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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2008, Volume 105, Issue 12, pp. 4826 - 4831
To enhance therapeutic efficacy and reduce adverse effects, practitioners of traditional Chinese medicine (TCM) prescribe a combination of plant... 
Up regulation | Acute promyelocytic leukemia | Arsenic | Medical treatment | Leukemia | Cell cycle | Cell lines | Mice | Cellular differentiation | Blood | Traditional Chinese medicine | Active ingredient | Systems biology | Synergism | Tetraarsenic tetrasulfide | systems biology | NB4 | APOPTOSIS | PHOSPHORYLATION | active ingredient | MULTIDISCIPLINARY SCIENCES | tetraarsenic tetrasulfide | traditional chinese medicine | ARSENIC TRIOXIDE AS2O3 | IN-VITRO | RETINOIC ACID | THERAPY | GENE | synergism | CELL-CYCLE | EXPRESSION | Transcription, Genetic - drug effects | Oncogene Proteins, Fusion - metabolism | Leukemia, Promyelocytic, Acute - pathology | Humans | Drugs, Chinese Herbal - pharmacology | G1 Phase - drug effects | Aquaporins - genetics | Arsenicals - therapeutic use | Ubiquitination - drug effects | Protein Processing, Post-Translational - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Drugs, Chinese Herbal - therapeutic use | Disease Models, Animal | Sulfides - pharmacology | Medicine, Chinese Traditional | Arsenicals - pharmacology | Aquaporins - metabolism | Sulfides - therapeutic use | Drug Synergism | Up-Regulation - drug effects | Animals | Cell Differentiation - drug effects | Cell Line, Tumor | Resting Phase, Cell Cycle - drug effects | Leukemia, Promyelocytic, Acute - genetics | Leukemia, Promyelocytic, Acute - drug therapy | Evaluation | Medicine, Chinese | Research | Drug synergism | Chinese medicine | Flowers & plants | Cells | Effectiveness studies | Index Medicus | Biological Sciences
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2011, Volume 286, Issue 21, pp. 19127 - 19137
Trastuzumab resistance emerges to be a major issue in anti-human epidermal growth factor receptor 2 (HER2) therapy for breast cancers. Here, we demonstrated... 
INVASION | CARCINOMA CELLS | POOR-PROGNOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | IN-VIVO | GROWTH | TUMOR-SUPPRESSOR GENE | DOWN-REGULATION | PTEN | MICRORNA-21 TARGETS | EXPRESSION | Carcinoma, Ductal, Breast - genetics | Gene Silencing - drug effects | Oligodeoxyribonucleotides, Antisense - pharmacology | Receptor, ErbB-2 - genetics | Humans | Receptor, ErbB-2 - metabolism | Antibodies, Monoclonal - therapeutic use | Antineoplastic Agents - therapeutic use | G1 Phase - drug effects | Breast Neoplasms - metabolism | Antibodies, Monoclonal, Humanized | Carcinoma, Ductal, Breast - drug therapy | Female | Antineoplastic Agents - pharmacology | Receptor, ErbB-2 - antagonists & inhibitors | G1 Phase - genetics | Gene Expression Regulation, Neoplastic - drug effects | Tumor Cells, Cultured | Carcinoma, Ductal, Breast - metabolism | PTEN Phosphohydrolase - genetics | Antibodies, Monoclonal - pharmacology | PTEN Phosphohydrolase - biosynthesis | MicroRNAs - biosynthesis | Up-Regulation - genetics | Breast Neoplasms - drug therapy | S Phase - genetics | Gene Expression Regulation, Enzymologic | Up-Regulation - drug effects | Breast Neoplasms - genetics | RNA, Neoplasm - biosynthesis | RNA, Neoplasm - genetics | MicroRNAs - genetics | S Phase - drug effects | Trastuzumab | Drug Resistance, Neoplasm - drug effects | Index Medicus | Gene Regulation | RNA Silencing | MicroRNA | Gene Therapy | Tumor Therapy | Breast Cancer
Journal Article
Cancer Cell, ISSN 1535-6108, 2007, Volume 11, Issue 2, pp. 175 - 189
In response to DNA damage, eukaryotic cells activate ATM-Chk2 and/or ATR-Chk1 to arrest the cell cycle and initiate DNA repair. We show that, in the absence of... 
SIGNALING | CANCER-CELLS | ONCOLOGY | 7-HYDROXYSTAUROSPORINE UCN-01 | MITOTIC CATASTROPHE | SECKEL-SYNDROME | MAPKAP KINASE-2 | NUCLEAR EXPORT | INDUCED CYTOTOXICITY | 14-3-3 PROTEIN-BINDING | RADIOSENSITIZING AGENT | PHOSPHORYLATION SITES | Protein Kinases - genetics | Antibiotics, Antineoplastic - pharmacology | Humans | DNA Repair - radiation effects | Staurosporine - analogs & derivatives | Bone Neoplasms - pathology | G2 Phase - drug effects | Bone Neoplasms - metabolism | DNA-Binding Proteins - metabolism | Neoplasms, Experimental - pathology | Tumor Suppressor Protein p53 - physiology | Ultraviolet Rays | Antineoplastic Agents - pharmacology | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | cdc25 Phosphatases - metabolism | Osteosarcoma - metabolism | Protein-Serine-Threonine Kinases - metabolism | DNA Damage - drug effects | DNA Repair - drug effects | Tumor Suppressor Proteins - metabolism | Mitosis - radiation effects | G2 Phase - radiation effects | Signal Transduction | Cell Survival | Cell Cycle Proteins - metabolism | Cells, Cultured | Cell Division - radiation effects | Intracellular Signaling Peptides and Proteins | Protein Kinase C - antagonists & inhibitors | Ataxia Telangiectasia Mutated Proteins | Cisplatin - pharmacology | Cell Division - drug effects | Mice, Knockout | Phosphorylation - radiation effects | S Phase - radiation effects | Animals | Mitosis - drug effects | Mice, Nude | Protein Kinases - physiology | Mice | S Phase - drug effects | Neoplasms, Experimental - metabolism | DNA Damage - radiation effects | Doxorubicin - pharmacology | Osteosarcoma - pathology | Staurosporine - pharmacology | DNA | Index Medicus
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 12/2010, Volume 70, Issue 24, pp. 10310 - 10320
Journal Article
Journal Article
Nature Structural and Molecular Biology, ISSN 1545-9993, 04/2017, Volume 24, Issue 4, pp. 353 - 361
Journal Article