Articles

Search Articles

Advanced search
Help

Catalogue

Articles

Databases

X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
index medicus (811) 811
s100 calcium-binding protein a4 (625) 625
humans (615) 615
animals (424) 424
s100 proteins - metabolism (354) 354
s100 proteins - genetics (287) 287
mice (275) 275
female (261) 261
s100a4 (248) 248
metastasis (247) 247
male (237) 237
expression (198) 198
oncology (195) 195
cell line, tumor (181) 181
calcium-binding protein (175) 175
immunohistochemistry (157) 157
biochemistry & molecular biology (146) 146
middle aged (141) 141
proteins (132) 132
cancer (127) 127
s100 proteins (127) 127
aged (118) 118
cells (115) 115
cell biology (114) 114
adult (111) 111
gene expression (106) 106
gene expression regulation, neoplastic (103) 103
prognosis (102) 102
rats (93) 93
e-cadherin (90) 90
neoplasm invasiveness (90) 90
neoplasm metastasis (88) 88
gene (86) 86
cells, cultured (83) 83
cell proliferation (82) 82
protein binding (82) 82
research (82) 82
analysis (78) 78
prognostic-significance (78) 78
pathology (72) 72
s100 calcium-binding protein a4 - metabolism (72) 72
tumor cells, cultured (71) 71
s100 proteins - biosynthesis (70) 70
transfection (70) 70
blotting, western (69) 69
carcinoma (69) 69
tumors (68) 68
molecular sequence data (67) 67
signal transduction (67) 67
cell movement (66) 66
calcium-binding proteins - genetics (62) 62
disease models, animal (62) 62
mice, inbred c57bl (62) 62
fibrosis (61) 61
breast-cancer (59) 59
epithelial-mesenchymal transition (59) 59
rna, messenger - metabolism (59) 59
calcium-binding proteins - metabolism (58) 58
aged, 80 and over (57) 57
human breast-cancer (57) 57
reverse transcriptase polymerase chain reaction (57) 57
s100 calcium-binding protein a4 - genetics (57) 57
gene-expression (56) 56
invasion (56) 56
s100 proteins - chemistry (56) 56
amino acid sequence (55) 55
nonmuscle myosin (55) 55
s100 proteins - analysis (54) 54
article (53) 53
fibroblasts - metabolism (53) 53
s100a4 protein (53) 53
cell line (52) 52
mice, transgenic (52) 52
in-vitro (51) 51
s100 proteins - physiology (50) 50
fibroblasts (49) 49
growth (49) 49
phenotype (49) 49
progression (49) 49
protein (49) 49
rna, messenger - genetics (49) 49
family (48) 48
up-regulation (48) 48
abridged index medicus (47) 47
physiological aspects (47) 47
protein s100a4 (47) 47
binding sites (46) 46
development and progression (46) 46
disease progression (46) 46
neurosciences (46) 46
inflammation (45) 45
p9ka (45) 45
research article (45) 45
rna interference (45) 45
apoptosis (44) 44
base sequence (44) 44
breast cancer (44) 44
activation (43) 43
genetic aspects (43) 43
mts1 gene (43) 43
more...
Library Location Library Location
Language Language
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


1. Full Text Nuclear factor of activated T cells 5 maintained by Hotair suppression of miR-568 upregulates S100 calcium binding protein A4 to promote breast cancer metastasis
by Li, Jun-Tang and Wang, Li-Feng and Zhao, Ya-Li and Yang, Tao and Li, Wei and Zhao, Jing and Yu, Feng and Wang, Lei and Meng, Yan-Ling and Liu, Ning-Ning and Zhu, Xiao-Shan and Gao, Chun-Fang and Jia, Lin-Tao and Yang, An-Gang
Breast Cancer Research, ISSN 1465-5411, 2014, Volume 16, Issue 5, pp. 454 - 454
Introduction: The onset of distal metastasis, which underlies the high mortality of breast cancers, warrants substantial studies to depict its molecular basis.... 
