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Biochemical journal, ISSN 0264-6021, 01/2010, Volume 425, Issue 1, pp. 215 - 223
The transcription factor SREBP1c (sterol-regulatory-element-binding protein 1c) is highly expressed in adipose tissue and plays a central role in several aspects of adipocyte development including the induction of PPAR gamma... 
Adipocyte | CCAAT/enhancer-binding protein(C/EBP) | Sterol-regulatory-element-binding protein 1c(SREBP1c) | Transcription | Lipid | Adipogenesis | Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Humans | Adipocytes - cytology | DNA-Binding Proteins - metabolism | Chromatin Immunoprecipitation | RNA Interference | CCAAT-Enhancer-Binding Protein-delta - metabolism | CCAAT-Enhancer-Binding Proteins - genetics | Sterol Regulatory Element Binding Protein 1 - metabolism | CCAAT-Enhancer-Binding Proteins - metabolism | Proto-Oncogene Proteins - metabolism | Promoter Regions, Genetic | CCAAT-Enhancer-Binding Protein-beta - genetics | Cells, Cultured | Gene Expression Regulation | Proto-Oncogene Proteins - genetics | Transcription Factors - genetics | 3T3-L1 Cells | DNA-Binding Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | CCAAT-Enhancer-Binding Protein-beta - metabolism | RUNX1 Translocation Partner 1 Protein | Transcription Factors - metabolism | Animals | Sterol Regulatory Element Binding Protein 1 - genetics | CCAAT-Enhancer-Binding Protein-delta - genetics | Adipocytes - metabolism | CCAAT-Enhancer-Binding Protein-alpha - genetics | Protein Binding | Mice | Index Medicus
Journal Article
PLoS genetics, ISSN 1553-7404, 07/2017, Volume 13, Issue 7, pp. e1006887 - e1006887
MicroRNA cluster mirn23a has previously been shown to promote myeloid development at the expense of lymphoid development in overexpression and knockout mouse... 
Guanylate Kinases - genetics | NIH 3T3 Cells | Up-Regulation | Cell Proliferation | MicroRNAs - metabolism | Mitochondrial Proteins - genetics | Phosphatidylinositol 3-Kinases - metabolism | Mitochondrial Proteins - metabolism | Trans-Activators - genetics | Cell Cycle Proteins - genetics | Cell Differentiation | Membrane Proteins - metabolism | B-Lymphocytes - metabolism | Cell Line | Forkhead Box Protein O1 - metabolism | Promoter Regions, Genetic | Signal Transduction | Membrane Proteins - genetics | Down-Regulation | Mice, Inbred C57BL | Cell Cycle Proteins - metabolism | Hematopoiesis - genetics | Phosphatidylinositol 3-Kinases - genetics | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Guanylate Kinases - metabolism | Trans-Activators - metabolism | Mice | MicroRNAs - genetics | Forkhead Box Protein O1 - genetics | Apoptosis | RNA sequencing | MicroRNA | Hematopoiesis | Physiological aspects | Genetic aspects | Genetic transcription | Observations | Methods | Cell culture | Flow cytometry | Polarization | Smad protein | Animal models | Transcription factors | Gene regulation | AKT protein | Kinases | Pax5 protein | Proteins | Signal transduction | Immunology | Pathways | FOXO1 protein | Bone marrow | Clusters | Bone morphogenetic proteins | miRNA | Buffers | Medical research | Transplants | Cell lineage | Runx1 protein | Pharmacology | Ribonucleic acid--RNA | 1-Phosphatidylinositol 3-kinase | Hemopoiesis | Cytometry | Inhibitors | Lymphocytes B | MicroRNAs | Stem cells | In vivo methods and tests | Differentiation | In vitro methods and tests | Cancer | Index Medicus | RNA | Ribonucleic acid
Journal Article
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 10/2001, Volume 21, Issue 19, pp. 6470 - 6483
Journal Article
PloS one, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, pp. e55481 - e55481
