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Journal of Immunology, ISSN 0022-1767, 10/2009, Volume 183, Issue 8, pp. 5418 - 5427
IFN-beta-1a has been used over the past 15 years as a primary therapy for relapsing-remitting multiple sclerosis (MS). However, the immunomodulatory mechanisms... 
EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | GROWTH-FACTOR-BETA | T(H)17 CELLS | MULTIPLE-SCLEROSIS | DENDRITIC CELLS | MESSENGER-RNA EXPRESSION | CENTRAL-NERVOUS-SYSTEM | INTERFERON-BETA | IMMUNOLOGY | UP-REGULATION | T-CELLS | Receptors, CCR6 - immunology | Interleukin-12 Subunit p35 - metabolism | Interleukin-23 Subunit p19 - antagonists & inhibitors | Dendritic Cells - immunology | Humans | Receptors, CCR6 - antagonists & inhibitors | CD4-Positive T-Lymphocytes - immunology | Receptors, Interleukin - metabolism | Interleukin-23 Subunit p19 - metabolism | STAT1 Transcription Factor - metabolism | Interleukin-1beta - metabolism | Receptors, Retinoic Acid - immunology | Phosphorylation - immunology | Phosphorylation - drug effects | Receptors, Thyroid Hormone - immunology | Interleukin-1beta - antagonists & inhibitors | Nuclear Receptor Subfamily 1, Group F, Member 3 | Receptors, Interleukin - immunology | Receptors, Interleukin - antagonists & inhibitors | Receptors, Retinoic Acid - metabolism | Cell Differentiation - immunology | Interferon beta-1a | Up-Regulation - immunology | Interleukin-23 Subunit p19 - immunology | STAT3 Transcription Factor - immunology | Interleukin-10 - immunology | Interleukin-17 - immunology | STAT1 Transcription Factor - drug effects | T-Lymphocytes, Helper-Inducer - drug effects | T-Lymphocytes, Helper-Inducer - immunology | Receptors, CCR6 - metabolism | Interleukin-10 - metabolism | Dendritic Cells - drug effects | Interleukin-10 - agonists | STAT3 Transcription Factor - metabolism | Cells, Cultured | Interleukin-1beta - immunology | Interleukin-12 Subunit p35 - agonists | Down-Regulation - drug effects | STAT1 Transcription Factor - immunology | Receptors, Thyroid Hormone - metabolism | Up-Regulation - drug effects | Interferon-beta - pharmacology | Interleukin-12 Subunit p35 - immunology | Cell Differentiation - drug effects | Down-Regulation - immunology | STAT3 Transcription Factor - drug effects | Multiple Sclerosis - immunology | CD4-Positive T-Lymphocytes - drug effects | Index Medicus | Abridged Index Medicus
Journal Article
British Journal of Cancer, ISSN 0007-0920, 01/2016, Volume 114, Issue 2, pp. 177 - 187
Background: Oestrogen receptor-negative (ER-) breast cancer is intrinsically sensitive to chemotherapy. However, tumour response is often incomplete, and... 
CELLS | REDUCES TUMOR-GROWTH | STAT1 | DNA-DAMAGE | chemotherapy | IFN | THERAPY | NEOADJUVANT CHEMOTHERAPY | ONCOLOGY | ER-negative breast cancer | GENE-EXPRESSION | RESISTANCE | PATHWAY ANALYSIS | predictive signature | XENOGRAFTS | Caspase 7 - metabolism | Immunohistochemistry | Receptors, Estrogen - metabolism | Cytoskeletal Proteins - genetics | Humans | Intracellular Signaling Peptides and Proteins - drug effects | Caspase 3 - metabolism | Gene Expression Profiling | Intracellular Signaling Peptides and Proteins - metabolism | Interferon-gamma - metabolism | Carrier Proteins - drug effects | Mitochondrial Proteins - drug effects | STAT1 Transcription Factor - metabolism | Mitochondrial Proteins - metabolism | Caspase 3 - genetics | Interferon-beta - genetics | Cytokines - genetics | Intracellular Signaling Peptides and Proteins - genetics | Real-Time Polymerase Chain Reaction | Ubiquitins - metabolism | Caspase 7 - drug effects | Antigens - drug effects | Caspase 7 - genetics | Membrane Proteins - genetics | Myxovirus Resistance Proteins - drug effects | Capecitabine - pharmacology | STAT1 Transcription Factor - genetics | Breast Neoplasms - drug therapy | Blotting, Western | Breast Neoplasms - genetics | Interferon-beta - metabolism | Signal Transduction - drug effects | Mice, Nude | Membrane Proteins - drug effects | Mice | Myxovirus Resistance Proteins - genetics | Ubiquitins - drug effects | Antigens - genetics | Neoplasm Transplantation | Phosphorylation | Ubiquitins - genetics | Gene Expression Regulation, Neoplastic | Interferon-gamma - drug effects | STAT1 Transcription Factor - drug effects | Mitochondrial Proteins - genetics | Interferon-beta - drug effects | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Breast Neoplasms - metabolism | In Situ Hybridization | Caspase 3 - drug effects | Cytoskeletal Proteins - metabolism | Female | Cytoskeletal Proteins - drug effects | Membrane Proteins - metabolism | Interferon-gamma - genetics | Cytokines - metabolism | Antigens - metabolism | Cisplatin - pharmacology | Xenograft Model Antitumor Assays | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Cytokines - drug effects | Myxovirus Resistance Proteins - metabolism | Index Medicus | Translational Therapeutics
Journal Article
Journal Article
Arthritis & Rheumatology, ISSN 2326-5191, 07/2017, Volume 69, Issue 7, pp. 1495 - 1501
Journal Article
Arthritis & Rheumatology, ISSN 2326-5191, 10/2016, Volume 68, Issue 10, pp. 2492 - 2502
ObjectiveAntinuclear antibodies (ANAs) are detected in approximate to 18% of females, yet autoimmune disease develops in only 5-8%. Immunologic differences... 
