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Journal Article
IEEE Transactions on Biomedical Engineering, ISSN 0018-9294, 05/2017, Volume 64, Issue 5, pp. 990 - 1002
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2016, Volume 36, Issue 1, pp. 19 - 24
OBJECTIVE—Ocular neovascularization (ONV) is a pathological feature of sight-threatening human diseases, such as diabetic retinopathy and age-related macular... 
myeloid cells | diabetic retinopathy | choroidal neovascularization | vascular endothelial growth factor A | macular degeneration | CELLS | ANGIOGENESIS | OXYGEN-INDUCED RETINOPATHY | VEGF | MOUSE | VITREAL MACROPHAGES | ROLES | EXPERIMENTAL CHOROIDAL NEOVASCULARIZATION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | RETINAL-PIGMENT EPITHELIUM | Vascular Endothelial Growth Factor A - deficiency | Retinal Neovascularization - metabolism | Vascular Endothelial Growth Factor A - metabolism | Retinopathy of Prematurity - genetics | Vascular Endothelial Growth Factor A - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - deficiency | Retinal Vessels - pathology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Choroidal Neovascularization - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Retinal Neovascularization - genetics | Basic Helix-Loop-Helix Transcription Factors - deficiency | Disease Models, Animal | Animals, Newborn | Oxygen | Basic Helix-Loop-Helix Transcription Factors - genetics | Retinal Vessels - metabolism | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Retinopathy of Prematurity - chemically induced | Mice, Knockout | Animals | Choroidal Neovascularization - metabolism | Myeloid Cells - metabolism | Retinal Neovascularization - chemically induced | Retinopathy of Prematurity - metabolism | Myeloid Cells - pathology | Choroidal Neovascularization - genetics | Retinal Neovascularization - pathology | Retinopathy of Prematurity - pathology | Index Medicus | 10011 | 10033 | 10012 | 10014 | Basic Sciences | 10028
Journal Article
Vascular Pharmacology, ISSN 1537-1891, 2016, Volume 90, pp. 19 - 26
Abstract Reactive oxygen species (ROS) as well as vascular endothelial growth factor (VEGF) play important roles in pathologic retinal neovascularization. We... 
Cardiovascular | Oxygen induced retinopathy | Reactive oxygen species | Retinal neovascularization | Betaine | Vascular endothelial growth factor | DIABETIC-RETINOPATHY | NADPH OXIDASE | CELLS | ANGIOGENESIS | VEGF | INDUCED RETINOPATHY | MACULAR DEGENERATION | NAD(P)H OXIDASE | IN-VITRO | PHARMACOLOGY & PHARMACY | PREMATURITY | Phosphorylation | Reactive Oxygen Species - metabolism | Intravitreal Injections | Humans | Astrocytes - pathology | Vascular Endothelial Growth Factor A - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Dose-Response Relationship, Drug | Retinal Vessels - pathology | Angiogenesis Inhibitors - administration & dosage | Betaine - administration & dosage | Hyperoxia - complications | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Animals, Newborn | Retinopathy of Prematurity - etiology | Astrocytes - drug effects | Retinal Vessels - metabolism | Endothelial Cells - metabolism | Mice, Inbred C57BL | Cells, Cultured | Angiogenesis Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Retinal Vessels - drug effects | Betaine - pharmacology | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Retinopathy of Prematurity - prevention & control | Cell Proliferation - drug effects | Retinopathy of Prematurity - metabolism | Endothelial Cells - pathology | Astrocytes - metabolism | Endothelial Cells - drug effects | Retinal Neovascularization | Retinopathy of Prematurity - pathology | Antimitotic agents | Neovascularization | Antineoplastic agents | Analysis | Animal experimentation | Medical research | Medical colleges | RNA | Blindness | Medicine, Experimental | Research institutes | Ophthalmology | Cell proliferation | Brain | Retinopathy | Transcription | Toxicity | Cytotoxicity | Retina | AKT protein | Vascularization | Angiogenesis | Cell growth | Eye (anatomy) | Growth factors | Oxygen | Astrocytes | Extracellular signal-regulated kinase | Chemical compounds | Endothelial cells | Vascular endothelial growth factor receptors | Signaling | Antiangiogenics | Microvasculature | Cell migration | Index Medicus
Journal Article
Ophthalmology, ISSN 0161-6420, 2011, Volume 118, Issue 8, pp. 1580 - 1587
Journal Article
Nature, ISSN 0028-0836, 12/2017, Volume 552, Issue 7684, pp. 248 - 252
Diabetic retinopathy is an important cause of blindness in adults(1,2), and is characterized by progressive loss of vascular cells and slow dissolution of... 
