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Cancer Science, ISSN 1347-9032, 08/2017, Volume 108, Issue 8, pp. 1584 - 1593
Chemokine ( CC motif) ligand 18 ( CCL 18) is involved in remodeling of the tumor microenvironment and plays critical roles in oncogenesis, invasiveness, and... 
epithelial–mesenchymal transition | OSCC | CSC | chemokine | motif) ligand 18 | 18 | oral squamous cell carcinoma | Slug | Cancer stem(‐like) cell | CCL | EMT | chemokine (CC motif) ligand 18 (CCL18) | epithelial–mesenchymal transition (EMT) | oral squamous cell carcinoma (OSCC) | Cancer stem(-like) cell (CSC) | MIGRATION | DIAGNOSIS | HEAD | BREAST-CANCER METASTASIS | CCL18 PROMOTES | MARKERS | EPITHELIAL-MESENCHYMAL TRANSITION | epithelial-mesenchymal transition (EMT) | INVASION | ONCOLOGY | SIGNALING PATHWAY | Up-Regulation | Snail Family Transcription Factors - metabolism | Signal Transduction | TOR Serine-Threonine Kinases - metabolism | Neoplasm Invasiveness | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Mouth Neoplasms - metabolism | Chemokines, CC - genetics | Epithelial-Mesenchymal Transition | Cell Line, Tumor | Female | Aged | Mouth Neoplasms - genetics | Cell Movement | Chemokines, CC - metabolism | Immunohistochemistry | Squamous cell carcinoma | Metastasis | Analysis | Stem cells | TOR protein | Transcription factors | Mesenchyme | Kinases | Medical diagnosis | Cancer therapies | Metastases | Cell adhesion & migration | Proteins | Signal transduction | CCL18 protein | Tumorigenesis | Invasiveness | RNA-mediated interference | Rapamycin | Breast cancer | Oral squamous cell carcinoma | Cell lines | Ligands | Chemokines | Cell migration | Tumors | Index Medicus | Cancer stem(‐like) cell (CSC) | Original
Journal Article
The Prostate, ISSN 0270-4137, 06/2015, Volume 75, Issue 9, pp. 957 - 968
BACKGROUNDMetastasis is the primary cause of prostate cancer (PCa) lethality and poses a huge clinical obstacle. Lipocalin 2 (LCN2), a member of the lipocalin... 
LCN2 | prostate cancer | SLUG | EMT | ERK | CARCINOMA-CELLS | NEUTROPHIL GELATINASE | GELATINASE-ASSOCIATED LIPOCALIN | EPITHELIAL-MESENCHYMAL TRANSITIONS | E-CADHERIN | BREAST-CANCER | TUMOR-METASTASIS | ENDOCRINOLOGY & METABOLISM | UROLOGY & NEPHROLOGY | UP-REGULATION | PROGRESSION | SNAIL | Lipocalins - biosynthesis | Nitriles - pharmacology | Humans | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Male | Acute-Phase Proteins - genetics | Extracellular Signal-Regulated MAP Kinases - metabolism | Proto-Oncogene Proteins - biosynthesis | Cell Movement - physiology | MAP Kinase Signaling System | RNA - genetics | Heterografts | Prostatic Neoplasms - genetics | Lipocalins - blood | Lipocalins - genetics | Epithelial-Mesenchymal Transition | Prostatic Neoplasms - blood | Snail Family Transcription Factors | Prostatic Neoplasms - pathology | Butadienes - pharmacology | Neoplasm Invasiveness | Enzyme Inhibitors - pharmacology | Proto-Oncogene Proteins - genetics | Transcription Factors - biosynthesis | Transcription Factors - genetics | RNA - chemistry | Reverse Transcriptase Polymerase Chain Reaction | Animals | Lipocalin-2 | Mice, Nude | Histocytochemistry | Cell Line, Tumor | Mice | Proto-Oncogene Proteins - blood | Acute-Phase Proteins - biosynthesis | Development and progression | Prostate cancer | Index Medicus
Journal Article
Genes and Development, ISSN 0890-9369, 03/2008, Volume 22, Issue 6, pp. 756 - 769
Expression of Snail1 in epithelial cells triggers an epithelial-mesenchymal transition (EMT). Here, we demonstrate that the synthesis of Zeb2, a... 
