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Journal Article
Journal Article
Cancer Cell, ISSN 1535-6108, 04/2018, Volume 33, Issue 4, pp. 752 - 769.e8
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2016, Volume 113, Issue 16, pp. 4476 - 4481
Inhibition of the vascular endothelial growth factor (VEGF) pathway has failed to improve overall survival of patients with glioblastoma (GBM). We previously... 
Anti-angiogenic therapy | Macrophage polarization | Tumor microenvironment | Microglia reprogramming | Anti-tumor immunity | anti-angiogenic therapy | BEVACIZUMAB | MULTIDISCIPLINARY SCIENCES | macrophage polarization | RANDOMIZED-TRIAL | ANGIOPOIETIN-2 | CANCER | THERAPY | microglia reprogramming | TIE2-EXPRESSING MONOCYTES | BLOOD-VESSELS | TUMOR-ASSOCIATED MACROPHAGES | tumor microenvironment | NORMALIZATION | anti-tumor immunity | ENDOTHELIAL GROWTH-FACTOR | Macrophages - pathology | Antibodies, Bispecific - pharmacology | Vesicular Transport Proteins - metabolism | Humans | Neoplasm Proteins - antagonists & inhibitors | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Neoplasm Proteins - metabolism | Neoplasms, Experimental - pathology | Xenograft Model Antitumor Assays | Macrophages - metabolism | Vesicular Transport Proteins - antagonists & inhibitors | Animals | Antibodies, Neoplasm - pharmacology | Ribonuclease, Pancreatic - metabolism | Glioblastoma - pathology | Cell Line, Tumor | Glioblastoma - metabolism | Antineoplastic Agents - pharmacology | Mice | Neoplasms, Experimental - metabolism | Glioblastoma - drug therapy | Ribonuclease, Pancreatic - antagonists & inhibitors | Neoplasms, Experimental - drug therapy | Viral antibodies | Care and treatment | Antibodies | Physiological aspects | Models | Macrophages | Vascular endothelial growth factor | Health aspects | Glioblastoma multiforme | Genotype & phenotype | Survival analysis | Protein expression | Cells | Tumors | Cancer | Index Medicus | Biological Sciences
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2016, Volume 113, Issue 16, pp. 4470 - 4475
Glioblastomas (GBMs) rapidly become refractory to anti-VEGF therapies. We previously demonstrated that ectopic overexpression of angiopoietin-2 (Ang-2)... 
Anti-angiogenic therapy | Colony-stimulating factor 1 | Tumor immunity | Tumor microenvironment | Macrophage | anti-angiogenic therapy | ANGIOPOIETIN-2 FUNCTIONS | ANGIOGENESIS | MULTIDISCIPLINARY SCIENCES | macrophage | BEVACIZUMAB PLUS IRINOTECAN | RANDOMIZED-TRIAL | ANTIBODY | tumor immunity | KINASE INHIBITOR | ANTIANGIOGENIC THERAPY | PHASE-II TRIAL | colony-stimulating factor 1 | tumor microenvironment | EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | Macrophages - pathology | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - metabolism | Neovascularization, Pathologic - pathology | Neoplasms, Experimental - pathology | Macrophages - metabolism | Animals | Antibodies, Neoplasm - pharmacology | Receptors, Vascular Endothelial Growth Factor - metabolism | Ribonuclease, Pancreatic - metabolism | Neovascularization, Pathologic - drug therapy | Glioblastoma - pathology | Cell Line, Tumor | Receptors, Vascular Endothelial Growth Factor - antagonists & inhibitors | Glioblastoma - metabolism | Mice | Neovascularization, Pathologic - metabolism | Neoplasms, Experimental - metabolism | Glioblastoma - drug therapy | Quinazolines - pharmacology | Ribonuclease, Pancreatic - antagonists & inhibitors | Neoplasms, Experimental - drug therapy | Drug Screening Assays, Antitumor | Survival analysis | Gene expression | Kinases | Cells | Tumors | Cancer | Index Medicus | Biological Sciences
Journal Article
Gut, ISSN 0017-5749, 06/2014, Volume 63, Issue 6, pp. 903 - 910
Objective Although polymorphisms of the NOD2 gene predispose to the development of ileal Crohn's disease, the precise mechanisms of this increased... 
