X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (1727) 1727
Publication (279) 279
Book Review (33) 33
Book Chapter (11) 11
Magazine Article (5) 5
Conference Proceeding (3) 3
Book / eBook (1) 1
Data Set (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (1376) 1376
index medicus (735) 735
rhinovirus (584) 584
rhinovirus - drug effects (534) 534
antiviral agents - pharmacology (395) 395
asthma (389) 389
animals (365) 365
male (358) 358
female (357) 357
viruses (316) 316
immunology (293) 293
adult (250) 250
virology (250) 250
infections (236) 236
respiratory system (232) 232
hela cells (228) 228
health aspects (227) 227
pharmacology & pharmacy (227) 227
children (197) 197
common cold (190) 190
virus replication - drug effects (185) 185
infection (175) 175
antiviral agents (171) 171
cells, cultured (167) 167
middle aged (167) 167
rhinovirus - physiology (167) 167
allergy (164) 164
microbiology (160) 160
biochemistry & molecular biology (156) 156
respiratory syncytial virus (156) 156
rhinovirus infection (153) 153
virus diseases (150) 150
adolescent (147) 147
inflammation (147) 147
cell line (146) 146
infectious diseases (146) 146
child (145) 145
mice (144) 144
research article (141) 141
rhinovirus - immunology (134) 134
antiviral agents - therapeutic use (132) 132
research (131) 131
common cold - drug therapy (129) 129
structure-activity relationship (122) 122
analysis (117) 117
respiratory tract infections - virology (117) 117
rhinovirus - isolation & purification (117) 117
chemistry, medicinal (116) 116
in-vitro (116) 116
viral infections (115) 115
child, preschool (113) 113
virus (113) 113
human rhinovirus (112) 112
antiviral agents - chemistry (109) 109
rhinovirus - genetics (109) 109
respiratory tract diseases (108) 108
aged (107) 107
article (106) 106
infant (106) 106
models, molecular (105) 105
epithelial cells - virology (104) 104
picornaviridae infections - virology (103) 103
expression (102) 102
influenza (101) 101
antiviral agents - chemical synthesis (100) 100
asthma - immunology (98) 98
poliovirus (96) 96
picornaviridae infections - drug therapy (95) 95
exacerbations (94) 94
rhinovirus - metabolism (94) 94
asthma - drug therapy (93) 93
risk factors (93) 93
rhinovirus - pathogenicity (91) 91
abridged index medicus (90) 90
obstructive pulmonary-disease (90) 90
rhinoviruses (90) 90
cytokines (88) 88
nf-kappa-b (87) 87
proteins (87) 87
viral-infections (87) 87
cell biology (86) 86
double-blind method (84) 84
epithelial cells - drug effects (84) 84
asthma - virology (83) 83
double-blind (81) 81
replication (81) 81
time factors (81) 81
picornaviridae infections - immunology (76) 76
medicine (74) 74
microbial sensitivity tests (72) 72
rhinovirus 3c protease (72) 72
allergy and immunology (71) 71
rna (71) 71
dose-response relationship, drug (70) 70
rhinovirus - growth & development (70) 70
bronchial epithelial-cells (69) 69
chronic obstructive pulmonary disease (69) 69
drug design (69) 69
epithelial cells - metabolism (69) 69
multidisciplinary sciences (69) 69
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (1692) 1692
German (17) 17
Russian (7) 7
Italian (6) 6
Chinese (5) 5
French (4) 4
Japanese (4) 4
Spanish (2) 2
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


PLoS Pathogens, ISSN 1553-7366, 10/2012, Volume 8, Issue 10, p. e1002976
Viruses use a limited set of host pathways for infection. These pathways represent bona fide antiviral targets with low likelihood of viral resistance. We... 
RAT-LIVER | LOW-PH | CELLS | PLECONARIL | VIRUS | ENTRY | HUMAN RHINOVIRUSES | MICROBIOLOGY | NITAZOXANIDE | RESPIRATORY SYNDROME CORONAVIRUS | REPLICATION | VIROLOGY | PARASITOLOGY | Influenza viruses | Physiological aspects | Antiviral agents | Amides | Rhinoviruses | Health aspects | Proteins | Antibiotics | Microscopy | Viruses | Infections | Genomes | FDA approval | Drug dosages | Viral infections
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 01/2015, Volume 191, Issue 1, pp. 34 - 44
A first step in primary disease prevention is identifying common, modifiable risk factors that contribute to a significant proportion of disease development.... 
