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12/2016, ISBN 0128036907, 319
Neuroprotection in Alzheimer's Disease offers a translational point-of-view from both basic and clinical standpoints, putting it on the cusp for further... 
Alzheimer's disease | Biology, life sciences | Neuroprotective agents
eBook
NeuroImage, ISSN 1053-8119, 04/2019, Volume 189, pp. 180 - 191
White matter abnormalities, revealed by Diffusion Tensor Imaging (DTI), are observed in patients with Alzheimer's Disease (AD), representing neural network... 
Retrograde degeneration | EpoD | Amyloid beta | p301L tau | Diffusion tensor imaging | Retinal ganglion cell | NEUROFIBRILLARY TANGLES | ALZHEIMERS-DISEASE | WALLERIAN DEGENERATION | PATHOLOGY | MILD COGNITIVE IMPAIRMENT | NEUROSCIENCES | NEUROIMAGING | EPOTHILONE D | MAGNETIC-RESONANCE | DIFFUSION | WHITE-MATTER HYPERINTENSITIES | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | SYNTHETIC TAU FIBRILS | Tubulin Modulators - pharmacology | Amyloid beta-Peptides - pharmacology | Peptide Fragments - administration & dosage | Epothilones - pharmacology | Tauopathies - pathology | White Matter - drug effects | Nerve Degeneration - diagnostic imaging | Peptide Fragments - pharmacology | Geniculate Bodies - drug effects | Nerve Degeneration - chemically induced | Diffusion Tensor Imaging | White Matter - diagnostic imaging | Animals | Nerve Degeneration - prevention & control | Tauopathies - diagnostic imaging | Epothilones - administration & dosage | Tubulin Modulators - administration & dosage | Mice | Tauopathies - chemically induced | Amyloid beta-Peptides - administration & dosage | Tauopathies - drug therapy | Nerve Degeneration - drug therapy | Retinal Ganglion Cells - drug effects | Disease Models, Animal | Medical colleges | Neurosciences | Neural networks | Anisotropy | Physiological aspects | Models | Genetic engineering | Alzheimer's disease | Phosphorylation | Peptides | Cognitive ability | Retina | Visual system | Neurodegeneration | Drug dosages | Multimedia | Medical imaging | Optic nerve | Neurodegenerative diseases | Transgenic mice | Histology | Injection | Substantia alba | Patients | Optic tract | Studies | Pathology | Retinal ganglion cells | Axons | Brain research | Magnetic resonance imaging | Tau protein | Lateral geniculate nucleus | Laboratory animals | Alzheimers disease | Animal cognition
Journal Article
2009, ISBN 0195326695, xii, 580
Disorders of the nervous and vascular systems continue to burden the planet's population not only with increasing morbidity and mortality, but also with a... 
Other branches of medicine | Pathology, Cellular | neuroscience | Inflammatory cells | Degenerative diseases | Cell biology | Nervous system | Cellular pathways | Longevity | Vascular system | Healthy aging | Cellular function | Inflammation | Pathology, Molecular
Book
PLoS ONE, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, p. e34724
Journal Article
Experimental Eye Research, ISSN 0014-4835, 2009, Volume 89, Issue 1, pp. 32 - 41
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2002, Volume 99, Issue 19, pp. 12432 - 12437
Phenotypic knockout of nerve growth factor (NGF) activity in transgenic anti-NGF mice (AD11 mice) results in a progressive neurodegenerative phenotype... 
Cholinergics | Biological Sciences | Phenotypes | Neuroscience | Neurons | Transgenic animals | Antibodies | Pharmacology | Amyloids | Behavioral neuroscience | Hippocampus | BASAL FOREBRAIN NEURONS | MESSENGER-RNA | NUCLEUS BASALIS | ALZHEIMERS-DISEASE | RETROGRADE TRANSPORT | CHOLINERGIC NEURONS | MULTIDISCIPLINARY SCIENCES | SPATIAL MEMORY | AMYLOID PRECURSOR PROTEIN | NICOTINIC ACETYLCHOLINE-RECEPTORS | TRANSGENIC MICE | Alzheimer Disease - etiology | Phosphorylation | Entorhinal Cortex - metabolism | Humans | tau Proteins - metabolism | Choline O-Acetyltransferase - metabolism | Galantamine - pharmacology | Nerve Growth Factor - antagonists & inhibitors | Nerve Growth Factor - physiology | Nerve Degeneration - prevention & control | Nerve Growth Factor - immunology | Amyloid beta-Peptides - metabolism | Amyloid beta-Protein Precursor - metabolism | Cholinergic Agonists - pharmacology | Disease Models, Animal | Nerve Growth Factor - pharmacology | Mice, Transgenic | Entorhinal Cortex - drug effects | Recombinant Proteins - pharmacology | Nerve Degeneration - pathology | Mice, Knockout | Phenotype | Animals | Antibodies - genetics | Mice | Entorhinal Cortex - pathology | Nerve Degeneration - etiology | Nerve Degeneration - drug therapy | Genetically modified mice | Usage | Physiological aspects | Nerve growth factor | Nervous system | Degeneration | Acetylcholine | Research | Cytochemistry | Molecular biology
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, p. e65389
Oxidative stress has been implicated in neurodegenerative diseases, including glaucoma. However, due to the lack of clinically relevant models and expense of... 
