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Journal of allergy and clinical immunology, ISSN 0091-6749, 2015, Volume 136, Issue 3, pp. 769 - 780
Background Inflammation and oxidative stress play critical roles in patients with chronic obstructive pulmonary disease (COPD). Mitochondrial oxidative stress... 
Allergy and Immunology | airway hyperresponsiveness | antioxidant | proliferation | MitoQ | inflammation | mitochondria | airway smooth muscle | Ozone | oxidative stress | chronic obstructive pulmonary disease | ASTHMA FEATURES | MECHANISMS | IMMUNOLOGY | FRAGMENTATION | HYPERRESPONSIVENESS | BETA | RESPONSES | ALLERGY | ENERGY-METABOLISM | EXPRESSION | COPD | Respiratory System - pathology | Reactive Oxygen Species - metabolism | Humans | Middle Aged | Male | Pulmonary Disease, Chronic Obstructive - pathology | Bronchial Hyperreactivity - genetics | Ubiquinone - pharmacology | Muscle, Smooth - drug effects | Smoking - physiopathology | Organophosphorus Compounds - pharmacology | Myocytes, Smooth Muscle - drug effects | Pneumonia - genetics | Pulmonary Disease, Chronic Obstructive - genetics | Bronchial Hyperreactivity - chemically induced | Signal Transduction | Mitochondria - pathology | Smoking - metabolism | Bronchial Hyperreactivity - pathology | Reactive Oxygen Species - antagonists & inhibitors | Electron Transport Chain Complex Proteins - metabolism | Pulmonary Disease, Chronic Obstructive - chemically induced | Respiratory System - drug effects | Airway Remodeling - genetics | Mice | Oxidative Stress - drug effects | Bronchial Hyperreactivity - drug therapy | Myocytes, Smooth Muscle - pathology | Pneumonia - pathology | Membrane Potential, Mitochondrial - drug effects | Pneumonia - chemically induced | Adult | Female | Pulmonary Disease, Chronic Obstructive - metabolism | Myocytes, Smooth Muscle - metabolism | Ubiquinone - analogs & derivatives | Gene Expression Regulation | Hydrogen Peroxide - pharmacology | Electron Transport Chain Complex Proteins - genetics | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - drug effects | Muscle, Smooth - metabolism | Animals | Pneumonia - drug therapy | Respiratory System - metabolism | Aged | Muscle, Smooth - pathology | Oxidative stress | Care and treatment | Lung diseases, Obstructive | Inflammation | Pharmaceutical industry | Biomedical research | Disease | Laboratories | Mortality | Colleges & universities | Smooth muscle | Mitochondrial DNA | Metabolism | Defects | Antioxidants | Mitochondria | Hospitals | Biopsy | Chronic obstructive pulmonary disease | Apoptosis | Smoking | NO, Nitric oxide | ΔΨm, Mitochondrial membrane potential | ASM, Airway smooth muscle | NAC, N-acetylcysteine | RL, Lung resistance | BAL, Bronchoalveolar lavage | OCR, Oxygen consumption rate | dTPP, Decyltriphenylphosphonium bromide | AHR, Airway hyperresponsiveness | logPC100, Concentration of acetylcholine that increased lung resistance by 100 | GOLD, Global Initiative for Chronic Obstructive Lung Disease | ATP, Adenosine triphosphate | KC, Keratinocyte-derived cytokine | JC-1, 5,5′,6,6′-Tetrachloro-1,1′,3,3′-tetraethylbenzimidazolylcarbocyanine iodide | COPD, Chronic obstructive pulmonary disease | ROS, Reactive oxygen species | Mechanisms of Allergy and Clinical Immunology
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2013, Volume 33, Issue 1, pp. 67 - 75
Objective-Aldosterone (Aldo) is involved in arterial stiffness and heart failure, but the mechanisms have remained unclear. Galectin-3 (Gal-3), a... 
