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Science, ISSN 0036-8075, 03/2012, Volume 335, Issue 6075, pp. 1503 - 1506
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2008, Volume 283, Issue 36, pp. 24770 - 24780
Accumulation of indigestible lipofuscin and decreased mitochondrial energy production are characteristic age-related changes of post-mitotic retinal pigment... 
LIPOFUSCIN FLUOROPHORE | RAT | DYSTROPHY | DNA | BIOCHEMISTRY & MOLECULAR BIOLOGY | MACULAR DEGENERATION | COMPONENT | RPE CELLS | N-RETINYLIDENE ETHANOLAMINE | BINDING | PHOTORECEPTORS | Retinoids - metabolism | Epithelial Cells - metabolism | Lipofuscin - pharmacology | Antioxidants - metabolism | Humans | Lysosomes - genetics | Rats, Long-Evans | Adenosine Triphosphate - genetics | Phagocytosis - genetics | Membrane Potential, Mitochondrial - drug effects | Mitosis - genetics | Oxidative Phosphorylation - drug effects | DNA, Mitochondrial - genetics | Lysosomes - metabolism | Aging - genetics | Cell Death - genetics | Mitochondria - genetics | Adenosine Triphosphate - metabolism | Membrane Potential, Mitochondrial - genetics | Pyruvic Acid - metabolism | Lysosomes - pathology | Cell Death - drug effects | Cell Line | Phagocytosis - drug effects | DNA, Mitochondrial - metabolism | Rats | Epithelial Cells - pathology | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - pathology | Aging - pathology | Pigment Epithelium of Eye - metabolism | Macular Degeneration - metabolism | Point Mutation | Pyridinium Compounds - pharmacology | Animals | Mitosis - drug effects | Macular Degeneration - genetics | Glucose - metabolism | Pigment Epithelium of Eye - pathology | Pyridinium Compounds - metabolism | Lipofuscin - metabolism | Retinoids - pharmacology | Aging - metabolism | Macular Degeneration - pathology | Index Medicus | Molecular Basis of Cell and Developmental Biology
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2015, Volume 10, Issue 11, pp. e0143852 - e0143852
Understanding the mechanisms in the generation of neural stem cells from pluripotent stem cells is a fundamental step towards successful management of... 
HUMAN ES | RETINOIC ACID | NUCLEAR-FACTOR | MULTIDISCIPLINARY SCIENCES | SWITCH CONTROLS | TRANSCRIPTION | GROWTH | NEURONS | RECEPTOR | PROLIFERATION | DIFFERENTIATION | Tyrosine 3-Monooxygenase - metabolism | Retinoid X Receptor alpha - metabolism | TOR Serine-Threonine Kinases - metabolism | Homeodomain Proteins - metabolism | Humans | tau Proteins - metabolism | Stem Cells - cytology | Stem Cells - metabolism | Dopaminergic Neurons - cytology | Morphogenesis | Cell Differentiation - genetics | Multiprotein Complexes - metabolism | Parkinson Disease - metabolism | Trophoblasts - cytology | Proto-Oncogene Proteins c-akt - metabolism | Calcium Signaling | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Disease Models, Animal | Cell Line | Nuclear Pore Complex Proteins - metabolism | Gene Expression Regulation | Axons - metabolism | Rats | Receptors, Retinoic Acid - metabolism | Parkinson Disease - genetics | Glycogen Synthase Kinase 3 - metabolism | beta Catenin - metabolism | Protein Transport | Transcription Factors - metabolism | Cyclic AMP Response Element-Binding Protein - genetics | Tyrosine 3-Monooxygenase - genetics | Animals | Receptor, trkA - metabolism | Signal Transduction - drug effects | Cell Differentiation - drug effects | Models, Biological | Cyclic AMP Response Element-Binding Protein - metabolism | Protein Binding | Stem Cells - drug effects | GTP-Binding Proteins - metabolism | Stem cell research | Analysis | Influence | Cellular signal transduction | G proteins | Research | Gene expression | Flow cytometry | Parkinson's disease | Substantia nigra | Genes | Stem cell transplantation | Kinases | Tyrosine 3-monooxygenase | Proteins | β-catenin | Signal transduction | Pathways | Rodents | TrkA protein | TrkA receptors | Movement disorders | Dopamine receptors | Tyrosine | Dopamine | Neurodegenerative diseases | Neurons | Axonogenesis | Hydroxylase | Implantation | Progenitor cells | Neurological diseases | Regeneration | Signaling | Brain-derived neurotrophic factor | Microscopy | Stem cells | Regulatory mechanisms (biology) | Cells (biology) | Epigenetics | Neural stem cells | Protein interaction | Differentiation | Retinoic acid | Pluripotency | Index Medicus
Journal Article
Cellular Physiology and Biochemistry, ISSN 1015-8987, 04/2018, Volume 46, Issue 1, pp. 187 - 202
Background/Aims: All-trans retinoic acid (ATRA) has protective effects against obesity and metabolic syndrome. We here aimed to gain further insight into the... 
