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PLoS ONE, ISSN 1932-6203, 03/2016, Volume 11, Issue 3, p. e0150893
Serum amyloid A (SAA) is an evolutionary highly conserved acute phase protein that is predominantly secreted by hepatocytes. However, its role in liver injury... 
APOPTOSIS | HUMAN NEUTROPHILS | ACUTE-PHASE REACTANT | RAT-LIVER FIBROSIS | MULTIDISCIPLINARY SCIENCES | SECRETION | MONOCYTE CHEMOTACTIC PROTEIN-1 | A SAA PROTEIN | NF-KAPPA-B | EXPRESSION | INTESTINAL EPITHELIAL-CELLS | Cholestasis - genetics | Cholestasis - metabolism | Serum Amyloid A Protein - pharmacology | Carbon Tetrachloride | Humans | Hepatic Stellate Cells - metabolism | Male | NF-kappa B - metabolism | Liver Cirrhosis - chemically induced | Proto-Oncogene Proteins c-akt - genetics | Matrix Metalloproteinase 9 - metabolism | Ligation | MAP Kinase Kinase 4 - metabolism | I-kappa B Kinase - metabolism | Matrix Metalloproteinase 9 - genetics | Mitogen-Activated Protein Kinase 1 - genetics | Liver Cirrhosis - metabolism | Chemokine CCL2 - metabolism | Chemokine CCL5 - metabolism | Cell Death - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Liver Cirrhosis - genetics | Hepatic Stellate Cells - pathology | Disease Models, Animal | Hepatic Stellate Cells - drug effects | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Gene Expression Regulation | Rats | Chemokine CCL2 - genetics | Inflammation | I-kappa B Kinase - genetics | Rats, Sprague-Dawley | Cholestasis - pathology | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | NF-kappa B - genetics | Chemokine CCL5 - genetics | Liver Cirrhosis - pathology | MAP Kinase Kinase 4 - genetics | Cell Proliferation - drug effects | Mice, Inbred BALB C | Primary Cell Culture | Mitogen-Activated Protein Kinase 1 - metabolism | Biochemistry | RNA | Cell death | Luciferase | Cell proliferation | Animal models | Transcription factors | Transcription | RANTES | Liver | Carbon tetrachloride | Cytotoxicity | AKT protein | Kinases | Caspase-3 | Proteins | Signal transduction | Immunology | Rodents | Hepatology | Gastroenterology | Fibroblasts | Amyloid | NF-κB protein | Stellate cells | Cytokines | Mortality | Neutrophils | Extracellular signal-regulated kinase | c-Jun protein | Caspase | Poly(ADP-ribose) polymerase | CCL4 protein | Bile duct | Gelatinase B | Medicine | Antibiotics | Hepatocytes | Surgeons | Skin | Annexin V | Monocyte chemoattractant protein 1 | Apoptosis
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2011, Volume 56, Issue 1, pp. 184 - 191
Background & Aims Mosaic G-protein alpha-subunit ( GNAS )-activating mutations are responsible for the McCune–Albright (MCA) syndrome. This oncogene that... 
