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Diabetes/Metabolism Research and Reviews, ISSN 1520-7552, 05/2019, Volume 35, Issue 4, pp. e3129 - n/a
Background To evaluate the association between fasting plasma glucose (FPG) and mortality by gender. Methods A total of 17 248 eligible participants from a... 
mortality | cohort study | fasting plasma glucose | sex specific | EVENTS | BLOOD-GLUCOSE | FOLLOW-UP | DEATH | LEVEL | RISK-FACTOR | DISEASE | ENDOCRINOLOGY & METABOLISM | CARDIOVASCULAR MORTALITY | HYPOGLYCEMIA | WOMENS HEALTH | Diabetics | Low density lipoproteins | Analysis | Mortality | Diabetes | Glucose | Health aspects | Dextrose | Fasting | Regression analysis | Diabetes mellitus
Journal Article
Scientific Reports, ISSN 2045-2322, 01/2016, Volume 6, Issue 1, p. 19079
This study was to determine the association between several single nucleotide polymorphisms (SNPs) in the dedicator of cytokinesis 7 (DOCK7), proprotein... 
SEX-SPECIFIC ASSOCIATION | RISK-FACTORS | LIFE-STYLE | VARIANTS | MULTIDISCIPLINARY SCIENCES | CARDIOVASCULAR-DISEASE | DIETARY INTERVENTION | ALCOHOL-CONSUMPTION | HAN POPULATIONS | MONOGENIC DYSLIPIDEMIAS | ENVIRONMENTAL-FACTORS | Haplotypes | Dyslipidemias - genetics | Cardiovascular Diseases - ethnology | Dyslipidemias - complications | Dyslipidemias - ethnology | Humans | Middle Aged | Male | Cardiovascular Diseases - genetics | Dyslipidemias - blood | Ethnic Groups | China | N-Acetylgalactosaminyltransferases - genetics | Proprotein Convertase 9 - blood | Cardiovascular Diseases - blood | Adult | Cholesterol, LDL - blood | Female | Lipid Metabolism - genetics | Cardiovascular Diseases - etiology | N-Acetylgalactosaminyltransferases - blood | Genome-Wide Association Study | Gene Frequency | Risk Factors | GTPase-Activating Proteins - blood | Gene Expression Regulation | Proprotein Convertase 9 - genetics | Linkage Disequilibrium | Alleles | Triglycerides - blood | Cholesterol, HDL - blood | Aged | Polymorphism, Single Nucleotide | GTPase-Activating Proteins - genetics | Lipoproteins (low density) | N-acetylgalactosaminyltransferase | Lipids | Subtilisin | Single-nucleotide polymorphism | Polypeptide N-acetylgalactosaminyltransferase | Cytokinesis | Apolipoproteins | Low density lipoprotein | Cholesterol | Kexin | Genotyping | Gene frequency | Lipoproteins (high density) | Polymorphism
Journal Article
Genes and Immunity, ISSN 1466-4879, 07/2010, Volume 11, Issue 5, pp. 406 - 415
The single nucleotide polymorphism 1858C> T in the PTPN22 gene is associated with type 1 diabetes (T1D) in several populations. Earlier reports have suggested... 
RHEUMATOID-ARTHRITIS | CAUCASIAN POPULATIONS | insulin autoantibodies | GLUTAMIC-ACID DECARBOXYLASE | ICA | IA-2 autoantibodies | SINGLE-NUCLEOTIDE POLYMORPHISM | IMMUNOLOGY | autoimmune disease | islet cell antigen | SEX-SPECIFIC ASSOCIATION | C1858T POLYMORPHISM | R620W FUNCTIONAL VARIANT | AUTOIMMUNE-DISEASES | GENETICS & HEREDITY | islet cell autoantibodies | ISLET-CELL-ANTIBODIES | PROTEIN-TYROSINE-PHOSPHATASE | Autoantibodies - metabolism | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Age Factors | Risk Assessment | Humans | Protein Tyrosine Phosphatase, Non-Receptor Type 22 - genetics | Child, Preschool | Diabetes Mellitus, Type 1 - genetics | Genotype | Infant | Male | HLA Antigens - genetics | Case-Control Studies | Autoantibodies - immunology | Sweden | Adolescent | Polymorphism, Single Nucleotide - genetics | Adult | Female | Glutamate Decarboxylase - immunology | Odds Ratio | Child | Infant, Newborn | Autoantibodies | Histocompatibility antigens | Type 1 diabetes | HLA histocompatibility antigens | Physiological aspects | Genetic aspects | Research | Single nucleotide polymorphisms | Risk factors | Autoimmune disease | Medical and Health Sciences | Medicin och hälsovetenskap | islet | Klinisk medicin | cell autoantibodies | Clinical Medicine | Endokrinologi och diabetes | Endocrinology and Diabetes
Journal Article
Journal Article
Diabetologia, ISSN 0012-186X, 1/2019, Volume 62, Issue 1, pp. 199 - 200
Journal Article
Environment International, ISSN 0160-4120, 02/2016, Volume 87, pp. 56 - 65
Brain growth and structural organization occurs in stages beginning prenatally. Toxicants may impact neurodevelopment differently dependent upon exposure... 
Neurodevelopment | Sensitive windows | Air pollution | Sex-specific associations | Particulate matter | Prenatal exposure | OXIDATIVE STRESS | POLYCYCLIC AROMATIC-HYDROCARBONS | BEHAVIOR | VULNERABILITY | POSTNATAL EXPOSURE | NONLINEAR MODELS | ENVIRONMENTAL SCIENCES | ATTENTION-DEFICIT/HYPERACTIVITY DISORDER | MEMORY | BRAIN | LIFE | Memory - drug effects | Particulate Matter - toxicity | Air Pollutants - analysis | Prenatal Exposure Delayed Effects - epidemiology | United States | Humans | Child, Preschool | Male | Urban Population | Neurodevelopmental Disorders - chemically induced | Attention - drug effects | Maternal Exposure - adverse effects | Female | Child Development - drug effects | Child | Models, Theoretical | Neurodevelopmental Disorders - epidemiology | Neurodevelopmental Disorders - physiopathology | Prenatal Exposure Delayed Effects - physiopathology | Gestational Age | Pregnancy | Particulate Matter - analysis | Sex Factors | Intelligence - drug effects | Air Pollutants - toxicity | Prenatal Exposure Delayed Effects - chemically induced | Smoking | Environmental aspects | Pregnant women | Index Medicus | Errors | Education | Sex | Mathematical models | Exposure | Children | Time measurements | Age | particulate matter | prenatal exposure | air pollution | sex-specific associations | neurodevelopment | sensitive windows
Journal Article