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Journal Article
JOURNAL OF NEUROSCIENCE, ISSN 0270-6474, 08/2012, Volume 32, Issue 34, pp. 11820 - 11834
In Alzheimer disease (AD), the perturbation of the endoplasmic reticulum (ER) calcium (Ca2+) homeostasis has been linked to presenilins, the catalytic core in... 
DANTROLENE | PRESENILINS | INFLUX | CA2+ RELEASE | CALCIUM-RELEASE | INCREASES | CHANNELS | PRECURSOR PROTEIN | N-TERMINUS | NEUROSCIENCES | I-CRAC | Amyloid Precursor Protein Secretases - genetics | Calcium - metabolism | Embryo, Mammalian | Humans | Calcium Channel Blockers - therapeutic use | Memory Disorders - metabolism | Aminophenols - therapeutic use | RNA, Messenger - metabolism | Phosphorylation - genetics | Endoplasmic Reticulum - drug effects | Muscle Relaxants, Central - pharmacology | Amyloid beta-Peptides - metabolism | Exploratory Behavior - drug effects | Caffeine - pharmacology | Purines - therapeutic use | Phosphorylation - drug effects | Disease Models, Animal | Membrane Potentials - drug effects | Mice, Transgenic | Dantrolene - pharmacology | Mutation - genetics | Enzyme Inhibitors - therapeutic use | Amyloid Precursor Protein Secretases - metabolism | Patch-Clamp Techniques | Memory Disorders - etiology | Analysis of Variance | Ryanodine Receptor Calcium Release Channel - genetics | Cytosol - metabolism | Plaque, Amyloid - metabolism | Mice | Inositol 1,4,5-Trisphosphate Receptors - genetics | Membrane Potentials - genetics | Alzheimer Disease - complications | Cytosol - drug effects | Endoplasmic Reticulum - metabolism | Ryanodine Receptor Calcium Release Channel - metabolism | Endoplasmic Reticulum - pathology | Maleimides - therapeutic use | Transfection | Inositol 1,4,5-Trisphosphate Receptors - metabolism | Reaction Time - drug effects | Amyloid beta-Protein Precursor - metabolism | Neurons - physiology | Membrane Proteins - metabolism | Neurons - drug effects | Neuroblastoma - pathology | Brain - cytology | Peptide Fragments - metabolism | Plaque, Amyloid - pathology | Gene Expression Regulation - genetics | Cells, Cultured | Maze Learning - drug effects | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Amyloid beta-Protein Precursor - genetics | Animals | Memory Disorders - drug therapy | Recognition (Psychology) - drug effects | Alzheimer Disease - metabolism | Alzheimer Disease - genetics
Journal Article
Neurology, ISSN 0028-3878, 04/2019, Volume 92, Issue 14, pp. e1600 - e1609
OBJECTIVETo identify the genetic defect causing a distal calf myopathy with cores. METHODSFamilies with a genetically undetermined calf-predominant myopathy... 
GENE | RYR1-RELATED MYOPATHIES | RECESSIVE MUTATIONS | DEPLETION | CONGENITAL MYOPATHY | DOMINANT | COMMON-CAUSE | MYALGIA | CLINICAL NEUROLOGY | CENTRAL CORE DISEASE
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 09/2013, Volume 62, pp. 189 - 202
Journal Article
Brain Research, ISSN 0006-8993, 2017, Volume 1670, pp. 1 - 5
Highlights • D4R activation induced a large calcium transient. • D4R mediated calcium transient is not from extracellular calcium influx. • IP3 receptor... 
Neurology | Ryanodine | Dopamine | Intracellular calcium | IP3 receptor | DR4 | receptor | VARIANTS | SUBTYPES | KINASE | MECHANISMS | NEUROSCIENCES | PREFRONTAL CORTEX | TRANSMISSION | D-4 RECEPTORS | OSCILLATIONS | BRAIN | MODULATION
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 04/2011, Volume 162, Issue 7, pp. 1618 - 1638
BACKGROUND AND PURPOSE P2X receptors mediate sympathetic control and autoregulation of the renal circulation triggering contraction of renal vascular smooth... 
