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Autophagy, ISSN 1554-8627, 12/2014, Volume 10, Issue 12, pp. 2171 - 2192
SNCA/α-synuclein aggregation plays a crucial role in synucleinopathies such as Parkinson disease and dementia with Lewy bodies. Aggregating and nonaggregating... 
GFAP, glial fibrillary acidic protein | HSPA8/Hsc70, heat shock 70kDa protein 8 | lysosomal degradation | BafA1, bafilomycinA | CASP3/aCasp3, caspase-3 | ESCRT, endosomal sorting complex required for transport | PF, particle fraction | ANXA5, annexin A5 | SNCA-T, tagged α-synuclein | PDGFB/PDGFb, platelet-derived growth factor subunit b | PD, Parkinson disease | S100B/S100b, S100 calcium-binding protein B | HRP, horseradish peroxidase | TUBB3/b-III-Tub, tubulin, β 3 class III | AIF1/Iba1, allograft inflammatory factor 1 | α-synuclein | CA1, cornu ammonis field1 | RT, room temperature | ILVs, intraluminal vesicles | LAMP2A/Lamp2a, lysosomal-associated membrane protein 2, isoform A | MAP2, microtubule-associated protein 2 | SYP, synaptophysin | CD63, CD63 molecule | LN, Lewy neuritis | ALP, autophagy-lysosomal pathway | RAB11A/rab11, member RAS oncogene family | 1 | secretion | EV, empty vector | protein aggregation | TNF/TNFa, tumor necrosis factor α | WT-SNCA, wild-type α-synuclein | CSF, cerebrospinal fluid | RBFOX3/NeuN, RNA binding protein, fox-1 homolog (C. elegans) 3 | DLB, dementia with Lewy bodies | CM, conditioned medium | tg, transgenic | Parkinson disease | ML, molecular layer | ACTB/bAct, actin, β | ER, endoplasmatic reticulum | synucleinopathies | SL, stratum lacunosum; SNCA/aSyn | SNCAIP/Sph1, synphilin-1 | CMA, chaperone-mediated autophagy | inflammation | Neoctx, neocortex | UPS, ubiquitin proteasome system | IL6/IL-6, interleukin-6 | N, neuron | LB, Lewy bodies | MVBs, multivesicular bodies | PS, phosphatidylserine | Hippo, hippocampus | AK, adenylate kinase | Protein aggregation | Lysosomal degradation | Secretion | Synucleinopathies | Inflammation | CALCIUM-DEPENDENT MANNER | CHAPERONE-MEDIATED AUTOPHAGY | LEWY BODY DISEASE | alpha-synuclein | CELL-DEATH | CELL BIOLOGY | TRANSMISSION | MULTIVESICULAR BODIES | PROTEIN-DEGRADATION | NEURONS | PARKINSONS-DISEASE | Animals | Rats, Wistar | Humans | Cells, Cultured | Mice, Transgenic | Signal Transduction - physiology | Autophagy - physiology | Mice | Neurons - metabolism | Parkinson Disease - metabolism | Lewy Bodies - metabolism | alpha-Synuclein - metabolism
Journal Article
The EMBO Journal, ISSN 0261-4189, 08/2007, Volume 26, Issue 16, pp. 3868 - 3878
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, p. e33606
Background: Sports-related head trauma is common but still there is no established laboratory test used in the diagnostics of minimal or mild traumatic brain... 
MILD | FOOTBALL | SPORTS | DIFFUSE AXONAL INJURY | FIBRILLARY ACIDIC PROTEIN | MULTIDISCIPLINARY SCIENCES | CHRONIC BRAIN-DAMAGE | CEREBROSPINAL-FLUID | CONCUSSION | S100B | LEAD | Prognosis | Prospective Studies | Follow-Up Studies | S100 Calcium Binding Protein beta Subunit | Humans | S100 Proteins - cerebrospinal fluid | Craniocerebral Trauma - diagnosis | Male | tau Proteins - cerebrospinal fluid | Case-Control Studies | Craniocerebral Trauma - etiology | Glial Fibrillary Acidic Protein - cerebrospinal fluid | Young Adult | Boxing - injuries | Transcription Factors - cerebrospinal fluid | Craniocerebral Trauma - cerebrospinal fluid | Adolescent | Adult | Female | Nerve Growth Factors - cerebrospinal fluid | Surveys and Questionnaires | Biomarkers - cerebrospinal fluid | Olympic athletes | Brain | Sports injuries | Boxing | Diagnosis | Biological markers | Injuries | Neurosciences | Nuclear magnetic resonance--NMR | Traumatic brain injury | Laboratories | S100b protein | Cerebrospinal fluid | Medical diagnosis | Subgroups | Proteins | Rest | Head injuries | Neurodegeneration | Questionnaires | Tomography | Physiology | Degeneration | Bioindicators | Injury analysis | Neurochemistry | Head | Cytokines | Soccer | Glial fibrillary acidic protein | Health risks | Trauma | Pathology | Hospitals | Concussion | Biomarkers | Brain damage | Marathons | Alzheimers disease | Medical and Health Sciences | MEDICINE | Medicin och hälsovetenskap | MEDICIN | Nuclear magnetic resonance | NMR
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 06/2015, Volume 19, Issue 6, pp. 1234 - 1244
Six‐transmembrane epithelial antigen of prostate 4 (Steap4)‐knockout mice develop hyperglycaemia and inflammation whereas Steap4 overexpression attenuates... 
