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British Journal of Pharmacology, ISSN 0007-1188, 01/2017, Volume 174, Issue 2, pp. 119 - 125
Zn2+, the second most prevalent trace element in the body, is essential for supporting a wide range of biological functions. While the majority of Zn2+ in the... 
VESICULAR ZINC | INHIBITION | TEMPORAL-LOBE EPILEPSY | CONCISE GUIDE | NMDA RECEPTORS | MOUSE | SERUM ZINC LEVELS | PHARMACOLOGY & PHARMACY | CONVULSIONS | ION CHANNELS | PHARMACOLOGY 2015/16 | Disease susceptibility | Seizures (Medicine) | Protein binding | Seizing | Brain | Animal models | Excitability | Homeostasis | Cerebrospinal fluid | Zinc | Proteins | Synaptic vesicles | Trace elements | Children | Supplementation | Seizures | Review
Journal Article
The Journal of neuroscience : the official journal of the Society for Neuroscience, ISSN 0270-6474, 11/2018, Volume 38, Issue 47, pp. 10220 - 10235
Synaptic AMPAR expression controls the strength of excitatory synaptic transmission and plasticity. An excess of synaptic AMPARs leads to epilepsy in response... 
epilepsy | Glua | Seizure | GluA, seizure | AMPAR | palmitoylation | seizure | HIPPOCAMPAL HYPEREXCITABILITY | POSTTRANSLATIONAL MODIFICATIONS | SYNAPTIC PLASTICITY | NEUROSCIENCES | GluA | DENDRITIC SPINES | GLUTAMATE RECEPTORS | RECEPTOR TRAFFICKING | LONG-TERM POTENTIATION | PROTEIN PALMITOYLATION | TRANSGENIC MICE
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 05/2019, Volume 176, Issue 10, pp. 1506 - 1523
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 10/2017, Volume 37, Issue 43, pp. 10278 - 10289
Creutzfeldt-Jakob disease (CJD) is a neurodegenerative disorder caused by prion protein (PrP) misfolding, clinically recognized by cognitive and motor... 
Prion | IL-1 receptor antagonist | Creutzfeldt-Jakob disease | Synaptic plasticity | Transgenic mice | Seizures | HIPPOCAMPAL SLICES | TEMPORAL-LOBE EPILEPSY | STATUS EPILEPTICUS | NEUROSCIENCES | CELLULAR PRION PROTEIN | transgenic mice | prion | seizures | RAT HIPPOCAMPUS | synaptic plasticity | LONG-TERM POTENTIATION | KAINATE-INDUCED SEIZURES | INFLAMMATORY CYTOKINES | NEURONAL EXCITABILITY | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Synaptic Transmission - physiology | Humans | Receptors, N-Methyl-D-Aspartate - metabolism | Seizures - drug therapy | Neuronal Plasticity - drug effects | Male | Seizures - metabolism | Hippocampus - drug effects | N-Methylaspartate - antagonists & inhibitors | Neuronal Plasticity - physiology | Interleukin-1beta - metabolism | Female | Synaptic Transmission - drug effects | Interleukin 1 Receptor Antagonist Protein - pharmacology | Disease Models, Animal | Interleukin-1beta - antagonists & inhibitors | Disease Susceptibility | N-Methylaspartate - metabolism | Mice, Inbred C57BL | Mice, Transgenic | Random Allocation | Mice, Knockout | Creutzfeldt-Jakob Syndrome - metabolism | Hippocampus - metabolism | Creutzfeldt-Jakob Syndrome - drug therapy | Animals | Interleukin 1 Receptor Antagonist Protein - therapeutic use | Signal Transduction - drug effects | Signal Transduction - physiology | Mice | Creutzfeldt–Jakob disease
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 07/2008, Volume 28, Issue 27, pp. 6904 - 6913
There are critical postnatal periods during which even subtle interventions can have long-lasting effects on adult physiology. We asked whether an immune... 
Development | Lipopolysaccharide | Seizure | Interleukin-1β | Tumor necrosis factor α | Cytokine | cytokine | development | ACID-INDUCED SEIZURES | seizure | TEMPORAL-LOBE EPILEPSY | NECROSIS-FACTOR-ALPHA | KAINIC ACID | FEBRILE SEIZURES | LIFE IMMUNE CHALLENGE | NEUROSCIENCES | interleukin-1 beta | tumor necrosis factor alpha | STEREOLOGICAL ESTIMATION | EPILEPTIFORM ACTIVITY | RECEPTOR TRAFFICKING | TNF-ALPHA | lipopolysaccharide | Electric Stimulation | Age Factors | Injections, Intraventricular | Nerve Degeneration - physiopathology | Gliosis - chemically induced | Male | Gliosis - physiopathology | Time | Epilepsy - physiopathology | Nerve Degeneration - chemically induced | Lipopolysaccharides | Afferent Pathways - immunology | Disease Susceptibility - immunology | Epilepsy - immunology | Tumor Necrosis Factor-alpha - immunology | Afferent Pathways - drug effects | Pyramidal Cells - drug effects | Gliosis - immunology | Hippocampus - growth & development | Action Potentials - drug effects | Organ Culture Techniques | Potassium Channel Blockers - pharmacology | Animals, Newborn | Hippocampus - immunology | Rats | Encephalitis - immunology | Nerve Degeneration - immunology | Rats, Sprague-Dawley | Action Potentials - physiology | Tumor Necrosis Factor-alpha - pharmacology | Animals | Pyramidal Cells - immunology | Convulsants - pharmacology | Encephalitis - chemically induced | Hippocampus - physiopathology | Index Medicus | tumor necrosis factor α | interleukin-1β
Journal Article
Nature Reviews Neuroscience, ISSN 1471-003X, 05/2014, Volume 15, Issue 5, pp. 283 - 283
Journal Article
Journal Article
Neuroscience, ISSN 0306-4522, 2009, Volume 159, Issue 2, pp. 842 - 849
Journal Article
The Journal of Neuroscience, ISSN 0270-6474, 11/2018, Volume 38, Issue 47, p. 10220
Synaptic AMPAR expression controls the strength of excitatory synaptic transmission and plasticity. An excess of synaptic AMPARs leads to epilepsy in response... 
Seizing | Brain | Membranes | Phosphorylation | Stability | Spine | Palmitoylation | Epilepsy | Trafficking | Long-term potentiation | α-Amino-3-hydroxy-5-methyl-4-isoxazole propionic acid receptors | Proteins | Receptors | Convulsions & seizures | Control | Plasticity (synaptic) | Synaptic transmission | Rodents | Cerebrum | Mice | Protein transport | Synapses | Seizures | Enlargement
Journal Article
Journal Article