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Trends in Immunology, ISSN 1471-4906, 2011, Volume 32, Issue 9, pp. 395 - 401
T helper (Th)17 cells have been proposed to represent a new CD4+ T cell lineage that is important for host defense against fungi and extracellular bacteria,... 
Allergy and Immunology | HYPER-IGE SYNDROME | GROWTH-FACTOR-BETA | TGF-BETA | T(H)17 CELLS | ROR-GAMMA-T | IN-VIVO | HUMAN TH17 CELLS | CENTRAL-NERVOUS-SYSTEM | IMMUNOLOGY | T-H-17 CELLS | GENOME-WIDE ASSOCIATION | Autoimmune Diseases - physiopathology | Species Specificity | Humans | Interleukin-17 - immunology | STAT5 Transcription Factor - immunology | Mycoses - microbiology | Cell Differentiation - genetics | STAT5 Transcription Factor - metabolism | Signal Transduction - immunology | Interleukin-23 Subunit p19 - biosynthesis | Th17 Cells - metabolism | Bacterial Infections - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Th17 Cells - cytology | Genetic Variation - immunology | Autoimmune Diseases - pathology | Bacterial Infections - microbiology | STAT3 Transcription Factor - metabolism | Transforming Growth Factor beta - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | Immunity, Cellular - genetics | Interleukin-17 - biosynthesis | Cell Differentiation - immunology | Animals | Th17 Cells - immunology | Mice | Interleukin-23 Subunit p19 - immunology | STAT3 Transcription Factor - immunology | Mycoses - immunology | Transforming Growth Factor beta - metabolism | Cell Lineage - immunology | T cells | Pathology | Transcription factors | Lymphocytes | Genes | Rodents | Rheumatoid arthritis | Autoimmune diseases | Kinases | Immune system | Crohns disease
Journal Article
International Immunology, ISSN 0953-8178, 11/2011, Volume 23, Issue 11, pp. 701 - 712
Rheumatoid arthritis (RA) is a chronic inflammatory disease that causes irreversible joint damage and significant disability. However, the fundamental... 
Joint destruction | Chronic inflammation | Collagen-induced arthritis | Rheumatoid arthritis | IL-6 | RHEUMATOID-ARTHRITIS | INTERLEUKIN-1 RECEPTOR ANTAGONIST | STAT3 | MONOCLONAL-ANTIBODY | IMMUNOLOGY | rheumatoid arthritis | OSTEOCLASTOGENESIS | RODENT MODELS | TUMOR-NECROSIS-FACTOR | collagen-induced arthritis | REGULATORY T-CELLS | DOUBLE-BLIND | joint destruction | NF-KAPPA-B | chronic inflammation | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Arthritis, Experimental - drug therapy | RANK Ligand - metabolism | Humans | Arthritis, Rheumatoid - complications | Interleukin-1beta - adverse effects | Arthritis, Experimental - pathology | RANK Ligand - immunology | Inflammation - complications | Signal Transduction - immunology | Osteoclasts - immunology | Arthritis, Rheumatoid - drug therapy | Inflammation - drug therapy | Interleukin-1beta - metabolism | Piperidines | Gene Expression - immunology | STAT3 Transcription Factor - deficiency | Tumor Necrosis Factor-alpha - immunology | Mice, Inbred DBA | Pyrroles - therapeutic use | Interleukin-6 - metabolism | Joints - metabolism | STAT3 Transcription Factor - genetics | Joints - drug effects | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Interleukin-1beta - immunology | Joints - pathology | Arthritis, Experimental - immunology | Inflammation - immunology | Pyrimidines - pharmacology | Arthritis, Rheumatoid - pathology | Pyrroles - pharmacology | Animals | Tumor Necrosis Factor-alpha - adverse effects | Interleukin-6 - pharmacology | RANK Ligand - antagonists & inhibitors | Pyrimidines - therapeutic use | Fibroblasts - drug effects | Interleukin-6 - immunology | Fibroblasts - immunology | Mice | STAT3 Transcription Factor - immunology | Arthritis, Rheumatoid - immunology | STAT3 Transcription Factor - antagonists & inhibitors | Chronic Disease | Osteoclasts - drug effects | Animal models | Transcription | Leukemia inhibitory factor | Stat3 protein | Data processing | Inflammation | Joint diseases | Osteoblasts | Inflammatory diseases | Interleukin 6 | Osteoclastogenesis | Collagen | Interleukin 1 | TRANCE protein | Tumor necrosis factor- alpha
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2014, Volume 192, Issue 7, pp. 3345 - 3354
Nerve growth factor (NGF) levels are highly increased in inflamed tissues, but their role is unclear. We show that NGF is part of a regulatory loop in... 
