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JOURNAL OF BIOLOGICAL CHEMISTRY, ISSN 0021-9258, 04/2014, Volume 289, Issue 16, pp. 11219 - 11229
Background: Steroid receptor coactivator-3 (SRC-3) is frequently overexpressed in human urinary bladder cancer. Results: SRC-3 promotes urinary bladder cancer... 
Tumor Metabolism | LACTATE-DEHYDROGENASE | ACTIVATION | Cell Proliferation | UROTHELIAL CARCINOMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | AIB1 | Urinary Bladder Cancer | PROLIFERATION | TRANSCRIPTIONAL COACTIVATOR | HIF1 | BREAST | Transcription Coactivators | OVEREXPRESSION | Glycolysis | GENE-EXPRESSION | SRC-3 COACTIVATOR | SRC-3 | Cancer
Journal Article
Journal Article
Oncogenesis, ISSN 2157-9024, 01/2015, Volume 4, Issue 2, pp. e137 - e137
We have previously described novel histone acetyltransferase (HAT) inhibitors that block neuroblastoma cell growth in vitro. Here we show that two selected... 
EPIGENETICS | ASSAY | CHROMATIN | ONCOLOGY | ACETYLATION | HAT INHIBITORS | P300 | IDENTIFICATION | CANCER | SRC-3 | TOOLS | Original
Journal Article
Molecular Cell, ISSN 1097-2765, 2010, Volume 37, Issue 3, pp. 321 - 332
EGF induces signal transduction between EGFR and FAK, and FAK is required for EGF-induced cell migration. It is unknown, however, what factor mediates the... 
FAK | SRC-3Δ4 | PAK1 | EGF | metastasis | cell migration | phosphorylation | EGFR
Journal Article
International Journal of Biochemistry and Cell Biology, ISSN 1357-2725, 01/2018, Volume 94, pp. 125 - 132
•NEAT1 interacted with SRC3 in the prostate cancer cell.•NEAT1 activated the AKT phosphorylation through SRC3/IGF1R pathway.•NEAT1 is an oncogenic ncRNA in... 
SRC3 | NEAT1 | Prostate cancer | IGF1R | ANDROGEN RECEPTOR | ACTIVATION | COMPLEX | BIOCHEMISTRY & MOLECULAR BIOLOGY | LINES | CELL BIOLOGY | BREAST-CANCER | MALAT-1 | RESISTANCE | STEROID-RECEPTOR COACTIVATOR | SRC-3 | PROGRESSION | Development and progression | RNA | Growth
Journal Article
Oncogene, ISSN 0950-9232, 01/2013, Volume 32, Issue 4, pp. 514 - 527
Journal Article
The EMBO Journal, ISSN 0261-4189, 02/2006, Volume 25, Issue 4, pp. 739 - 751
Nuclear retinoic acid (RA) receptors (RARs) activate gene expression through dynamic interactions with coregulators in coordination with the ligand and... 
proteasome | nuclear receptor | retinoic acid | phosphorylation | coactivator | SRC‐3/AIB1 | Coactivator | Nuclear receptor | Phosphorylation | SRC-3/AIB1 | Retinoic acid | Proteasome | RECRUITMENT | SIGNALING PATHWAYS | ACTIVATION | NUCLEAR RECEPTORS | BIOCHEMISTRY & MOLECULAR BIOLOGY | ESTROGEN-RECEPTOR | AF-1 DOMAIN | CELL BIOLOGY | CROSS-TALK | TRANSACTIVATION | RETINOIC ACID RECEPTOR | AIB1 | SRC-3
Journal Article
by Mao, I and Liu, J and Li, X and Luo, H
Cellular and Molecular Life Sciences, ISSN 1420-682X, 12/2008, Volume 65, Issue 24, pp. 3971 - 3980
REGγ, a member of the 11S proteasome activators, has been shown to bind and activate the 20S proteasome to promote proteasome-dependent degradation of... 
Biochemistry, general | Proteasome activator | ubiquitin- and ATP-independent protein degradation | Biomedicine general | p21 | Cell Biology | p53 | Life Sciences | viral pathogenesis | Life Sciences, general | REGγ | cancer | SRC-3 | Ubiquitin- and ATP-independent protein degradation | Viral pathogenesis | Cancer
Journal Article
Asian Pacific Journal of Cancer Prevention, ISSN 1513-7368, 2013, Volume 14, Issue 6, pp. 3847 - 3850
The three homologous members of the p160 SRC family (SRC-1, SRC-2 and SRC-3) mediate the transcriptional functions of nuclear receptors and other transcription... 
CXCR4 | Bladder cancer | SRC-3 | COREGULATOR | METASTASIS | MARKER | BREAST-CANCER | LUNG-CANCER | ONCOLOGY | bladder cancer | RESISTANCE | CHEMOKINE | SRC-3 COACTIVATOR | EXPRESSION | PROGRESSION
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 10/2016, Volume 789, pp. 46 - 59
Gambogic acid (GA), the active ingredient from gamboges, has been verified as a potent anti-tumor agent in many cancer cells. Nevertheless, its function in... 
Gambogic acid | Non-Hodgkin's lymphoma | Histone | Deacetylation | SRC-3 | Apoptosis | INDUCED APOPTOSIS | AIB1 | MYELOID-LEUKEMIA CELLS | PROLIFERATION | TRANSCRIPTIONAL COACTIVATOR | ANTITUMOR-ACTIVITY | CANCER | INHIBITION | PHARMACOLOGY & PHARMACY | SRC-3 COACTIVATOR | NF-KAPPA-B | Oncogene Proteins - genetics | Nuclear Receptor Coactivator 3 - deficiency | Apoptosis - drug effects | Histones - chemistry | Humans | NF-kappa B - metabolism | Antineoplastic Agents - therapeutic use | Lymphoma, B-Cell - genetics | Molecular Targeted Therapy | Xanthones - pharmacology | Antineoplastic Agents - pharmacology | Lysine - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Lymphoma, B-Cell - drug therapy | Xanthones - therapeutic use | Lymphoma, B-Cell - metabolism | Gene Silencing | Nuclear Receptor Coactivator 3 - genetics | Down-Regulation - drug effects | Xenograft Model Antitumor Assays | Acetylation - drug effects | Animals | Signal Transduction - drug effects | Nuclear Receptor Coactivator 3 - metabolism | Lymphoma, B-Cell - pathology | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Histones - metabolism | G1 Phase Cell Cycle Checkpoints - drug effects | S Phase Cell Cycle Checkpoints - drug effects | Histone Deacetylase 1 - metabolism | Medical colleges | Lymphomas | Metastasis | Gene expression | Lysine | Ligases | Index Medicus
Journal Article
by Sha, LY and Zhang, Y and Wang, W and Sui, X and Liu, SK and Wang, T and Zhang, H
EUROPEAN REVIEW FOR MEDICAL AND PHARMACOLOGICAL SCIENCES, ISSN 1128-3602, 06/2016, Volume 20, Issue 11, pp. 2201 - 2208
OBJECTIVE: MiR-18a is a miRNA that is aberrantly overexpressed in triple-negative breast cancer (TNBC). However, its biophysical function in TNBC is still not... 
APOPTOSIS | Dicer | MICRORNA-18A | PROLIFERATION | CELL-GROWTH | TUMORS | miR-18a | BIOGENESIS | Paclitaxel | DROSHA | PHARMACOLOGY & PHARMACY | SRC-3 | Triple negative breast cancer | PROGRESSION
Journal Article