Respiratory syncytial virus | RSV | Asthma | Rhinovirus | AIRWAY EPITHELIAL-CELLS | COMMUNITY-ACQUIRED PNEUMONIA | SYNCYTIAL VIRUS-INFECTION | REDUCED LUNG-FUNCTION | RV | INNATE IMMUNE-RESPONSE | asthma | PRETERM INFANTS | ENVIRONMENTAL TOBACCO-SMOKE | rhinovirus | VITAMIN-D DEFICIENCY | RESPIRATORY SYSTEM | HUMAN RHINOVIRUS | respiratory syncytial virus | MATERNAL SMOKING | CRITICAL CARE MEDICINE | Asthma - prevention & control | Epigenesis, Genetic | Humans | Respiratory Syncytial Viruses - immunology | Microbiota - drug effects | Child, Preschool | Infant | Respiratory Syncytial Virus Infections - immunology | Palivizumab | Respiratory Syncytial Virus Infections - drug therapy | Asthma - immunology | Respiratory Tract Infections - complications | Asthma - virology | Respiratory Tract Infections - virology | Rhinovirus - immunology | Respiratory Syncytial Virus Infections - complications | Rhinovirus - drug effects | Severity of Illness Index | Genetic Predisposition to Disease | Antibodies, Monoclonal, Humanized - therapeutic use | Microbiota - immunology | Antiviral Agents - therapeutic use | Respiratory Syncytial Viruses - drug effects | Risk Factors | Respiratory Syncytial Virus Infections - genetics | Respiratory Tract Infections - drug therapy | Asthma - genetics | Environmental Exposure - adverse effects | Tobacco Smoke Pollution - adverse effects | Diet | Microbiota - genetics | Respiratory Syncytial Viruses - genetics | Primary Prevention - methods | Respiratory Tract Infections - immunology | Rhinovirus - genetics | Pulmonary
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e86755
Vitamin D has been linked to reduced risk of viral respiratory illness. We hypothesized that vitamin D could directly reduce rhinovirus (RV) replication in... 
DEFENSE | ANTAGONIZES | CYTOKINES | MULTIDISCIPLINARY SCIENCES | DISEASE | D DEFICIENCY | SQUAMOUS METAPLASIA | ANTIMICROBIAL PEPTIDES | INDUCTION | PROLINE-RICH PROTEIN | RESPIRATORY-TRACT INFECTION | Bronchi - virology | Respiratory Mucosa - drug effects | Epithelial Cells - drug effects | Humans | Respiratory Mucosa - virology | Rhinovirus - physiology | Cornified Envelope Proline-Rich Proteins - agonists | Bronchi - drug effects | Respiratory Mucosa - pathology | Chemokine CXCL10 - biosynthesis | Cornified Envelope Proline-Rich Proteins - genetics | Interleukin-8 - metabolism | Bronchi - pathology | Wound Healing - drug effects | Rhinovirus - drug effects | Virus Replication - drug effects | Gene Expression | Interleukin-8 - biosynthesis | Epithelial Cells - ultrastructure | Epithelial Cells - pathology | Keratin-5 - genetics | Cell Shape - drug effects | Cell Differentiation - drug effects | Calcitriol - pharmacology | Epithelial Cells - virology | Cornified Envelope Proline-Rich Proteins - metabolism | Keratin-5 - metabolism | Cell Proliferation - drug effects | Keratin-5 - agonists | Primary Cell Culture | Chemokine CXCL10 - metabolism | Vitamin D | Analysis | Calcifediol | Respiratory tract diseases | Alfacalcidol | Gene expression | Enzyme-linked immunosorbent assay | Cell proliferation | Immunohistochemistry | Cytokeratin | Pediatrics | Peptides | Epithelial cells | Cytology | Proline | Smooth muscle | Infections | Experiments | Cell morphology | Respiratory tract | Protein folding | Rodents | Chronic obstructive pulmonary disease | Supplementation | Thickening | Enzymes | Wound healing | Cytokines | Secretion | Risk reduction | Vitamin deficiency | Antimicrobial agents | Epithelium | Asthma | Metaplasia | Acids | Morphology | CXCL10 protein | Replication | Chemokines | Viral infections
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2016, Volume 113, Issue 6, pp. 1642 - 1647
Clinical observations link respiratory virus infection and colonization in chronic lung disease, including cystic fibrosis (CF) and chronic obstructive... 
