoplasm - genetics | Small Cell Lung Carcinoma - pathology | Afatinib | Retinoblastoma Protein - genetics | Erlotinib Hydrochloride - pharmacology | Lung - drug effects | Cell Line, Tumor | Protein Kinase Inhibitors - pharmacology | Carcinoma, Non-Small-Cell Lung - drug therapy | Mutation | Quinazolines - pharmacology | bcl-X Protein - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | Drug Resistance, Neoplasm - drug effects | Index Medicus
Journal Article
Cell, ISSN 0092-8674, 2005, Volume 123, Issue 6, pp. 1001 - 1011
Our understanding of cancer has largely come from the analysis of aberrations within the tumor cell population. Yet it is increasingly clear that the tumor... 
PROSTATE | CELLS | TARGETED DISRUPTION | UROGENITAL SINUS | CARCINOGENESIS | MICRODISSECTED STROMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | G CONTROL | TUMOR-SUPPRESSOR GENE | CANCER STROMA | BREAST | CELL BIOLOGY | Prostatic Neoplasms - metabolism | Adenocarcinoma - pathology | Cell Proliferation | Epithelial Cells - metabolism | S100 Calcium-Binding Protein A4 | Stromal Cells - pathology | Male | Prostate - metabolism | Tumor Suppressor Protein p53 - genetics | Loss of Heterozygosity - genetics | Prostate - pathology | Keratins - analysis | Adenocarcinoma - metabolism | Prostatic Neoplasms - genetics | Actins - analysis | Gene Deletion | Adenocarcinoma - genetics | Disease Models, Animal | Fibroblasts - metabolism | Prostatic Neoplasms - pathology | Antigens, Polyomavirus Transforming - genetics | Connective Tissue - pathology | Paracrine Communication | Retinoblastoma Protein - metabolism | Stromal Cells - metabolism | Tumor Suppressor Protein p53 - metabolism | Epithelial Cells - pathology | Genotype | Mice, Transgenic | Fibroblasts - pathology | Mutation - genetics | S100 Proteins - analysis | Mice, Knockout | Animals | Models, Biological | Keratin-8 | Mice | Genetic research | Tumor suppressor genes | Research | Epithelial tumors | Gene mutations | Tumor proteins | Prostate cancer | Analysis | Tumors | Index Medicus
Journal Article
The Journal of Pathology, ISSN 0022-3417, 03/2017, Volume 241, Issue 4, pp. 488 - 500
Journal Article
Journal Article