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1. Full Text Losartan reverses impaired osseointegration in spontaneously hypertensive rats
by Mulinari-Santos, Gabriel and Souza Batista, Fábio Roberto and Kirchweger, Franziska and Tangl, Stefan and Gruber, Reinhard and Okamoto, Roberta
Clinical Oral Implants Research, ISSN 0905-7161, 11/2018, Volume 29, Issue 11, pp. 1126 - 1134
Objective Hypertension is not only associated with cardiovascular diseases but also with alterations in bone quality. Hypertension therefore might be a risk... 
bone | losartan | osseointegration | spontaneously hypertensive rats | renin‐angiotensin system | renin-angiotensin system | ALVEOLAR BONE | BONE HEALING-PROCESS | ENGINEERING, BIOMEDICAL | PERI-IMPLANT BONE | ANGIOTENSIN | OSTEOPOROSIS | METABOLISM | MODELS | RECEPTOR BLOCKER LOSARTAN | DENTISTRY, ORAL SURGERY & MEDICINE | CALCIUM | NORMOTENSIVE RATS | Hypertension | Medicine, Experimental | Medical research | Angiotensin | Osseointegration | Torque | Parameters | Rats | Implantation | Stability analysis | Risk analysis | Immunosuppressive agents | Risk factors | Bone growth | Computed tomography | Tibia | Rodents | Tomography | Bone | Angiotensin II | Cardiovascular diseases | Heart diseases | Bone (medullary) | Osteogenesis
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2. Full Text Alamandine attenuates hypertension and cardiac hypertrophy in hypertensive rats
by Liu, Chi and Yang, Chuan-Xi and Chen, Xi-Ru and Liu, Bo-Xun and Li, Yong and Wang, Xiao-Zhi and Sun, Wei and Li, Peng and Kong, Xiang-Qing
Amino Acids, ISSN 0939-4451, 8/2018, Volume 50, Issue 8, pp. 1071 - 1081
Oral administration of the peptide alamandine has antihypertensive and anti-fibrotic effects in rats. This work aimed to determine whether subcutaneous... 
Life Sciences | Biochemistry, general | Hypertension | Protein kinase A | Alamandine | Analytical Chemistry | Life Sciences, general | Biochemical Engineering | Proteomics | Neurobiology | Cardiac hypertrophy | Spontaneously hypertensive rats | OVERLOAD | RENIN-ANGIOTENSIN SYSTEM | BIOCHEMISTRY & MOLECULAR BIOLOGY | BLOOD-PRESSURE | Rats, Inbred SHR | Cell Line | Antihypertensive Agents - pharmacology | Echocardiography | Angiotensins - pharmacology | Cardiomegaly - diagnostic imaging | Cardiomegaly - drug therapy | Antihypertensive Agents - administration & dosage | Rats | Hypertension - drug therapy | Male | Receptors, G-Protein-Coupled - biosynthesis | Heart Ventricles - diagnostic imaging | Rats, Sprague-Dawley | Infusions, Subcutaneous | Animals | Myocytes, Cardiac - drug effects | Heart Ventricles - physiopathology | Injections, Subcutaneous | Nerve Tissue Proteins - biosynthesis | Oligopeptides - administration & dosage | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Oligopeptides - pharmacology | Measurement | Heart | RNA | Heart enlargement | Blood pressure | G proteins | Protein kinases | Membrane proteins | Cardiovascular agents | Natriuretic peptides | Neonates | Brain | Peptides | Ventricles (cerebral) | Atrial natriuretic peptide | mRNA | Kinases | Proteins | Infusion | Rodents | Attenuation | Inhibition | Angiotensin II | Brain natriuretic peptide | Oral administration | Cardiomyocytes | Gene expression | Fibrosis | Angiotensin | Pressure measurement | Ventricle | Hypertrophy | Original
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3. Full Text Decreased expression of Na+-H+ exchanger isoforms 1 and 3 in denervated spontaneously hypertensive rat kidney
by Li, Jianling and He, Qiaoling and Li, Qingjie and Huang, Rongjie and Wei, Xiaoyan and Pan, Xiaofeng and Wu, Weifeng
Clinical and Experimental Hypertension, ISSN 1064-1963, 05/2018, Volume 41, Issue 3, pp. 1 - 9
To determine whether the sympathetic nerve plays a role in the regulation of Na+-H+ exchange (NHE) in the kidney of spontaneously hypertensive rats (SHR), we... 