protein interaction | high glucose | S100B | Steap4 | Protein interaction | High glucose | PATHWAYS | MEDICINE, RESEARCH & EXPERIMENTAL | PROTEIN | QUANTITATIVE PROTEOMICS | SIGNALS | RECEPTOR | ALPHA | DIABETIC-NEPHROPATHY | CELL BIOLOGY | GENE | EXPRESSION | GLYCATION END-PRODUCTS | Transcription, Genetic - drug effects | Diabetes Mellitus, Experimental - genetics | Immunoblotting | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Phosphatidylinositol 3-Kinases - metabolism | S100 Calcium Binding Protein beta Subunit - pharmacology | Extracellular Signal-Regulated MAP Kinases - metabolism | Mesangial Cells - drug effects | Dose-Response Relationship, Drug | Time Factors | Janus Kinase 2 - metabolism | Protein Binding - drug effects | S100 Calcium Binding Protein beta Subunit - genetics | Cell Membrane - metabolism | Membrane Proteins - metabolism | S100 Calcium Binding Protein beta Subunit - metabolism | Diabetes Mellitus, Experimental - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Cell Membrane - drug effects | STAT3 Transcription Factor - metabolism | Cell Line | Membrane Proteins - genetics | Glucose - pharmacology | Mesangial Cells - metabolism | Fibronectins - metabolism | Mice, Inbred ICR | Animals | Transforming Growth Factor beta - genetics | Smad Proteins - metabolism | Transforming Growth Factor beta - metabolism | Fibronectins | COX-2 inhibitors | Hyperglycemia | Streptozocin | Collagen | Atherosclerosis | Bone morphogenetic proteins | Glucose | Transforming growth factors | Mass spectrometry | Protein-protein interactions | Dextrose | Antigens | Kidneys | Diabetes | Cellular biology | Membranes | Transforming growth factor | Collagens | S100b protein | AKT protein | Adipocytes | Kinases | Fibronectin | Prostaglandin endoperoxide synthase | Proteins | Rodents | Transcription activation | Mesangial cells | Attenuation | Collagen (type IV) | Growth factors | Immunoglobulins | Cytokines | Diabetes mellitus | Stat3 protein | Mass spectroscopy | Glycosylation | Cell membranes | Gene expression | Plasmids | Arteriosclerosis | Protein expression | Mice | Gifts | Mutation | Cyclooxygenase-2 | Prostate | Original
Journal Article
Colloids and Surfaces B: Biointerfaces, ISSN 0927-7765, 05/2016, Volume 141, pp. 558 - 564
Journal Article
Biochemistry, ISSN 0006-2960, 09/2012, Volume 51, Issue 36, pp. 7189 - 7201
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2010, Volume 285, Issue 35, pp. 27487 - 27498
The S100B-p53 protein complex was discovered in C8146A malignant melanoma, but the consequences of this interaction required further study. When S100B... 
WILD-TYPE P53 | UV-RADIATION | CANCER-CELLS | P53-MEDIATED APOPTOSIS | P53-INDUCED APOPTOSIS | CARCINOMA-CELLS | MDM2 GENE | DIFFERENTIAL EXPRESSION | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUMOR-SUPPRESSOR PROTEIN | PIFITHRIN-ALPHA | RNA, Small Interfering - genetics | Caspase 9 - genetics | Caspase 9 - metabolism | Inhibitor of Apoptosis Proteins - genetics | Membrane Potential, Mitochondrial - radiation effects | Humans | Caspase 3 - metabolism | Cell Survival - genetics | Multiprotein Complexes - genetics | Nerve Growth Factors - metabolism | fas Receptor - metabolism | Death Domain Receptor Signaling Adaptor Proteins | Multiprotein Complexes - metabolism | RNA, Messenger - biosynthesis | Phosphorylation - genetics | S100 Proteins - genetics | Ultraviolet Rays | Caspase 3 - genetics | fas Receptor - genetics | Biomarkers, Tumor - metabolism | Membrane Potential, Mitochondrial - genetics | Inhibitor of Apoptosis Proteins - metabolism | S100 Calcium Binding Protein beta Subunit | Down-Regulation | S100 Proteins - metabolism | Melanoma - pathology | Enzyme Activation - radiation effects | Phosphorylation - radiation effects | RNA, Neoplasm - biosynthesis | Nerve Growth Factors - genetics | Cell Line, Tumor | RNA, Neoplasm - genetics | Biomarkers, Tumor - genetics | Mutation | Transcription, Genetic - genetics | Enzyme Activation - genetics | Caspase 8 - metabolism | Gene Expression Regulation, Neoplastic | Transcription, Genetic - radiation effects | Cytochromes c - genetics | Tumor Suppressor Protein p53 - genetics | Caspase 8 - genetics | Melanoma - genetics | Melanoma - metabolism | Cytochromes c - metabolism | RNA, Messenger - genetics | Tumor Suppressor Protein p53 - metabolism | Cell Survival - radiation effects | Carrier Proteins - genetics | Carrier Proteins - metabolism | Apoptosis | Molecular Bases of Disease | S100 Proteins | Calcium-binding Proteins | S100B | Cell Biology | p53 | Protein-Protein Interactions | Tumor Suppressor
Journal Article
Acta Crystallographica Section F, ISSN 2053-230X, 04/2017, Volume 73, Issue 4, pp. 215 - 221
S100A1 is a member of the S100 family of Ca2+‐binding proteins and regulates several cellular processes, including those involved in Ca2+ signaling and cardiac... 