NFAT5 | LONG NONCODING RNA | ONCOLOGY | TUMOR-METASTASIS | MECHANISMS | TRANSCRIPTION FACTOR | EXPRESSION | CARCINOMA | BLOOD-PRESSURE | PROGRESSION | INSIGHTS | Neoplasm Transplantation | Up-Regulation | S100 Calcium-Binding Protein A4 | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Breast Neoplasms - metabolism | Human Umbilical Vein Endothelial Cells - physiology | S100 Proteins - genetics | Lung Neoplasms - secondary | RNA Interference | Base Sequence | Epithelial-Mesenchymal Transition | Female | Binding Sites | Lung Neoplasms - genetics | S100 Proteins - metabolism | Lymphatic Metastasis | RNA, Long Noncoding - genetics | Transcription Factors - genetics | Vascular Endothelial Growth Factor C - genetics | Transcription Factors - metabolism | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Mice, Nude | Vascular Endothelial Growth Factor C - metabolism | Cell Line, Tumor | Mice, Inbred BALB C | MicroRNAs - genetics | Development and progression | Breast cancer | Genetic transcription | Metastasis | Health aspects | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
2. Full Text The microRNA miR‐124 inhibits vascular smooth muscle cell proliferation by targeting S100 calcium‐binding protein A4 (S100A4)
by Choe, Nakwon and Kwon, Duk‐Hwa and Shin, Sera and Kim, Yong Sook and Kim, Young‐Kook and Kim, Jaetaek and Ahn, Youngkeun and Eom, Gwang H and Kook, Hyun
FEBS Letters, ISSN 0014-5793, 04/2017, Volume 591, Issue 7, pp. 1041 - 1052
S100 calcium‐binding protein A4 (S100A4) induces proliferation and migration of vascular smooth muscle cells ( VSMC s). We aimed to find the micro RNA... 
miR‐124 | S100A4 | neointima | vascular smooth muscle cell proliferation | carotid artery injury | miR-124 | METASTASIS | MARKER | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOWN-REGULATION | CELL BIOLOGY | BREAST-CANCER | TRANSITION | BIOPHYSICS | ENDOTHELIAL-CELLS | GROWTH | SUPPRESSES | DIFFERENTIATION | PROGRESSION | Immunohistochemistry | Cell Line | Cell Proliferation - genetics | Gene Expression Regulation | 3' Untranslated Regions - genetics | Male | Muscle, Smooth, Vascular - cytology | Reverse Transcriptase Polymerase Chain Reaction | Neointima - metabolism | Rats, Sprague-Dawley | Blotting, Western | Sequence Homology, Amino Acid | Neointima - genetics | Animals | RNA Interference | S100 Calcium-Binding Protein A4 - genetics | Time Factors | Base Sequence | Carotid Artery Injuries - genetics | MicroRNAs - genetics | Myocytes, Smooth Muscle - metabolism | S100 Calcium-Binding Protein A4 - metabolism | Carotid Artery Injuries - metabolism | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
3. Full Text Serum S100 calcium binding protein A4 improves the diagnostic accuracy of transient elastography for assessing liver fibrosis in hepatitis B
by Yan, Li-Bo and Zhang, Qing-Bo and Zhu, Xia and He, Min and Tang, Hong
Clinics and Research in Hepatology and Gastroenterology, ISSN 2210-7401, 02/2018, Volume 42, Issue 1, pp. 64 - 71
The diagnostic performance of Fibroscan might be improved when combined with other serum fibrosis related markers. Previous study has demonstrated that S100A4... 
Liver stiffness measurement | S100A4 | Chronic hepatitis B | Significant fibrosis | CELLS | CIRRHOSIS | BIOPSY | MARKERS | GASTROENTEROLOGY & HEPATOLOGY | Elasticity Imaging Techniques - methods | Liver Cirrhosis - etiology | Reproducibility of Results | Algorithms | Hepatitis B, Chronic - complications | Liver Cirrhosis - diagnostic imaging | Humans | Adult | Female | Male | Liver Cirrhosis - blood | S100 Calcium-Binding Protein A4 - blood | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
4. Full Text Prognostic significance of heme oxygenase-1, S100 calcium-binding protein A4, and syndecan-1 expression in primary non–muscle-invasive bladder cancer
by Kim, Joo Heon, MD, PhD and Park, Jinsung, MD, PhD
Human Pathology, ISSN 0046-8177, 2014, Volume 45, Issue 9, pp. 1830 - 1838
Summary We investigated the prognostic significance of heme oxygenase-1 (HO-1), S100 calcium-binding protein A4 (S100A4), and syndecan-1 (SYND1) expression in... 