Sympathetic nerve | kidney | exchange | hypertension | spontaneously hypertensive rats | NHE1 | Na+-H+ exchange | SYMPATHETIC-NERVOUS-SYSTEM | MECHANISM | MEMBRANE | FAMILY | RENAL PROXIMAL TUBULE | NHERF-1 | K-ATPASE | PHARMACOLOGY & PHARMACY | PERIPHERAL VASCULAR DISEASE | MEDIATED INHIBITION | TRANSEPITHELIAL HCO3-ABSORPTION
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4. Full Text Farrerol Modulates Aorta Gene Expression Profile in Spontaneously Hypertensive Rats
by Qin, Xiaojiang and Hou, Xiaomin and Zhang, Kun and Li, Qingshan
Planta Medica, ISSN 0032-0943, 03/2018, Volume 84, Issue 5, pp. 296 - 303
Abstract Farrerol, a typical natural flavanone and major active component in Rhododendron dauricum var. ciliatum , has been shown to possess vasoactive ability... 
Biological and Pharmacological Activity | farrerol | flavanone | spontaneously hypertensive rat | aorta | gene expression profile | Ericaceae | Rhododendron dauricum | CELLS | CHEMISTRY, MEDICINAL | FIBROBLAST | FLAVONOIDS | PLANT SCIENCES | PRESSURE | REDUCTION | INTEGRATIVE & COMPLEMENTARY MEDICINE | VASCULAR FUNCTION | PHARMACOLOGY & PHARMACY | EXTRACT
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5. Full Text Development of structural kidney damage in spontaneously hypertensive rats
by Hultström, Michael and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Uppsala universitet and Institutionen för medicinsk cellbiologi and Integrativ Fysiologi
Journal of Hypertension, ISSN 0263-6352, 06/2012, Volume 30, Issue 6, pp. 1087 - 1091
The spontaneously hypertensive rat (SHR) is one of the major models of hypertension. This article describes the current state of knowledge about the mechanism... 
media hypertrophy | tubular atrophy | glomerulosclerosis | rat | nephrosclerosis | spontaneously hypertensive rat | afferent arteriole | cilia | kidney damage | hypertension | OXIDATIVE STRESS | PROGNOSTIC-FACTORS | RENAL DAMAGE | MYOGENIC RESPONSE | ANGIOTENSIN-II | BLOOD-PRESSURE | GLOMERULAR-FILTRATION-RATE | MATRIX METALLOPROTEINASES | JUXTAMEDULLARY CORTEX | PERIPHERAL VASCULAR DISEASE | ANTIHYPERTENSIVE THERAPY | Rats, Inbred SHR | Rats | Animals | Kidney - pathology | Hypertension - physiopathology | Kidney - physiopathology | Medical and Health Sciences | Medicin och hälsovetenskap
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6. Full Text Heart failure-induced skeletal myopathy in spontaneously hypertensive rats
by Damatto, R.L and Martinez, P.F and Lima, A.R.R and Cezar, M.D.M and Campos, D.H.S and Oliveira Junior, S.A and Guizoni, D.M and Bonomo, C and Nakatani, B.T and Dal Pai Silva, M and Carvalho, R.F and Okoshi, K and Okoshi, M.P
International Journal of Cardiology, ISSN 0167-5273, 2012, Volume 167, Issue 3, pp. 698 - 703
Abstract Background Although skeletal muscle atrophy and changes in myosin heavy chain (MyHC) isoforms have often been observed during heart failure, their... 
