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Journal of Cell Science, ISSN 0021-9533, 03/2018, Volume 131, Issue 5, pp. e0502 - e0502
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 10/2013, Volume 127, Issue 2, pp. 149 - 151
Read the full article ‘Amyloid beta1–42 (Aβ42) up‐ regulates the expression of sortilin via the p75NTR/RhoA signaling pathway’ on doi: 10.1111/jnc.12383 
RhoA protein
Journal Article
Nature Genetics, ISSN 1061-4036, 11/2019, Volume 51, Issue 11, pp. 1660 - 1660
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, p. e101330
The loss of microRNA-122 (miR-122) expression is strongly associated with increased invasion and metastasis, and poor prognosis of hepatocellular carcinoma... 
CANCER-CELLS | LIPID-METABOLISM | METASTASIS | MULTIDISCIPLINARY SCIENCES | IN-VIVO | LIVER | DOWN-REGULATION | CYCLE ARREST | TUMOR-SUPPRESSOR | NUCLEAR FACTOR 4-ALPHA | MIR-122 EXPRESSION | Up-Regulation | MicroRNAs - antagonists & inhibitors | Cadherins - metabolism | alpha Catenin - metabolism | Vimentin - metabolism | Humans | Oligonucleotides, Antisense - metabolism | MicroRNAs - metabolism | rhoA GTP-Binding Protein - metabolism | rhoA GTP-Binding Protein - genetics | Transfection | Base Sequence | Epithelial-Mesenchymal Transition | Liver Neoplasms - pathology | 3' Untranslated Regions | Hepatocyte Nuclear Factor 4 - metabolism | rhoA GTP-Binding Protein - chemistry | Signal Transduction | Down-Regulation | Sequence Alignment | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Line, Tumor | MicroRNAs - genetics | Carcinoma, Hepatocellular - metabolism | Cell Movement | Fibronectins | MicroRNA | Hepatoma | Gene therapy | Intermediate filament proteins | Stem cells | Vimentin | Motility | Mesenchyme | Genes | Hepatocellular carcinoma | Metastasis | Inactivation | E-cadherin | Fibronectin | Metastases | Proteins | Liver cancer | Rodents | Hepatology | Cell adhesion | Inhibition | α-catenin | Medical research | Deactivation | Invasiveness | MiRNA | RhoA protein | Suppressors | Zonula occludens-1 protein | Rho-associated kinase | Overexpression | Ribonucleic acids | Plasmids | MicroRNAs | Medical prognosis | Cell lines | Cytoskeleton | Laboratory animals | Binding sites | Cell migration
Journal Article
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 8/2014, Volume 34, Issue 8, pp. 1258 - 1269
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2010, Volume 107, Issue 22, pp. 9944 - 9949
Actomyosin contractility affects cellular organization within tissues in part through the generation of mechanical forces at sites of cell—matrix and cell—cell... 
Cell growth | Microinjections | Epithelial cells | Bowties | Cell adhesion | Cadherins | Adherens junctions | Focal adhesions | Physiological regulation | Endothelial cells | Mechanotransduction | Traction force | Myosin | Adherens junction | PDMS | CONTACTS | ACTIVATION | mechanotransduction | MULTIDISCIPLINARY SCIENCES | MYOSIN IIB | myosin | EXTENSION | FOCAL ADHESIONS | traction force | SHAPE | adherens junction | RAC | GROWTH | RHO | VASCULAR ENDOTHELIAL-CADHERIN | Myosin Type II - physiology | rhoA GTP-Binding Protein - administration & dosage | Cadherins - metabolism | Mechanotransduction, Cellular - drug effects | Humans | Green Fluorescent Proteins - genetics | rhoA GTP-Binding Protein - physiology | rhoA GTP-Binding Protein - genetics | Mechanotransduction, Cellular - physiology | Antigens, CD - genetics | Antigens, CD - metabolism | Endothelial Cells - ultrastructure | Thrombin - pharmacology | Lysophospholipids - pharmacology | rac GTP-Binding Proteins - physiology | Cadherins - genetics | Endothelial Cells - physiology | Recombinant Proteins - metabolism | Green Fluorescent Proteins - metabolism | Cells, Cultured | Recombinant Proteins - genetics | rho GTP-Binding Proteins - physiology | Recombinant Proteins - administration & dosage | Sphingosine - pharmacology | Biomechanical Phenomena | Sphingosine - analogs & derivatives | Adherens Junctions - physiology | Adherens Junctions - drug effects | Adherens Junctions - ultrastructure | Amino Acid Substitution | Endothelial Cells - drug effects | Junctional complexes (Epithelium) | Cell junctions | Properties | Biological Sciences | Physical Sciences
Journal Article
Laboratory Investigation, ISSN 0023-6837, 10/2015, Volume 95, Issue 10, pp. 1145 - 1156
Hepatocellular carcinoma (HCC) carries a poor prognosis with no effective treatment available other than liver transplantation for selected patients. Vascular... 