Myokines | Obesity | AMPK | Muscle | PPARβ/δ | Nuclear receptors | Intramyocellular lipids | Retinoids/Vitamin A | Fatty acid/oxidation | PPAR beta/delta | PHYSIOLOGY | LIPID-METABOLISM | VITAMIN-A SUPPLEMENTATION | ACTIVATED RECEPTOR | CELL BIOLOGY | INSULIN-RESISTANCE | Retinoids/vitamin A | GENE-EXPRESSION | LEPTIN EXPRESSION | UNCOUPLING PROTEIN-3 | MICE | ADIPOSE-TISSUE | WHITE ADIPOCYTES | AMP-Activated Protein Kinases - metabolism | Liver - pathology | Male | Muscle, Skeletal - metabolism | Muscle, Skeletal - cytology | Fibroblast Growth Factors - metabolism | Liver - drug effects | PPAR-beta - agonists | Muscle, Skeletal - drug effects | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - metabolism | Fatty Acids - metabolism | Interleukin-6 - metabolism | Retinoid X Receptors - metabolism | Tretinoin - pharmacology | Neuropeptides - analysis | Cell Line | Enzyme-Linked Immunosorbent Assay | PPAR-beta - metabolism | Liver - metabolism | Neuropeptides - metabolism | Fibronectins - blood | Fibronectins - metabolism | Triglycerides - metabolism | Animals | Retinoid X Receptors - agonists | PPAR delta - agonists | Lipid Metabolism - drug effects | Glucose - metabolism | Fibronectins - genetics | Mice | Muscle, Skeletal - pathology | PPAR delta - metabolism | Liver | Homeostasis | Cytotoxicity | Lipids | Adipocytes | Glucose | Metabolism | Kinases | Fatty acids | Insulin | Proteins | Musculoskeletal system | Vitamin A | Antibiotics | Energy | Rodents | Oxidation | Diabetes | Index Medicus
Journal Article
Cerebral Cortex, ISSN 1047-3211, 01/2015, Volume 25, Issue 1, pp. 75 - 83
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 03/2010, Volume 298, Issue 3, pp. G364 - G374
Shen Z, Liang X, Rogers CQ, Rideout D, You M. Involvement of adiponectin-SIRT1-AMPK signaling in the protective action of rosiglitazone against alcoholic fatty... 
