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International Journal of Radiation Oncology, Biology, Physics, ISSN 0360-3016, 11/2011, Volume 81, Issue 3, pp. 698 - 705
Purpose Our hypothesis was that pretreatment inflammation in the lung makes pulmonary tissue more susceptible to radiation damage. The relationship between... 
Radiology | Hematology, Oncology and Palliative Medicine | Radiation-induced lung toxicity (RILT) | Positron emission tomography (PET) | Pneumonitis | Dyspnea | Fluorodeoxyglucose (FDG) | Lung | MANAGEMENT | TISSUE DOSE CONSTRAINTS | INDUCED PNEUMONITIS | DISORDERS | RISK | CHEMOTHERAPY | POSITRON-EMISSION-TOMOGRAPHY | PRESCRIPTION | ONCOLOGY | INFECTION | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | PET | Carcinoma, Non-Small-Cell Lung - radiotherapy | Fluorodeoxyglucose F18 - pharmacokinetics | Age Factors | Humans | Lung Neoplasms - metabolism | Middle Aged | Lung Neoplasms - radiotherapy | Male | Tomography, X-Ray Computed | Dyspnea - etiology | Lung - diagnostic imaging | Positron-Emission Tomography | Chemoradiotherapy | Radiation Pneumonitis - metabolism | Multimodal Imaging - methods | Aged, 80 and over | Adult | Female | Lung - radiation effects | Lung - metabolism | Retrospective Studies | Odds Ratio | Radiopharmaceuticals - pharmacokinetics | Carcinoma, Non-Small-Cell Lung - metabolism | Radiation Pneumonitis - diagnostic imaging | Logistic Models | Lung Neoplasms - diagnostic imaging | Aged | Carcinoma, Non-Small-Cell Lung - diagnostic imaging | Cancer patients | Lung cancer, Non-small cell | Radiotherapy | Developmental biology | Risk factors | Radiation | Toxicity | Lung cancer | Tomography | Inflammation | Respiration | Age | Tumors | Index Medicus | TOMOGRAPHY | HOURS LIVING RADIOISOTOPES | EMISSION COMPUTED TOMOGRAPHY | LUNGS | RADIATION EFFECTS | FLUORODEOXYGLUCOSE | RADIATION DOSES | MEDICINE | CAT SCANNING | ANTIMETABOLITES | FLUORINE ISOTOPES | THERAPY | NANOSECONDS LIVING RADIOISOTOPES | POSITRON COMPUTED TOMOGRAPHY | HYPOTHESIS | DRUGS | BETA DECAY RADIOISOTOPES | RADIOLOGY AND NUCLEAR MEDICINE | PNEUMONITIS | RADIOTHERAPY | LIGHT NUCLEI | RADIOLOGY | BETA-PLUS DECAY RADIOISOTOPES | DIAGNOSTIC TECHNIQUES | NEOPLASMS | ISOMERIC TRANSITION ISOTOPES | RADIOISOTOPES | TOXICITY | SYMPTOMS | COMPUTERIZED TOMOGRAPHY | NUCLEAR MEDICINE | ISOTOPES | ORGANS | DISEASES | NUCLEI | DOSES | RESPIRATORY SYSTEM | INFLAMMATION | ODD-ODD NUCLEI | BODY | PATHOLOGICAL CHANGES | FLUORINE 18 | HAZARDS
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, pp. e60317 - e60317
Aberrant expression of microRNA-146a (miR-146a) has been reported to be involved in the development and progression of various types of cancers. However, its... 
SUPPRESSOR | ONCOGENESIS | RNA | MULTIDISCIPLINARY SCIENCES | CERVICAL-CANCER | INTERFERENCE | MICRORNAS | NF-KAPPA-B | FACTOR RECEPTOR | EXPRESSION | CONTRIBUTES | Lung Neoplasms - drug therapy | Humans | Lung Neoplasms - metabolism | Middle Aged | Gene Expression Regulation, Neoplastic | Lung Neoplasms - pathology | Male | MicroRNAs - metabolism | NF-kappa B - metabolism | Receptor, Epidermal Growth Factor - metabolism | Antibodies, Monoclonal, Humanized - pharmacology | Aged, 80 and over | Adult | Female | Antineoplastic Agents - pharmacology | Lung - metabolism | Cetuximab | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Lung - pathology | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Lung - drug effects | Cell Line, Tumor | Aged | Cell Proliferation - drug effects | MicroRNAs - genetics | Carcinoma, Non-Small-Cell Lung - drug therapy | Apoptosis | Cell Movement | MicroRNA | Growth | Monoclonal antibodies | Lung cancer, Small cell | Development and progression | Lung cancer, Non-small cell | Health aspects | Cell proliferation | Laboratories | Lung | Lung cancer | Oncology | Drug delivery | Kinases | Paraffin | Cancer therapies | Metastases | Signal transduction | Cell growth | Bioindicators | Inhibition | Gefitinib | Sensitizing | NF-κB protein | Phenotypes | Cytokines | Epidermal growth factor receptors | MiRNA | Non-small cell lung carcinoma | siRNA | Breast cancer | Gene expression | Ribonucleic acid--RNA | Patients | Pathology | Signaling | Chemotherapy | Thyroid cancer | MicroRNAs | Medical prognosis | Pancreatic cancer | Cell lines | Biomarkers | Mutation | Cell migration | Cervical cancer | Tumors | Cancer | Index Medicus | Ribonucleic acid
Journal Article
Journal Article
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 2015, Volume 6, Issue 1, pp. 6377 - 6377
Tyrosine kinase inhibitors are effective treatments for non-small-cell lung cancers (NSCLCs) with epidermal growth factor receptor (EGFR) mutations. However,... 