Biofilm | Respiratory Syncytial Virus | Pseudomonas Aeruginosa | Cystic Fibrosis | Nutritional Immunity | MULTIDISCIPLINARY SCIENCES | RHINOVIRUS | COLONIZATION | AIRWAY IRON | TRANSITION | nutritional immunity | EPITHELIAL-CELLS | IRON HOMEOSTASIS | TRANSFERRIN | cystic fibrosis | biofilm | Pseudomonas aeruginosa | BACTERIAL BIOFILM | CYSTIC-FIBROSIS | respiratory syncytial virus | PNEUMONIAE | Bronchoalveolar Lavage Fluid | Respiratory Syncytial Viruses - physiology | Epithelial Cells - drug effects | Humans | Biofilms - growth & development | Cystic Fibrosis - pathology | Nutritional Physiological Phenomena | Pseudomonas aeruginosa - physiology | Immunity | Homeostasis - drug effects | Iron - pharmacology | Bronchi - pathology | Antiviral Agents - pharmacology | Cystic Fibrosis - microbiology | Respiratory Syncytial Viruses - drug effects | Pseudomonas aeruginosa - drug effects | Microbial Interactions - drug effects | Animals | Interferon-beta - pharmacology | Signal Transduction - drug effects | Models, Biological | Epithelial Cells - microbiology | Epithelial Cells - virology | Mice | Respiratory Syncytial Virus Infections - pathology | Transferrin - metabolism | Host-bacteria relationships | Respiratory syncytial virus | Immune response | Host-virus relationships | Observations | Health aspects | Biological Sciences
Journal Article
Journal of Antimicrobial Chemotherapy, ISSN 0305-7453, 10/2016, Volume 71, Issue 10, pp. 2767 - 2781
Background: Exacerbations of asthma and COPD are triggered by rhinoviruses. Uncontrolled inflammatory pathways, pathogenic bacterial burden and impaired... 
INFECTIOUS DISEASES | MICROBIOLOGY | BLOOD MONONUCLEAR-CELLS | OBSTRUCTIVE PULMONARY-DISEASE | ANTIVIRAL RESPONSES | HUMAN MXA PROTEIN | ASTHMA EXACERBATIONS | IMMUNE-RESPONSES | PHARMACOLOGY & PHARMACY | ENDOPLASMIC-RETICULUM | ANTIBIOTIC-TREATMENT | HEPATITIS-C | AMPHIPATHIC ALPHA-HELIX | Interferons - biosynthesis | Interferon-beta - immunology | Gram-Positive Bacteria - drug effects | Haemophilus influenzae - drug effects | Epithelial Cells - drug effects | Escherichia coli - drug effects | Humans | Anti-Inflammatory Agents, Non-Steroidal - chemistry | Anti-Inflammatory Agents, Non-Steroidal - isolation & purification | Interferons - immunology | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Bronchi - drug effects | Anti-Bacterial Agents - therapeutic use | Macrolides - chemistry | Antiviral Agents - chemistry | Anti-Bacterial Agents - chemistry | Macrolides - pharmacology | Interleukin-8 - metabolism | Drug Evaluation, Preclinical | Bronchi - cytology | Interleukin-6 - metabolism | Rhinovirus - drug effects | Virus Replication - drug effects | Antiviral Agents - pharmacology | Antiviral Agents - therapeutic use | Cells, Cultured | Anti-Bacterial Agents - isolation & purification | Pseudomonas aeruginosa - drug effects | Asthma - drug therapy | Drug Discovery | Proteins - genetics | Gram-Negative Bacteria - drug effects | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Interleukin-6 - immunology | Anti-Bacterial Agents - pharmacology | Antiviral Agents - isolation & purification | Interleukin-8 - immunology | Macrolides - therapeutic use | Myxovirus Resistance Proteins - genetics | Index Medicus | Original Research
Journal Article
Respiratory Research, ISSN 1465-9921, 03/2015, Volume 16, Issue 1, p. 42
Background and purpose: The persistent influx of neutrophils into the lung and subsequent tissue damage are characteristics of COPD, cystic fibrosis and acute... 
