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Nature, ISSN 0028-0836, 02/2018, Volume 554, Issue 7692, pp. 373 - 377
Both microbial and host genetic factors contribute to the pathogenesis of autoimmune diseases(1-4). There is accumulating evidence that microbial species that... 
INTESTINAL INFLAMMATION | RESPONSES | IMMUNITY | MULTIDISCIPLINARY SCIENCES | IN-VIVO | DISEASE | HUMAN MICROBIOTA | MICE | DIFFERENTIATION | EXPRESSION | ANTIGEN | Bioengineering | T-Lymphocytes, Regulatory - metabolism | Nuclear Receptor Subfamily 1, Group F, Member 3 - antagonists & inhibitors | Homeostasis | Colitis - pathology | Male | Intestines - immunology | T-Lymphocytes, Regulatory - immunology | Forkhead Transcription Factors - metabolism | Helicobacter hepaticus - pathogenicity | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | T-Lymphocytes, Regulatory - cytology | Female | Th17 Cells - cytology | Colitis - immunology | Helicobacter hepaticus - genetics | Interleukin-10 - deficiency | Helicobacter hepaticus - immunology | Immune Tolerance | Host-Pathogen Interactions | Animals | Colitis - microbiology | Intestines - microbiology | Proto-Oncogene Proteins c-maf - deficiency | Th17 Cells - immunology | Interleukin-10 - biosynthesis | Proto-Oncogene Proteins c-maf - metabolism | Mice | Interleukin-10 - immunology | Host-bacteria relationships | Inflammatory bowel diseases | Helicobacter infections | Physiological research | Research | T cells | Immunological tolerance | Pathogenesis | Helper cells | Lymphocytes T | Inactivation | Small intestine | Accumulation | Microbiota | Microorganisms | Immunology | Lymphocytes | Rodents | Bacteria | Foxp3 protein | Genetic factors | Deactivation | Immunoregulation | T cell receptors | c-Maf protein | Inflammatory bowel disease | Large intestine | Interleukin 10 | Colitis | Autoimmune diseases | Differentiation | Colonization
Journal Article
Cell Cycle, ISSN 1538-4101, 06/2016, Volume 15, Issue 11, pp. 1405 - 1409
Journal Article
Gene, ISSN 0378-1119, 06/2018, Volume 660, pp. 51 - 61
The small Mafs, MafF, MafG and MafK play critical roles in morphogenesis and homeostasis through associating with Cap “n” Collar family of transcription... 
Microcystin | Cristaria plicata | Small Mafs | Sigma-class glutathione S-Transferases1, 2 | High-affinity | RNA | Glutathione transferase | Escherichia coli | Animal genetic engineering | Genetic aspects | DNA binding proteins | Genetic transcription | Recombinant proteins | Index Medicus
Journal Article
Blood, ISSN 0006-4971, 2009, Volume 113, Issue 18, pp. 4309 - 4318
CS1 is highly expressed on tumor cells from the majority of multiple myeloma (MM) patients regardless of cytogenetic abnormalities or response to current... 
CD2 | IMMUNOGLOBULIN SUPERFAMILY | OVEREXPRESSION | BIOLOGY | ANTIBODY | PROLIFERATION | HEMATOLOGY | CYTOTOXICITY | EXPRESSION | BIOCONDUCTOR | LYMPHOCYTES | Cell Membrane | Phosphorylation | Cell Proliferation | Oligonucleotide Array Sequence Analysis | Stromal Cells - pathology | Humans | Transcriptional Activation | Immunoblotting | Gene Expression Profiling | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Flow Cytometry | Transfection | Bone Marrow - metabolism | Antigens, Surface - metabolism | Biomarkers, Tumor - metabolism | Lentivirus - genetics | Colony-Forming Units Assay | Tumor Cells, Cultured | Culture Media, Serum-Free - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | STAT3 Transcription Factor - metabolism | Enzyme-Linked Immunosorbent Assay | Cell Survival | Proto-Oncogene Proteins c-maf - genetics | RNA, Small Interfering - pharmacology | Stromal Cells - metabolism | Antigens, Surface - genetics | Antigens, Surface - chemistry | Mice, SCID | Multiple Myeloma - metabolism | Xenograft Model Antitumor Assays | Multiple Myeloma - pathology | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Cell Adhesion - physiology | Bone Marrow - pathology | Proto-Oncogene Proteins c-maf - metabolism | Biomarkers, Tumor - genetics | Mice | Multiple Myeloma - prevention & control | Mitogen-Activated Protein Kinase 1 - metabolism | Lymphoid Neoplasia
Journal Article
European Journal of Immunology, ISSN 0014-2980, 04/2010, Volume 40, Issue 4, pp. 1174 - 1184
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 01/2011, Volume 31, Issue 1, pp. 151 - 162
Journal Article
Cancer Science, ISSN 1347-9032, 04/2011, Volume 102, Issue 4, pp. 890 - 894
Latent infection of human T‐cell leukemia virus type 1 (HTLV‐1) is considered to be preferentially associated with CCR4 +  CD4 + T cells. Here we report that... 
TRANSFORMATION | TRANSCRIPTIONAL ACTIVITY | HTLV-I | ACTIVATION | POLYMERASE-CHAIN-REACTION | ONCOLOGY | GENE-REGULATION | CHEMOKINE | INFECTION | EXPRESSION | LYMPHOCYTES | RNA, Small Interfering - genetics | Luciferases - metabolism | Gene Products, tax - antagonists & inhibitors | Humans | Transcriptional Activation | Retroviridae Proteins, Oncogenic - metabolism | Gene Products, tax - genetics | Th2 Cells | Gene Products, env - metabolism | CREB-Binding Protein - genetics | CREB-Binding Protein - metabolism | Receptors, CCR4 - metabolism | Gene Products, tax - metabolism | HTLV-I Infections - metabolism | Virion | Gene Products, env - genetics | Human T-lymphotropic virus 1 - genetics | CD4-Positive T-Lymphocytes - virology | HTLV-I Infections - genetics | Promoter Regions, Genetic | Jurkat Cells | Leukemia-Lymphoma, Adult T-Cell - genetics | Proto-Oncogene Proteins c-maf - genetics | RNA, Messenger - genetics | Receptors, CCR4 - genetics | CD4-Positive T-Lymphocytes - metabolism | Leukemia-Lymphoma, Adult T-Cell - virology | Reverse Transcriptase Polymerase Chain Reaction | HTLV-I Infections - virology | Blotting, Western | Leukemia-Lymphoma, Adult T-Cell - metabolism | Human T-lymphotropic virus 1 - metabolism | Proto-Oncogene Proteins c-maf - metabolism | Retroviridae Proteins, Oncogenic - genetics | Proto-Oncogene Proteins c-maf - antagonists & inhibitors | Cell Transformation, Viral | Leukemia | Universities and colleges | T cells | Gene expression | Binding proteins | Health aspects | Medicine, Preventive | Preventive health services
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