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PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, ISSN 0027-8424, 02/2011, Volume 108, Issue 5, pp. 1919 - 1924
Epstein-Barr virus (EBV) nuclear antigen 3C (EBNA3C) and EBNA3A are each essential for EBV conversion of primary human B lymphocytes into continuously... 
B-LYMPHOCYTES | SIGNAL-BINDING-PROTEIN | HUMAN-PAPILLOMAVIRUS TYPE-16 | cell cycle | RBP-J-KAPPA | MULTIDISCIPLINARY SCIENCES | lymphoma | Notch | PROTHYMOSIN-ALPHA | CYCLIN-A | DEPENDENT KINASE-ACTIVITY | TRANSFORMATION IN-VITRO | polycomb protein | DEMETHYLASE JMJD3 CONTRIBUTES | HISTONE DEACETYLASE | CtBP
Journal Article
Journal of Virology, ISSN 0022-538X, 05/2018, Volume 92, Issue 9
Epstein-Barr virus nuclear antigen (EBNA) leader protein (EBNALP) is one of the first viral genes expressed upon B-cell infection. EBNALP is essential for... 
Coactivation | EBNALP | EP300 | Epstein-Barr virus | Transcription factor | transcription factor | SIGNAL-BINDING-PROTEIN | GENETIC-ANALYSIS | coactivation | P300 | SARCOMA-ASSOCIATED HERPESVIRUS | LYMPHOBLASTOID CELL-LINES | TRANSCRIPTIONAL COACTIVATION | NUCLEAR-PROTEIN-2 ACIDIC DOMAIN | VIROLOGY | EBNA-LP | NF-KAPPA-B | LYMPHOCYTE-TRANSFORMATION
Journal Article
Nature Genetics, ISSN 1061-4036, 05/2018, Volume 50, Issue 5, pp. 699 - 707
Explaining the genetics of many diseases is challenging because most associations localize to incompletely characterized regulatory regions. Using new... 
REED-STERNBERG CELLS | SYSTEMIC-LUPUS-ERYTHEMATOSUS | RISK LOCI | SIGNAL-BINDING-PROTEIN | GENETICS & HEREDITY | BARR-VIRUS INFECTION | MOLECULAR MIMICRY | NF-KAPPA-B | HUMAN GENE-EXPRESSION | TRAIT LOCI | GENOME-WIDE ASSOCIATION | Epstein-Barr Virus Nuclear Antigens - immunology | Cell Line | Herpesvirus 4, Human - genetics | Epstein-Barr Virus Nuclear Antigens - genetics | Humans | Regulatory Sequences, Nucleic Acid | Viral Proteins - immunology | Autoimmunity - genetics | Viral Proteins - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Maximum Tolerated Dose | Phenotype | Neoplasms - genetics | Lupus Erythematosus, Systemic - genetics | Cell Line, Tumor | Protein Binding | Autoimmunity | Multiple sclerosis | Genes | Arthritis | B cells | DNA binding proteins | Gene expression | Rheumatoid factor | Systemic lupus erythematosus | Analysis | Genetic aspects | Epstein-Barr virus | Prostate cancer | Transcription factors | Inflammatory bowel diseases | Disease | Downstream effects | Viruses | Genomes | Diabetes mellitus (insulin dependent) | Regulatory sequences | Datasets | Proteins | Consortia | Intestine | Genetics | Deoxyribonucleic acid--DNA | Binding | Lupus | Phenotypes | Nominations | Diabetes mellitus | Health risks | Breast cancer | Loci | Celiac disease | Chronic conditions | Androgens | Rheumatoid arthritis | Computer applications | GATA-3 protein | Lymphomas | Autoimmune diseases | Prostate | Cancer
Journal Article
Cell Host & Microbe, ISSN 1931-3128, 10/2017, Volume 22, Issue 4, pp. 561 - 573.e4
Epstein-Barr virus (EBV) transforms B cells to continuously proliferating lymphoblastoid cell lines (LCLs), which represent an experimental model for... 
EBV regulome | CDKN2A/B | Epstein-Barr virus | ChIA-PET | MYC | EBV super-enhancers | TRANSCRIPTION FACTORS | SIGNAL-BINDING-PROTEIN | C-MYC | MICROBIOLOGY | HUMAN B-LYMPHOCYTES | KAPPA-B | VIROLOGY | SUPER-ENHANCERS | NUCLEAR-PROTEIN EBNA3C | BURKITTS-LYMPHOMA | GROWTH | GENE-EXPRESSION | PARASITOLOGY | Chromatin | B cells | Gene expression | Analysis | Antigens | Pets | Anopheles | Microbiology | Genes | Genomics | DNA binding proteins
Journal Article
Seminars in Cancer Biology, ISSN 1044-579X, 12/2002, Volume 12, Issue 6, pp. 431 - 441
Journal Article
Journal of Virology, ISSN 0022-538X, 10/2017, Volume 91, Issue 20
Epstein-Barr virus (EBV) latency and its associated carcinogenesis are regulated by dynamic changes in DNA methylation of both virus and host genomes. We show... 