Pathology | Immunohistochemistry | Biomarkers | Recurrence | Prognosis | Bladder cancer | BACILLUS-CALMETTE-GUERIN | PATHOLOGY | TUMORS | LUNG-CANCER | MOLECULAR MARKERS | DISEASE | REDUCED SURVIVAL | PREDICTING RECURRENCE | PROGRESSION | CLINICAL-SIGNIFICANCE | PEROXIREDOXIN-1 | Heme Oxygenase-1 - metabolism | Follow-Up Studies | S100 Calcium-Binding Protein A4 | Humans | Middle Aged | Male | Carcinoma, Transitional Cell - therapy | Neoplasm Recurrence, Local - pathology | Urinary Bladder Neoplasms - pathology | Aged, 80 and over | Adult | Female | Urinary Bladder Neoplasms - metabolism | Biomarkers - metabolism | Syndecan-1 - metabolism | Neoplasm Recurrence, Local - metabolism | Neoplasm Recurrence, Local - therapy | Kaplan-Meier Estimate | Proportional Hazards Models | S100 Proteins - metabolism | Carcinoma, Transitional Cell - metabolism | Disease Progression | Disease-Free Survival | Carcinoma, Transitional Cell - pathology | Urinary Bladder Neoplasms - therapy | Aged | Binding proteins | Analysis | Calmodulin | Heme | Protein binding | Calcium | Breast cancer | Multivariate analysis | Cancer therapies | Patients | Proteins | Studies | Confidence intervals | Variables | Medical prognosis | Tumors | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
5. Full Text S100 calcium-binding protein A4 is a novel independent prognostic factor for the poor prognosis of gastric carcinomas
by ZHAO, YING and ZHANG, TIANBIAO and WANG, QIANG
Oncology Reports, ISSN 1021-335X, 7/2013, Volume 30, Issue 1, pp. 111 - 118
Overexpression of the S100 calcium-binding protein A4 (S100A4) is involved in epithelial-to-mesenchymal transition, oncogenic transformation, angiogenesis,... 
progression | pathogenesis | S100A4 | gastric carcinoma | prognosis | Progression | Gastric carcinoma | Prognosis | Pathogenesis | METASTASIS | TUMOR PROGRESSION | HYPOMETHYLATION-INDUCED EXPRESSION | E-CADHERIN | INVASIVENESS | INVASION | MESSENGER-RNA | GENE | ONCOLOGY | COLORECTAL-CANCER | SQUAMOUS-CELL CARCINOMA | Calcium-Binding Proteins - metabolism | Up-Regulation | Calcium-Binding Proteins - biosynthesis | Gastritis - metabolism | Neoplasm Invasiveness | S100 Calcium-Binding Protein A4 | Tissue Array Analysis | Humans | Middle Aged | Stomach Neoplasms - metabolism | Male | S100 Proteins - metabolism | Lymphatic Metastasis | Carcinogenesis | Survival | Disease Progression | Adenocarcinoma - metabolism | Biomarkers, Tumor - metabolism | Female | S100 Proteins - biosynthesis | Stomach Neoplasms - mortality | Antigens | Lymphatic system | Genes | Mortality | Metastasis | Multivariate analysis | Stomach cancer | Proteins | Medical prognosis | Rodents | Cell cycle | Classification | Tumors | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
6. Protective effect and mechanism of rat recombinant S100 calcium‑binding protein A4 on oxidative stress injury of rat vascular endothelial cells
by Meng, Xiangyan and Gao, Xiujie and Zhang, Zhiqing and Zhou, Xuesi and Wu, Lei and Yang, Miaomiao and Wang, Kun and Ren, Hanlin and Sun, Bei and Wang, Tianhui
Oncology Letters, ISSN 1792-1074, 09/2018, Volume 16, Issue 3, pp. 3614 - 3622
The aim of the present study was to examine the protective effects and mechanisms of S100 calcium-binding protein A4 (S100A4) on endothelial cell apoptosis... 