AML1-ETO fusion protein (AE) is generated by t(8;21)(q22;q22) chromosomal translocation, which is one of the most frequently observed structural abnormalities in acute myeloid leukemia (AML... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Oncogene Proteins, Fusion - metabolism | p300-CBP Transcription Factors - antagonists & inhibitors | Apoptosis - drug effects | Humans | Leukemia, Myeloid, Acute - metabolism | Proto-Oncogene Proteins c-kit - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | p300-CBP Transcription Factors - genetics | Leukemia, Myeloid, Acute - drug therapy | Female | Proto-Oncogene Proteins c-kit - genetics | Hematopoietic Stem Cells - drug effects | Core Binding Factor Alpha 2 Subunit - metabolism | Leukemia, Myeloid, Acute - pathology | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Gene Expression Regulation, Leukemic - drug effects | Hematopoietic Stem Cells - metabolism | RUNX1 Translocation Partner 1 Protein | Granulocyte Colony-Stimulating Factor - pharmacology | p300-CBP Transcription Factors - metabolism | Acetylation - drug effects | Animals | Histones - genetics | Signal Transduction - drug effects | Cell Cycle Checkpoints - drug effects | Oncogene Proteins, Fusion - genetics | Hematopoietic Stem Cells - cytology | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Histones - metabolism | Core Binding Factor Alpha 2 Subunit - genetics | Proto-Oncogene Proteins c-bcl-2 - genetics | Leukemia, Myeloid, Acute - genetics | Physiological aspects | Genetic aspects | Research | Transferases | Cell proliferation | Transcription | Bcl-2 protein | Leukemia | Event-related potentials | Kinases | Proteins | Cell growth | Allografts | Granulocyte colony-stimulating factor | Peripheral blood | Cell cycle | Colony-stimulating factor | Inhibition | Acetylation | Fusion protein | G1 phase | Translocation | Hematology | Colonies | Myeloid leukemia | Abnormalities | Melanoma | Gene expression | Histone acetyltransferase | c-Kit protein | Patients | AML1 protein | Inhibitors | Stem cells | Leukemogenesis | Leukocytes (granulocytic) | Laboratory animals | Acute myeloid leukemia | Prostate cancer | Histone H3 | Apoptosis | Index Medicus
Journal Article
Nature communications, ISSN 2041-1723, 07/2018, Volume 9, Issue 1, pp. 2733 - 18
ASXL1 mutations occur frequently in myeloid neoplasms and are associated with poor prognosis. However, the mechanisms by which mutant ASXL1 induces... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Whole-Body Irradiation | Homeodomain Proteins - metabolism | Humans | Leukemia, Myeloid - genetics | Interferon Regulatory Factors - metabolism | Male | Carcinogenesis - metabolism | Leukemia, Myeloid - pathology | Ubiquitination | Bone Marrow - metabolism | Tumor Suppressor Proteins - genetics | Bone Marrow Transplantation | HEK293 Cells | Ubiquitin Thiolesterase - metabolism | Female | RUNX1 Translocation Partner 1 Protein - genetics | Leukemia, Myeloid - mortality | RUNX1 Translocation Partner 1 Protein - metabolism | Repressor Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Core Binding Factor Alpha 2 Subunit - metabolism | Signal Transduction | Mice, Inbred C57BL | Carcinogenesis - genetics | Interferon Regulatory Factors - genetics | Repressor Proteins - genetics | Gene Expression Regulation, Leukemic | Ubiquitin Thiolesterase - genetics | Gene Editing | Homeodomain Proteins - genetics | Carcinogenesis - pathology | Animals | Leukemia, Myeloid - metabolism | Bone Marrow - pathology | CRISPR-Cas Systems | Survival Analysis | Mice | HeLa Cells | Mutation | Core Binding Factor Alpha 2 Subunit - genetics | Myeloid leukemia | Leukemia | Runx1 protein | Gene expression | Progenitor cells | Neoplasms | Cells (biology) | Stem cells | Tumor suppressor genes | HOXA gene | Catalysis | Tumors | Index Medicus
Journal Article
Scientific reports, ISSN 2045-2322, 09/2016, Volume 6, Issue 1, pp. 32474 - 32474
Journal Article
Journal of cellular biochemistry, ISSN 0730-2312, 03/2018, Volume 119, Issue 3, pp. 2750 - 2762
Journal Article