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Journal Article
Journal Article
Journal Article
Gut, ISSN 0017-5749, 06/2014, Volume 63, Issue 6, pp. 903 - 910
Objective Although polymorphisms of the NOD2 gene predispose to the development of ileal Crohn's disease, the precise mechanisms of this increased... 
Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Defensins - metabolism | Defensins - genetics | Escherichia coli - drug effects | Peptides - genetics | Ileum - metabolism | Nod2 Signaling Adaptor Protein - genetics | RNA, Messenger - metabolism | alpha-Defensins - genetics | Microbial Sensitivity Tests | Salmonella enterica - drug effects | Feces - microbiology | Lectins, C-Type - metabolism | Peptides - metabolism | Antigens, Neoplasm - metabolism | Biomarkers, Tumor - metabolism | Transcription, Genetic | alpha-Defensins - metabolism | Ileum - cytology | Paneth Cells - metabolism | Protein Precursors - genetics | Mice, Inbred C57BL | Paneth Cells - cytology | Ribonuclease, Pancreatic - genetics | Muramidase - metabolism | Pancreatitis-Associated Proteins | Mice, Knockout | Protein Precursors - metabolism | alpha-Defensins - pharmacology | Animals | Nod2 Signaling Adaptor Protein - metabolism | Ribonuclease, Pancreatic - metabolism | Analysis | Antimicrobial peptides | Crohn's disease | Genetic aspects | Research | Gene expression | Risk factors | Genetic polymorphisms | Studies | Pathogenesis | Rodents | Antimicrobial agents | Inflammation | Clinical medicine | Laboratory animals | Crohns disease | Index Medicus | Abridged Index Medicus | IBD - GENETICS | INTESTINAL EPITHELIUM | BACTERIAL INTERACTIONS | CROHN'S DISEASE | Inflammatory Bowel Disease | 1506
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 04/2016, Volume 113, Issue 16, pp. 4470 - 4475
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 04/2016, Volume 113, Issue 16, pp. 4476 - 4481
Inhibition of the vascular endothelial growth factor (VEGF) pathway has failed to improve overall survival of patients with glioblastoma (GBM). We previously... 
Anti-angiogenic therapy | Macrophage polarization | Tumor microenvironment | Microglia reprogramming | Anti-tumor immunity | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Macrophages - pathology | Antibodies, Bispecific - pharmacology | Vesicular Transport Proteins - metabolism | Humans | Neoplasm Proteins - antagonists & inhibitors | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Neoplasm Proteins - metabolism | Neoplasms, Experimental - pathology | Xenograft Model Antitumor Assays | Macrophages - metabolism | Vesicular Transport Proteins - antagonists & inhibitors | Animals | Antibodies, Neoplasm - pharmacology | Ribonuclease, Pancreatic - metabolism | Glioblastoma - pathology | Cell Line, Tumor | Glioblastoma - metabolism | Antineoplastic Agents - pharmacology | Mice | Neoplasms, Experimental - metabolism | Glioblastoma - drug therapy | Ribonuclease, Pancreatic - antagonists & inhibitors | Neoplasms, Experimental - drug therapy | Viral antibodies | Care and treatment | Antibodies | Physiological aspects | Models | Macrophages | Vascular endothelial growth factor | Health aspects | Glioblastoma multiforme | Genotype & phenotype | Survival analysis | Protein expression | Cells | Tumors | Cancer | Index Medicus | Biological Sciences | anti-angiogenic therapy | macrophage polarization | tumor microenvironment | microglia reprogramming | anti-tumor immunity
Journal Article