DNA methylation | EBV | Epigenetics | TET2 | EBNA2 | Epstein-Barr virus | Hydroxymethylation | Epigenome | TRANSFORMATION | epigenome | SIGNAL-BINDING-PROTEIN | RBP-J-KAPPA | TRANSCRIPTION | epigenetics | hydroxymethylation | 5-METHYLCYTOSINE OXIDATION | VIROLOGY | CELL SELF-RENEWAL | TRANSACTIVATION | TARGETS | LYMPHOMA | PROMOTER
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2011, Volume 108, Issue 36, pp. 14902 - 14907
Epstein-Barr virus nuclear antigen 2 (EBNA2) regulation of transcription through the cell transcription factor RBPJ is essential for resting B-lymphocyte (RBL)... 
T lymphocytes | Up regulation | Chromatin | B lymphocytes | DNA | Nucleosomes | Gene expression regulation | Epstein Barr virus infections | Human herpesvirus 4 | Binding sites | Development | Lymphoma | Leukemia | Notch | leukemia | development | SIGNAL-BINDING-PROTEIN | CIS-ELEMENT | MULTIDISCIPLINARY SCIENCES | lymphoma | C-MYC | NUCLEAR ANTIGEN-2 | FATE | GENOME | LONG-RANGE INTERACTION | GENE | PROMOTER | J-KAPPA | Cell Proliferation | Herpesvirus 4, Human - genetics | Proto-Oncogene Proteins c-ets - genetics | Genome, Viral - genetics | Humans | Transcription Factor RelA - genetics | Viral Proteins - metabolism | Trans-Activators - genetics | B-Lymphocytes - virology | Epstein-Barr Virus Infections - genetics | Core Binding Factor alpha Subunits - metabolism | Transcription, Genetic | Proto-Oncogene Proteins c-ets - metabolism | Epstein-Barr Virus Nuclear Antigens - metabolism | Immunoglobulin J Recombination Signal Sequence-Binding Protein - genetics | B-Lymphocytes - metabolism | Proto-Oncogene Proteins - metabolism | Immunoglobulin J Recombination Signal Sequence-Binding Protein - metabolism | Epstein-Barr Virus Nuclear Antigens - genetics | Response Elements | Nucleosomes - metabolism | Viral Proteins - genetics | Nucleosomes - genetics | Proto-Oncogene Proteins - genetics | Proto-Oncogene Proteins c-myc - metabolism | Core Binding Factor alpha Subunits - genetics | Transcription Factor RelA - metabolism | Cell Line, Tumor | Trans-Activators - metabolism | Proto-Oncogene Proteins c-myc - genetics | Herpesvirus 4, Human - metabolism | Epstein-Barr Virus Infections - metabolism | Physiological aspects | Transcription factors | Epstein-Barr virus | Genetic aspects | Lymphocytes | Health aspects | Cell growth | Deoxyribonucleic acid--DNA | Biological Sciences
Journal Article
BLOOD, ISSN 0006-4971, 02/2008, Volume 111, Issue 4, pp. 2280 - 2289
The oncogene latent membrane protein 1 (LMP1) of Epstein-Barr virus (EBV) without a ligand drives proliferation of EBV-infected B cells. Its levels vary in... 
SIGNAL-BINDING-PROTEIN | RBP-J-KAPPA | TRANSCRIPTION FACTOR XBP-1 | RETICULUM-RESIDENT KINASE | ENDOPLASMIC-RETICULUM | HEMATOLOGY | PLASMA-CELL DIFFERENTIATION | NF-KAPPA-B | EPSTEIN-BARR-VIRUS | LATENT MEMBRANE PROTEIN-1 | LYMPHOCYTE GROWTH TRANSFORMATION
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2017, Volume 140, Issue 4, pp. 1079 - 1089
Background Allergic asthma is characterized by a TH 2 response induced by dendritic cells (DCs) that present inhaled allergen. Although the mechanisms by which... 
Allergy and Immunology | Allergic asthma | house dust mite | Notch signaling | TH2 immunity | Gata-3 | recombination signal–binding protein for immunoglobulin Jκ region | cytokines | Jagged | TH1 immunity | T | 2 immunity | 1 immunity | TH2 IMMUNITY | RESPONSIVENESS | recombination signal-binding protein for immunoglobulin J(k) region | T(H)1 immunity | INDUCTION | IMMUNOLOGY | T(H)2 immunity | HOUSE-DUST MITE | RESPONSES | MEDIATED-IMMUNITY | FOLLICULAR-HELPER-CELLS | ALLERGY | GATA3 EXPRESSION | TYPE-2 IMMUNITY | DIFFERENTIATION | Jagged-1 Protein - metabolism | Signal Transduction | Receptors, Notch - metabolism | Dendritic Cells - immunology | Humans | Mice, Inbred C57BL | Cells, Cultured | Hypersensitivity - immunology | Male | Th2 Cells - immunology | Jagged-1 Protein - genetics | Jagged-2 Protein - genetics | Mice, Knockout | Antigens, Dermatophagoides - immunology | Animals | Female | Mice | Disease Models, Animal | Jagged-2 Protein - metabolism | Pyroglyphidae - immunology | Allergens | Dendritic cells | T cells | Asthma | Analysis | Albumin | Inflammation | Protein binding | Recombination | Downstream effects | Lymphocytes T | Respiratory tract | Signal transduction | Cell activation | Clonal deletion | Lymphocytes | Rodents | Deletion | Respiratory tract diseases | Antigens | Cytokines | Leukocytes (eosinophilic) | Hypersensitivity | House dust | T cell receptors | Allergies | Ligands | In vivo methods and tests | Notch protein
Journal Article