MRS 2578 | renal vascular smooth muscle cells | channels | inositol 1,4,5‐trisphosphate receptor | meATP | ryanodine receptor | P2X receptor | signalling | sarcoplasmic reticulum | NF 279 | voltage‐gated Ca | voltage-gated Ca2+channels | 5-trisphosphate receptor | PIG VENTRICULAR MYOCYTES | INDUCED CALCIUM-RELEASE | PHOSPHOLIPASE-C | MOUSE URINARY-BLADDER | alpha beta-meATP | SARCOPLASMIC-RETICULUM FUNCTION | PORTAL-VEIN MYOCYTES | RYANODINE RECEPTORS | INOSITOL 1,4,5-TRISPHOSPHATE RECEPTORS | PHARMACOLOGY & PHARMACY | SMOOTH-MUSCLE-CELLS | Ca2+signalling | inositol 1 | VASCULAR MYOCYTES | Kidney - blood supply | Calcium Channels - metabolism | Muscle, Smooth, Vascular - metabolism | Calcium - metabolism | Muscle Cells - metabolism | Rats | Rats, Inbred WKY | Male | Muscle, Smooth, Vascular - cytology | Purinergic P2X Receptor Agonists - pharmacology | Adenosine Triphosphate - analogs & derivatives | Adenosine Triphosphate - pharmacology | Animals | Ryanodine Receptor Calcium Release Channel - genetics | Inositol 1,4,5-Trisphosphate Receptors - metabolism | Adenosine Triphosphate - metabolism | Receptors, Purinergic P2X - metabolism | Renal Artery - metabolism | Sarcoplasmic Reticulum - metabolism | Myocytes, Smooth Muscle - metabolism | Inositol 1,4,5-Trisphosphate Receptors - genetics | Research Papers | voltage-gated Ca2+ channels | inositol 1,4,5-trisphosphate receptor | Ca2+ signalling | αβ-meATP
Journal Article
The EMBO Journal, ISSN 0261-4189, 06/1997, Volume 16, Issue 11, pp. 3078 - 3088
Stimulation of B‐cell antigen receptor (BCR) induces a rapid increase in cytoplasmic free calcium due to its release from intracellular stores and influx from... 
apoptosis | calcium mobilization | B‐cell antigen receptor | muscarinic receptor | inositol 1,4,5‐trisphosphate receptor | Inositol 1,4,5-trisphosphate receptor | Calcium mobilization | Muscarinic receptor | B-cell antigen receptor | Apoptosis | CYCLIC ADP-RIBOSE | BIOCHEMISTRY & MOLECULAR BIOLOGY | INTRACELLULAR CA2+ STORES | SEA-URCHIN EGGS | SURFACE IG-RECEPTOR | FUNCTIONAL-CHARACTERIZATION | TYROSINE KINASES | CELL BIOLOGY | CAPACITATIVE CALCIUM-ENTRY | ACTIVATED T-CELLS | RYANODINE RECEPTOR | K CQ BIOCHEMISTRY & MOLECULAR BIOLOGY | K DR CELL BIOLOGY | inositol 1,4,5-trisphosphate receptor | NF-KAPPA-B | Calcium Channels - metabolism | Calcium - metabolism | Myristoylated Alanine-Rich C Kinase Substrate | Type C Phospholipases - metabolism | Receptors, Muscarinic - metabolism | Receptors, Antigen, B-Cell - metabolism | Receptors, Cytoplasmic and Nuclear - classification | Biological Transport | Isoenzymes - metabolism | Phospholipase C gamma | Inositol 1,4,5-Trisphosphate Receptors | Calcium Channels - genetics | Cell Line | Signal Transduction | Calcium Channels - classification | Intracellular Signaling Peptides and Proteins | Receptors, Cytoplasmic and Nuclear - genetics | Receptor, Muscarinic M1 | Membrane Proteins | Proteins - metabolism | Thapsigargin - pharmacology | Enzyme Activation | Mutation | Inositol 1,4,5-Trisphosphate - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism
Journal Article
Nature Neuroscience, ISSN 1097-6256, 05/2004, Volume 7, Issue 5, pp. 525 - 533
Synaptic inhibition is a vital component in the control of cell excitability within the brain. Here we report a newly identified form of inhibitory synaptic... 
