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Journal of Clinical Investigation, ISSN 0021-9738, 12/2012, Volume 122, Issue 12, pp. 4621 - 4634
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2013, Volume 110, Issue 34, pp. 13839 - 13844
The adult mammalian heart has limited potential for regeneration. Thus, after injury, cardiomyocytes are permanently lost, and contractility is diminished. In... 
Heart | Regeneration | Transcription factors | Ligation | Alleles | Fibrosis | Myocardium | Photoreceptors | Left coronary artery | Cardiac output | Cardiac fibrosis | Cardiomyopathy | Cell cycle | TRANSCRIPTION FACTORS | F-ACTIN | cell cycle | ORGAN SIZE CONTROL | MULTIDISCIPLINARY SCIENCES | CARDIOMYOCYTE PROLIFERATION | ZEBRAFISH HEART REGENERATION | MIR-15 FAMILY | TEAD/TEF FAMILY | cardiac fibrosis | SIGNALING PATHWAY | IN-VIVO | cardiomyopathy | HOLT-ORAM-SYNDROME | Echocardiography | Heart - physiology | Myocardial Contraction - physiology | Mice, Transgenic | DNA Primers - genetics | Phosphoproteins - genetics | Transcription Factors - genetics | Phosphoproteins - metabolism | Regeneration - physiology | Blotting, Western | Mutation, Missense - genetics | Tetrazolium Salts | Transcription Factors - metabolism | Myocardial Contraction - genetics | Animals | Histological Techniques | Myocytes, Cardiac - physiology | Adaptor Proteins, Signal Transducing - genetics | Myocytes, Cardiac - metabolism | Signal Transduction - physiology | Mice | Adaptor Proteins, Signal Transducing - metabolism | Protein-Serine-Threonine Kinases - metabolism | Proteins | Molecular genetics | Heart cells | Cellular control mechanisms | Physiological aspects | Genetic aspects | Regeneration (Biology) | Research | Index Medicus | Biological Sciences
Journal Article
Circulation Research, ISSN 0009-7330, 10/2012, Volume 111, Issue 9, pp. 1125 - 1136
Journal Article
Nature, ISSN 0028-0836, 01/2018, Volume 553, Issue 7686, pp. 96 - 100
Journal Article
Journal Article
Nature, ISSN 0028-0836, 07/2011, Volume 475, Issue 7354, pp. 106 - 110
Reactive oxygen species (ROS) are mutagenic and may thereby promote cancer(1). Normally, ROS levels are tightly controlled by an inducible antioxidant program... 
TRANSFORMATION | OXIDATIVE STRESS | ACTIVATION | INHIBITION | K-RAS | PATHWAY | MULTIDISCIPLINARY SCIENCES | PANCREATIC-CANCER | HEME OXYGENASE-1 | MUTATIONS | EXPRESSION | NIH 3T3 Cells | Cell Proliferation | Pancreatic Neoplasms - metabolism | Reactive Oxygen Species - metabolism | Cytoskeletal Proteins - genetics | Proto-Oncogene Proteins p21(ras) - genetics | Antioxidants - metabolism | Humans | JNK Mitogen-Activated Protein Kinases - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | MAP Kinase Signaling System | Mitogen-Activated Protein Kinase Kinases - metabolism | Cell Transformation, Neoplastic - genetics | Cytoskeletal Proteins - metabolism | NF-E2-Related Factor 2 - genetics | Intracellular Signaling Peptides and Proteins - genetics | Kelch-Like ECH-Associated Protein 1 | Proto-Oncogene Proteins B-raf - metabolism | Fibroblasts - metabolism | Proto-Oncogene Proteins p21(ras) - metabolism | Oncogenes - genetics | Oxidation-Reduction | Pancreatic Neoplasms - pathology | Cells, Cultured | Pancreatic Neoplasms - genetics | NF-E2-Related Factor 2 - deficiency | Cell Transformation, Neoplastic - metabolism | Animals | Proto-Oncogene Proteins B-raf - genetics | Genes, myc - genetics | NF-E2-Related Factor 2 - metabolism | Adaptor Proteins, Signal Transducing - genetics | Alleles | Cell Line, Tumor | Mice | Adaptor Proteins, Signal Transducing - metabolism | Cell Transformation, Neoplastic - pathology | Polymerase chain reaction | Usage | Reactive oxygen species | Physiological aspects | Research | Gene expression | Oncogenes | Studies | Mass spectrometry | Rodents | Evacuations & rescues | Cancer
Journal Article
by Zhou, Q and Wang, X and Yu, Z and Wu, X and Chen, X and Li, J and Zhu, Z and Liu, B and Su, L
Oncogene, ISSN 0950-9232, 03/2017, Volume 36, Issue 13, pp. 1873 - 1886
Journal Article