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Circulation Research, ISSN 0009-7330, 10/2017, Volume 121, Issue 9, pp. 1058 - 1068
RATIONALE:Downregulation of the pacemaking ion channel, HCN4 (hyperpolarization-activated cyclic nucleotide gated channel 4), and the corresponding ionic... 
INTRONIC MICRORNAS | HEART-FAILURE | sinoatrial node | DOWN-REGULATION | athletes | micro-RNAs | sinus bradycardia | O-GLCNAC TRANSFERASE | HIGH VAGAL TONE | ATRIAL-FIBRILLATION | ion channel remodeling | HOST GENES | ENDURANCE EXERCISE | exercise training | CARDIOMYOCYTE DIFFERENTIATION | APOPTOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | RECEPTOR | PROLIFERATION | MICRORNAS | TBX5 | PERIPHERAL VASCULAR DISEASE | HEART-RATE | HEMATOLOGY | EXPRESSION | REVEALS | Gene Targeting - methods | Bradycardia - genetics | Humans | Physical Conditioning, Animal - methods | Male | MicroRNAs - metabolism | Gene Knockdown Techniques - methods | Potassium Channels - metabolism | Young Adult | Bradycardia - metabolism | Muscle Proteins - metabolism | Adult | Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels - metabolism | Mice, Inbred C57BL | Sinoatrial Node - physiopathology | Bradycardia - physiopathology | Physical Conditioning, Animal - physiology | Potassium Channels - genetics | Muscle Proteins - genetics | Animals | Adolescent | Sinoatrial Node - metabolism | Exercise - physiology | Mice | MicroRNAs - genetics | Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels - genetics | Bradycardia | Exercise | Physiological aspects | Ion channels | Research | Nucleotide sequencing | Athletes | Health aspects | Risk factors | DNA sequencing | Cardiac arrhythmia | Animal models | Physical training | Reverse transcription | Polymerase chain reaction | Heart rate | Sinuses | Sinus | Computer applications | miRNA | Fitness training programs | Nkx2.5 protein | Hyperpolarization | Ion channels (cyclic nucleotide-gated) | 10063 | 10032 | 10035 | Molecular Medicine | 10001 | 10003
Journal Article
Journal Article
Molecular Medicine Reports, ISSN 1791-2997, 03/2016, Volume 13, Issue 3, pp. 2677 - 2682
Alterations to the expression L-type calcium channels (LTCCs) and Kv4.3 potassium channels form the possible basis of atrial electrical remodeling during rapid... 
Mitogen-activated protein kinase | Ion channel | Rapid pacing | FIBRILLATION | MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | rapid pacing | CARDIAC MYOCYTES | MUSCLE | MECHANISMS | mitogen-activated protein kinase | ONCOLOGY | ion channel | CALCIUM-RELEASE | AFTERDEPOLARIZATIONS | ACTIVATED PROTEIN-KINASES
Journal Article
Journal Article
Journal Article
Journal Article
Arrhythmia & electrophysiology review, ISSN 2050-3369, 08/2014, Volume 3, Issue 2, pp. 101 - 106
Pulmonary vein isolation (PVI) has been demonstrated to be a highly effective treatment option for patients with paroxysmal atrial fibrillation (AF), but less... 
Diagnostic Electrophysiology & Ablation | catheter ablation | reverse electrical remodelling | antiarrhythmic drug therapy | Atrial fibrillation
Journal Article
JACC. Cardiovascular imaging, ISSN 1936-878X, 07/2019
This study assessed the impact of right-atrial (RA) pacing on left-atrial (LA) physiology and clinical outcome. Data for the effects of RA pacing on LA... 
Journal Article
Journal Article
JOURNAL OF INTERVENTIONAL CARDIAC ELECTROPHYSIOLOGY, ISSN 1383-875X, 12/2019, Volume 56, Issue 3, pp. 291 - 297
Purpose Pulmonary vein isolation (PVI) by catheter ablation has reduced efficacy for the treatment of persistent atrial fibrillation (persAF), as compared to... 
CARDIAC & CARDIOVASCULAR SYSTEMS | Amiodarone | Ablation | Atrial reverse remodeling | Persistent atrial fibrillation | Pulmonary vein isolation | Cardiac arrhythmia | Survival analysis | Fibrillation | Veins | Arrhythmia | Medical instruments | Electrocardiography | Patients | Catheters
Journal Article
Circulation, ISSN 0009-7322, 03/2004, Volume 109, Issue 11, pp. 1379 - 1385
Background - Although modulation of inflammatory processes has been suggested as a new treatment modality in heart failure (HF), our knowledge about... 
Heart failure | Myocardium | Inflammation | Leukocytes | heart failure | CYTOKINES | CARDIAC & CARDIOVASCULAR SYSTEMS | inflammation | PHENOTYPE | leukocytes | PERIPHERAL VASCULAR DISEASE | myocardium | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | EXPRESSION | FOLLISTATIN | Myocardial Infarction - genetics | Protein Biosynthesis | Rats, Wistar | Tissue Inhibitor of Metalloproteinase-1 - biosynthesis | Humans | Middle Aged | Male | Monocytes - metabolism | Inhibin-beta Subunits - blood | Inhibin-beta Subunits - physiology | Heart Failure - blood | Transforming Growth Factor beta - biosynthesis | Activin Receptors, Type I - biosynthesis | T-Lymphocytes - metabolism | Matrix Metalloproteinase 9 - genetics | Atrial Natriuretic Factor - genetics | Myocardium - metabolism | Activin Receptors, Type II - biosynthesis | Ventricular Remodeling - genetics | Female | Models, Animal | Animals, Newborn | Matrix Metalloproteinase 9 - biosynthesis | Natriuretic Peptide, Brain - genetics | Transforming Growth Factor beta1 | Atrial Natriuretic Factor - biosynthesis | Gene Expression Regulation | Natriuretic Peptide, Brain - biosynthesis | Rats | Heart Failure - genetics | Heart Failure - pathology | Ventricular Remodeling - physiology | Reverse Transcriptase Polymerase Chain Reaction | Activin Receptors, Type I - genetics | Proteins - genetics | Tissue Inhibitor of Metalloproteinase-1 - genetics | Animals | Transforming Growth Factor beta - genetics | Activin Receptors, Type II - genetics | Myocytes, Cardiac - metabolism | Chemokine CCL2
Journal Article
European Thyroid Journal, ISSN 2235-0640, 06/2015, Volume 4, Issue 2, pp. 129 - 137
Background: Although hyperthyroidism predisposes to atrial fibrillation, previous trials have suggested decreased triiodothyronine (T3) concentrations to be... 
Clinical Thyroidology / Original Paper | 3,5-Diiodothyronine | Nonthyroidal illness syndrome | Reverse triiodothyronine | Clinical Thyroidology | TACITUS | Atrial fibrillation | Original Paper | Thyroid hormone
Journal Article