collagen type I | aldosterone | HEART-FAILURE | LEFT-VENTRICULAR DYSFUNCTION | INFLAMMATION | ENDOTHELIAL-CELLS | fibrosis | SMOOTH-MUSCLE-CELLS | EXTRACELLULAR-MATRIX | MICE | galectin-3 | vascular smooth muscle cells | EXPRESSION | INSIGHTS | CARDIOTROPHIN-1 | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Blood Pressure | Inflammation - chemically induced | Inflammation - pathology | Up-Regulation | Muscle, Smooth, Vascular - metabolism | Rats, Wistar | Humans | Galectin 3 - metabolism | Myocytes, Smooth Muscle - pathology | Male | Aldosterone | Muscle, Smooth, Vascular - physiopathology | Galectin 3 - deficiency | Inflammation - metabolism | Transfection | RNA Interference | Time Factors | Hypertension - chemically induced | Mineralocorticoid Receptor Antagonists - pharmacology | Hypertension - prevention & control | Hypertension - genetics | Myocytes, Smooth Muscle - drug effects | Vascular Stiffness | Myocytes, Smooth Muscle - metabolism | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Mice, Inbred C57BL | Cells, Cultured | Rats | Galectin 3 - genetics | Hypertension - pathology | Hypertension - physiopathology | Hypertension - metabolism | Mice, Knockout | Collagen Type I - biosynthesis | Muscle, Smooth, Vascular - pathology | Animals | Galectin 3 - antagonists & inhibitors | Fibrosis | Inflammation - genetics | Inflammation - prevention & control | Mice | Inflammation - physiopathology | Cellular Biology | Life Sciences | Muscle, Smooth, Vascular | Mineralocorticoid Receptor Antagonists | Hypertension | Hematology | Biochemistry, Molecular Biology | Collagen Type I | Inflammation | Human health and pathology | Myocytes, Smooth Muscle | Galectin 3
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 02/2014, Volume 34, Issue 2, pp. 355 - 364
OBJECTIVE—Vascular remodeling occurs after endothelial injury, resulting in smooth muscle cell (SMC) proliferation and vascular fibrosis. We previously... 
placental growth factor | receptors mineralocorticoid | vascular endothelial growth factor receptor-1 | aldosterone | myocytes smooth muscle | MORTALITY | OXIDATIVE STRESS | EVENTS | receptors | smooth muscle | myocytes | mineralocorticoid | BLOOD-PRESSURE | SPIRONOLACTONE | PERIPHERAL VASCULAR DISEASE | EPLERENONE | HYPERTENSION | HEMATOLOGY | EXPRESSION | BLOCKER | GENE-TRANSCRIPTION | Carotid Arteries - drug effects | Carotid Arteries - metabolism | Receptors, Mineralocorticoid - agonists | Receptors, Mineralocorticoid - genetics | Vascular Endothelial Growth Factor Receptor-1 - antagonists & inhibitors | Muscle, Smooth, Vascular - metabolism | Pregnancy Proteins - genetics | Myocytes, Smooth Muscle - pathology | Male | Pregnancy Proteins - metabolism | RNA, Messenger - metabolism | Receptors, Mineralocorticoid - deficiency | Time Factors | Carotid Artery Injuries - genetics | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Carotid Artery Injuries - metabolism | Carotid Artery Injuries - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Endothelial Cells - metabolism | Mice, Inbred C57BL | Aldosterone - pharmacology | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Mice, Knockout | Antibodies - pharmacology | Muscle, Smooth, Vascular - pathology | Animals | Fibrosis | Placenta Growth Factor | Carotid Arteries - pathology | Cell Proliferation - drug effects | Mice | Endothelial Cells - pathology | Endothelial Cells - drug effects | smooth muscle cells | mineralocorticoid receptor | vascular remodeling | VEGF
Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 2017, Volume 139, Issue 3, pp. 780 - 789
Journal Article
PloS one, ISSN 1932-6203, 02/2016, Volume 11, Issue 2, p. e0148657
Serotonergic anorexigens are the primary pharmacologic risk factor associated with pulmonary arterial hypertension (PAH), and the resulting PAH is clinically... 
