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Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 03/2018, Volume 191, Issue 3, pp. 363 - 372
Summary Blockade of the CD80/86‐CD28 pathway by belatacept after kidney transplantation is associated with an increased risk of rejection compared with... 
co‐stimulation | T cells | cytotoxicity | transplantation | co-stimulation | ACTIVATION | TOLERANCE | IMMUNOLOGY | COSTIMULATION BLOCKADE | TRIAL | BELATACEPT | KIDNEY-TRANSPLANTATION | REJECTION | TACROLIMUS | EXPRESSION | EXHAUSTION | T-Lymphocytes, Cytotoxic - immunology | Graft Rejection - drug therapy | Kidney Failure, Chronic - immunology | Cell Proliferation | Humans | Immunosuppressive Agents - therapeutic use | Middle Aged | Cells, Cultured | Programmed Cell Death 1 Receptor - metabolism | Male | Kidney Transplantation | Antigens, CD - metabolism | Kidney Failure, Chronic - therapy | Netherlands | Abatacept - therapeutic use | Isoantigens - immunology | Adult | Female | Aged | Graft Rejection - immunology | CD57 Antigens - metabolism | Cytotoxicity, Immunologic | Signaling Lymphocytic Activation Molecule Family - metabolism | Transplantation of organs, tissues, etc | Chronic kidney failure | Analysis | CD57 antigen | End-stage renal disease | Flow cytometry | PD-1 protein | Toxicity | CD8 antigen | Calcineurin | Calcineurin inhibitors | Fluorescence | Cytotoxicity | Stimulation | Leukocytes (mononuclear) | Transplantation | Lymphocytes T | CD223 antigen | CD28 antigen | Granzyme B | Lymphocytes | Peripheral blood mononuclear cells | Inhibition | CD80 antigen | Patients | CD4 antigen | Graft rejection | Rejection | Kidney diseases | Apoptosis | Kidney transplantation | Original
Journal Article
Immunology, ISSN 0019-2805, 2018, Volume 155, Issue 2, pp. 211 - 224
Journal Article
Advances in chronic kidney disease, ISSN 1548-5595, 2004
Journal
Journal Article
Journal Article
Nature medicine, ISSN 1546-170X, 2019, Volume 25, Issue 5, pp. 805 - 813
Chronic inflammation is postulated to be involved in the development of end-stage renal disease in diabetes, but which specific circulating inflammatory proteins contribute to this risk remain unknown... 
MEDICINE, RESEARCH & EXPERIMENTAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | FOLLOW-UP | NEPHROPATHY | AMERICAN-INDIANS | DECLINE | CELL BIOLOGY | BIOMARKERS | URINARY ALBUMIN EXCRETION | GENE-EXPRESSION | CHRONIC KIDNEY-DISEASE | TNF RECEPTORS 1 | PROGRESSION | Prognosis | Prospective Studies | Diabetic Nephropathies - etiology | Diabetes Mellitus, Type 2 - genetics | Humans | Middle Aged | Male | Diabetes Mellitus, Type 1 - complications | Diabetic Nephropathies - blood | Blood Proteins - metabolism | Blood Proteins - genetics | Receptors, Tumor Necrosis Factor - blood | Adult | Female | Diabetes Mellitus, Type 2 - complications | Receptors, Tumor Necrosis Factor - genetics | Risk Factors | Inflammation Mediators - blood | Diabetes Mellitus, Type 1 - genetics | Diabetic Nephropathies - genetics | Biomarkers - blood | Disease Progression | Kidney Failure, Chronic - genetics | Diabetes Mellitus, Type 2 - blood | Kidney Failure, Chronic - blood | Diabetes Mellitus, Type 1 - blood | Proteomics | Aged | Cohort Studies | Kidney Failure, Chronic - etiology | Complications and side effects | Chronic kidney failure | Tumor necrosis factor | Cytokine receptors | Development and progression | Diabetes | Health aspects | Risk factors | End-stage renal disease | Kidneys | Therapeutic applications | Medical treatment | Diabetes mellitus | Health risks | Risk | Diabetes mellitus (insulin dependent) | Inflammation | Proteins | Developmental stages | Biomarkers | Kidney diseases | Diabetes mellitus (non-insulin dependent)
Journal Article
PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 1, p. e0147329
Journal Article