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Atherosclerosis, ISSN 0021-9150, 04/2018, Volume 271, pp. 36 - 44
Journal Article
Kidney International, ISSN 0085-2538, 12/2017, Volume 92, Issue 6, pp. 1444 - 1457
Journal Article
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 07/2018, Volume 19, Issue 7, p. 1918
All-trans-retinoic acid (ATRA) and 1 alpha,25-dihydroxyvitamin D (1,25D) are potent inducers of differentiation of myeloid leukemia cells. During myeloid... 
CCAAT-enhancer-binding proteins | Retinoic acid receptor α | Vitamin D receptor | Differentiation | Nuclear receptors | Acute myeloid leukemia | CEBP genes | TRANSCRIPTION FACTORS | retinoic acid receptor alpha | differentiation | BIOCHEMISTRY & MOLECULAR BIOLOGY | CCAAT/ENHANCER-BINDING PROTEINS | GRANULOCYTE | CHEMISTRY, MULTIDISCIPLINARY | MICE LACKING | BETA | EPSILON | nuclear receptors | LIVER NUCLEAR-PROTEIN | C/EBP-ALPHA | INDUCED DIFFERENTIATION | acute myeloid leukemia | vitamin D receptor | D-2 ANALOGS | Retinoic Acid Receptor alpha - genetics | CCAAT-Enhancer-Binding Protein-beta - genetics | Humans | Leukemia, Myeloid, Acute - metabolism | Retinoic Acid Receptor alpha - metabolism | Gene Silencing | Receptors, Calcitriol - metabolism | Receptors, Calcitriol - genetics | Blotting, Western | CCAAT-Enhancer-Binding Protein-beta - metabolism | Glyceraldehyde-3-Phosphate Dehydrogenase (Phosphorylating) - genetics | Glyceraldehyde-3-Phosphate Dehydrogenase (Phosphorylating) - metabolism | Flow Cytometry | CCAAT-Enhancer-Binding Protein-delta - genetics | HL-60 Cells | CCAAT-Enhancer-Binding Protein-delta - metabolism | Leukemia, Myeloid, Acute - genetics | Regulators | Transcription factors | Data points | Myeloid leukemia | Redundancy | Leukemia | Genes | Dihydroxyvitamin D | CCAAT/enhancer-binding protein | Tumor cell lines | Gene expression | Proteins | Vitamin D | Developmental stages | Cell lines | Vitamin D receptors | Retinoic acid | Inducers | trans-Retinoic acid | Index Medicus | retinoic acid receptor α
Journal Article
Journal Article
Oncogene, ISSN 0950-9232, 2018, Volume 38, Issue 14, pp. 2482 - 2500
Journal Article
Oncogene, ISSN 0950-9232, 11/2017, Volume 36, Issue 44, pp. 6074 - 6084
Journal Article
Journal of Steroid Biochemistry and Molecular Biology, ISSN 0960-0760, 03/2019, Volume 187, pp. 118 - 123
Journal Article
by Hsu, YT and Li, J and Wu, D and Sudhof, TC and Chen, L
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, ISSN 0027-8424, 04/2019, Volume 116, Issue 14, pp. 7113 - 7122
Homeostatic synaptic plasticity is a stabilizing mechanism engaged by neural circuits in response to prolonged perturbation of network activity. The... 
Hebbian plasticity | MULTIDISCIPLINARY SCIENCES | STRENGTH | mTOR signaling | RAR-ALPHA | TRANSLATION | retinoic acid receptor | enriched environment | MEMORY | LONG-TERM DEPRESSION | homeostatic synaptic plasticity | MENTAL-RETARDATION PROTEIN | LTD | DORSAL | PLASTICITY | Synaptic Transmission - physiology | TOR Serine-Threonine Kinases - metabolism | Retinoic Acid Receptor alpha - metabolism | Neuronal Plasticity - drug effects | Receptors, Retinoic Acid - metabolism | Hippocampus - metabolism | Learning - physiology | Animals | Neuronal Plasticity - physiology | Signal Transduction - drug effects | Homeostasis - physiology | Long-Term Potentiation - physiology | Protein Biosynthesis - drug effects | Synaptic Transmission - drug effects | Mice | Receptors, AMPA - metabolism | Tretinoin - pharmacology | TOR protein | Potentiation | Plasticity (homeostatic) | Enrichment | Circuits | Spatial discrimination learning | Glutamic acid receptors | Plasticity (neural) | Proteins | Learning | Receptors | Clonal deletion | Synaptic transmission | α-Amino-3-hydroxy-5-methyl-4-isoxazole propionic acid | Plasticity | Deletion | Long-term potentiation | Protein biosynthesis | Rapamycin | Perturbation | Plasticity (hippocampal) | Ablation | Signaling | Plasticity (synaptic) | Plasticity (Hebbian) | Acids | Protein synthesis | Neural networks | Retinoic acid | Hippocampus | Synapses | Spatial memory | Index Medicus | Biological Sciences | PNAS Plus
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 3896 - 13
Journal Article