RHEUMATOID-ARTHRITIS | TOLL-LIKE-RECEPTORS | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | SIGNALING PATHWAYS | ACTIVATION | NEGATIVE REGULATION | IL-10 PRODUCTION | IMMUNOLOGY | NF-KAPPA-B | EXPRESSION | INNATE IMMUNITY | Leukocytes, Mononuclear - metabolism | Humans | NF-kappa B - immunology | Child, Preschool | Receptor, trkA - immunology | Infant | Monocytes - metabolism | NF-kappa B - metabolism | Monocytes - immunology | Interleukin-1beta - genetics | Lipopolysaccharides - immunology | Signal Transduction - immunology | Interleukin-1beta - metabolism | Leukocytes, Mononuclear - immunology | Nerve Growth Factor - immunology | Inflammation Mediators - metabolism | Interleukin-10 - metabolism | Toll-Like Receptors - metabolism | Child | Cytokines - genetics | Interleukin-6 - metabolism | Cytokines - immunology | Inflammation Mediators - immunology | Nerve Growth Factor - metabolism | Cytokines - metabolism | Enzyme-Linked Immunosorbent Assay | Interleukin-6 - genetics | Toll-Like Receptors - immunology | Cells, Cultured | Interleukin-1beta - immunology | Nerve Growth Factor - pharmacology | Down-Regulation - drug effects | Protein Binding - immunology | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Monocytes - drug effects | Receptor, trkA - metabolism | Arthritis, Juvenile - immunology | Down-Regulation - immunology | Interleukin-10 - genetics | Adolescent | Interleukin-6 - immunology | Lipopolysaccharides - pharmacology | Receptor, trkA - genetics | Interleukin-10 - immunology | Arthritis, Juvenile - pathology
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 06/2013, Volume 72, Issue 6, pp. 901 - 907
Journal Article
Lancet, The, ISSN 0140-6736, 2009, Volume 373, Issue 9678, pp. 1905 - 1917
Summary Glucocorticoid resistance or insensitivity is a major barrier to the treatment of several common inflammatory diseases—including chronic obstructive... 
Internal Medicine | RHEUMATOID-ARTHRITIS | MEDICINE, GENERAL & INTERNAL | OBSTRUCTIVE PULMONARY-DISEASE | HISTONE DEACETYLASE ACTIVITY | MIGRATION INHIBITORY FACTOR | ULCERATIVE-COLITIS | REGULATORY T-CELLS | RECEPTOR-BETA | RELATIVE CORTICOSTEROID INSENSITIVITY | BLOOD MONONUCLEAR-CELLS | RESPIRATORY-DISTRESS-SYNDROME | Transcription Factor AP-1 - drug effects | Glucocorticoids - therapeutic use | Transcriptional Activation - genetics | Transcription Factor AP-1 - genetics | Humans | Macrophage Migration-Inhibitory Factors - immunology | Transcriptional Activation - drug effects | Transcription Factor AP-1 - immunology | Histone Deacetylases - immunology | Transcriptional Activation - immunology | ATP-Binding Cassette, Sub-Family B, Member 1 - drug effects | Calcineurin Inhibitors | Macrophage Migration-Inhibitory Factors - genetics | Inflammation - drug therapy | Phosphodiesterase 4 Inhibitors | Anti-Inflammatory Agents - therapeutic use | Mitogen-Activated Protein Kinases - immunology | Genetic Predisposition to Disease - genetics | NF-kappa B - antagonists & inhibitors | Histone Deacetylases - genetics | Macrophage Migration-Inhibitory Factors - drug effects | Repressor Proteins - genetics | Histone Deacetylase 2 | Drug Resistance - drug effects | Inflammation - immunology | Asthma - drug therapy | ATP-Binding Cassette, Sub-Family B, Member 1 - immunology | Drug Resistance - genetics | ATP-Binding Cassette, Sub-Family B, Member 1 - genetics | Drug Resistance - immunology | Repressor Proteins - immunology | Histone Deacetylases - drug effects | Mitogen-Activated Protein Kinases - genetics | Repressor Proteins - drug effects | Mitogen-Activated Protein Kinases - drug effects | Pulmonary Disease, Chronic Obstructive - drug therapy | Corticosteroids | Dosage and administration | Inflammation | Research | Drug resistance | Drug therapy | Health aspects | Complications and side effects | Risk factors | Inflammatory bowel disease | Proteins | Respiratory distress syndrome | Asthma | Steroids
Journal Article
Journal of Autoimmunity, ISSN 0896-8411, 2012, Volume 39, Issue 3, pp. 222 - 228
Journal Article
Nature Immunology, ISSN 1529-2908, 06/2017, Volume 18, Issue 7, pp. 800 - 812
An imbalance in the lineages of immunosuppressive regulatory T cells (T cells) and the inflammatory T 17 subset of helper T cells leads to the development of... 
