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International Journal of Dermatology, ISSN 0011-9059, 03/2016, Volume 55, Issue 3, pp. 249 - 258
Nonmelanoma skin cancer (NMSC) is the most common cancer in patients and includes basal cell carcinoma (BCC) and squamous cell carcinoma (SCC). Treatments... 
CAPECITABINE THERAPY | GROWTH-FACTOR RECEPTOR | DOSE-ESCALATION | ORGAN TRANSPLANT RECIPIENTS | RETINOIC ACID | SQUAMOUS-CELL CARCINOMA | PREVENTION | PHASE-II | INHIBITOR | HEDGEHOG-PATHWAY | DERMATOLOGY | Capecitabine - administration & dosage | Skin Neoplasms - pathology | Receptor, IGF Type 1 - antagonists & inhibitors | Phthalazines - administration & dosage | Pyridines - administration & dosage | Skin Neoplasms - drug therapy | Administration, Oral | Carcinoma, Squamous Cell - pathology | Erlotinib Hydrochloride - administration & dosage | Carcinoma, Basal Cell - pathology | Antineoplastic Agents - administration & dosage | Hedgehog Proteins - antagonists & inhibitors | Tumor Burden | Molecular Targeted Therapy | Retinoids - administration & dosage | Carcinoma, Squamous Cell - drug therapy | Fluorouracil - administration & dosage | Anilides - administration & dosage | Biphenyl Compounds - administration & dosage | Podophyllotoxin - administration & dosage | Carcinoma, Basal Cell - drug therapy | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Quinazolines - administration & dosage | Podophyllotoxin - analogs & derivatives | Cancer patients | Care and treatment | Squamous cell carcinoma | Chemotherapy | Dermatology | Melanoma | Formulae, receipts, prescriptions | Dermatologic agents | Skin | Health aspects | Skin cancer | Cancer
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Japanese Journal of Clinical Oncology, ISSN 0368-2811, 04/2016, Volume 46, Issue 4, pp. 291 - 298
Epidermal growth factor receptor inhibition is a good target for the treatment of lung, colon, pancreatic and head and neck cancers. Epidermal growth factor... 
Minocycline | EGFR-TKI | Rash acneiform | Skin rash | 1ST-LINE TREATMENT | FACTOR-RECEPTOR GENE | OPEN-LABEL | METASTATIC COLORECTAL-CANCER | skin rash | CELL LUNG-CANCER | RANDOMIZED PHASE-3 TRIAL | TARGETED THERAPIES | ONCOLOGY | rash acneiform | minocycline | CLINICAL-IMPLICATIONS | PLACEBO-CONTROLLED TRIAL | ADVERSE EVENTS | Paronychia - prevention & control | Dermatologic Agents - administration & dosage | Humans | Antineoplastic Agents - therapeutic use | Administration, Cutaneous | Antineoplastic Agents - administration & dosage | Protein Kinase Inhibitors - adverse effects | Aspirin - administration & dosage | Sunscreening Agents - administration & dosage | Retinoids - administration & dosage | Antineoplastic Agents - adverse effects | Exanthema - chemically induced | Exanthema - prevention & control | Vitamin K - administration & dosage | Skin - pathology | Patient Education as Topic | Paronychia - chemically induced | Severity of Illness Index | Skin Diseases - chemically induced | Administration, Oral | Randomized Controlled Trials as Topic | Exanthema - epidemiology | Protein Kinase Inhibitors - administration & dosage | Quality of Life | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Skin - physiopathology | Anti-Bacterial Agents - administration & dosage | Skin - drug effects | Adrenal Cortex Hormones - administration & dosage | Protein-Tyrosine Kinases - antagonists & inhibitors | Index Medicus | Review
Journal Article
British Journal of Dermatology, ISSN 0007-0963, 04/2005, Volume 152, Issue 4, pp. 597 - 615
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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2006, Volume 103, Issue 31, pp. 11736 - 11741
We have demonstrated that cytokine thymic stromal lymphopoietin (TSLP), whose expression is rapidly induced upon keratinocyteselective ablation of retinoid X... 
T lymphocytes | Receptors | Ethanol | Vitamin D | Cytokines | Cholecalciferols | Keratinocytes | Skin | Mast cells | Inflammation | Retinoic acid receptor | Retinoid X receptor | Retinoic acid | Vitamin D receptor | retinoid X receptor | ALLERGIC INFLAMMATION | CELLS | MULTIDISCIPLINARY SCIENCES | skin | NUCLEAR RECEPTOR SUPERFAMILY | retinoic acid receptor | retinoic acid | GENE | ASTHMA | ABLATION | vitamin D receptor | 1,25-DIHYDROXYVITAMIN D-3 | MICE | ASSOCIATION | Skin - cytology | Retinoid X Receptor alpha - metabolism | Calcium - metabolism | Dermatologic Agents - administration & dosage | Humans | Calcitriol - adverse effects | Dermatologic Agents - adverse effects | Receptors, Calcitriol - genetics | Receptors, Retinoic Acid - genetics | Calcitriol - administration & dosage | Calcitriol - analogs & derivatives | Dermatitis, Atopic - chemically induced | Vitamins - immunology | Receptors, Retinoic Acid - agonists | Dermatitis, Atopic - immunology | Female | Cytokines - genetics | Dimerization | Skin - pathology | Cytokines - immunology | Retinoid X Receptor beta - metabolism | Vitamins - chemistry | Cells, Cultured | Retinoid X Receptor beta - genetics | Mice, Transgenic | Receptors, Retinoic Acid - metabolism | Keratinocytes - cytology | Receptors, Calcitriol - metabolism | Keratinocytes - immunology | Mice, Knockout | Retinoid X Receptor alpha - agonists | Vitamins - adverse effects | Animals | Retinoid X Receptor alpha - genetics | Mice | Dermatologic Agents - immunology | Administration, Topical | Retinoids - immunology | Skin - drug effects | Vitamins - administration & dosage | Calcitriol - immunology | Retinoid X Receptor beta - agonists | Physiological aspects | Causes of | Vitamin D3 | Atopic dermatitis | Tretinoin | Dermatitis, Atopic | Calcium | Calcitriol | Retinoid X Receptor alpha | Retinoids | Life Sciences | Retinoid X Receptor beta | Receptors, Retinoic Acid | Biochemistry, Molecular Biology | Vitamins | Dermatologic Agents | Receptors, Calcitriol | Molecular biology | Biological Sciences
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