TRANSMITTER RELEASE | ENDOGENOUS CANNABINOIDS | RAT CEREBELLUM | REBOUND POTENTIATION | INHIBITION | DEPOLARIZATION-INDUCED SUPPRESSION | NEURONS | SYNAPTIC CURRENTS | CLIMBING FIBER | CALCIUM STORES | NEUROSCIENCES | Evoked Potentials - radiation effects | Electric Stimulation | Synaptic Transmission - physiology | Calcium - metabolism | gamma-Aminobutyric Acid - metabolism | Receptors, N-Methyl-D-Aspartate - metabolism | Neuronal Plasticity - drug effects | Membrane Potentials - radiation effects | Patch-Clamp Techniques - methods | N-Methylaspartate - pharmacology | Dose-Response Relationship, Drug | Neural Inhibition - physiology | Drug Interactions | Neuronal Plasticity - physiology | Synapses - metabolism | Piperidines - pharmacology | Time Factors | Isoenzymes - metabolism | Egtazic Acid - analogs & derivatives | Tetrodotoxin - pharmacology | Ryanodine - pharmacology | Pyrazoles - pharmacology | Synaptophysin - metabolism | Animals, Newborn | Membrane Potentials - drug effects | Neuronal Plasticity - radiation effects | Purkinje Cells - metabolism | Cadmium - pharmacology | Rats | Sodium Channel Blockers - pharmacology | Excitatory Amino Acid Antagonists - pharmacology | Chelating Agents - pharmacology | Rats, Sprague-Dawley | Evoked Potentials - drug effects | Immunohistochemistry - methods | Animals | Egtazic Acid - pharmacology | Interneurons - metabolism | Cerebellum - cytology | Glutamate Decarboxylase - metabolism | Presynaptic Terminals - metabolism | In Vitro Techniques | Physiological aspects | GABA | Receptors | Ion channels | Research | Purkinje cells
Journal Article
Neuropharmacology, ISSN 0028-3908, 2011, Volume 60, Issue 6, pp. 944 - 952
Chronic activation or inhibition of cannabinoid receptors (CB1) leads to continuous suppression of neuronal plasticity in hippocampus and other brain regions,... 
Calcium | Protein kinase | Second messenger | Plasticity | Fluorescence | N-Methyl- d-aspartate | Cannabinoid | Glutamate | Confocal imaging | N-Methyl-d-aspartate | DEPENDENT PHOSPHORYLATION | N-Methyl-D-aspartate | PROTEIN-KINASE | SYNAPTIC-TRANSMISSION | GLYCINE SITE | NEUROSCIENCES | INHIBITION | NEURONS | RYANODINE-SENSITIVE STORES | PHARMACOLOGY & PHARMACY | RAT HIPPOCAMPUS | MODULATION | Cannabinoid Receptor Modulators - metabolism | Calcium - metabolism | Neuronal Plasticity - drug effects | Ryanodine Receptor Calcium Release Channel - metabolism | Hippocampus - drug effects | Receptor, Cannabinoid, CB1 - physiology | Cannabinoid Receptor Modulators - physiology | Neuronal Plasticity - physiology | Receptor, Cannabinoid, CB1 - agonists | Piperidines - pharmacology | 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology | Benzamides - pharmacology | Neurons - metabolism | Phosphorylation - drug effects | Endocannabinoids | Carbamates - pharmacology | Pyrazoles - pharmacology | Second Messenger Systems - drug effects | Morpholines - pharmacology | Rats | Rats, Sprague-Dawley | Receptors, N-Methyl-D-Aspartate - physiology | Arachidonic Acids - pharmacology | Naphthalenes - pharmacology | Hippocampus - metabolism | Animals | Glycine - pharmacology | Benzoxazines - pharmacology | Second Messenger Systems - physiology | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Hippocampus - physiology | In Vitro Techniques | Fatty-acid amide hydrolase | Firing pattern | Calcium (intracellular) | Glutamic acid receptors (ionotropic) | Neurons | Brain slice preparation | Ryanodine receptors | N-Methyl-D-aspartic acid receptors | Antagonists | Cannabinoid CB1 receptors | Plasticity (neural) | Second messengers | Ion channels | Glutamatergic transmission | Short term memory | Endoplasmic reticulum | Hippocampus | Synapses | n-methyl-d-aspartate | fluorescence | cannabinoid | confocal imaging | plasticity | second messenger | glutamate | protein kinase
Journal Article