Oxidative stress | Rat recombinant S100 calcium-binding protein A4 | Tumor protein 53 | Apoptosis | S100A4(MTS1) | METASTASIS | ANGIOGENESIS | MOUSE | apoptosis | rat recombinant S100 calcium-binding protein A4 | FAMILY | ONCOLOGY | ROLES | INFLAMMATION | INTERACTS | GROWTH | tumor protein 53 | NF-KAPPA-B | oxidative stress
Journal Article
Details down arrow
7. Full Text Fate Tracing Reveals the Pericyte and Not Epithelial Origin of Myofibroblasts in Kidney Fibrosis
by Humphreys, Benjamin D and Lin, Shuei-Liong and Kobayashi, Akio and Hudson, Thomas E and Nowlin, Brian T and Bonventre, Joseph V and Valerius, M. Todd and McMahon, Andrew P and Duffield, Jeremy S
American Journal of Pathology, The, ISSN 0002-9440, 2010, Volume 176, Issue 1, pp. 85 - 97
Understanding the origin of myofibroblasts in kidney is of great interest because these cells are responsible for scar formation in fibrotic kidney disease.... 
Pathology | TO-MESENCHYMAL TRANSITION | GROWTH-FACTOR-BETA | STEM-CELLS | IN-VITRO | TGF-BETA | MESANGIAL CELL | COLLAGEN-PRODUCING CELLS | BONE-MARROW | POSTISCHEMIC KIDNEY | ACUTE-RENAL-FAILURE | PATHOLOGY | Green Fluorescent Proteins | Up-Regulation | Epithelial Cells - metabolism | Kidney - pathology | S100 Calcium-Binding Protein A4 | Homeodomain Proteins - metabolism | Actins - metabolism | Promoter Regions, Genetic - genetics | Kidney - metabolism | Forkhead Transcription Factors - metabolism | Pericytes - pathology | Integrases - metabolism | Kidney Tubules - pathology | Kidney Tubules - metabolism | Disease Models, Animal | Fibroblasts - metabolism | Pericytes - metabolism | Cells, Cultured | Epithelial Cells - pathology | S100 Proteins - metabolism | Transcription Factors - genetics | Fibroblasts - pathology | Homeodomain Proteins - genetics | Cell Lineage | Animals | Fibrosis | Mesoderm - metabolism | Mice | Mesoderm - pathology | Cell Movement | Index Medicus | Abridged Index Medicus | Regular
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
8. Full Text Heart repair by reprogramming non-myocytes with cardiac transcription factors
by Song, Kunhua and Nam, Young-Jae and Luo, Xiang and Qi, Xiaoxia and Tan, Wei and Huang, Guo N and Acharya, Asha and Smith, Christopher L and Tallquist, Michelle D and Neilson, Eric G and Hill, Joseph A and Bassel-Duby, Rhonda and Olson, Eric N
Nature, ISSN 0028-0836, 2012, Volume 485, Issue 7400, pp. 599 - 604
The adult mammalian heart possesses little regenerative potential following injury. Fibrosis due to activation of cardiac fibroblasts impedes cardiac... 
MYOCARDIUM | CELLS | REGULATOR | DEFINED FACTORS | MULTIDISCIPLINARY SCIENCES | IN-VIVO | GENE-EXPRESSION | GENERATION | CARDIOMYOCYTES | FIBROBLASTS | ADULT | Fibroblasts - physiology | S100 Calcium-Binding Protein A4 | Tail - cytology | Heart - physiology | Regenerative Medicine - methods | Cellular Reprogramming | Myocardial Infarction - therapy | S100 Proteins - genetics | Basic Helix-Loop-Helix Transcription Factors - metabolism | Myocardial Infarction - pathology | Cell Transdifferentiation | Myocardial Infarction - physiopathology | Heart - physiopathology | Basic Helix-Loop-Helix Transcription Factors - genetics | Myocytes, Cardiac - cytology | Myocardium - pathology | S100 Proteins - metabolism | Transcription Factors - genetics | Myocardium - cytology | Transcription Factors - metabolism | Cell Lineage | Phenotype | Animals | Myocytes, Cardiac - physiology | Fibroblasts - cytology | Mice | Care and treatment | Heart cells | Physiological aspects | Regeneration (Biology) | Research | Genetic transcription | Heart attack | Proteins | Heart | Flow cytometry | Genotype & phenotype | Heart attacks | Cardiomyocytes | Gene expression | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
9. Full Text Calcium-binding protein S100A4 confers mesenchymal progenitor cell fibrogenicity in idiopathic pulmonary fibrosis
by Xia, Hong and Gilbertsen, Adam and Herrera, Jeremy and Racila, Emilian and Smith, Karen and Peterson, Mark and Griffin, Timothy and Benyumov, Alexey and Yang, Libang and Bitterman, Peter B and Henke, Craig A
Journal of Clinical Investigation, ISSN 0021-9738, 06/2017, Volume 127, Issue 7, pp. 2586 - 2597
Idiopathic pulmonary fibrosis (IPF) is a progressive disease with a prevalence of 1 million persons worldwide. The fibrosis spreads from affected alveoli into... 
