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The New England Journal of Medicine, ISSN 0028-4793, 10/2010, Volume 363, Issue 15, pp. 1397 - 1409
Journal Article
JACC: Cardiovascular Imaging, ISSN 1936-878X, 2015, Volume 8, Issue 5, pp. 501 - 510
Journal Article
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2015, Volume 65, Issue 4, pp. 375 - 377
Journal Article
Journal Article
Circulation. Arrhythmia and electrophysiology, ISSN 1941-3149, 11/2017, Volume 10, Issue 11
Journal Article
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 12/2008, Volume 295, Issue 6, pp. 2264 - 2272
Anesthetic agents prolong cardiac repolarization by blocking ion currents. However, the clinical relevance of this blockade in subjects with reduced... 
Repolarization reserve | Long QT syndrome types 1 and 2 | Sudden cardiac death | Ventricular fibrillation | ventricular fibrillation | TORSADES-DE-POINTES | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | LANGE-NIELSEN SYNDROME | POTASSIUM CHANNEL CURRENTS | repolarization reserve | sudden cardiac death | VENTRICULAR MYOCYTES | I-KS | PROPOFOL | INTRAVENOUS ANESTHETICS | Q-T INTERVAL | PERIPHERAL VASCULAR DISEASE | long QT syndrome types 1 and 2 | LONG-QT-SYNDROME | HEART-RATE | KCNQ1 Potassium Channel - genetics | Humans | KCNQ1 Potassium Channel - metabolism | Tachycardia, Ventricular - chemically induced | Ether-A-Go-Go Potassium Channels - metabolism | Male | ERG1 Potassium Channel | Long QT Syndrome - physiopathology | Action Potentials | Heart Rate - drug effects | Anesthetics - toxicity | Time Factors | KCNQ1 Potassium Channel - antagonists & inhibitors | Electrocardiography | Tachycardia, Ventricular - genetics | Romano-Ward Syndrome - physiopathology | Potassium Channel Blockers - toxicity | Romano-Ward Syndrome - genetics | Disease Models, Animal | Rabbits | Pharmacogenetics | Heart Conduction System - metabolism | Risk Assessment | Animals, Genetically Modified | Genotype | Heart Conduction System - drug effects | Ether-A-Go-Go Potassium Channels - genetics | Ether-A-Go-Go Potassium Channels - antagonists & inhibitors | Long QT Syndrome - metabolism | Phenotype | Tachycardia, Ventricular - physiopathology | Animals | Heart Conduction System - physiopathology | Romano-Ward Syndrome - metabolism | Long QT Syndrome - genetics | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 2015, Volume 10, Issue 5, p. e0122754
Remodeling of cardiac repolarizing currents, such as the downregulation of slowly activating K+ channels (IKs), could underlie ventricular fibrillation (VF) in... 
Ventricular Fibrillation - diagnostic imaging | Ventricular Fibrillation - metabolism | Calcium - metabolism | Heart Failure - physiopathology | Male | Heart Conduction System - abnormalities | Arrhythmias, Cardiac - diagnostic imaging | Arrhythmias, Cardiac - physiopathology | Potassium Channels, Voltage-Gated - metabolism | Muscular Diseases - physiopathology | Heart Conduction System - diagnostic imaging | Ventricular Fibrillation - physiopathology | Romano-Ward Syndrome - physiopathology | Ion Transport | Heart Failure - diagnostic imaging | Cardiac Conduction System Disease | Arrhythmias, Cardiac - metabolism | Rabbits | Echocardiography | Heart Conduction System - metabolism | Animals, Genetically Modified | Muscular Diseases - metabolism | Heart Failure - metabolism | Romano-Ward Syndrome - diagnostic imaging | Animals | Heart Conduction System - physiopathology | Romano-Ward Syndrome - metabolism | Brugada Syndrome | Muscular Diseases - diagnostic imaging | Heart | Cardiac arrhythmia | Conduction | Ryanodine | Restitution | Calcium | Cardiomyopathy | Transgenic | Electrophysiology | Remodeling | Kinases | Medical schools | Ventricular fibrillation | Nerve conduction | Fibrillation | Rodents | Mathematical models | Cardiology | Heart diseases | Heart failure | Computer simulation | Telemetry | Medicine | Hospitals | Ventricle | Gene mapping | Laboratory animals | Potassium | Myopathy
Journal Article