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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2016, Volume 113, Issue 36, pp. 10198 - 10203
Mutations in superoxide dismutase (SOD1) cause amyotrophic lateral sclerosis (ALS), a fatal neurodegenerative disease characterized by the loss of upper and... 
ALS|mutant sod1 mouse | Mutant sod1|misfolded sod1|mif | mutant SOD1 | MOTOR-NEURONS | WILD-TYPE | PROTEIN | MULTIDISCIPLINARY SCIENCES | mutant SOD1 mouse | MITOCHONDRIA | ALS | ALPHA-B-CRYSTALLIN | MONOCLONAL-ANTIBODY | MIF | AMYOTROPHIC-LATERAL-SCLEROSIS | SUPEROXIDE DISMUTASE-1 | DISEASE | misfolded SOD1 | REVEALS | Biological Sciences
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 7, p. e11552
Mutations in CuZn-superoxide dismutase (SOD1) cause amyotrophic lateral sclerosis (ALS) and are found in 6% of ALS patients. Non-native and aggregation-prone... 
MOTOR-NEURON DEGENERATION | IMMUNOREACTIVITY | MULTIDISCIPLINARY SCIENCES | MOLECULAR PATHOLOGY | DISEASE | CU/ZN SUPEROXIDE-DISMUTASE | LINKED SOD1 | GENE MUTATION | MUTANT SOD1 | MICE | AMYOTROPHIC-LATERAL-SCLEROSIS | Immunohistochemistry | Inclusion Bodies - immunology | Rabbits | Superoxide Dismutase - genetics | Spinal Cord - metabolism | Enzyme-Linked Immunosorbent Assay | Humans | Middle Aged | Ubiquitin - metabolism | Male | Blotting, Western | DNA-Binding Proteins - metabolism | Superoxide Dismutase - immunology | Animals | Chickens | Amyotrophic Lateral Sclerosis - metabolism | Aged, 80 and over | Antibodies - immunology | Female | Aged | Superoxide Dismutase-1 | Inclusion Bodies - metabolism | Microscopy, Fluorescence | Superoxide Dismutase - metabolism | Ubiquitin | Viral antibodies | Enzymes | Medical research | Peptides | Medicine, Experimental | Antibodies | Amyotrophic lateral sclerosis | Superoxide | Enzyme-linked immunosorbent assay | Protein binding | Spinal and bulbar muscular atrophy | Spinal cord | Pathogenesis | Phagosomes | Lysosomes | Confocal microscopy | Superoxide dismutase | Confocal | Inclusions | Atrophy | Cords | Mitochondria | Rodents | Deoxyribonucleic acid--DNA | Motor neurons | Markers | Patients | Microscopy | Mutation | Endoplasmic reticulum | Medical Biotechnology | Medical and Health Sciences | Medicin och hälsovetenskap | MEDICIN | amyotrophic-lateral-sclerosis; cu/zn superoxide-dismutase; motor-neuron degeneration; molecular pathology; gene mutation; linked SOD1; mutant SOD1; mice; disease; immunoreactivity | Medicinsk bioteknologi | MEDICINE | Medical Biotechnology (with a focus on Cell Biology (including Stem Cell Biology), Molecular Biology, Microbiology, Biochemistry or Biopharmacy) | Medicinsk bioteknologi (med inriktning mot cellbiologi (inklusive stamcellsbiologi), molekylärbiologi, mikrobiologi, biokemi eller biofarmaci) | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 07/2017, Volume 134, Issue 1, pp. 97 - 111
Journal Article
Journal Article
Neurotherapeutics, ISSN 1933-7213, 10/2012, Volume 9, Issue 4, pp. 814 - 826
Amyotrophic lateral sclerosis is a neurodegenerative disorder characterized by progressive weakness, muscle atrophy, and paralysis due to the loss of upper and... 
Neurology | Neurosciences | Biomedicine | Motoneuron disease | PRE-084 | Neurobiology | Amyotrophic lateral sclerosis | Neurosurgery | Sigma-1 receptor | SOD1 G93A mice | mice | SOD1 | SOD1(G93A) mice | AMYOTROPHIC-LATERAL-SCLEROSIS | SIGMA RECEPTOR | FRONTOTEMPORAL LOBAR DEGENERATION | CALCIUM DYSREGULATION | NEUROSCIENCES | CLINICAL NEUROLOGY | IN-VITRO | MOUSE MODEL | PHARMACOLOGY & PHARMACY | CENTRAL-NERVOUS-SYSTEM | ANIMAL-MODEL | MUTANT SOD1 | TRANSGENIC MICE | Immunohistochemistry | Superoxide Dismutase - genetics | Amyotrophic Lateral Sclerosis - physiopathology | Cell Survival | Humans | Electrophysiology | Morpholines - pharmacology | Motor Activity - drug effects | Male | Mice, Transgenic | Receptors, sigma - agonists | Amyotrophic Lateral Sclerosis - drug therapy | Blotting, Western | Motor Neurons - pathology | Motor Neurons - metabolism | Amyotrophic Lateral Sclerosis - pathology | Animals | Neuroprotective Agents - pharmacology | Amyotrophic Lateral Sclerosis - metabolism | Female | Mice | Superoxide Dismutase-1 | Motor Neurons - drug effects | Disease Models, Animal | Methyl aspartate | Nervous system diseases | Analysis | Cells | Motor neurons | Phosphorylation | Spinal cord | Neurodegenerative diseases | Glutamic acid receptors (ionotropic) | Central nervous system | Muscles | N-Methyl-D-aspartic acid receptors | Glutamic acid receptors | Survival | Calcium influx | Western blotting | Atrophy | Locomotion | Ischemia | Paralysis | Frontotemporal dementia | Age | SOD1G93A mice | Original
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