Signal transduction | Acetylation | Lipid metabolism | Transcriptional regulators | DIETARY SATURATED FAT | PHYSIOLOGY | ACTIVATED PROTEIN-KINASE | LIPID-METABOLISM | signal transduction | lipid metabolism | PHOSPHATIDATE PHOSPHOHYDROLASE | TNF-ALPHA PRODUCTION | INSULIN SENSITIVITY | acetylation | MAMMALIAN SIRTUIN-1 | ENERGY-EXPENDITURE | GENE-EXPRESSION | transcriptional regulators | GASTROENTEROLOGY & HEPATOLOGY | HEPATIC STEATOSIS | RNA, Small Interfering - genetics | Liver - pathology | Gene Expression - genetics | Tumor Necrosis Factor-alpha - genetics | Ion Channels - genetics | Male | Fatty Liver, Alcoholic - metabolism | Liver - drug effects | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Lipid Metabolism - genetics | Thiazolidinediones - pharmacology | PPAR gamma - genetics | Adiponectin - pharmacology | Liver - metabolism | Rats | Intra-Abdominal Fat - metabolism | Phosphatidate Phosphatase | Triglycerides - metabolism | Acetylation - drug effects | Signal Transduction - drug effects | Models, Biological | Cell Line, Tumor | Transaminases - blood | Mice | Transcription Factors | Histones - metabolism | AMP-Activated Protein Kinases - genetics | Acetyl-CoA Carboxylase - metabolism | Receptors, Adiponectin - genetics | Sirtuin 1 - metabolism | AMP-Activated Protein Kinases - metabolism | Gene Expression - drug effects | Mitochondrial Proteins - genetics | Adiponectin - physiology | Fatty Liver, Alcoholic - prevention & control | Sirtuin 1 - genetics | Fatty Liver, Alcoholic - blood | Thiazolidinediones - therapeutic use | Trans-Activators - genetics | Intra-Abdominal Fat - drug effects | Fatty Liver, Alcoholic - pathology | Nuclear Proteins - genetics | Mice, Inbred C57BL | Nuclear Proteins - metabolism | Ethanol - pharmacology | Animals | Lipid Metabolism - drug effects | Retinoid X Receptor alpha - genetics | Triglycerides - blood | Signal Transduction - physiology | Trans-Activators - metabolism | Uncoupling Protein 2 | Fatty liver | Physiological aspects | Cellular signal transduction | Research | Rosiglitazone maleate | Health aspects | Protein kinases | Ethanol | Metabolism | Fatty acids | Liver | Rodents | Index Medicus | Hormones and Signaling
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 02/2012, Volume 287, Issue 7, pp. 5059 - 5069
Compromised clearance of all-trans-retinal (atRAL), a component of the retinoid cycle, increases the susceptibility of mouse retina to acute light-induced... 
VISUAL CYCLE | OXIDATIVE STRESS | ACTIVATED PROTEIN-KINASE | 5-HT1A RECEPTORS | BIOCHEMISTRY & MOLECULAR BIOLOGY | PHOSPHOLIPASE-C | IN-VIVO | RECESSIVE RETINITIS-PIGMENTOSA | GENE ABCR | BINDING-PROPERTIES | NEUTROPHIL NADPH-OXIDASE | Reactive Oxygen Species - metabolism | Receptor, Serotonin, 5-HT2A - genetics | Calcium - metabolism | Humans | NADPH Oxidases - metabolism | Corneal Dystrophies, Hereditary - genetics | Type C Phospholipases - metabolism | Retinaldehyde - metabolism | Alcohol Oxidoreductases - genetics | ATP-Binding Cassette Transporters - genetics | Corneal Dystrophies, Hereditary - pathology | ATP-Binding Cassette Transporters - metabolism | NADPH Oxidases - genetics | Signal Transduction | Light - adverse effects | Receptor, Muscarinic M3 - metabolism | Alcohol Oxidoreductases - metabolism | Corneal Dystrophies, Hereditary - metabolism | Receptor, Muscarinic M3 - genetics | Serotonin 5-HT2 Receptor Antagonists - pharmacology | Mice, Knockout | Macular Degeneration - metabolism | Animals | Macular Degeneration - genetics | Type C Phospholipases - genetics | Mice | Inositol 1,4,5-Trisphosphate - genetics | Photoreceptor Cells, Vertebrate - metabolism | Receptor, Serotonin, 5-HT2A - metabolism | Inositol 1,4,5-Trisphosphate - metabolism | Photoreceptor Cells, Vertebrate - pathology | Macular Degeneration - pathology | Index Medicus | Molecular Bases of Disease | Vitamin A | Retinal metabolism | Vision | Reactive oxygen species (ROS) | Retinoid
Journal Article
Journal of Lipid Research, ISSN 0022-2275, 2005, Volume 46, Issue 6, pp. 1133 - 1149
Mitochondrial cytopathy has been associated with modifications of lipid metabolism in various situations, such as the acquisition of an abnormal adipocyte... 