MIGRATION | MEDICINE, RESEARCH & EXPERIMENTAL | APOPTOSIS | HEPATOCELLULAR-CARCINOMA | METASTASIS | CANCER CELLS | PATHWAY | INFLAMMATION | LIVER | PATHOLOGY | NF-KAPPA-B | EXPRESSION | Liver - pathology | Galectin 3 - metabolism | Male | Myosin-Light-Chain Kinase - antagonists & inhibitors | Neoplasm Proteins - antagonists & inhibitors | rhoA GTP-Binding Protein - metabolism | rhoA GTP-Binding Protein - genetics | Neoplasm Proteins - metabolism | rho-Associated Kinases - antagonists & inhibitors | Carcinoma, Hepatocellular - drug therapy | Liver - drug effects | Protein Processing, Post-Translational - drug effects | rho-Associated Kinases - metabolism | Actin Cytoskeleton - drug effects | Liver Neoplasms - pathology | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Neoplasm Proteins - genetics | Cardiac Myosins - metabolism | Myosin-Light-Chain Kinase - chemistry | Actin Cytoskeleton - metabolism | Myosin-Light-Chain Kinase - metabolism | Neoplasm Invasiveness | Liver - metabolism | Mice, Inbred C57BL | Gene Silencing | Liver Neoplasms - drug therapy | rhoA GTP-Binding Protein - antagonists & inhibitors | Enzyme Activation - drug effects | Galectin 3 - genetics | Mice, Knockout | Cell Movement - drug effects | Animals | Neoplasm Proteins - agonists | Galectin 3 - antagonists & inhibitors | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Line, Tumor | Myosin Light Chains - metabolism | Protein Kinase Inhibitors - pharmacology | rhoA GTP-Binding Protein - agonists | Actin Cytoskeleton - pathology | Carcinoma, Hepatocellular - metabolism | Index Medicus | Galectin 3 | cell migration | MLC2 | hepatocellular carcinoma | RhoA
Journal Article
Anti-Cancer Drugs, ISSN 0959-4973, 02/2017, Volume 28, Issue 5, pp. 514 - 521
GTPase RhoA and its downstream Rho-associated coiled-coil-containing protein kinases (ROCKs) are frequently overexpressed in human cancers. Inhibition of the... 
RhoA/ROCK signaling pathway | vasculogenic mimicry | fasudil | osteosarcoma | MIGRATION | RHO-KINASE INHIBITOR | POTENTIAL THERAPEUTIC TARGET | PROLIFERATION | RhoA | LUNG-CANCER | GTPASES | ONCOLOGY | GROWTH | PHARMACOLOGY & PHARMACY | ANTI-ANGIOGENIC THERAPY | MELANOMA-CELLS | NEOVASCULARIZATION | ROCK signaling pathway | Osteosarcoma - drug therapy | Cell Plasticity - drug effects | Humans | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - analogs & derivatives | rhoA GTP-Binding Protein - metabolism | rhoA GTP-Binding Protein - biosynthesis | Bone Neoplasms - pathology | rho-Associated Kinases - antagonists & inhibitors | Bone Neoplasms - metabolism | Neovascularization, Pathologic - pathology | rho-Associated Kinases - metabolism | Bone Neoplasms - blood supply | Osteosarcoma - blood supply | rho-Associated Kinases - biosynthesis | Bone Neoplasms - drug therapy | Osteosarcoma - metabolism | Cytoskeleton - pathology | rhoA GTP-Binding Protein - antagonists & inhibitors | Cell Movement - drug effects | Signal Transduction - drug effects | Neovascularization, Pathologic - drug therapy | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | Fluorescent Antibody Technique | Cell Line, Tumor | Cell Proliferation - drug effects | Neovascularization, Pathologic - metabolism | Protein Kinase Inhibitors - pharmacology | Cytoskeleton - drug effects | Osteosarcoma - pathology
Journal Article
Nature Methods, ISSN 1548-7091, 03/2013, Volume 10, Issue 3, pp. 249 - 252
We report an optogenetic method based on Arabidopsis thaliana cryptochrome 2 for rapid and reversible protein oligomerization in response to blue light. We... 
SYSTEM | SPATIOTEMPORAL CONTROL | LIGHT | BIOLOGY | BIOCHEMICAL RESEARCH METHODS | INDUCTION | LIVING CELLS | Wnt3A Protein - metabolism | NIH 3T3 Cells | Cryptochromes - radiation effects | Fluorescence Recovery After Photobleaching | Humans | Cryptochromes - genetics | Cytoplasm - metabolism | rhoA GTP-Binding Protein - metabolism | beta Catenin - radiation effects | rhoA GTP-Binding Protein - genetics | Recombinant Fusion Proteins - metabolism | Arabidopsis Proteins - metabolism | Cell Nucleus - metabolism | Light Signal Transduction | Transfection | Luminescent Proteins - radiation effects | Cell Nucleus - radiation effects | Recombinant Fusion Proteins - radiation effects | Light | HEK293 Cells | Wnt3A Protein - radiation effects | Transcription, Genetic | rhoA GTP-Binding Protein - radiation effects | Cell Culture Techniques | Wnt Signaling Pathway - radiation effects | Cryptochromes - metabolism | Protein Multimerization - radiation effects | Arabidopsis Proteins - genetics | Cytoplasm - radiation effects | Wnt3A Protein - genetics | Enzyme-Linked Immunosorbent Assay | beta Catenin - metabolism | Blotting, Western | beta Catenin - genetics | Arabidopsis Proteins - radiation effects | Animals | Mice | Luminescent Proteins - metabolism | Arabidopsis thaliana | Plant genetics | Physiological aspects | Protein biosynthesis | Genetic aspects | Research | Genetic transcription | Biochemistry | Mammals | Photobiology | Protein synthesis
Journal Article
Leukemia, ISSN 0887-6924, 03/2018, Volume 32, Issue 3, pp. 694 - 702
Journal Article