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Neuron (Cambridge, Mass.), ISSN 0896-6273, 02/2013, Volume 77, Issue 3, pp. 472 - 484
Major outputs of the neocortex are conveyed by corticothalamic axons (CTAs), which form reciprocal connections with thalamocortical axons, and... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Thyroid Nuclear Factor 1 | Age Factors | Embryo, Mammalian | Leukocyte L1 Antigen Complex - metabolism | Gene Expression Regulation, Developmental - genetics | Axons - physiology | Cerebral Cortex - cytology | Neural Pathways - physiology | DNA-Binding Proteins - metabolism | POU Domain Factors - genetics | tau Proteins - genetics | Thalamus - physiology | Contactin 2 - metabolism | Repressor Proteins - metabolism | Glycoproteins - genetics | Tumor Suppressor Proteins - metabolism | Wnt3A Protein - genetics | Membrane Proteins - genetics | Mice, Inbred C57BL | Mice, Transgenic | Nuclear Proteins - metabolism | Transcription Factors - genetics | Nerve Tissue Proteins - genetics | Homeodomain Proteins - genetics | Membrane Glycoproteins - genetics | Nerve Tissue Proteins - metabolism | S100 Calcium Binding Protein G - metabolism | Transcription Factors - metabolism | Animals | Calbindin 2 | Thalamus - cytology | Cerebral Cortex - physiology | Luminescent Proteins - genetics | Mice | Body Patterning - genetics | Luminescent Proteins - metabolism | Developmental biology | Neurons | Studies | Laboratories | Experiments | Index Medicus | Repressor Proteins | Cerebral Cortex | Cellular Biology | Neural Pathways | tau Proteins | Life Sciences | Contactin 2 | Gene Expression Regulation, Developmental | Body Patterning | Thalamus | Membrane Glycoproteins | Luminescent Proteins | DNA-Binding Proteins | POU Domain Factors | Calcium-Binding Protein, Vitamin D-Dependent | Glycoproteins | Nerve Tissue Proteins | Nuclear Proteins | Membrane Proteins | Axons | Homeodomain Proteins | Leukocyte L1 Antigen Complex | Transcription Factors | Wnt3A Protein | Tumor Suppressor Proteins | reciprocal connections | handshake | waiting period | PlexinD1 | axon guidance | Sema3E | thalamocortical | corticothalamic
Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 10/2003, Volume 40, Issue 3, pp. 501 - 514
Targeted deletion of focal adhesion kinase ( fak) in the developing dorsal forebrain resulted in local disruptions of the cortical basement membrane located... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Immunohistochemistry | Membrane Glycoproteins - metabolism | Embryo, Mammalian | Protein-Tyrosine Kinases - metabolism | Homeodomain Proteins - metabolism | Astrocytes - pathology | Cerebral Cortex - pathology | Glial Fibrillary Acidic Protein - metabolism | Muscular Dystrophies - genetics | Protein-Tyrosine Kinases - deficiency | Focal Adhesion Kinase 1 | Calbindins | Neurons - metabolism | Serine Endopeptidases | Disease Models, Animal | Fibroblasts - metabolism | Muscular Dystrophies - metabolism | Muscular Dystrophies - congenital | Silver Staining | Nerve Tissue Proteins | Basement Membrane - metabolism | Blotting, Western | Precipitin Tests | Mice, Knockout | Infection | Staining and Labeling | Mice | Mutation | Dura Mater | Astrocytes - metabolism | Neurons - pathology | Microtubule-Associated Proteins - metabolism | Otx Transcription Factors | Phosphopyruvate Hydratase - metabolism | Basement Membrane - pathology | Dystroglycans | Cerebral Cortex - metabolism | Cerebral Cortex - ultrastructure | Phosphotyrosine - metabolism | DNA-Binding Proteins - metabolism | Protein-Tyrosine Kinases - genetics | Lamins - metabolism | src-Family Kinases - metabolism | Cytoskeletal Proteins - metabolism | Cell Adhesion Molecules, Neuronal - metabolism | Cerebral Cortex - abnormalities | Extracellular Matrix Proteins - metabolism | Fibroblasts - virology | Cells, Cultured | Focal Adhesion Protein-Tyrosine Kinases | Microscopy, Electron | Muscular Dystrophies - pathology | S100 Calcium Binding Protein G - metabolism | Transcription Factors - metabolism | Animals | Carrier Proteins - metabolism | Calbindin 2 | Bacterial Proteins - metabolism | Heterozygote | Proteins | Neurons | Rodents | Protein expression | Kinases | Embryos | Muscular dystrophy | Deoxyribonucleic acid--DNA | Cell adhesion & migration | Index Medicus
Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 03/2010, Volume 65, Issue 5, pp. 597 - 611
.... miR-219 directly represses the expression of PDGFRα, Sox6, FoxJ3, and ZFP238 proteins, all of which... 