AMP-dependent kinase | Peroxisome proliferator- activated receptor γ | Phosphatidylinositol 3-kinase/Akt1/glycogen synthase kinase 3β | Muscle carnitine palmitoyl transferase-1 | Carbohydrate-responsive element binding protein | ACTIVATED PROTEIN-KINASE | LIPID-METABOLISM | INTRACELLULAR CALCIUM | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLYCOGEN-SYNTHASE KINASE-3 | ACETYL-COA CARBOXYLASE | SKELETAL-MUSCLE | carbohydrate-responsive element binding protein | TRANSPORT | muscle carnitine palmitoyl transferase-1 | CPT-I | phosphatidylinositol 3 kinase/Akt1/glycogen synthase kinase 3 beta AMP-dependent kinase | GENE-EXPRESSION | MULTIPLE SYMMETRICAL LIPOMATOSIS | peroxisome proliferator activated receptor gamma | Luciferases - metabolism | Retinoid X Receptor alpha - metabolism | Calcium - metabolism | Adipocytes - cytology | Phosphatidylinositol 3-Kinases - metabolism | PPAR gamma - metabolism | RNA, Messenger - metabolism | Oxygen - metabolism | Transfection | Time Factors | Muscle Proteins - metabolism | Cell Differentiation | Cell Membrane - metabolism | Fatty Acids - metabolism | Monosaccharide Transport Proteins - metabolism | Protein-Serine-Threonine Kinases - metabolism | Carnitine O-Palmitoyltransferase - biosynthesis | Proto-Oncogene Proteins - metabolism | Glucose Transporter Type 4 | Down-Regulation | Carbohydrate Metabolism | Cells, Cultured | Enzyme Inhibitors - pharmacology | Lipid Metabolism | Mitochondria - metabolism | DNA - metabolism | Mitochondria - pathology | 3T3-L1 Cells | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Protein Transport | Triglycerides - metabolism | Proto-Oncogene Proteins c-akt | Microscopy, Confocal | Phenotype | Animals | Muscles - enzymology | Models, Biological | Glucose - pharmacokinetics | Glucose - metabolism | Protein Binding | Mice | Microscopy, Fluorescence | Index Medicus
Journal Article
Hepatology, ISSN 0270-9139, 12/2012, Volume 56, Issue 6, pp. 2288 - 2296
Nuclear receptors (NRs) play crucial roles in the regulation of hepatic cholesterol synthesis, metabolism, and conversion to bile acids, but their actions in... 
