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Cancer Discovery, ISSN 2159-8274, 04/2017, Volume 7, Issue 4, pp. 400 - 409
Entrectinib, a potent oral inhibitor of the tyrosine kinases TRKA/B/C, ROS1, and ALK, was evaluated in two phase I studies in patients with advanced or... 
REARRANGEMENT | ONCOGENE | ONCOLOGY | ANALOG SECRETORY CARCINOMA | LANDSCAPE | KINASE FUSIONS | SARCOMAS | ETV6-NTRK3 GENE FUSION | CRIZOTINIB | GENOMIC ALTERATIONS | CLINICAL-RESPONSE | Benzamides - pharmacokinetics | Colorectal Neoplasms - genetics | Humans | Middle Aged | Receptor, trkA - antagonists & inhibitors | Male | Receptor, trkB - genetics | Indazoles - administration & dosage | Protein Kinase Inhibitors - adverse effects | Mammary Analogue Secretory Carcinoma - genetics | Dose-Response Relationship, Drug | Benzamides - administration & dosage | Membrane Glycoproteins - antagonists & inhibitors | Receptor, trkC - genetics | Anaplastic Lymphoma Kinase | Melanoma - genetics | Colorectal Neoplasms - drug therapy | Receptor, trkB - antagonists & inhibitors | Aged, 80 and over | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Adult | Female | Benzamides - adverse effects | Carcinoma, Non-Small-Cell Lung - pathology | Crizotinib | Protein Kinase Inhibitors - pharmacokinetics | Proto-Oncogene Proteins - antagonists & inhibitors | Pyridines - administration & dosage | Carcinoma, Non-Small-Cell Lung - genetics | Receptor, trkC - antagonists & inhibitors | Melanoma - pathology | Mammary Analogue Secretory Carcinoma - drug therapy | Membrane Glycoproteins - genetics | Protein Kinase Inhibitors - administration & dosage | Sequestosome-1 Protein - genetics | Pyrazoles - administration & dosage | Indazoles - pharmacokinetics | Receptor Protein-Tyrosine Kinases - genetics | Oncogene Proteins, Fusion - genetics | Melanoma - drug therapy | Adolescent | Receptor, trkA - genetics | Oncogene Proteins, Fusion - antagonists & inhibitors | Aged | Carcinoma, Non-Small-Cell Lung - drug therapy | Indazoles - adverse effects | Colorectal Neoplasms - pathology | Protein-Tyrosine Kinases - antagonists & inhibitors
Journal Article
Science, ISSN 0036-8075, 8/2010, Volume 329, Issue 5993, pp. 805 - 810
Therapeutic efforts to restore biosynthetic processing of the cystic fibrosis transmembrane conductance regulator lacking the F508 residue (ΔF508CFTR) are... 
Cellular metabolism | RESEARCH ARTICLES | Internalization | Small interfering RNA | HeLa cells | Immunoblotting | Cystic fibrosis | Cell membranes | Biosynthesis | Recycling | Physiological regulation | TRANSMEMBRANE CONDUCTANCE REGULATOR | CFTR-DELTA-F508 | CELLS | CHIP | RETICULUM-ASSOCIATED DEGRADATION | MULTIDISCIPLINARY SCIENCES | ENDOPLASMIC-RETICULUM | CYSTIC-FIBROSIS | HSC70 | E3 UBIQUITIN LIGASE | SECRETORY PATHWAY | Temperature | Molecular Chaperones - metabolism | HSC70 Heat-Shock Proteins - metabolism | Humans | Homeostasis | Ubiquitination | Lysosomes - metabolism | Cell Membrane - metabolism | Protein Stability | Ubiquitinated Proteins - metabolism | Recombinant Proteins - metabolism | HSC70 Heat-Shock Proteins - genetics | Ubiquitinated Proteins - chemistry | Ubiquitin-Protein Ligases - metabolism | Mutant Proteins - metabolism | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Glycosylation | Cystic Fibrosis Transmembrane Conductance Regulator - chemistry | Protein Folding | Protein Transport | Mutant Proteins - chemistry | HSP90 Heat-Shock Proteins - metabolism | Cystic Fibrosis Transmembrane Conductance Regulator - genetics | Protein Conformation | RNA, Small Interfering | HeLa Cells | Ubiquitin-Protein Ligases - genetics | Physiological aspects | Research | Membrane proteins | Proteins | Plasma | Membranes | Cellular biology
Journal Article
2017, Methods in molecular biology, ISBN 9781493972616, Volume 1662
Web Resource
2017, Methods in molecular biology, ISBN 9781493972616, Volume 1662
Web Resource
Journal Article
Journal Article
The FEBS Journal, ISSN 1742-464X, 01/2017, Volume 284, Issue 1, pp. 56 - 76
Apoptosis‐linked gene 2 ( ALG ‐2), which is a gene product of PDCD 6 , is a 22‐ kD a Ca 2+ ‐binding protein. Accumulating evidence points to a role for ALG ‐2... 
protein–protein interaction | Trk‐fused gene (TFG) protein | endoplasmic reticulum exit sites | ALG‐2 | binding protein | Trk-fused gene (TFG) protein | ALG-2 | CA2+-BINDING PROTEINS | COILED-COIL DOMAIN | ALTERNATIVELY SPLICED ISOFORM | BIOCHEMISTRY & MOLECULAR BIOLOGY | EARLY SECRETORY PATHWAY | TARGET RECOGNITION | Ca2+-binding protein | CA2+-DEPENDENT MANNER | MEMBRANE-FUSION | ESCRT-I | INTERACTS | ANNEXIN-XI | protein-protein interaction | Calcium - metabolism | Humans | Protein Multimerization | Endoplasmic Reticulum - metabolism | Green Fluorescent Proteins - genetics | Recombinant Fusion Proteins - metabolism | Time-Lapse Imaging | Endoplasmic Reticulum - drug effects | Escherichia coli - metabolism | HEK293 Cells | Apoptosis Regulatory Proteins - genetics | Binding Sites | Calcium-Binding Proteins - metabolism | Green Fluorescent Proteins - metabolism | Signal Transduction | Enzyme Inhibitors - pharmacology | Gene Expression Regulation | COP-Coated Vesicles - metabolism | Apoptosis Regulatory Proteins - metabolism | Proteins - genetics | Proteins - metabolism | Escherichia coli - genetics | Protein Binding | Recombinant Fusion Proteins - genetics | Thapsigargin - pharmacology | Luminescent Proteins - genetics | HeLa Cells | Calcium-Binding Proteins - genetics | Luminescent Proteins - metabolism | Genes | Genetic research | Fluorescence | Polymerization | Binding proteins | Calmodulin | Protein-protein interactions | Protein binding | Proteins | Calcium | Endoplasmic reticulum | Rodents | Calcium (intracellular) | Trafficking | Accumulation | Thapsigargin | Clonal deletion | Calcium-binding protein | Green fluorescent protein | Deletion | Protein transport | Localization | Calcium ions | Apoptosis
Journal Article
Protein Science, ISSN 0961-8368, 11/2017, Volume 26, Issue 11, pp. 2291 - 2301
Journal Article