NAT | Zeb2/Sip1 | IRES | EMT | Snail1 | RNA | INITIATION | SMAD-INTERACTING PROTEIN-1 | TUMOR-CELLS | DEVELOPMENTAL BIOLOGY | E-CADHERIN | TRANSLATION | CELL BIOLOGY | SIP1 | HUMAN GENOME | GENETICS & HEREDITY | REPRESSOR | SNAIL | RNA-Binding Proteins - genetics | Adenocarcinoma - pathology | Cadherins - metabolism | Homeodomain Proteins - metabolism | Humans | Gene Expression Regulation, Neoplastic | RNA, Messenger - metabolism | Zinc Finger E-box Binding Homeobox 2 | Case-Control Studies | Colonic Neoplasms - metabolism | Adenocarcinoma - metabolism | Colon | Cadherins - genetics | Snail Family Transcription Factors | Repressor Proteins - metabolism | Cell Line | Promoter Regions, Genetic | Zinc Fingers | RNA, Messenger - genetics | Cells, Cultured | Repressor Proteins - genetics | DNA Primers | Reverse Transcriptase Polymerase Chain Reaction | Nerve Tissue Proteins - genetics | Blotting, Western | Homeodomain Proteins - genetics | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | 5' Untranslated Regions | Plasmids | Colonic Neoplasms - pathology | Mesoderm - physiology | Epithelium - physiology | RNA, Antisense - physiology | RNA-Binding Proteins - metabolism | Synthesis | Epithelial cells | Zebra fish | Analysis | Physiological aspects | Research | Gene expression | Index Medicus | Sip1 | Zeb2 | Research Paper
Journal Article
Human Pathology, ISSN 0046-8177, 2011, Volume 42, Issue 4, pp. 482 - 488
Journal Article
Journal Article
Oncogene, ISSN 0950-9232, 02/2016, Volume 35, Issue 8, pp. 1049 - 1057
Journal Article
Cellular Signalling, ISSN 0898-6568, 12/2018, Volume 52, pp. 83 - 94
Tetraspanin membrane proteins form physical complexes with signaling molecules and have been suggested to influence the signaling events of associated... 
β-catenin | CD82 | Snail | Sp1 | E-cadherin | Cell adhesion | beta-catenin | CANCER-CELLS | POTENTIAL TARGET | CELLS IN-VITRO | TUMOR-CELLS | EPITHELIAL-MESENCHYMAL TRANSITIONS | CELL BIOLOGY | ADHESION | PROSTATE-CANCER | TRANSCRIPTIONAL REGULATION | KAI1 | METASTASIS SUPPRESSOR GENE
Journal Article
Journal Article
Molecular Cancer Research, ISSN 1541-7786, 12/2012, Volume 10, Issue 12, pp. 1597 - 1606
To understand the mechanisms leading to trastuzumab resistance in HER2-overexpressing breast tumors, we created trastuzumab-insensitive cell lines (SKBR3/100-8... 