Cardiovascular | Heart failure | Muscle atrophy | Myosin heavy chain | Skeletal myopathy | Spontaneously hypertensive rat | Fibrosis | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-FUNCTION | ATROPHY | MYOD | MUSCLE MASS | HEAVY-CHAIN EXPRESSION | CARDIAC-HYPERTROPHY | MYOSTATIN | GENE-EXPRESSION | MICE | FOLLISTATIN | Rats, Inbred SHR | Muscular Atrophy - metabolism | Species Specificity | Muscular Diseases - metabolism | Muscular Atrophy - pathology | Myogenic Regulatory Factors - antagonists & inhibitors | Myosin Heavy Chains - genetics | Rats | Rats, Inbred WKY | Follistatin-Related Proteins - antagonists & inhibitors | Heart Failure - genetics | Male | Muscle, Skeletal - metabolism | Muscular Atrophy - genetics | Heart Failure - metabolism | Muscular Diseases - pathology | Heart Failure - pathology | Follistatin-Related Proteins - biosynthesis | Myosin Heavy Chains - biosynthesis | Myogenic Regulatory Factors - biosynthesis | Animals | Muscle, Skeletal - pathology | Muscular Diseases - genetics | Hypertension | Muscles | Muscle proteins | Myosin
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7. Full Text Myocardial myostatin in spontaneously hypertensive rats with heart failure
by Damatto, R.L and Lima, A.R.R and Martinez, P.F and Cezar, M.D.M and Okoshi, K and Okoshi, M.P
International Journal of Cardiology, ISSN 0167-5273, 2015, Volume 215, pp. 384 - 387
Abstract Background Myostatin has been shown to regulate skeletal and cardiac muscle growth. However, its status on long-term hypertrophied myocardium has not... 
Cardiovascular | Cardiac failure | Myocardium | Myostatin | Follistatin | Spontaneously hypertensive rat | Cardiac remodeling | FIBROSIS | SKELETAL-MUSCLE | CARDIAC & CARDIOVASCULAR SYSTEMS | MASS | STRESS | EXPRESSION | Rats, Inbred SHR | Body Weight | Echocardiography | Follistatin - biosynthesis | Follistatin - metabolism | Rats | Rats, Inbred WKY | Male | Myocardium - pathology | Heart Failure - metabolism | Ventricular Dysfunction, Left - diagnostic imaging | Hypertension - metabolism | Ventricular Dysfunction, Left - metabolism | Animals | Myocardium - metabolism | Myostatin - biosynthesis | Heart Failure - diagnostic imaging | Myostatin - metabolism | Heart failure | Hypertension | Medical colleges | Collagen | Body weight
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8. Full Text Low-molecular procyanidin rich grape seed extract exerts antihypertensive effect in males spontaneously hypertensive rats
by Quiñones, M and Guerrero, L and Suarez, M and Pons, Z and Aleixandre, A and Arola, L and Muguerza, B
Food Research International, ISSN 0963-9969, 05/2013, Volume 51, Issue 2, pp. 587 - 595
Grapes are a good source of flavonoids, which have been previously demonstrated to exert beneficial healthy effects on cardiovascular diseases. The aims of... 
Hypertension | Polyphenols | Oxidative stress | Spontaneously hypertensive rats | Procyanidins | Grape seed procyanidin extract | ANTIOXIDANT ACTIVITY | PHENOLIC SUBSTANCES | ENDOTHELIAL DYSFUNCTION | NITRIC-OXIDE SYNTHASE | FOOD SCIENCE & TECHNOLOGY | BLOOD-PRESSURE | EGG-WHITE HYDROLYSATE | COCOA POWDER | RED WINE | CORONARY-ARTERY-DISEASE | Antioxidants | Isoflavones | Gallates | Seeds | Grapes | Rats | Blood pressure | Diseases
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9. Full Text Effects on Atrial Fibrillation in Aged Hypertensive Rats by Ca2+-Activated K+ Channel Inhibition
by Diness, Jonas G and Skibsbye, Lasse and Jespersen, Thomas and Bartels, Emil D and Sørensen, Ulrik S and Hansen, Rie S and Grunnet, Morten
Hypertension, ISSN 0194-911X, 06/2011, Volume 57, Issue 6, pp. 1129 - 1135
We have shown previously that inhibition of small conductance Ca-activated K (SK) channels is antiarrhythmic in models of acutely induced atrial fibrillation... 
ALPHA-ACTININ2 | MYOCYTES | HEART-FAILURE | FUNCTIONAL ROLES | RISK | ion channels | LEFT-VENTRICULAR HYPERTROPHY | ACTION-POTENTIAL PROLONGATION | atrial fibrillation | UCL1684 | spontaneously hypertensive rats | NS8593 | SK2 CHANNEL | PERIPHERAL VASCULAR DISEASE | antiarrhythmia agents | small conductance Ca2+-activated K+ channel | EXPRESSION
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10. Full Text A new approach to identifying hypertension-associated genes in the mesenteric artery of spontaneously hypertensive rats and stroke-prone spontaneously hypertensive rats
by Ikawa, Takashi and Watanabe, Yuko and Okuzaki, Daisuke and Goto, Naohisa and Okamura, Nobutaka and Yamanishi, Kyosuke and Higashino, Toshihide and Yamanishi, Hiromichi and Okamura, Haruki and Higashino, Hideaki
Journal of Hypertension, ISSN 0263-6352, 08/2019, Volume 37, Issue 8, pp. 1644 - 1656
OBJECTIVE:Hypertension is one of the most prevalent diseases in humans who live a modern lifestyle. Alongside more effective care, clarification of the genetic... 
PROTEIN | WKY | CANDIDATE GENES | spontaneously hypertensive rat | mesenteric artery | two kidneys and one clip renal hypertensive rat | BLOOD-PRESSURE | DNA microarray | stroke-prone spontaneously hypertensive rat | MODELS | PERIPHERAL VASCULAR DISEASE | gene expression profile | SHR | hypertension | one kidney and one clip renal hypertensive rat | EXPRESSION | GENOME-WIDE ASSOCIATION | ORIGINAL PAPERS | Experimental hypertension | two kidneys and one clip renal hypertensive rat, one kidney and one clip renal hypertensive rat
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11. Full Text Creatine kinase inhibition lowers systemic arterial blood pressure in spontaneously hypertensive rats: a randomized controlled trial
by Karamat, Fares A and Oudman, Inge and Haan, Yentl C and van Kuilenburg, Ane B. P and Leen, Rene and Danser, Jan A. H and Leijten, Frank P. J and Ris-Stalpers, Carrie and van Montfrans, Gert A and Clark, Joseph F and Brewster, Lizzy M
Journal of hypertension, ISSN 0263-6352, 2016, Volume 34, Issue 12, pp. 2418 - 2426
Creatine kinase is reported to be a main predictor of blood pressure (BP) in the general population, with a strong correlation between resistance artery... 
Hypertension | Spontaneously hypertensive rat | Creatine kinase | Antihypertensive treatment | Blood pressure | Drug development | Betaguanidinopropionic acid | blood pressure | POPULATION | antihypertensive treatment | GUANIDINOPROPIONIC ACID | drug development | MUSCLE | spontaneously hypertensive rat | FAILURE | HYPERTROPHY | THERAPY | METABOLISM | PERIPHERAL VASCULAR DISEASE | creatine kinase | AFRICAN | KIDNEY | hypertension | EXPRESSION | beta-guanidinopropionic acid | Creatine Kinase - antagonists & inhibitors | Propionates - pharmacology | Rats, Inbred SHR | Renin - metabolism | Rats | Guanidines - pharmacology | Hypertension - drug therapy | Male | Arteries - drug effects | Random Allocation | Diuresis - drug effects | Kidney - metabolism | Animals | Arteries - physiopathology | Blood Pressure - drug effects | Protein Kinase Inhibitors - pharmacology | Vasodilation - drug effects
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12. Full Text Sterile Inflammation after Permanent Distal MCA Occlusion in Hypertensive Rats
by Möller, Karoline and Boltze, Johannes and Pösel, Claudia and Seeger, Johannes and Stahl, Tobias and Wagner, Daniel-Christoph
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 2/2014, Volume 34, Issue 2, pp. 307 - 315
The pathophysiology of stroke is governed by immune reactions within and remote from the injured brain. Hypertension, a major cause and comorbidity of stroke,... 
DENDRITIC CELLS | BRAIN-INJURY | MONOCYTES | EAE | spontaneously hypertensive rat | NEUROSCIENCES | RESPONSES | inflammation | cerebral ischemia | ENDOCRINOLOGY & METABOLISM | flow cytometry | MICE | HEMATOLOGY | microglia | ISCHEMIC-STROKE | T-CELLS | BLOOD | Rats, Inbred SHR | Infarction, Middle Cerebral Artery - physiopathology | Inflammation - pathology | Leukocytes - pathology | Microglia - metabolism | Brain - physiopathology | Dendritic Cells - pathology | Rats | Infarction, Middle Cerebral Artery - pathology | Infarction, Middle Cerebral Artery - metabolism | Inflammation - etiology | Hypertension - pathology | Hypertension - physiopathology | Hypertension - metabolism | Brain - metabolism | Brain - blood supply | Inflammation - metabolism | Animals | Infarction, Middle Cerebral Artery - complications | Brain - pathology | Microglia - pathology | Leukocytes - metabolism | Dendritic Cells - metabolism | Inflammation - physiopathology | Brain | Original
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13. Full Text Chronic inhibition of chemokine receptor CXCR2 attenuates cardiac remodeling and dysfunction in spontaneously hypertensive rats
by Zhang, Yun-Long and Geng, Chi and Yang, Jie and Fang, Jiao and Yan, Xiao and Li, Hui-Hua and Li, Pang-Bo and Zou, Lei-Xin and Chen, Chen and Guo, Shu-Bin and Liu, Ying
BBA - Molecular Basis of Disease, ISSN 0925-4439, 12/2019, Volume 1865, Issue 12, p. 165551
System hypertension is a major risk factor for cardiac hypertrophy and heart failure. Our recent findings reveal that the ablation or inhibition of C-X-C... 
Macrophage infiltration | Oxidative stress | Spontaneously hypertensive rats | Inflammation | Cardiac remodeling | Chemokine receptor CXCR2
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14. Full Text The immunomodulatory mechanism of brain injury induced by hyperhomocysteinemia in spontaneously hypertensive rats
by Zhang, Yu and Wang, Lin and Zhou, Xin and Geng, Jie and Li, Xin
Journal of Cellular Biochemistry, ISSN 0730-2312, 06/2019, Volume 120, Issue 6, pp. 9421 - 9429
A immunomodulatory mechanism of brain injury induced by hyperhomocysteinemia in rat model is proposed. The mechanism relies on upregulated inflammation and... 
homocysteine | immune mechanism | brain injury | spontaneously hypertensive rats | RISK-FACTORS | BIOCHEMISTRY & MOLECULAR BIOLOGY | PATHOLOGY | FOLIC-ACID | VITAMIN-B12 DEFICIENCY | CELL BIOLOGY | AUTOIMMUNE | IL-17 | REGULATORY T-CELLS | PLASMA HOMOCYSTEINE | TH17 CELLS | ARTERY-DISEASE | Hypertension | Brain | Enzymes | Inflammation | Permeability | Transforming growth factors | T cells | Folic acid | Brain research | Interleukins | Brain damage | Bone morphogenetic proteins | Homocysteine | Injuries | Enzyme-linked immunosorbent assay | Immunohistochemistry | Traumatic brain injury | Body weight | Transforming growth factor-b | Interleukin | Brain slice preparation | Membrane permeability | Inflammatory response | Lymphocytes T | Body weight loss | Tissues | Head injuries | Lymphocytes | Rodents | Peripheral blood | Foxp3 protein | Blood pressure | Lesions | Growth factors | Immune system | Immune response | Cytokines | Immunoregulation | Immunomodulation | Methionine | Weight loss | Polymerase chain reaction | Diagnostic systems | Brain injury | Vitamin B6
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