CROSS-TALK | CELLS | SMOOTH-MUSCLE | CONTRACTION | SRC | TRANSGENIC MOUSE | MULTIDISCIPLINARY SCIENCES | DISEASE | 5-HYDROXYTRYPTAMINE TRANSPORTER GENE | KNOCKOUT MICE | 5-HT2B RECEPTORS | Phosphorylation | Oligonucleotide Array Sequence Analysis | Cytoskeletal Proteins - genetics | Receptor, Serotonin, 5-HT2B - metabolism | Myocytes, Smooth Muscle - pathology | Gene Expression Profiling | Hypertension, Pulmonary - prevention & control | Receptor, Serotonin, 5-HT2B - genetics | Urea - analogs & derivatives | Muscle Proteins - metabolism | src-Family Kinases - metabolism | Cytoskeletal Proteins - metabolism | Indoles - pharmacology | Lung - metabolism | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Lung - pathology | Bone Morphogenetic Protein Receptors, Type II - genetics | Signal Transduction | src-Family Kinases - antagonists & inhibitors | Bone Morphogenetic Protein Receptors, Type II - deficiency | Gene Expression Regulation | Hypertension, Pulmonary - genetics | Mice, Transgenic | Hypertension, Pulmonary - metabolism | Serotonin Antagonists - pharmacology | Protein Transport | Muscle Proteins - genetics | Cell Movement - drug effects | Animals | Muscle Contraction - drug effects | Lung - drug effects | Vascular Stiffness - drug effects | Mice | Mutation | src-Family Kinases - genetics | Hypertension, Pulmonary - pathology | Urea - pharmacology | Prevention | Serotonin | Physiological aspects | Genetic aspects | Research | Gene expression | Pulmonary hypertension | Atomic force microscopy | Disease | Critical care | Smooth muscle | Antagonists | Activation | Kinases | Contraction | Risk factors | Recruitment | Engineering | Rodents | Stiffness | Inhibition | Protein-tyrosine kinase | Biomedical engineering | Animal care | Hypertension | Tyrosine | Bone morphogenetic protein receptor type II | Blood vessels | Principal components analysis | Pharmacology | Inflammation | Muscle contraction | Medicine | Microscopy | Lungs | Cytoskeleton | Aberration
Journal Article
Circulation research, ISSN 1524-4571, 2017, Volume 121, Issue 3, pp. 220 - 233
Rationale: Mitochondrial changes occur during cell differentiation and cardiovascular disease. DRP1 (dynamin-related protein 1) is a key regulator of... 
atherosclerosis | collagen | myocytes, smooth muscle | mitochondria | oxidative stress | INFANTILE ENCEPHALOPATHY | CARDIAC & CARDIOVASCULAR SYSTEMS | ATHEROSCLEROTIC PLAQUES | VASCULAR CALCIFICATION | smooth muscle | OSTEOBLASTIC DIFFERENTIATION | MATRIX VESICLES | SMOOTH-MUSCLE-CELL | MAMMALIAN-CELLS | myocytes | MITOCHONDRIAL FISSION | RETICULUM EXIT SITES | PERIPHERAL VASCULAR DISEASE | ENDOPLASMIC-RETICULUM | HEMATOLOGY | Carotid Artery Diseases - prevention & control | RNA, Small Interfering - genetics | Humans | Oxidative Stress - physiology | Myocytes, Smooth Muscle - pathology | Male | GTP Phosphohydrolases - antagonists & inhibitors | Mice, 129 Strain | Vascular Calcification - metabolism | Carotid Artery Diseases - pathology | Myocytes, Smooth Muscle - drug effects | Carotid Artery Diseases - metabolism | Myocytes, Smooth Muscle - metabolism | Mice, Inbred C57BL | Mitochondrial Proteins - antagonists & inhibitors | Cells, Cultured | Vascular Calcification - prevention & control | Mice, Transgenic | Mitochondrial Proteins - biosynthesis | Microtubule-Associated Proteins - antagonists & inhibitors | Collagen - secretion | Microtubule-Associated Proteins - biosynthesis | Vascular Calcification - pathology | Animals | Mice | Oxidative Stress - drug effects | RNA, Small Interfering - administration & dosage | GTP Phosphohydrolases - biosynthesis | Calcification (ectopic) | Immunohistochemistry | Oxidative stress | Alkaline phosphatase | Femur | Calcium | Smooth muscle | Osteoblasts | Proteins | Mitochondria | Clonal deletion | Mineralization | Aorta | Bone density | Carotid artery | Bone loss | RNA-mediated interference | Exploration | Subtilisin | Kexin | Arteriosclerosis | Collagen | Dynamin | Calcification | Cytoskeleton | Cardiovascular diseases | aortic valve calcification | Mechanisms | Oxidant Stress | Vascular Biology | dynamin-related protein 1 | calcification | DNM1L | Basic Science Research
Journal Article
Circulation research, ISSN 0009-7330, 08/2008, Volume 103, Issue 5, pp. e28 - e34
Journal Article
Journal Article