INTESTINAL INFLAMMATION | HOMEOSTASIS | INFLAMMATORY-BOWEL-DISEASE | PEPTIDES | MICE | MICROBIOTA | DIVERSITY | GASTROENTEROLOGY & HEPATOLOGY | ILEAL CROHNS-DISEASE | ALPHA-DEFENSIN EXPRESSION | INNATE IMMUNITY | Defensins - metabolism | Defensins - genetics | Escherichia coli - drug effects | Peptides - genetics | Ileum - metabolism | Nod2 Signaling Adaptor Protein - genetics | RNA, Messenger - metabolism | alpha-Defensins - genetics | Microbial Sensitivity Tests | Salmonella enterica - drug effects | Feces - microbiology | Lectins, C-Type - metabolism | Peptides - metabolism | Antigens, Neoplasm - metabolism | Biomarkers, Tumor - metabolism | Transcription, Genetic | alpha-Defensins - metabolism | Ileum - cytology | Paneth Cells - metabolism | Protein Precursors - genetics | Mice, Inbred C57BL | Paneth Cells - cytology | Ribonuclease, Pancreatic - genetics | Muramidase - metabolism | Pancreatitis-Associated Proteins | Mice, Knockout | Protein Precursors - metabolism | alpha-Defensins - pharmacology | Animals | Nod2 Signaling Adaptor Protein - metabolism | Ribonuclease, Pancreatic - metabolism | Analysis | Antimicrobial peptides | Crohn's disease | Genetic aspects | Research | Gene expression | Risk factors | Genetic polymorphisms | Studies | Pathogenesis | Rodents | Antimicrobial agents | Inflammation | Clinical medicine | Laboratory animals | Crohns disease | Index Medicus | Abridged Index Medicus | IBD - GENETICS | INTESTINAL EPITHELIUM | BACTERIAL INTERACTIONS | CROHN'S DISEASE | Inflammatory Bowel Disease | 1506
Journal Article
Journal Article
Science, ISSN 0036-8075, 3/2005, Volume 307, Issue 5715, pp. 1630 - 1634
Antigen-presenting cells (APCs) internalize antigens and present antigen-derived peptides to T cells. Although APCs have been thought to exhibit a... 
Spleen | T lymphocytes | Antigens | Dextrans | Plant growth regulators | B lymphocytes | Lysosomes | Reports | Biosynthesis | Ova | Lymph nodes | Lymphoid Tissue - enzymology | Membrane Glycoproteins - metabolism | Dendritic Cells - immunology | Lysosomes - enzymology | Ribonucleases - metabolism | Lymphoid Tissue - immunology | Endocytosis | Antigen Presentation | Ribonuclease, Pancreatic - immunology | Horseradish Peroxidase - immunology | Lysosome-Associated Membrane Glycoproteins | B-Lymphocytes - metabolism | Dendritic Cells - metabolism | Macrophages - immunology | Peptide Hydrolases - metabolism | Green Fluorescent Proteins - metabolism | Antigen-Presenting Cells - metabolism | B-Lymphocytes - enzymology | Cells, Cultured | Antigens - metabolism | Antigen-Presenting Cells - immunology | Mice, Inbred C3H | Macrophages - enzymology | Lysosomes - ultrastructure | Lymphoid Tissue - cytology | Macrophages - metabolism | Animals | B-Lymphocytes - immunology | Green Fluorescent Proteins - immunology | Histocompatibility Antigens Class II - immunology | Ribonuclease, Pancreatic - metabolism | Dendritic Cells - enzymology | Ribonucleases - immunology | Horseradish Peroxidase - metabolism | Mice | Antigen-Presenting Cells - enzymology | Antigen presenting cells | Research | Proteolysis | Proteins | Antigen presentation | Index Medicus
Journal Article
Biomaterials, ISSN 0142-9612, 2016, Volume 85, pp. 88 - 98
Abstract Magnetism has recently been implicated to play significant roles in the regulation of cell responses. Allowing cells to experience a magnetic field... 
Advanced Basic Science | Dentistry | Osteoblastic differentiation | Magnetic scaffolds | Angiogenesis | Static magnetic field | Bone regeneration | CELLS | MATRIX | DESIGN | MATERIALS SCIENCE, BIOMATERIALS | ENGINEERING, BIOMEDICAL | BASIC APPROACH | ELECTROMAGNETIC-FIELDS | POLYCAPROLACTONE | PROLIFERATION | ODONTOBLASTIC DIFFERENTIATION | NANOPARTICLES | IN-VITRO | Paxillin - metabolism | Focal Adhesion Kinase 1 - genetics | Up-Regulation | Paxillin - genetics | Nanocomposites - chemistry | Integrins - genetics | NF-kappa B - metabolism | Integrins - metabolism | Tissue Scaffolds - chemistry | Female | Osteoblasts - cytology | Focal Adhesion Kinase 1 - metabolism | Tissue Engineering | Signal Transduction | rho GTP-Binding Proteins - genetics | Osteoblasts - drug effects | Osteogenesis - drug effects | Ribonuclease, Pancreatic - genetics | Mice, Inbred ICR | Polyesters - chemistry | Animals | NF-kappa B - genetics | Ribonuclease, Pancreatic - metabolism | Cell Differentiation - drug effects | rho GTP-Binding Proteins - metabolism | Mitogen-Activated Protein Kinases - genetics | Mice | Magnetic Fields | Mitogen-Activated Protein Kinases - metabolism | Phosphatases | Endothelial growth factors | Analysis | Bone morphogenetic proteins | Magnetism | Magnetic fields | Mitogens | Magnetic properties | Integrins | Endothelium | Index Medicus | Biomedical materials | Biocompatibility | Bones | Formations | Kinases | Scaffolds
Journal Article
Journal Article