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Neuron, ISSN 0896-6273, 2005, Volume 47, Issue 6, pp. 817 - 831
The molecular mechanisms controlling the differentiation of neural progenitors into distinct subtypes of neurons during neocortical development are unknown.... 
AREA IDENTITY | NEOCORTICAL NEURONS | CELL FATE SPECIFICATION | CEREBRAL CORTICAL DEVELOPMENT | LAYER NEURONS | CENTRAL-NERVOUS-SYSTEM | SUBCORTICAL TARGETS | ZINC-FINGER GENE | SUBVENTRICULAR ZONE | NEUROSCIENCES | ZEBRAFISH FOREBRAIN | Membrane Glycoproteins - metabolism | Embryo, Mammalian | Homeodomain Proteins - metabolism | Nerve Tissue Proteins - deficiency | S100 Proteins - genetics | POU Domain Factors - genetics | Forkhead Transcription Factors - metabolism | In Situ Hybridization - methods | Repressor Proteins - metabolism | Gene Expression Regulation, Developmental - physiology | POU Domain Factors - metabolism | Animals, Newborn | DNA-Binding Proteins - physiology | Motor Neurons - physiology | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Cell Cycle Proteins - metabolism | S100 Proteins - metabolism | In Situ Nick-End Labeling - methods | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Immunohistochemistry - methods | Annexin A2 - genetics | Cell Death - physiology | Green Fluorescent Proteins - biosynthesis | Mice | Annexin A2 - metabolism | Age Factors | Phosphopyruvate Hydratase - metabolism | Cell Movement - physiology | DNA-Binding Proteins - deficiency | DNA-Binding Proteins - metabolism | Amino Acids - metabolism | Motor Neurons - cytology | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Membrane Proteins - metabolism | Bromodeoxyuridine - metabolism | Cell Differentiation - physiology | Pyramidal Tracts - physiology | Nerve Tissue Proteins - physiology | Neocortex - growth & development | Nuclear Proteins - metabolism | DNA-Binding Proteins - genetics | Dopamine and cAMP-Regulated Phosphoprotein 32 - metabolism | Nerve Tissue Proteins - genetics | Homeodomain Proteins - genetics | Membrane Glycoproteins - genetics | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Cell Count - methods | Neocortex - cytology | Receptors, Antigen, T-Cell, alpha-beta - metabolism | Neurosciences | Neurons | Developmental biology | Stem cells | Studies | Brain | Ultrasonic imaging | Transcription factors | Hybridization | Neurogenesis | Index Medicus
Journal Article
Neuron, ISSN 0896-6273, 02/2013, Volume 77, Issue 3, pp. 472 - 484
Major outputs of the neocortex are conveyed by corticothalamic axons (CTAs), which form reciprocal connections with thalamocortical axons, and... 
MUTANT MICE | CORTICAL AXONS | THALAMOCORTICAL AXONS | GROWTH | SEMAPHORIN 3E | CENTRAL-NERVOUS-SYSTEM | GUIDANCE | CHICK HINDLIMB | CAJAL-RETZIUS CELLS | NEUROSCIENCES | CEREBRAL-CORTEX | Thyroid Nuclear Factor 1 | Age Factors | Embryo, Mammalian | Leukocyte L1 Antigen Complex - metabolism | Gene Expression Regulation, Developmental - genetics | Axons - physiology | Cerebral Cortex - cytology | Neural Pathways - physiology | DNA-Binding Proteins - metabolism | POU Domain Factors - genetics | tau Proteins - genetics | Thalamus - physiology | Contactin 2 - metabolism | Repressor Proteins - metabolism | Glycoproteins - genetics | Tumor Suppressor Proteins - metabolism | Wnt3A Protein - genetics | Membrane Proteins - genetics | Mice, Inbred C57BL | Mice, Transgenic | Nuclear Proteins - metabolism | Transcription Factors - genetics | Nerve Tissue Proteins - genetics | Homeodomain Proteins - genetics | Membrane Glycoproteins - genetics | Nerve Tissue Proteins - metabolism | S100 Calcium Binding Protein G - metabolism | Transcription Factors - metabolism | Animals | Calbindin 2 | Thalamus - cytology | Cerebral Cortex - physiology | Luminescent Proteins - genetics | Mice | Body Patterning - genetics | Luminescent Proteins - metabolism | Developmental biology | Neurons | Studies | Laboratories | Experiments | Index Medicus | Repressor Proteins | Cerebral Cortex | Cellular Biology | Neural Pathways | tau Proteins | Life Sciences | Contactin 2 | Gene Expression Regulation, Developmental | Body Patterning | Thalamus | Membrane Glycoproteins | Luminescent Proteins | DNA-Binding Proteins | POU Domain Factors | Calcium-Binding Protein, Vitamin D-Dependent | Glycoproteins | Nerve Tissue Proteins | Nuclear Proteins | Membrane Proteins | Axons | Homeodomain Proteins | Leukocyte L1 Antigen Complex | Transcription Factors | Wnt3A Protein | Tumor Suppressor Proteins | reciprocal connections | handshake | waiting period | PlexinD1 | axon guidance | Sema3E | thalamocortical | corticothalamic
Journal Article
Neuron, ISSN 0896-6273, 2010, Volume 65, Issue 5, pp. 597 - 611
To investigate the role of microRNAs in regulating oligodendrocyte (OL) differentiation and myelination, we utilized transgenic mice in which microRNA... 