MIGRATION | STEM-CELLS | INVASION | GROWTH-FACTOR-RECEPTOR | MECHANISM | LIGANDS | ONCOLOGY | HERCEPTIN | MESENCHYMAL TRANSITION | BETA-CATENIN | CONTRIBUTES | CELL BIOLOGY | Receptor, Epidermal Growth Factor - genetics | Cadherins - metabolism | Receptor, ErbB-2 - genetics | Humans | Transcriptional Activation | Receptor, ErbB-2 - metabolism | Drug Resistance, Neoplasm | Cadherins - biosynthesis | Wnt3 Protein - genetics | Breast Neoplasms - metabolism | Wnt3 Protein - biosynthesis | beta Catenin - biosynthesis | Breast Neoplasms - enzymology | Receptor, Epidermal Growth Factor - metabolism | Nuclear Proteins - biosynthesis | Antibodies, Monoclonal, Humanized - pharmacology | Female | Wnt3 Protein - metabolism | Twist-Related Protein 1 - biosynthesis | Cadherins - genetics | Nuclear Proteins - genetics | Snail Family Transcription Factors | Wnt Signaling Pathway | Receptor, Epidermal Growth Factor - biosynthesis | Receptor, ErbB-2 - biosynthesis | Organic Cation Transport Proteins - metabolism | Nuclear Proteins - metabolism | Transcription Factors - biosynthesis | Transcription Factors - genetics | Organic Cation Transport Proteins - biosynthesis | beta Catenin - metabolism | beta Catenin - genetics | Transcription Factors - metabolism | Phenotype | Breast Neoplasms - genetics | Twist-Related Protein 1 - genetics | Cell Line, Tumor | Organic Cation Transport Proteins - genetics | Twist-Related Protein 1 - metabolism | Trastuzumab | Index Medicus | trastuzumab | β-catenin | EMT | Breast Cancer | Wnt3
Journal Article
Oncogene, ISSN 0950-9232, 08/2010, Volume 29, Issue 31, pp. 4436 - 4448
Epithelial to mesenchymal transition (EMT) is a key step toward metastasis. MCF7 breast cancer cells conditionally expressing the EMT master regulator SNAI1... 
SNAI1 | StarD10 | breast cancer cell invasion | Nectin-1 | EMT | miR-661 | METASTASIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION FACTOR SNAIL | PHENOTYPE | E-CADHERIN | MIR-200 FAMILY | REPRESSORS ZEB1 | SIGNATURE | CELL BIOLOGY | ONCOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | RECEPTORS | MICRORNA | Cell Adhesion Molecules - genetics | Epithelial Cells - metabolism | Oligonucleotide Array Sequence Analysis | Humans | MicroRNAs - metabolism | Gene Expression Profiling | Phosphoproteins - metabolism | RNA, Messenger - metabolism | Nectins | Breast Neoplasms - metabolism | Epithelial Cells - physiology | Gene Expression - physiology | Female | Gene Expression Regulation, Neoplastic - drug effects | Tumor Cells, Cultured | Gene Expression Regulation, Neoplastic - physiology | Snail Family Transcription Factors | Mesenchymal Stromal Cells - physiology | Transcription Factors - physiology | Cell Dedifferentiation - physiology | Cell Dedifferentiation - drug effects | Neoplasm Invasiveness | RNA, Messenger - genetics | RNA, Small Interfering - pharmacology | Mesenchymal Stromal Cells - metabolism | Phosphoproteins - genetics | Transcription Factors - genetics | Cell Adhesion Molecules - metabolism | Transcription Factors - metabolism | Breast Neoplasms - genetics | Validation Studies as Topic | Breast Neoplasms - pathology | MicroRNAs - genetics | MicroRNAs - physiology | Cell Dedifferentiation - genetics | Messenger RNA | Physiological aspects | Development and progression | Genetic aspects | Breast cancer | Metastasis | Research | Risk factors | Proteins | Gene expression | Cell adhesion & migration | Index Medicus | Gene Expression | Cell Adhesion Molecules | Gene Expression Regulation, Neoplastic | Cell Dedifferentiation | Epithelial Cells | Breast Neoplasms | Mesenchymal Stem Cells | Life Sciences | Phosphoproteins | MicroRNAs | RNA, Small Interfering | Transcription Factors | RNA, Messenger | Cancer | pathology | genetics | pharmacology | physiology | drug effects | metabolism
Journal Article