Gastroenterology and Hepatology | CRP | interleukine 6 signal transducer | GTP | C-reactive protein | jun N-terminal kinase | serum amyloid A | JNK | macroregenerative nodule | complementary deoxyribonucleic acid | MRN | mitogen activated proteins kinase | cAMP | glycoprotein 130 | neuroendocrine secretory protein 55 | hepatocyte nuclear factor 1A | G-protein alpha stimulatory subunit | cDNA | gp130 | protein kinase A | hepatocellular carcinoma | β catenin | IHCA | McCune-Albright syndrome | HCA | PKA | HCC | MAP kinase | MCA | HNF1A | inflammatory hepatocellular adenoma | SAA | cyclic adenosine monophosphate | hepatocellular adenoma | GNAS | CTNNB1 | NESP55 | guanosine-5′-triphosphate | IL6ST | INACTIVATION | PATHWAY | HEPATOCELLULAR ADENOMA | GS-ALPHA | GROWTH | CLASSIFICATION | HUMAN CANCERS | MCCUNE-ALBRIGHT-SYNDROME | GASTROENTEROLOGY & HEPATOLOGY | G-PROTEIN | BINDING | Liver Neoplasms - classification | Humans | Middle Aged | Adenoma, Liver Cell - pathology | Male | Fibrous Dysplasia, Polyostotic - genetics | GTP-Binding Protein alpha Subunits, Gs - genetics | Chromogranins | DNA Mutational Analysis | Base Sequence | Carcinoma, Hepatocellular - genetics | Adult | Female | Liver Neoplasms - pathology | Adenoma, Liver Cell - genetics | STAT3 Transcription Factor - metabolism | Liver Neoplasms - genetics | Signal Transduction | Adenoma, Liver Cell - metabolism | Models, Biological | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Aged | DNA, Neoplasm - genetics | Mutation | Carcinoma, Hepatocellular - metabolism | Liver cancer | Genetic aspects | Nucleic acids | Adenylic acid | Mitogens | Protein kinases | Index Medicus | GTP-Binding Protein alpha Subunits, Gs | Fibrous Dysplasia, Polyostotic | Liver Neoplasms | Life Sciences | Adenoma, Liver Cell | DNA, Neoplasm | STAT3 Transcription Factor | Carcinoma, Hepatocellular | Cancer
Journal Article
Fish and Shellfish Immunology, ISSN 1050-4648, 06/2017, Volume 65, pp. 267 - 277
Journal Article
2012, Progress in Molecular Biology and Translational Science, ISBN 9780123945969, Volume 105, 38
Book Chapter
Developmental and Comparative Immunology, ISSN 0145-305X, 2001, Volume 25, Issue 8, pp. 725 - 743
Tissue trauma or invasion by pathogens or parasites induce changes in the quantities of several macromolecules in animal body fluids. These changes comprise... 
CRP | Acute phase protein | SAA | Transferrin | Pre-cerebellin-like protein | Acute phase response | Fibrinogen | Fish | Innate immunity | Inflammation | TCBP1 | acute phase response | C-REACTIVE PROTEIN | RAINBOW-TROUT | fibrinogen | AEROMONAS-SALMONICIDA | transferrin | IMMUNOLOGY | CATFISH ICTALURUS-PUNCTATUS | CARP CYPRINUS-CARPIO | PLEURONECTES-PLATESSA L | SERUM CONCENTRATIONS | ZOOLOGY | pre-cerebellin-like protein | innate immunity | inflammation | fish | AMYLOID-P-COMPONENT | TROUT ONCORHYNCHUS-MYKISS | acute phase protein | ATLANTIC SALMON | Serum Amyloid P-Component - immunology | alpha-Macroglobulins - physiology | Acute-Phase Reaction - immunology | alpha-Macroglobulins - immunology | alpha-Macroglobulins - genetics | Molecular Sequence Data | Serum Amyloid P-Component - physiology | Serum Amyloid A Protein - genetics | Transferrin - physiology | Acute-Phase Proteins - genetics | Transferrin - genetics | Acute-Phase Proteins - physiology | Fishes - immunology | Transferrin - immunology | Apolipoproteins - genetics | Fishes - physiology | Amino Acid Sequence | Fishes - genetics | Environmental Monitoring | Complement C3 - metabolism | C-Reactive Protein - genetics | Muramidase - metabolism | Apolipoproteins - immunology | C-Reactive Protein - physiology | Serum Amyloid A Protein - immunology | Sequence Homology, Amino Acid | Serum Amyloid P-Component - genetics | Animals | Apolipoproteins - physiology | C-Reactive Protein - immunology | Serum Amyloid A Protein - physiology | Acute-Phase Proteins - immunology
Journal Article