DEVBIO | MOLNEURO | TRANSCRIPTION FACTORS | NERVOUS-SYSTEM | GLIAL PROGENITOR-CELL | PROTEIN | CYCLE EXIT | NEURONS | GROWTH-FACTOR | IDENTIFICATION | MICRORNA EXPRESSION | LINEAGE | NEUROSCIENCES | Central Nervous System - metabolism | MicroRNAs - metabolism | Green Fluorescent Proteins - genetics | S100 Proteins - genetics | Oligodendroglia - drug effects | Basic Helix-Loop-Helix Transcription Factors - metabolism | Animals, Newborn | Basic Helix-Loop-Helix Transcription Factors - genetics | Receptor, Platelet-Derived Growth Factor alpha - metabolism | S100 Calcium Binding Protein beta Subunit | Oligonucleotide Array Sequence Analysis - methods | Rats | Mice, Transgenic | Oligodendrocyte Transcription Factor 2 | Myelin Proteins - genetics | Rats, Sprague-Dawley | Nerve Growth Factors - genetics | Mice | MicroRNAs - genetics | Myelin Proteins - metabolism | SOXD Transcription Factors - genetics | Optic Nerve - metabolism | Age Factors | SOXD Transcription Factors - metabolism | Gene Expression Regulation, Developmental - genetics | Sciatic Nerve - growth & development | Myelin Sheath - metabolism | Central Nervous System - growth & development | DNA-Binding Proteins - metabolism | Sciatic Nerve - metabolism | Oligodendroglia - physiology | Transfection | DEAD-box RNA Helicases - metabolism | Cell Differentiation - physiology | Optic Nerve - growth & development | Brain - cytology | 2',3'-Cyclic-Nucleotide Phosphodiesterases - genetics | Ribonuclease III - genetics | Ribonuclease III - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Profiling - methods | Transcription Factors - genetics | 2',3'-Cyclic-Nucleotide Phosphodiesterases - metabolism | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | DEAD-box RNA Helicases - genetics | Animals | Cell Differentiation - drug effects | Receptor, Platelet-Derived Growth Factor alpha - genetics | Stem Cells - drug effects | Stem Cells - physiology | Proteins | Genotype & phenotype | Gene expression | Experiments | Rodents | Cell cycle | Index Medicus
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 03/2011, Volume 286, Issue 9, pp. 7214 - 7226
The Ca2+-binding protein of the EF-hand type, S100B, is abundantly expressed in and secreted by astrocytes, and release of S100B from damaged astrocytes occurs... 
CYTOPLASMIC DOMAIN | KAPPA-B ACTIVATION | IN-VITRO | NEURITE OUTGROWTH | INFLAMMATORY RESPONSES | BRAIN-INJURY | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | NITRIC-OXIDE | GROWTH-FACTOR | GLYCATION END-PRODUCTS | Microglia - metabolism | Chemokine CCL3 - immunology | Nerve Growth Factors - metabolism | S100 Proteins - immunology | Cell Movement - immunology | S100 Proteins - genetics | Chemokine CXCL12 - genetics | Microglia - immunology | Chemokine CCL3 - genetics | Calcium-Binding Proteins - immunology | Cattle | Encephalitis - metabolism | Chemokine CCL3 - metabolism | Chemokine CCL5 - metabolism | Receptors, Immunologic - immunology | Receptor for Advanced Glycation End Products | Chemokine CXCL12 - immunology | Chemokines - immunology | Calcium-Binding Proteins - metabolism | Microglia - cytology | Recombinant Proteins - metabolism | Cell Line | S100 Calcium Binding Protein beta Subunit | Mice, Inbred C57BL | Rats | Encephalitis - immunology | S100 Proteins - metabolism | Nerve Growth Factors - immunology | Recombinant Proteins - genetics | Chemokines - genetics | Animals | Carrier Proteins - metabolism | Chemokine CXCL12 - metabolism | Recombinant Proteins - immunology | Up-Regulation - immunology | Nerve Growth Factors - genetics | Chemokine CCL5 - genetics | Cytoskeleton - metabolism | Chemokines - metabolism | Mice | Receptors, Immunologic - genetics | Chemokine CCL5 - immunology | Receptors, Immunologic - metabolism | Calcium-Binding Proteins - genetics | Index Medicus | Immunology | Defensins | Signal Transduction | Neurodegeneration | Migration | Calcium-binding Proteins | Innate Immunity | RAGE | S100B | Chemokines | Microglia
Journal Article
Neurobiology of Aging, ISSN 0197-4580, 2008, Volume 31, Issue 4, pp. 578 - 590
Abstract Astrocyte pathology occurs in association with Alzheimer's disease (AD) and in brain ageing, but is poorly characterised. We sought to define the... 