MEDICINE, RESEARCH & EXPERIMENTAL | MOLECULAR-PATTERNS | METASTASIS | TUMOR-SUPPRESSOR | LUNG INJURY | CLASSIFICATION | PROLIFERATION | MICE | FIBROBLASTS | CANCER STEM-CELLS | P53 | Idiopathic Pulmonary Fibrosis - genetics | Humans | Mesenchymal Stromal Cells - metabolism | Male | Mice, Transgenic | Idiopathic Pulmonary Fibrosis - chemically induced | Idiopathic Pulmonary Fibrosis - metabolism | Animals | S100 Calcium-Binding Protein A4 - genetics | Idiopathic Pulmonary Fibrosis - pathology | Female | Mice | Mesenchymal Stromal Cells - pathology | S100 Calcium-Binding Protein A4 - metabolism | Disease Models, Animal | Development and progression | Care and treatment | Genetic aspects | Pulmonary fibrosis | Binding proteins | Health aspects | Cell proliferation | Motility | Transcription | Mesenchyme | Pathogenesis | p53 Protein | Methyltransferase | S100A4 protein | Metastasis | Proteins | Cell growth | Bleomycin | Rodents | Calcium-binding protein | Fibroblasts | Alveoli | Invasiveness | Lung diseases | Injection | Gene expression | Studies | Stem cells | Fibrosis | Death | Cytoplasm | Cancer | Index Medicus | Abridged Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
10. Full Text Discovery of endothelial to mesenchymal transition as a source for carcinoma-associated fibroblasts
by Zeisberg, Elisabeth M and Potenta, Scott and Xie, Liang and Zeisberg, Michael and Kalluri, Raghu
Cancer Research, ISSN 0008-5472, 11/2007, Volume 67, Issue 21, pp. 10123 - 10128
Activated fibroblasts are associated with many different tumors. Myofibroblasts, activated fibroblasts, and perivascular mesenchymal cells such as pericytes... 
FIBROSIS | CELLS | TGF-BETA | ONCOLOGY | MICE | CANCER | TUMSTATIN | TUMORS | S100 Proteins | S100 Calcium-Binding Protein A4 | Calcium-Binding Proteins - analysis | Mice, Inbred C57BL | Pancreatic Neoplasms - pathology | Melanoma, Experimental - pathology | Platelet Endothelial Cell Adhesion Molecule-1 - analysis | Mice, Transgenic | Fibroblasts - pathology | Animals | Cell Differentiation | Mice | Endothelial Cells - pathology | Neoplasms - pathology | Mesoderm - pathology | Transforming Growth Factor beta1 - pharmacology | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
11. Full Text MicroRNA Induced Cardiac Reprogramming In Vivo: Evidence for Mature Cardiac Myocytes and Improved Cardiac Function
by Jayawardena, Tilanthi M and Finch, Elizabeth A and Zhang, Lunan and Zhang, Hengtao and Hodgkinson, Conrad P and Pratt, Richard E and Rosenberg, Paul B and Mirotsou, Maria and Dzau, Victor J
Circulation Research, ISSN 0009-7330, 01/2015, Volume 116, Issue 3, pp. 418 - 424
RATIONALE:A major goal for the treatment of heart tissue damaged by cardiac injury is to develop strategies for restoring healthy heart muscle through the... 