IRIS ATROPHY | OXIDATIVE STRESS | TRABECULAR MESHWORK | PROTECTIVE ROLE | MULTIDISCIPLINARY SCIENCES | NITRIC-OXIDE | NEURODEGENERATION | KAPPA-B | DBA/2J MOUSE MODEL | RAT MODEL | GLYCATION END-PRODUCTS | Retina - drug effects | Thioctic Acid - administration & dosage | Up-Regulation | Retina - metabolism | Intraocular Pressure - drug effects | Oxidative Stress | Axons - physiology | Nerve Degeneration - prevention & control | Mice, Inbred DBA | Cell Death - drug effects | Drug Evaluation, Preclinical | Receptor for Advanced Glycation End Products | Gene Expression | Retinal Ganglion Cells - physiology | Oxidation-Reduction | Administration, Oral | Mice, Inbred C57BL | Axons - drug effects | Glaucoma - drug therapy | Treatment Outcome | Animals | Antioxidants - administration & dosage | Glaucoma - pathology | Mice | DNA Damage | Lipid Peroxidation | Dietary Supplements | Retina - pathology | Receptors, Immunologic - metabolism | Retinal Ganglion Cells - drug effects | Nitric Oxide Synthase Type II - metabolism | Glaucoma | Intervention | Lipoic acid | Therapy | Oxidative stress | Diabetic retinopathy | Animal models | Disease | Pathogenesis | DNA damage | Medical services | Lipid peroxidation | Retina | Lipids | mRNA | Macular degeneration | Proteins | Antioxidants | Prevention | Oxidation resistance | Metabolites | Neurodegeneration | Rodents | Oxidation | Age | Deoxyribonucleic acid--DNA | Peroxidation | Hypertension | Free radicals | Neurodegenerative diseases | Metabolism | Retrograde transport | Gene expression | Neurological diseases | Retinal ganglion cells | Acids | Diet | Cell death | Nitric oxide | Diabetes | Transport | Axonal transport | Deoxyribonucleic acid | DNA
Journal Article
Experimental Neurology, ISSN 0014-4886, 2008, Volume 211, Issue 2, pp. 574 - 584
Treatment of degenerating basal forebrain cholinergic neurons with nerve growth factor (NGF) in Alzheimer's disease has long been contemplated, but an... 
Neurotrophin | Nucleus basalis of Meynert | Gene delivery | Adeno-associated virus | Dose–response | CERE-110 | Trophic activity | Nerve growth factor | Basal forebrain cholinergic neurons | Alzheimer's disease | Dose-response | IMPROVES SPATIAL MEMORY | DOWNS-SYNDROME | ALZHEIMERS-DISEASE | trophic activity | COGNITIVE IMPAIRMENT | adeno-associated virus | INTRACEREBROVENTRICULAR INFUSION | NEUROSCIENCES | NERVE GROWTH-FACTOR | gene delivery | basal forebrain cholinergic neurons | nerve growth factor | RETROGRADE TRANSPORT | dose-response | nucleus basalis of Meynert | FACTOR PREVENTS DEGENERATION | neurotrophin | GENE-TRANSFER | AGED RATS | Nerve Growth Factor - metabolism | Genetic Vectors - administration & dosage | Cholinergic Fibers - virology | Humans | Rats, Inbred F344 | Genetic Vectors - metabolism | Nerve Degeneration - genetics | Rats | Male | Rats, Sprague-Dawley | Nerve Growth Factor - therapeutic use | Cholinergic Fibers - metabolism | Nerve Growth Factor - administration & dosage | Marmota | Animals | Nerve Degeneration - prevention & control | Prosencephalon - cytology | Adenoviridae - genetics | Prosencephalon - virology | Adenoviridae - metabolism | Prosencephalon - metabolism | Drug Delivery Systems - methods | Nerve Degeneration - drug therapy | Neurosciences | Neurons | Health aspects | Alzheimer’s disease
Journal Article
Journal Article
2011, 1st ed., ISBN 0123849136, xix, 520
Inflammation is a central mechanism in many neurological diseases, including stroke, multiple sclerosis, and brain trauma as well as meningitis and contributes... 
Nervous system | Clinical & internal medicine
Book