Neurology | Internal Medicine | Aging | Alzheimer's type pathology | Excitatory amino acid transporter | Temporal cortex | Astrocyte | Glial fibrillary acidic protein | OXIDATIVE STRESS | AMYLOID PLAQUES | FIBROUS ASTROCYTES | NEUROSCIENCES | NEURODEGENERATIVE DISEASES | COGNITIVE FUNCTION | GERIATRICS & GERONTOLOGY | IN-VITRO | CEREBROSPINAL-FLUID S100B | SENILE-DEMENTIA | FIBRILLARY ACIDIC PROTEIN | GLUTAMATE TRANSPORTER | Immunohistochemistry | Predictive Value of Tests | Prospective Studies | Humans | Astrocytes - pathology | Nerve Tissue Proteins - analysis | tau Proteins - metabolism | Male | Gliosis - physiopathology | Nerve Growth Factors - metabolism | Glial Fibrillary Acidic Protein - metabolism | Alzheimer Disease - pathology | Brain - metabolism | Gliosis - pathology | Amyloid beta-Peptides - metabolism | Aged, 80 and over | Female | Biomarkers - metabolism | Severity of Illness Index | Alzheimer Disease - physiopathology | Plaque, Amyloid - pathology | S100 Calcium Binding Protein beta Subunit | Brain - physiopathology | Glial Fibrillary Acidic Protein - analysis | Gliosis - metabolism | Biomarkers - analysis | S100 Proteins - metabolism | Excitatory Amino Acid Transporter 1 - metabolism | Excitatory Amino Acid Transporter 2 - analysis | Excitatory Amino Acid Transporter 2 - metabolism | S100 Proteins - analysis | Nerve Growth Factors - analysis | Nerve Tissue Proteins - metabolism | Phenotype | Alzheimer Disease - metabolism | Amyloid beta-Peptides - analysis | Brain - pathology | Plaque, Amyloid - metabolism | Aged | tau Proteins - analysis | Excitatory Amino Acid Transporter 1 - analysis | Longitudinal Studies | Astrocytes - metabolism | Cohort Studies | Amino acids | Genetic aspects | Alzheimer's disease | Intermediate filament proteins | Public health | Index Medicus
Journal Article
Science, ISSN 0036-8075, 9/2010, Volume 329, Issue 5998, pp. 1537 - 1541
The serotonin transporter (SERT) ensures the recapture of serotonin and is the pharmacological target of selective serotonin reuptake inhibitor (SSRI)... 
Receptors | MicroRNA | Serotonin agents | Neurons | Antidepressants | Serotonin receptors | REPORTS | Cell lines | Serotonin plasma membrane transport proteins | Adrenergic neurons | Locus coeruleus | LOCUS-COERULEUS | CELLS | RECEPTOR | RECOGNITION | RAT | MULTIDISCIPLINARY SCIENCES | Up-Regulation | Protein Biosynthesis | Humans | MicroRNAs - metabolism | Nerve Growth Factors - metabolism | Antidepressive Agents, Second-Generation - administration & dosage | Raphe Nuclei - metabolism | Serotonin Plasma Membrane Transport Proteins - genetics | Neurons - metabolism | 3' Untranslated Regions | Neurons - drug effects | Serotonin Uptake Inhibitors - metabolism | Cell Line | Fluoxetine - pharmacology | S100 Calcium Binding Protein beta Subunit | MicroRNAs - administration & dosage | Depression | S100 Proteins - metabolism | Antidepressive Agents, Second-Generation - pharmacology | Serotonin Plasma Membrane Transport Proteins - metabolism | Animals | Norepinephrine - metabolism | Raphe Nuclei - drug effects | Serotonin Uptake Inhibitors - administration & dosage | Serotonin - metabolism | Fluoxetine - administration & dosage | Fluoxetine - metabolism | Mice | MicroRNAs - genetics | HeLa Cells | Antidepressive Agents, Second-Generation - metabolism | Serotonin Uptake Inhibitors - pharmacology | Locus Coeruleus - metabolism | Pharmacogenetics | Serotonin | Physiological aspects | Serotonin uptake inhibitors | Dosage and administration | Research | Health aspects | Pharmacology | Index Medicus
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 07/2010, Volume 19, Issue 20, pp. 3919 - 3935
Although a direct causative pathway from the gene mutation to the selective neostriatal neurodegeneration remains unclear in Huntington's disease (HD), one... 