NPC1L1 | PATHWAY | CELLULAR CHOLESTEROL | MACROPHAGES | GENE-EXPRESSION | ALPHA | MICE | ABSORPTION | SECRETION | GASTROENTEROLOGY & HEPATOLOGY | MODULATION | RNA-Binding Proteins - genetics | Steroid Hydroxylases - metabolism | Receptors, Estrogen - metabolism | Receptors, Steroid - metabolism | Bile Ducts - metabolism | Gene Expression Profiling | Hepatocytes - metabolism | Orphan Nuclear Receptors - metabolism | PPAR gamma - metabolism | Receptors, Calcitriol - genetics | Bile Ducts - cytology | Receptors, Thyroid Hormone - genetics | Liver X Receptors | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | ATP-Binding Cassette Transporters - metabolism | Membrane Transport Proteins - metabolism | ATP Binding Cassette Transporter 1 | Retinoid X Receptors - genetics | Retinoid X Receptors - metabolism | Steroid Hydroxylases - genetics | PPAR delta - genetics | PPAR gamma - genetics | Hepatocyte Nuclear Factor 4 - metabolism | Receptors, Estrogen - genetics | Cells, Cultured | Rats | Hepatocyte Nuclear Factor 4 - genetics | Receptors, Calcitriol - metabolism | Transcription Factors - genetics | Cholesterol - metabolism | Orphan Nuclear Receptors - genetics | Receptors, Thyroid Hormone - metabolism | Transcription Factors - metabolism | Animals | Receptors, Steroid - genetics | PPAR delta - metabolism | Homeostasis - genetics | RNA-Binding Proteins - metabolism | Index Medicus
Journal Article
FASEB Journal, ISSN 0892-6638, 06/2009, Volume 23, Issue 6, pp. 1643 - 1654
Late-onset Alzheimer's disease is often connected with nutritional misbalance, such as enhanced cholesterol intake, deficiency in polyunsaturated fatty acids,... 
Shedding | Vitamin A | Nuclear receptors | Alzheimer's disease | Amyloid precursor protein | vitamin A | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | AMYLOID-PRECURSOR-PROTEIN | shedding | X-RECEPTOR | BETA-PEPTIDE | NEUROBLASTOMA-CELLS | HIPPOCAMPAL-NEURONS | VITAMIN-A | CELL BIOLOGY | nuclear receptors | RESPONSE ELEMENT | amyloid precursor protein | BIOLOGY | LONG-TERM POTENTIATION | EXPRESSION | Amyloid Precursor Protein Secretases - genetics | Up-Regulation | Tretinoin - chemistry | Humans | Male | PPAR gamma - metabolism | Receptors, Calcitriol - genetics | Receptors, Retinoic Acid - genetics | DNA-Binding Proteins - metabolism | Keratolytic Agents - metabolism | Receptors, Thyroid Hormone - genetics | Tretinoin - metabolism | Liver X Receptors | Acitretin - metabolism | Amyloid beta-Protein Precursor - metabolism | Membrane Proteins - metabolism | Molecular Structure | Orphan Nuclear Receptors | Retinoid X Receptors - genetics | Retinoid X Receptors - metabolism | PPAR gamma - genetics | Cell Line | Promoter Regions, Genetic | Membrane Proteins - genetics | Gene Expression Regulation | ADAM10 Protein | Mice, Transgenic | Receptors, Retinoic Acid - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Receptors, Calcitriol - metabolism | DNA-Binding Proteins - genetics | Acitretin - chemistry | Receptors, Thyroid Hormone - metabolism | ADAM Proteins - metabolism | Amyloid Precursor Protein Secretases - metabolism | Amyloid beta-Protein Precursor - genetics | Animals | Alzheimer Disease - metabolism | Mice | ADAM Proteins - genetics | Receptors, Cytoplasmic and Nuclear - metabolism | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2015, Volume 10, Issue 12, pp. e0145467 - e0145467
Alzheimer's disease (AD) is the major cause of dementia worldwide. The pharmacological activation of nuclear receptors (Liver X receptors: LXRs or Retinoid X... 