DEVBIO | MOLNEURO | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Central Nervous System - metabolism | MicroRNAs - metabolism | Green Fluorescent Proteins - genetics | S100 Proteins - genetics | Oligodendroglia - drug effects | Basic Helix-Loop-Helix Transcription Factors - metabolism | Animals, Newborn | Basic Helix-Loop-Helix Transcription Factors - genetics | Receptor, Platelet-Derived Growth Factor alpha - metabolism | S100 Calcium Binding Protein beta Subunit | Oligonucleotide Array Sequence Analysis - methods | Rats | Mice, Transgenic | Oligodendrocyte Transcription Factor 2 | Myelin Proteins - genetics | Rats, Sprague-Dawley | Nerve Growth Factors - genetics | Mice | MicroRNAs - genetics | Myelin Proteins - metabolism | SOXD Transcription Factors - genetics | Optic Nerve - metabolism | Age Factors | SOXD Transcription Factors - metabolism | Gene Expression Regulation, Developmental - genetics | Sciatic Nerve - growth & development | Myelin Sheath - metabolism | Central Nervous System - growth & development | DNA-Binding Proteins - metabolism | Sciatic Nerve - metabolism | Oligodendroglia - physiology | Transfection | DEAD-box RNA Helicases - metabolism | Cell Differentiation - physiology | Optic Nerve - growth & development | Brain - cytology | 2',3'-Cyclic-Nucleotide Phosphodiesterases - genetics | Ribonuclease III - genetics | Ribonuclease III - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Profiling - methods | Transcription Factors - genetics | 2',3'-Cyclic-Nucleotide Phosphodiesterases - metabolism | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | DEAD-box RNA Helicases - genetics | Animals | Cell Differentiation - drug effects | Receptor, Platelet-Derived Growth Factor alpha - genetics | Stem Cells - drug effects | Stem Cells - physiology | Proteins | Genotype & phenotype | Gene expression | Experiments | Rodents | Cell cycle | Index Medicus
Journal Article
Genes chromosomes & cancer, ISSN 1045-2257, 02/2014, Volume 53, Issue 2, pp. 183 - 193
Journal Article
Neurobiology of aging, ISSN 0197-4580, 04/2010, Volume 31, Issue 4, pp. 578 - 590
... is histopathologically characterised by the extracellular deposition of β-amyloid protein in senile plaques, intracellular neurofibrillary tangles (NFT) of hyperphosphorylated tau... 
Neurology | Internal Medicine | Aging | Alzheimer's type pathology | Excitatory amino acid transporter | Temporal cortex | Astrocyte | Glial fibrillary acidic protein | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Geriatrics & Gerontology | Science & Technology | Immunohistochemistry | Predictive Value of Tests | Prospective Studies | Humans | Astrocytes - pathology | Nerve Tissue Proteins - analysis | tau Proteins - metabolism | Male | Gliosis - physiopathology | Nerve Growth Factors - metabolism | Glial Fibrillary Acidic Protein - metabolism | Alzheimer Disease - pathology | Brain - metabolism | Gliosis - pathology | Amyloid beta-Peptides - metabolism | Aged, 80 and over | Female | Biomarkers - metabolism | Severity of Illness Index | Alzheimer Disease - physiopathology | Plaque, Amyloid - pathology | S100 Calcium Binding Protein beta Subunit | Brain - physiopathology | Glial Fibrillary Acidic Protein - analysis | Gliosis - metabolism | Biomarkers - analysis | S100 Proteins - metabolism | Excitatory Amino Acid Transporter 1 - metabolism | Excitatory Amino Acid Transporter 2 - analysis | Excitatory Amino Acid Transporter 2 - metabolism | S100 Proteins - analysis | Nerve Growth Factors - analysis | Nerve Tissue Proteins - metabolism | Phenotype | Alzheimer Disease - metabolism | Amyloid beta-Peptides - analysis | Brain - pathology | Plaque, Amyloid - metabolism | Aged | tau Proteins - analysis | Excitatory Amino Acid Transporter 1 - analysis | Longitudinal Studies | Astrocytes - metabolism | Cohort Studies | Amino acids | Genetic aspects | Alzheimer's disease | Intermediate filament proteins | Public health | Index Medicus
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2011, Volume 141, Issue 2, pp. 621 - 632
.... Epithelial cells are distinct in that they are highly polarized; this polarity is, at least in part, established by the epithelium-specific polarized sorting factor adaptor protein (AP)-1B... 