GROWTH-FACTOR RECEPTOR | DRUG-SENSITIVITY | GEFITINIB | AMPLIFICATION | MODELS | GENE | MULTIDISCIPLINARY SCIENCES | MUTATION | ACQUIRED-RESISTANCE | KINASE INHIBITORS | CHEMOTHERAPY | Lung Neoplasms - drug therapy | Recurrence | Adenocarcinoma - pathology | Adenocarcinoma of Lung | Humans | Lung Neoplasms - metabolism | ErbB Receptors - genetics | Gene Expression Regulation, Neoplastic | bcl-X Protein - genetics | Lung Neoplasms - pathology | Small Cell Lung Carcinoma - drug therapy | Small Cell Lung Carcinoma - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Adenocarcinoma - metabolism | Adenocarcinoma - genetics | Antineoplastic Agents - pharmacology | Lung - metabolism | Gefitinib | ErbB Receptors - deficiency | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Lung - pathology | Aniline Compounds - pharmacology | ErbB Receptors - antagonists & inhibitors | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Retinoblastoma Protein - deficiency | Small Cell Lung Carcinoma - genetics | Sulfonamides - pharmacology | Adenocarcinoma - drug therapy | Disease Progression | Drug Resistance, Neoplasm - genetics | Small Cell Lung Carcinoma - pathology | Afatinib | Retinoblastoma Protein - genetics | Erlotinib Hydrochloride - pharmacology | Lung - drug effects | Cell Line, Tumor | Protein Kinase Inhibitors - pharmacology | Carcinoma, Non-Small-Cell Lung - drug therapy | Mutation | Quinazolines - pharmacology | bcl-X Protein - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | Drug Resistance, Neoplasm - drug effects | Index Medicus
Journal Article
2005, Hematology/oncology clinics of North America, ISBN 9781416027737, Volume 19, no. 2., xii, p. 209-426
Book
PLoS ONE, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, pp. e35065 - e35065
Background: Lysine specific demethylase 1 (LSD1) has been identified and biochemically characterized in epigenetics, but the pathological roles of its... 
BREAST-CANCER | HISTONE DEMETHYLASES | PROGRAMS | BIOLOGY | Immunohistochemistry | RNA, Small Interfering - genetics | Histone Demethylases - antagonists & inhibitors | Prognosis | Humans | Lung Neoplasms - metabolism | Middle Aged | Gene Expression Regulation, Neoplastic | Lung Neoplasms - pathology | Male | Epithelial-Mesenchymal Transition - genetics | Histone Demethylases - genetics | Cell Movement - genetics | Adult | Female | Lung - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Lung - pathology | Carcinoma, Non-Small-Cell Lung - genetics | Neoplasm Invasiveness | Carcinoma, Non-Small-Cell Lung - metabolism | Survival Rate | Carcinoma, Non-Small-Cell Lung - mortality | Cell Movement - drug effects | Lung - drug effects | Cell Line, Tumor | Pargyline - pharmacology | Aged | Biomarkers, Tumor - genetics | Cell Proliferation - drug effects | Epigenetic inheritance | RNA | Analysis | Oncology, Experimental | Lung cancer, Small cell | Research | Lung cancer, Non-small cell | T cells | Health aspects | Cancer | Cell proliferation | Mesenchyme | Lung | Lung cancer | Biology | mRNA | Statistical methods | Metastasis | Neuroblastoma | Assaying | Cancer therapies | Nuclei | Phase transitions | Western blotting | Cell cycle | DNA methylation | Medical research | Enzymes | Statistical analysis | Cell survival | Non-small cell lung carcinoma | siRNA | Breast cancer | Gene expression | Biological assays | Patients | Medicine | Studies | Androgens | Overexpression | Lysine | Epigenetics | Nuclei (cytology) | Immunofluorescence | Pargyline | Prostate cancer | Cell migration | Index Medicus
Journal Article
Journal of Thoracic Oncology, ISSN 1556-0864, 09/2011, Volume 6, Issue 9, pp. 1496 - 1504
Journal Article