S100A1 | S100 | crystal structure | calcium‐binding proteins | S100B | cardiomyopathy | Alzheimer's disease | calcium-binding proteins | DYNAMIC PROPERTIES | SYSTEM | CA2+ RELEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | NUCLEAR-MAGNETIC-RESONANCE | BIOCHEMICAL RESEARCH METHODS | CRYSTALLOGRAPHY | CA2+-BINDING PROTEIN S100A1 | PEPTIDE | BIOPHYSICS | RYANODINE RECEPTOR | SMALL-MOLECULE INHIBITORS | CALMODULIN | BINDING-SITE | Calcium - metabolism | Humans | Protein Multimerization | Crystallography, X-Ray | Calcium - chemistry | S100 Proteins - chemistry | S100 Proteins - genetics | Protein Isoforms - metabolism | Protein Isoforms - chemistry | Cloning, Molecular | Escherichia coli - metabolism | S100 Calcium Binding Protein beta Subunit - genetics | Protein Interaction Domains and Motifs | S100 Calcium Binding Protein beta Subunit - metabolism | Binding Sites | Recombinant Proteins - metabolism | Amino Acid Sequence | Protein Conformation, alpha-Helical | Gene Expression | S100 Calcium Binding Protein beta Subunit - chemistry | Genetic Vectors - chemistry | Genetic Vectors - metabolism | Models, Molecular | Recombinant Proteins - chemistry | S100 Proteins - metabolism | Recombinant Proteins - genetics | Sequence Alignment | Cations, Divalent | Escherichia coli - genetics | Protein Binding | Structural Homology, Protein | Protein Isoforms - genetics | Proteins | Pathology | Brain | Progressions | Muscles | Biology | Crystal structure
Journal Article
Journal Article
Neurobiology of Aging, ISSN 0197-4580, 2008, Volume 31, Issue 4, pp. 578 - 590
Abstract Astrocyte pathology occurs in association with Alzheimer's disease (AD) and in brain ageing, but is poorly characterised. We sought to define the... 
Neurology | Internal Medicine | Aging | Alzheimer's type pathology | Excitatory amino acid transporter | Temporal cortex | Astrocyte | Glial fibrillary acidic protein | OXIDATIVE STRESS | AMYLOID PLAQUES | FIBROUS ASTROCYTES | NEUROSCIENCES | NEURODEGENERATIVE DISEASES | COGNITIVE FUNCTION | GERIATRICS & GERONTOLOGY | IN-VITRO | CEREBROSPINAL-FLUID S100B | SENILE-DEMENTIA | FIBRILLARY ACIDIC PROTEIN | GLUTAMATE TRANSPORTER | Immunohistochemistry | Predictive Value of Tests | Prospective Studies | Humans | Astrocytes - pathology | Nerve Tissue Proteins - analysis | tau Proteins - metabolism | Male | Gliosis - physiopathology | Nerve Growth Factors - metabolism | Glial Fibrillary Acidic Protein - metabolism | Alzheimer Disease - pathology | Brain - metabolism | Gliosis - pathology | Amyloid beta-Peptides - metabolism | Aged, 80 and over | Female | Biomarkers - metabolism | Severity of Illness Index | Alzheimer Disease - physiopathology | Plaque, Amyloid - pathology | S100 Calcium Binding Protein beta Subunit | Brain - physiopathology | Glial Fibrillary Acidic Protein - analysis | Gliosis - metabolism | Biomarkers - analysis | S100 Proteins - metabolism | Excitatory Amino Acid Transporter 1 - metabolism | Excitatory Amino Acid Transporter 2 - analysis | Excitatory Amino Acid Transporter 2 - metabolism | S100 Proteins - analysis | Nerve Growth Factors - analysis | Nerve Tissue Proteins - metabolism | Phenotype | Alzheimer Disease - metabolism | Amyloid beta-Peptides - analysis | Brain - pathology | Plaque, Amyloid - metabolism | Aged | tau Proteins - analysis | Excitatory Amino Acid Transporter 1 - analysis | Longitudinal Studies | Astrocytes - metabolism | Cohort Studies | Amino acids | Genetic aspects | Alzheimer's disease | Intermediate filament proteins | Public health
Journal Article