Adhesion molecules | VAP-1/SSAO | Lung inflammation | Neutrophils | COPD | INDUCED ASTHMA | RECRUITMENT | VASCULAR ADHESION PROTEIN-1 | AIRWAY INFLAMMATION | SENSITIVE AMINE OXIDASE | CXCR2 ANTAGONIST | INDUCED LUNG INFLAMMATION | RESPIRATORY SYSTEM | IN-VIVO | INFECTION | CYSTIC-FIBROSIS | Respiratory Tract Infections - enzymology | Cell Adhesion Molecules - antagonists & inhibitors | Rhinovirus - pathogenicity | Cecum - microbiology | Picornaviridae Infections - virology | Lipopolysaccharides | Lung - enzymology | Cecum - surgery | Bronchoconstriction - drug effects | Asthma - immunology | Pneumonia - immunology | Respiratory Tract Infections - etiology | Asthma - virology | Neutrophil Infiltration - drug effects | Disease Models, Animal | Klebsiella Infections - enzymology | Asthma - physiopathology | Anti-Inflammatory Agents - pharmacology | Pneumonia - enzymology | Asthma - enzymology | Allylamine - analogs & derivatives | Enzyme Inhibitors - pharmacology | Lung - physiopathology | Cell Adhesion Molecules - metabolism | Picornaviridae Infections - enzymology | Lung - drug effects | Respiratory Tract Infections - immunology | Picornaviridae Infections - physiopathology | Mice, Inbred BALB C | Endothelial Cells - enzymology | Leukocyte Rolling - drug effects | Rats, Wistar | Benzamides - pharmacokinetics | Picornaviridae Infections - drug therapy | Dose-Response Relationship, Drug | Ligation | Amine Oxidase (Copper-Containing) - antagonists & inhibitors | Enzyme Inhibitors - pharmacokinetics | Allylamine - pharmacology | Anti-Inflammatory Agents - pharmacokinetics | Benzamides - pharmacology | Klebsiella Infections - drug therapy | Picornaviridae Infections - immunology | Pneumonia - etiology | Mice, Inbred C57BL | Punctures | Respiratory Tract Infections - drug therapy | Klebsiella Infections - microbiology | Asthma - drug therapy | Amine Oxidase (Copper-Containing) - metabolism | Endothelial Cells - immunology | Animals | Pneumonia - drug therapy | Klebsiella Infections - immunology | Klebsiella pneumoniae - pathogenicity | Allylamine - pharmacokinetics | Lung - immunology | Endothelial Cells - drug effects | Health aspects | Analysis | Endothelium | VAP-1 | SSAO | Research
Journal Article
Immunological Reviews, ISSN 0105-2896, 07/2011, Volume 242, Issue 1, pp. 205 - 219
The adoption of the concept that asthma is primarily a disease most frequently associated with elaboration of T‐helper 2 (Th2)‐type inflammation has led to the... 
remodeling | environmental injury | phenotypes | inflammation | origins | progression | asthma | epithelium | Progression | Origins | Phenotypes | Environmental injury | Inflammation | Epithelium | Remodeling | Asthma | RETICULAR BASEMENT-MEMBRANE | CHEMOKINE RECEPTOR 4 | DENDRITIC CELLS | INCREASED EXPRESSION | IMMUNOLOGY | CHILDHOOD ASTHMA | THYMIC STROMAL LYMPHOPOIETIN | TIGHT JUNCTIONS | EPIDERMAL-GROWTH-FACTOR | GOBLET CELL HYPERPLASIA | BRONCHIAL-ASTHMA | Epithelial Cells - metabolism | Epithelial Cells - drug effects | Humans | Hypersensitivity - immunology | Infant | Th2 Cells - immunology | Rhinovirus - physiology | Hypersensitivity - drug therapy | Airway Remodeling - immunology | Picornaviridae Infections - virology | Epithelium - immunology | Signal Transduction - immunology | Allergens - immunology | Asthma - immunology | Anti-Inflammatory Agents - therapeutic use | Adult | Respiratory Tract Infections - virology | Rhinovirus - immunology | Child | Respiratory System - immunology | Cytokines - immunology | Adaptive Immunity | Epithelium - drug effects | Picornaviridae Infections - immunology | Asthma - physiopathology | Epithelium - metabolism | Inflammation - immunology | Immunity, Innate | Asthma - drug therapy | Th2 Cells - metabolism | Mice, Knockout | Animals | Hypersensitivity - physiopathology | Epithelial Cells - immunology | Respiratory System - drug effects | Respiratory Tract Infections - immunology | Respiratory System - physiopathology | Mice | Mucus - immunology | Allergens | Allergy | Dendritic cells | Asthma in children | Development and progression | Disease susceptibility | Health aspects | Allergic reaction | Hypersensitivity | Helper cells | Lymphocytes T | Atopy | Respiratory tract | Bronchoconstriction | Respiratory tract diseases | Microenvironments | Children | Pollutants | Growth factors | Adoption
Journal Article
AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY, ISSN 1044-1549, 02/2017, Volume 56, Issue 2, pp. 242 - 251
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 2017, Volume 312, Issue 1, pp. L89 - L99
the aim of this study is to elucidate the role of TRAIL during rhinovirus (RV) infection in vivo. Naive wild-type and tumor necrosis factor-related... 