Gastroenterology and Hepatology | IgA | Inflammatory Bowel Disease | T Cell | Immune Response | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Tumor Necrosis Factor-alpha - metabolism | Intestinal Mucosa - metabolism | Adaptor Protein Complex mu Subunits - metabolism | Epithelial Cells - metabolism | Adaptor Protein Complex beta Subunits - deficiency | Humans | Cell Membrane Permeability | Crohn Disease - metabolism | Cell Membrane - physiology | Ribonucleases - metabolism | Adaptor Protein Complex 1 - metabolism | Th17 Cells - metabolism | Intestinal Mucosa - immunology | alpha-Defensins - metabolism | Adaptor Protein Complex beta Subunits - immunology | Cell Membrane - metabolism | beta-Defensins - metabolism | Colon | Receptors, Cytokine - metabolism | Colitis - immunology | Adaptor Protein Complex 1 - deficiency | Lipocalins - metabolism | Adaptor Protein Complex 1 - immunology | Immunoglobulin A - metabolism | Signal Transduction | Acute-Phase Proteins - metabolism | Down-Regulation | Oncogene Proteins - metabolism | Homeostasis - immunology | Epithelial Cells - pathology | S100 Proteins - metabolism | Muramidase - metabolism | Intestinal Mucosa - microbiology | Mice, Knockout | Interleukin-17 - metabolism | Adaptor Protein Complex beta Subunits - metabolism | Animals | Colitis - microbiology | Proteins - metabolism | Ribonuclease, Pancreatic - metabolism | Lipocalin-2 | Cathelicidins - metabolism | Epithelial Cells - immunology | Receptors, Cytokine - immunology | Mice | Intestinal Mucosa - pathology | Medical colleges | Fc receptors | Gastrointestinal diseases | Homeostasis | Biological response modifiers | Immunoglobulin A | T cells | Index Medicus | Abridged Index Medicus
Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 2005, Volume 47, Issue 6, pp. 817 - 831
The molecular mechanisms controlling the differentiation of neural progenitors into distinct subtypes of neurons during neocortical development are unknown.... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Membrane Glycoproteins - metabolism | Embryo, Mammalian | Homeodomain Proteins - metabolism | Nerve Tissue Proteins - deficiency | S100 Proteins - genetics | POU Domain Factors - genetics | Forkhead Transcription Factors - metabolism | In Situ Hybridization - methods | Repressor Proteins - metabolism | Gene Expression Regulation, Developmental - physiology | POU Domain Factors - metabolism | Animals, Newborn | DNA-Binding Proteins - physiology | Motor Neurons - physiology | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Cell Cycle Proteins - metabolism | S100 Proteins - metabolism | In Situ Nick-End Labeling - methods | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Immunohistochemistry - methods | Annexin A2 - genetics | Cell Death - physiology | Green Fluorescent Proteins - biosynthesis | Mice | Annexin A2 - metabolism | Age Factors | Phosphopyruvate Hydratase - metabolism | Cell Movement - physiology | DNA-Binding Proteins - deficiency | DNA-Binding Proteins - metabolism | Amino Acids - metabolism | Motor Neurons - cytology | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Membrane Proteins - metabolism | Bromodeoxyuridine - metabolism | Cell Differentiation - physiology | Pyramidal Tracts - physiology | Nerve Tissue Proteins - physiology | Neocortex - growth & development | Nuclear Proteins - metabolism | DNA-Binding Proteins - genetics | Dopamine and cAMP-Regulated Phosphoprotein 32 - metabolism | Nerve Tissue Proteins - genetics | Homeodomain Proteins - genetics | Membrane Glycoproteins - genetics | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Cell Count - methods | Neocortex - cytology | Receptors, Antigen, T-Cell, alpha-beta - metabolism | Neurons | Developmental biology | Stem cells | Studies | Brain | Ultrasonic imaging | Transcription factors | Hybridization | Neurogenesis | Index Medicus
Journal Article
Scientific reports, ISSN 2045-2322, 02/2016, Volume 6, Issue 1, pp. 22112 - 22112
The consequence of a loss of balance between G-protein activation and deactivation in cancers has been interrogated by studying infrequently occurring mutants of trimeric G-protein alpha-subunits and GPCRs... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Neoplastic Cells, Circulating - pathology | Vesicular Transport Proteins - metabolism | Humans | Intracellular Signaling Peptides and Proteins - metabolism | DNA-Binding Proteins - metabolism | S100 Calcium-Binding Protein A4 - genetics | Guanine Nucleotide Exchange Factors - metabolism | Biomarkers, Tumor - metabolism | Microfilament Proteins - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Microfilament Proteins - genetics | Calcium-Binding Proteins - metabolism | Colorectal Neoplasms - mortality | Protein Structure, Tertiary | Gene Expression | GTP-Binding Protein alpha Subunits - metabolism | GTP-Binding Protein alpha Subunits - genetics | Transcription Factors - genetics | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Disease-Free Survival | Neoplasm Metastasis - genetics | Neoplasm Metastasis - pathology | Biomarkers, Tumor - genetics | Colorectal Neoplasms - pathology | S100 Calcium-Binding Protein A4 - metabolism | Proteins | G protein-coupled receptors | Deactivation | Tumor cells | Colorectal carcinoma | Medical prognosis | S100A4 protein | Colorectal cancer | Metastasis | Guanine nucleotide exchange factor | Guanine | Metastases | Index Medicus
Journal Article