CYTOCHROME-C | TOXIN 3-NITROPROPIONIC ACID | COMPONENT FIS1P | DEGENERATIVE CHANGES | BUDDING YEAST | BIOCHEMISTRY & MOLECULAR BIOLOGY | MUTANT HUNTINGTIN | GENETICS & HEREDITY | HEREDITARY OPTIC NEUROPATHY | TRANSCRIPTION FACTOR | NEURODEGENERATIVE DISEASES | TRANSGENIC MICE | Neurons - pathology | Neostriatum - ultrastructure | Microtubule-Associated Proteins - metabolism | Humans | Huntington Disease - pathology | Electron Transport Complex IV - analysis | Gene Expression Profiling | DNA-Binding Proteins - metabolism | Calbindins | Mitochondrial Membrane Transport Proteins | DNA, Mitochondrial - genetics | Mitochondria - genetics | Mitochondrial Proteins - metabolism | Membrane Potential, Mitochondrial | Polymerase Chain Reaction | Membrane Transport Proteins - metabolism | Neostriatum - metabolism | Nuclear Proteins - genetics | S100 Calcium Binding Protein G - analysis | Gene Expression | Superoxide Dismutase - analysis | DNA, Mitochondrial - metabolism | Neurons - chemistry | Cytochromes c - immunology | Mitochondria - metabolism | Cytochromes c - analysis | Mitochondria - pathology | Huntington Disease - metabolism | Nerve Tissue Proteins - genetics | Transcription Factors - metabolism | Huntingtin Protein | Superoxide Dismutase - immunology | GTP Phosphohydrolases - metabolism | Energy Metabolism | Fluorescent Antibody Technique | Peroxisome Proliferator-Activated Receptors - metabolism | Huntington Disease - genetics | Index Medicus
Journal Article
Genes, Chromosomes and Cancer, ISSN 1045-2257, 02/2014, Volume 53, Issue 2, pp. 183 - 193
Journal Article
Cell Stem Cell, ISSN 1934-5909, 11/2016, Volume 19, Issue 5, pp. 613 - 627
Mesenchymal niche cells may drive tissue failure and malignant transformation in the hematopoietic system, but the underlying molecular mechanisms and... 
microenvironment | leukemia | inflammation | mesenchymal | hematopoietic stem cell | shwachman-diamond syndrome | niche | cancer | myelodysplastic syndrome (MDS) | S100A8 | PROGENITOR CELLS | OXIDATIVE DNA-DAMAGE | GENE | SBDS | MUTATIONS | SHWACHMAN-DIAMOND-SYNDROME | MYELODYSPLASTIC CELLS | STROMAL CELLS | EXPRESSION | DEFICIENCY | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Bone and Bones - pathology | Inflammation - pathology | Leukemia - pathology | Oxidative Stress | Humans | Hematopoietic Stem Cells - pathology | Precancerous Conditions - metabolism | Leukemia - metabolism | S100 Proteins - genetics | Lipomatosis - pathology | Gene Deletion | Integrases - metabolism | Precancerous Conditions - pathology | Toll-Like Receptors - metabolism | Stem Cell Niche | Pathogen-Associated Molecular Pattern Molecules - metabolism | Signal Transduction | Exocrine Pancreatic Insufficiency - pathology | Risk Factors | Tumor Suppressor Protein p53 - metabolism | Mesenchymal Stromal Cells - metabolism | S100 Proteins - metabolism | Treatment Outcome | Hematopoietic Stem Cells - metabolism | Mitochondria - metabolism | Bone Marrow Diseases - pathology | Disease Progression | Transcription Factors - metabolism | Animals | Proteins - metabolism | DNA Repair | Sp7 Transcription Factor | Mice | DNA Damage | Mesenchymal Stromal Cells - pathology | Bone and Bones - abnormalities | Index Medicus
Journal Article