Cellular reprogramming | microRNAs | Myocardial infarction | Excitation-contraction coupling | Cardiac | Myocytes | Guided tissue regeneration | CARDIAC & CARDIOVASCULAR SYSTEMS | cellular reprogramming | myocardial infarction | MUSCLE | myocytes | FIBROBLASTS | guided tissue regeneration | GROWTH | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | excitation-contraction coupling | cardiac | Myocytes, Cardiac - cytology | S100 Calcium-Binding Protein A4 | Male | MicroRNAs - metabolism | S100 Proteins - metabolism | Myocardial Infarction - metabolism | Cellular Reprogramming | Myocardial Infarction - therapy | S100 Proteins - genetics | Animals | Myocytes, Cardiac - metabolism | Fibroblasts - cytology | Mice | MicroRNAs - genetics | Guided Tissue Regeneration | Fibroblasts - metabolism | Index Medicus | Direct reprogramming | tissue regeneration | cardiac myocytes | cardiac differentiation | cardiac injury | myocardial regeneration
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
12. Full Text Adipocyte-derived fibroblasts promote tumor progression and contribute to the desmoplastic reaction in breast cancer
by Bochet, Ludivine and Lehuédé, Camille and Dauvillier, Stéphonies and Wang, Yuan Yuan and Dirat, Béatrice and Laurent, Victor and Dray, Cédric and Guiet, Romain and Maridonneau-Parini, Isabelle and Gonidec, Sophie Le and Couderc, Bettina and Escourrou, Ghislaine and Valet, Philippe and Muller, Catherine
Cancer Research, ISSN 0008-5472, 09/2013, Volume 73, Issue 18, pp. 5657 - 5668
Cancer-associated fibroblasts (CAF) comprise the majority of stromal cells in breast cancers, yet their precise origins and relative functional contributions... 
INVASION | MICROENVIRONMENT | ONCOLOGY | EXHIBIT | DIFFERENTIATION | SECRETION | BETA-CATENIN | CELL-LINES | ADIPOGENESIS | EXPRESSION | ADIPOSE-TISSUE | Cell Proliferation | S100 Calcium-Binding Protein A4 | Stromal Cells - pathology | Coculture Techniques | Humans | Spheroids, Cellular - pathology | Tumor Microenvironment | Wnt Proteins - metabolism | Immunoenzyme Techniques | Breast Neoplasms - metabolism | S100 Proteins - genetics | Wnt Proteins - genetics | Female | Real-Time Polymerase Chain Reaction | Fibroblasts - metabolism | RNA, Messenger - genetics | Stromal Cells - metabolism | Cells, Cultured | S100 Proteins - metabolism | Fibroblasts - pathology | Reverse Transcriptase Polymerase Chain Reaction | Adipocytes - pathology | beta Catenin - metabolism | Blotting, Western | Fibronectins - metabolism | beta Catenin - genetics | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Mice, Nude | Adipocytes - metabolism | Fluorescent Antibody Technique | Fibronectins - genetics | Mice | Cell Movement | Index Medicus | Life Sciences | Cancer
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
13. Full Text Inhibition and role of let-7d in idiopathic pulmonary fibrosis
by Pandit, Kusum V and Corcoran, David and Yousef, Hanadie and Yarlagadda, Manohar and Tzouvelekis, Argyris and Gibson, Kevin F and Konishi, Kazuhisa and Yousem, Samuel A and Singh, Mandal and Handley, Daniel and Richards, Thomas and Selman, Moises and Watkins, Simon C and Pardo, Annie and Ben-Yehudah, Ahmi and Bouros, Demosthenes and Eickelberg, Oliver and Ray, Prabir and Benos, Panayiotis V and Kaminski, Naftali
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 07/2010, Volume 182, Issue 2, pp. 220 - 229
Rationale: Idiopathic pulmonary fibrosis (IPF) is a chronic, progressive, and usually lethal fibrotic lung disease characterized by profound changes in... 
CANCER-CELLS | TRANSFORMATION | MICRORNA GENES | HMGA2 (high-mobility group AT-hook 2) | MIR-200 FAMILY | REPRESSORS ZEB1 | transforming growth factor-beta | CAENORHABDITIS-ELEGANS | RESPIRATORY SYSTEM | IN-VIVO | DISEASE | GENE-EXPRESSION | microRNA | epithelial-mesenchymal transition | CRITICAL CARE MEDICINE | Epithelial Cells - metabolism | Cadherins - metabolism | S100 Calcium-Binding Protein A4 | Vimentin - metabolism | Down-Regulation | Humans | Mice, Inbred C57BL | Actins - metabolism | Cells, Cultured | Transforming Growth Factor beta - physiology | MicroRNAs - metabolism | S100 Proteins - metabolism | Smad3 Protein - metabolism | Idiopathic Pulmonary Fibrosis - metabolism | Animals | In Situ Hybridization | HMGA2 Protein - metabolism | Pulmonary Alveoli - metabolism | Polymerase Chain Reaction | Idiopathic Pulmonary Fibrosis - pathology | Lung - metabolism | Mice | Index Medicus | Abridged Index Medicus | epithelial–mesenchymal transition | transforming growth factor-β | F. Interstitial Lung Disease
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights