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humans (353) 353
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retinoblastoma-like protein p107 (252) 252
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Journal Article
Journal Article
STEM CELLS, ISSN 1066-5099, 05/2014, Volume 32, Issue 5, pp. 1323 - 1336
Thermogenic (beige and brown) adipocytes protect animals against obesity and metabolic disease. However, little is known about the mechanisms that commit stem... 
Beige or brite adipocytes | Brown adipocytes | Lineage commitment | p107 | PRDM16 | Mesenchymal stem cells | PROGENITOR CELLS | DIET-INDUCED THERMOGENESIS | ADULT HUMANS | BROWN ADIPOSE-TISSUE | UNCOUPLING PROTEIN GENE | CELL & TISSUE ENGINEERING | CELL BIOLOGY | WHITE FAT | ENERGY-EXPENDITURE | RETINOBLASTOMA PROTEIN | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | TRANSCRIPTIONAL CONTROL | HEMATOLOGY | TRANSGENIC MICE | Adipocytes, Brown - metabolism | Adipose Tissue, White - metabolism | Adipocytes - cytology | Retinoblastoma-Like Protein p107 - genetics | Stem Cells - cytology | Stem Cells - metabolism | DNA-Binding Proteins - metabolism | Cell Differentiation - genetics | Mesenchymal Stromal Cells - cytology | Adipose Tissue, White - cytology | Cell Lineage - genetics | Fibroblasts - metabolism | Gene Expression | Retinoblastoma-Like Protein p107 - metabolism | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Mice, Inbred C3H | Mice, Knockout | Transcription Factors - metabolism | Adipose Tissue, Brown - cytology | Animals | Embryo, Mammalian - cytology | Adipocytes - metabolism | Adipose Tissue, Brown - metabolism | Fibroblasts - cytology | Mice, Inbred BALB C | Adipocytes, Brown - cytology | Stem cells | Medical research | Adipocytes | Metabolic disorders
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2013, Volume 288, Issue 34, pp. 24809 - 24824
Melanoma antigen-A11 (MAGE-A11) is a low-abundance, primate-specific steroid receptor coregulator in normal tissues of the human reproductive tract that is... 
BIOCHEMISTRY & MOLECULAR BIOLOGY | ANDROGEN-RECEPTOR | PROSTATE-CANCER | ADENOVIRUS E1A | TUMOR-SUPPRESSOR | CELL-CYCLE | MOLECULAR-CLONING | DEPENDENT KINASE INHIBITOR | VIRAL ONCOPROTEINS | TRANSCRIPTION FACTOR | RECEPTOR COREGULATOR MAGE-11 | Prostatic Neoplasms - metabolism | Humans | E1A-Associated p300 Protein - genetics | Cercopithecus aethiops | Male | Retinoblastoma-Like Protein p107 - genetics | E1A-Associated p300 Protein - metabolism | Neoplasm Proteins - metabolism | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Phosphorylation - genetics | Prostatic Neoplasms - genetics | Adenovirus E1A Proteins - metabolism | Receptors, Androgen - biosynthesis | Antigens, Neoplasm - metabolism | Transcription, Genetic | Protein Stability | Neoplasm Proteins - genetics | Antigens, Neoplasm - genetics | Prostatic Neoplasms - pathology | Retinoblastoma-Like Protein p107 - metabolism | E2F1 Transcription Factor - metabolism | Animals | Receptors, Androgen - genetics | Adenovirus E1A Proteins - genetics | HeLa Cells | E2F1 Transcription Factor - genetics | COS Cells | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Gene Regulation | Gene Transcription | Melanoma Antigen-A11 | Androgen Receptor | E2F Transcription Factor | Retinoblastoma (Rb) | Steroid Receptor Coregulator | Cell Cycle Regulation | Prostate Cancer | p107 | MAGE-A11
Journal Article
Journal Article
Journal Article
Journal Article
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Journal Article
Journal Article
Oncogene, ISSN 0950-9232, 01/2014, Volume 33, Issue 5, pp. 556 - 566
Retinoblastoma (Rb) protein is a tumor suppressor that is dysregulated in a majority of human cancers. Rb functions to inhibit cell cycle progression in part... 
Metabolism | Cancer | Glutamine | Retinoblastoma Protein | Tumor Suppressor | retinoblastoma protein | APOPTOSIS | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | G CONTROL | tumor suppressor | CELL-PROLIFERATION | FACTOR-I | glutamine | CELL BIOLOGY | GLYCOLYSIS | INHIBITION | ONCOLOGY | GENETICS & HEREDITY | metabolism | cancer | GROWTH-FACTOR | ENERGY-METABOLISM | Cell Proliferation | Reactive Oxygen Species - metabolism | Glutamate-Cysteine Ligase - biosynthesis | Minor Histocompatibility Antigens | Glutamine - metabolism | Retinoblastoma-Like Protein p107 - genetics | Adenosine Triphosphate - biosynthesis | Oxygen - metabolism | Glutathione - biosynthesis | RNA Interference | Retinoblastoma-Like Protein p130 - metabolism | Fibroblasts | Biological Transport - genetics | Amino Acid Transport System ASC - genetics | Ketoglutaric Acids - metabolism | Glutaminase - biosynthesis | Promoter Regions, Genetic | Retinoblastoma-Like Protein p107 - metabolism | Retinoblastoma Protein - metabolism | Cells, Cultured | Amino Acid Transport System ASC - metabolism | Glutaminase - metabolism | Mice, Knockout | Animals | E2F3 Transcription Factor - biosynthesis | Cell Cycle | Retinoblastoma Protein - genetics | E2F3 Transcription Factor - metabolism | Mice | RNA, Small Interfering | Retinoblastoma-Like Protein p130 - genetics | Amino Acid Transport System ASC - biosynthesis | Glutaminase - genetics | Oncology, Experimental | Physiological aspects | Genetic research | Tumor suppressor genes | Genetic aspects | Research | Studies | Oncology | Tumors
Journal Article
Genes and Development, ISSN 0890-9369, 08/2011, Volume 25, Issue 16, pp. 1734 - 1745
The miR-17~92 cluster is a potent microRNA-encoding oncogene. Here, we show that miR-17~92 synergizes with loss of Rb family members to promote retinoblastoma.... 
MiR-17;92 | Retina | MicroRNA | Retinoblastoma | Cancer | Multigene Family | Retina - metabolism | Cell Proliferation | Retina - growth & development | Oligonucleotide Array Sequence Analysis | Retinoblastoma - genetics | Humans | Male | Retinoblastoma-Like Protein p107 - genetics | Gene Expression Profiling | Retina - embryology |