BLOOD-RETINAL BARRIER | ACID | ANGIOGENESIS | CHOLESTEROL | STABILITY | MULTIDISCIPLINARY SCIENCES | ENDOTHELIAL-CELLS | DEPENDENT ENDOCYTOSIS | MEMBRANES | VE-CADHERIN | ASSOCIATION | Retina - drug effects | Retina - metabolism | Cadherins - metabolism | Pericytes - drug effects | Humans | Capillary Permeability - drug effects | Male | Diabetic Retinopathy - pathology | Antigens, CD - metabolism | Vitreous Body - metabolism | Pericytes - pathology | Retina - enzymology | Intercellular Junctions - pathology | Female | Cell Membrane - drug effects | Disease Models, Animal | Ependymoglial Cells | Cell Survival - drug effects | Endothelial Cells - metabolism | Mice, Inbred C57BL | Solubility | Pancreatic Elastase - metabolism | Fatty Acids, Unsaturated - metabolism | Diabetic Retinopathy - metabolism | Disease Progression | Cell Movement - drug effects | Diabetic Retinopathy - prevention & control | Animals | Carrier Proteins - metabolism | Diabetic Retinopathy - enzymology | Intercellular Junctions - drug effects | Presenilin-1 - metabolism | Mice | Endothelial Cells - pathology | Epoxide Hydrolases - antagonists & inhibitors | Retina - pathology | Docosahexaenoic Acids - metabolism | Endothelial Cells - drug effects | Prevention | Physiological aspects | Enzymes | Diabetic retinopathy | Hydrolases | Epoxy compounds | Retinopathy | Pathogenesis | Cell junctions | Retina | Glial cells | Cell interactions | Kinases | Docosahexaenoic acid | Epoxide hydrolase | Vascularization | Proteins | Signal transduction | Hyperglycemia | N-Cadherin | Rodents | Inhibition | Growth factors | Edema | Abnormalities | Diabetes mellitus | Permeability | Cadherin | Cholesterol | Endothelial cells | Acids | Presenilin 1 | Nitric oxide | Blindness | Vitreous humour | Adults | Infiltration | Diabetes | Staphylococcal enterotoxin H | Index Medicus
Journal Article
Graefe's Archive for Clinical and Experimental Ophthalmology, ISSN 0721-832X, 12/2010, Volume 248, Issue 12, pp. 1713 - 1718
Journal Article
Diabetic Medicine, ISSN 0742-3071, 03/2019, Volume 36, Issue S1, pp. 155 - 156
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, pp. e65691 - e65691
Background: Retinal vasculopathies, including diabetic retinopathy (DR), threaten the vision of over 100 million people. Retinal pericytes are critical for... 
DIABETIC-RETINOPATHY | ENDOTHELIAL PROGENITOR CELLS | ACTIVATION | OXYGEN-INDUCED RETINOPATHY | MULTIDISCIPLINARY SCIENCES | GROWTH | SMOOTH-MUSCLE-CELLS | CONTRACTILE PHENOTYPE | MODEL | STROMAL CELLS | CHOROIDAL NEOVASCULARIZATION | Retina - metabolism | Retinal Vessels - metabolism | Humans | Pericytes - cytology | Pericytes - metabolism | Adipocytes - cytology | Oxygen - adverse effects | Stem Cells - cytology | Diabetic Retinopathy - pathology | Stem Cells - metabolism | Diabetic Retinopathy - metabolism | Retinal Vessels - pathology | Animals | Adipocytes - metabolism | Stem Cells - drug effects | Mice | Neovascularization, Pathologic - metabolism | Retina - pathology | Transforming Growth Factor beta1 - pharmacology | Blood circulation disorders | Stem cells | Blindness | Bone morphogenetic proteins | Muscle proteins | Transforming growth factors | Endothelium | Therapy | Diabetic retinopathy | Animal models | Retinopathy | Transplants & implants | Pathogenesis | Transforming growth factor-b | Medical services | Paracrine signalling | Stem cell transplantation | Smooth muscle | Retina | Macular degeneration | Angiogenesis | Reduction | Vascular diseases | Allografts | Actin | Bone marrow | Physiology | Growth factors | Biomedical engineering | Diabetes mellitus | Contractility | Muscles | Blood vessels | Regrowth | Pharmacology | Ablation | Muscle contraction | Endothelial cells | Morphology | Pericytes | Hypoxia | Microvasculature | Diabetes | Destabilization | Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 03/2015, Volume 116, Issue 7, pp. 1143 - 1156
Journal Article
Microvascular Research, ISSN 0026-2862, 05/2013, Volume 87, pp. 65 - 74
The purpose of the study was to evaluate the effects of hesperetin (Hsp) on diabetes-induced retinal oxidative stress, neuroinflammation and apoptosis in rats.... 
INDUCED ACTIVATION | TUMOR-NECROSIS-FACTOR | OPTIC-NERVE | EXPERIMENTAL GALACTOSEMIA | RETINOPATHY | PERIPHERAL VASCULAR DISEASE | CENTRAL-NERVOUS-SYSTEM | INFLAMMATORY RESPONSE | MULLER CELLS | CELL-DEATH | BASEMENT-MEMBRANE | Immunohistochemistry | Retina - drug effects | Tumor Necrosis Factor-alpha - metabolism | Diabetic Retinopathy - drug therapy | Inflammation - pathology | Diabetes Mellitus, Experimental - drug therapy | Retina - metabolism | Rats, Wistar | Apoptosis - drug effects | Glutathione - metabolism | Streptozocin | Caspase 3 - metabolism | Body Weight - drug effects | Male | Retina - immunology | Diabetic Retinopathy - pathology | Glial Fibrillary Acidic Protein - metabolism | Inflammation - metabolism | Retina - ultrastructure | Time Factors | Inflammation - drug therapy | Interleukin-1beta - metabolism | Inflammation Mediators - metabolism | Female | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Superoxide Dismutase - metabolism | Microscopy, Electron, Transmission | Diabetic Retinopathy - immunology | Anti-Inflammatory Agents - pharmacology | Rats | Inflammation - immunology | Antioxidants - pharmacology | Diabetic Retinopathy - metabolism | Blood Glucose - drug effects | Catalase - metabolism | Diabetes Mellitus, Experimental - immunology | Animals | Aquaporin 4 - metabolism | Diabetes Mellitus, Experimental - pathology | Hesperidin - pharmacology | Oxidative Stress - drug effects | Blood Glucose - metabolism | Diabetic retinopathy | Oxidative stress | Enzymes | Aquaporins | Heat shock proteins | Superoxide | Intermediate filament proteins | Restaurants | Apoptosis | Index Medicus
Journal Article