Inflammation | TRAIL | Tumor necrosis factor-related apoptosis-inducing ligand | Asthma | Rhinovirus | PHYSIOLOGY | VIRUS-INFECTION | CHRONIC HEPATITIS-B | ALLERGIC AIRWAYS DISEASE | asthma | PROTEIN PHOSPHATASE 2A | rhinovirus | PLASMACYTOID DENDRITIC CELLS | VIRAL-INFECTIONS | APOPTOSIS-INDUCING LIGAND | ANTIVIRAL DEFENSE | NK CELLS | inflammation | RESPIRATORY SYSTEM | tumor necrosis factor-related apoptosis-inducing ligand | T-CELLS | Inflammation - pathology | Epithelial Cells - metabolism | Epithelial Cells - drug effects | Humans | Phosphoprotein Phosphatases - metabolism | Male | Rhinovirus - physiology | RNA, Messenger - metabolism | Picornaviridae Infections - virology | Hypersensitivity - metabolism | T-Lymphocytes - metabolism | Hypersensitivity - parasitology | T-Lymphocytes - drug effects | Pyroglyphidae - drug effects | Inflammation Mediators - metabolism | TNF-Related Apoptosis-Inducing Ligand - deficiency | Interferon Type I - metabolism | Dendritic Cells - drug effects | Dendritic Cells - metabolism | Disease Models, Animal | Picornaviridae Infections - pathology | Rhinovirus - drug effects | Virus Replication - drug effects | Antiviral Agents - pharmacology | Picornaviridae Infections - immunology | RNA, Messenger - genetics | Inflammation - virology | TNF-Related Apoptosis-Inducing Ligand - metabolism | Pyroglyphidae - physiology | Proteins - genetics | Bronchial Hyperreactivity - pathology | Animals | Proteins - metabolism | Signal Transduction - drug effects | Hypersensitivity - pathology | Killer Cells, Natural - drug effects | Mice, Inbred BALB C | HeLa Cells | Killer Cells, Natural - metabolism
Journal Article
Immunological Reviews, ISSN 0105-2896, 07/2011, Volume 242, Issue 1, pp. 186 - 204
The airway epithelial cell is the initial cell type impacted both by inhaled environmental factors, such as pathogens, allergens, and pollutants, and inhaled... 
innate immunity | epithelial cell | inflammation | airway remodeling | host defense | Epithelial cell | Innate immunity | Inflammation | Airway remodeling | Host defense | COLONY-STIMULATING FACTOR | INCREASED EXPRESSION | NITRIC-OXIDE SYNTHASE | PHOSPHATIDYLINOSITOL 3-KINASE | PROINFLAMMATORY CYTOKINES | IMMUNOLOGY | THYMIC STROMAL LYMPHOPOIETIN | IN-VITRO | HUMAN RHINOVIRUS INFECTION | NF-KAPPA-B | MOLECULAR-MECHANISMS | Bronchodilator Agents - therapeutic use | Epithelial Cells - metabolism | Epithelial Cells - drug effects | Humans | Hypersensitivity - immunology | Cell Adhesion Molecules - immunology | Rhinovirus - physiology | Hypersensitivity - drug therapy | Airway Remodeling - immunology | Picornaviridae Infections - virology | Epithelium - immunology | Signal Transduction - immunology | Allergens - immunology | Asthma - immunology | Respiratory Tract Infections - virology | Rhinovirus - immunology | Child | Respiratory System - immunology | Cytokines - immunology | Epithelium - drug effects | Picornaviridae Infections - immunology | Asthma - physiopathology | Epithelium - metabolism | Inflammation - immunology | Immunity, Innate | Asthma - drug therapy | Mice, Knockout | Animals | Hypersensitivity - physiopathology | Epithelial Cells - immunology | Respiratory System - drug effects | Respiratory Tract Infections - immunology | Respiratory System - physiopathology | Mice | Intercellular Signaling Peptides and Proteins - immunology | Allergens | Respiratory agents | Universities and colleges | Drugs | Pathogens | Immune response | Epithelial cells | Immunoregulation | Environmental factors | Epithelium | Asthma | Reviews | Respiratory tract diseases | Pollutants | Structure-function relationships
Journal Article