WILD-TYPE | AMYLOID BETA-PEPTIDE | ADULT NEUROGENESIS | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | SUBVENTRICULAR ZONE | TRANSGENIC MODEL | DENTATE GYRUS | HIPPOCAMPAL NEUROGENESIS | APOLIPOPROTEIN-E | Benzoates - therapeutic use | Cerebral Cortex - pathology | tau Proteins - metabolism | Cognition Disorders - metabolism | Male | Hippocampus - drug effects | Orphan Nuclear Receptors - metabolism | Apolipoproteins E - metabolism | Excitatory Postsynaptic Potentials - drug effects | ATP Binding Cassette Transporter 1 - metabolism | Cerebral Cortex - metabolism | Alzheimer Disease - pathology | Gliosis - pathology | Dentate Gyrus - drug effects | Long-Term Potentiation - drug effects | Liver X Receptors | Amyloid beta-Peptides - metabolism | Female | Cerebral Cortex - drug effects | Biomarkers - metabolism | Alzheimer Disease - physiopathology | Cognition Disorders - physiopathology | Dentate Gyrus - metabolism | Orphan Nuclear Receptors - agonists | Neural Stem Cells - drug effects | Alzheimer Disease - drug therapy | Mice, Transgenic | Nuclear Proteins - metabolism | Hippocampus - pathology | Cognition Disorders - drug therapy | Gliosis - complications | Nerve Tissue Proteins - metabolism | Nestin - metabolism | Up-Regulation - drug effects | Hippocampus - metabolism | Cognition Disorders - complications | Animals | Alzheimer Disease - metabolism | Fluorescent Antibody Technique | Benzoates - pharmacology | Protein Biosynthesis - drug effects | Cell Proliferation - drug effects | Benzylamines - pharmacology | Benzylamines - therapeutic use | Dentate Gyrus - pathology | Neural Stem Cells - metabolism | Brain | Advertising executives | Cognition | Apolipoproteins | Alzheimer's disease | Liver | Amyloidogenesis | Peptides | Cognitive ability | Proteins | Receptors | Neurodegeneration | Physiology | Chemical synthesis | Neurodegenerative diseases | Cortex | Protein biosynthesis | Behavioral sciences | Metabolism | Retinoid X receptors | Nuclear receptors | Dentate gyrus | Gliosis | Molecular modelling | Tau protein | Protein synthesis | Stem cells | Ligands | ATP-binding protein | Hippocampus | Animal models | Cerebral cortex | Lipids | Kinases | Neurogenesis | ABCA1 protein | Reduction | Metabolites | Apolipoprotein E | Rodents | Dementia disorders | Liver X receptors | Neurons | Pharmacology | Cholesterol | Cortex (entorhinal) | Plasticity (synaptic) | Presenilin 1 | Laboratory animals | Alzheimers disease | Animal cognition | Index Medicus
Journal Article
Cell Metabolism, ISSN 1550-4131, 03/2014, Volume 19, Issue 3, pp. 512 - 526
Insulin resistance is a major cause of diabetes and is highly associated with adipose tissue (AT) inflammation in obesity. RBP4, a retinol transporter, is... 
UNIQUE POPULATION | DENDRITIC CELLS | PLASMA RETINOL | OB/OB MICE | ENDOCRINOLOGY & METABOLISM | REGULATORY T-CELLS | RETINOL-BINDING-PROTEIN | RETINOL-BINDING-PROTEIN-4 | DIET-INDUCED OBESITY | EXPRESSION | MACROPHAGE ACTIVATION | CELL BIOLOGY | Mannose-Binding Lectins - metabolism | Cell Proliferation | Antigen-Presenting Cells - cytology | Humans | Transcriptome | Adipose Tissue - cytology | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Recombinant Proteins - biosynthesis | Glucose Intolerance | CD4-Positive T-Lymphocytes - immunology | Th1 Cells - metabolism | Adipose Tissue - metabolism | Lectins, C-Type - metabolism | Retinol-Binding Proteins, Plasma - metabolism | Antigen-Presenting Cells - metabolism | Signal Transduction | Bone Marrow Cells - cytology | CD4-Positive T-Lymphocytes - cytology | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Insulin Resistance | Receptors, Cell Surface - metabolism | Recombinant Proteins - genetics | Macrophages - cytology | Recombinant Proteins - pharmacology | Retinol-Binding Proteins, Plasma - pharmacology | Macrophages - metabolism | Animals | Macrophages - drug effects | Liver - cytology | Mice | Th1 Cells - cytology | Retinol-Binding Proteins, Plasma - genetics | Adipose tissues | Antigens | Glucose metabolism | Physiological aspects | Insulin resistance | Inflammation | Retinoids | Glucose | T cells | Dextrose | Index Medicus
Journal Article