Forkhead Transcription Factors - immunology | Humans | Encephalomyelitis, Autoimmune, Experimental - immunology | Sjogren's Syndrome - immunology | Immunoblotting | Intracellular Signaling Peptides and Proteins - immunology | Case-Control Studies | T-Lymphocytes, Regulatory - immunology | DNA-Binding Proteins - metabolism | Flow Cytometry | Chromatin Immunoprecipitation | Forkhead Transcription Factors - metabolism | Histone Acetyltransferases - metabolism | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | HEK293 Cells | Adaptor Proteins, Signal Transducing - immunology | Acetylation | Colitis - immunology | Lysine Acetyltransferase 5 | Transcription Factors - immunology | Real-Time Polymerase Chain Reaction | STAT3 Transcription Factor - metabolism | Cytokines - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | DNA-Binding Proteins - immunology | Smad Proteins - immunology | Mice, Transgenic | Mice, Knockout | Transcription Factors - metabolism | Cell Differentiation - immunology | Microscopy, Confocal | Animals | Adaptor Proteins, Signal Transducing - genetics | Th17 Cells - immunology | Trans-Activators - metabolism | Mice | HeLa Cells | Proteasome Endopeptidase Complex - metabolism | STAT3 Transcription Factor - immunology | Arthritis, Rheumatoid - immunology | Smad Proteins - metabolism | Microscopy, Fluorescence
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2010, Volume 120, Issue 6, pp. 2131 - 2143
Enteropathy-associated T cell lymphoma is a severe complication of celiac disease (CD). One mechanism suggested to underlie its development is chronic exposure... 
INTESTINAL INFLAMMATION | RHEUMATOID-ARTHRITIS | MEDICINE, RESEARCH & EXPERIMENTAL | MOLECULAR TARGETS | NATURAL-KILLER-CELLS | INTERLEUKIN-15 | BCL-X | RECEPTOR | REFRACTORY SPRUE | CD8(+) T-CELLS | TRANSGENIC MICE | Phosphorylation | Apoptosis - drug effects | Enterocytes - metabolism | Humans | Intestines - metabolism | Leukemia - metabolism | STAT3 Transcription Factor - pharmacology | Interleukin-15 - immunology | Intestines - immunology | Inflammation - complications | Signal Transduction - immunology | Inflammation - metabolism | Lymphocytes - immunology | Celiac Disease - immunology | Leukemia - complications | Celiac Disease - complications | Adult | Janus Kinase 3 - metabolism | Intestine, Small - immunology | STAT3 Transcription Factor - metabolism | Cytokines - immunology | Janus Kinase 3 - immunology | Lymphocytes - metabolism | Cytokines - metabolism | Inflammation - immunology | Protein Binding - immunology | Enterocytes - immunology | Interleukin-15 - pharmacology | Apoptosis - immunology | Signal Transduction - drug effects | Leukemia - immunology | Interleukin-15 - metabolism | Intestine, Small - metabolism | STAT3 Transcription Factor - immunology | Celiac Disease - metabolism | Cytokines - pharmacology | Care and treatment | Lymphocytes | Development and progression | Interleukin-15 | Inflammation | Non-Hodgkin's lymphomas | T cells | Properties | Health aspects
Journal Article