SYSTEMIC-LUPUS-ERYTHEMATOSUS | INTERFERON | CYTOKINE PRODUCTION | RISK-FACTORS | DENDRITIC CELLS | ANTINUCLEAR ANTIBODIES | RHEUMATOLOGY | EXPRESSION | SLE | IG-LIKE RECEPTOR-1 | T-CELLS | Interferon-alpha - pharmacology | Multivariate Analysis | Phosphoproteins - drug effects | Interferon-beta - immunology | Antibodies, Antinuclear - immunology | Humans | Middle Aged | Interleukin-17 - immunology | STAT1 Transcription Factor - drug effects | Centromere Protein B - immunology | Male | Monocytes - immunology | Ribosomal Proteins - immunology | Healthy Volunteers | Case-Control Studies | Leukocytes - immunology | Young Adult | Flow Cytometry | Autoimmunity - immunology | Lupus Erythematosus, Systemic - immunology | Ribonucleoproteins - immunology | Tumor Necrosis Factor-alpha - immunology | Aged, 80 and over | Adult | B-Lymphocyte Subsets - immunology | Female | Interleukin-12 Subunit p40 - immunology | Cytokines - immunology | Interleukin 1 Receptor Antagonist Protein - immunology | Immunophenotyping | Logistic Models | Nuclear Proteins - immunology | STAT1 Transcription Factor - immunology | Stem Cell Factor - immunology | Interferon-alpha - immunology | Autoantibodies - immunology | Interferon-gamma - immunology | STAT3 Transcription Factor - drug effects | Sex Factors | Aged | Granulocyte Colony-Stimulating Factor - immunology | STAT3 Transcription Factor - immunology | Plasma Cells - immunology | Index Medicus | Abridged Index Medicus | Systemic Lupus Erythematosus
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2010, Volume 184, Issue 7, pp. 3478 - 3486
Journal Article
Nature Neuroscience, ISSN 1097-6256, 03/2004, Volume 7, Issue 3, pp. 229 - 235
The generation of distinct cell types during development depends on the competence of progenitor populations to differentiate along specific lineages. Here we... 
RNA-POLYMERASE-II | RECOMBINANT RETROVIRUS | X-INACTIVATION | STEM-CELLS | METHYLTRANSFERASE | RAT CEREBRAL-CORTEX | TRANSCRIPTION | DEVELOPMENTAL-CHANGES | CENTRAL-NERVOUS-SYSTEM | NEUROSCIENCES | HISTONE H3 METHYLATION | Trans-Activators - drug effects | Fibroblast Growth Factor 2 - pharmacology | Gene Expression Regulation, Developmental - genetics | Chromatin Assembly and Disassembly - genetics | Stem Cells - cytology | Glial Fibrillary Acidic Protein - metabolism | Promoter Regions, Genetic - drug effects | Stem Cells - metabolism | Cerebral Cortex - cytology | Promoter Regions, Genetic - genetics | Cerebral Cortex - metabolism | Chromatin Assembly and Disassembly - drug effects | Transcription Factors - drug effects | DNA-Binding Proteins - metabolism | Cell Differentiation - genetics | Trans-Activators - genetics | Fibroblast Growth Factor 2 - metabolism | Fetus | Lysine - metabolism | Ciliary Neurotrophic Factor - pharmacology | Astrocytes - cytology | Astrocytes - drug effects | Cells, Cultured | DNA-Binding Proteins - drug effects | Gene Expression Regulation, Developmental - drug effects | Rats | Signal Transduction - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Histones - drug effects | Ciliary Neurotrophic Factor - metabolism | Animals | Histones - genetics | Cerebral Cortex - embryology | Methylation - drug effects | Signal Transduction - drug effects | Cell Differentiation - drug effects | Glial Fibrillary Acidic Protein - drug effects | STAT1 Transcription Factor | Stem Cells - drug effects | Trans-Activators - metabolism | Histones - metabolism | Astrocytes - metabolism | Physiological aspects | Fibroblast growth factors | Research | Gene expression | Astrocytes | Index Medicus
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 2008, Volume 45, Issue 7, pp. 950 - 963
Journal Article
Journal Article
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 07/2006, Volume 126, Issue 7, pp. 1574 - 1581
Toll-like receptor (TLR)3 is a receptor for virus-associated double-stranded RNA, and triggers antiviral immune responses during viral infection. Epidermal... 
TOLL-LIKE RECEPTOR-3 | TRANSCRIPTION FACTORS | DOUBLE-STRANDED-RNA | ANTIVIRAL RESPONSES | EPITHELIAL-CELLS | IRF FAMILY | FACTOR-KAPPA-B | EPIDERMAL-KERATINOCYTES | INTERFERON REGULATORY FACTOR-7 | VIRAL-INFECTION | DERMATOLOGY | Humans | Interferon Regulatory Factor-3 - drug effects | STAT1 Transcription Factor - analysis | STAT1 Transcription Factor - drug effects | Suppressor of Cytokine Signaling 1 Protein | Interferon Regulatory Factor-7 - genetics | Keratinocytes - chemistry | Repressor Proteins - analysis | Toll-Like Receptor 3 - analysis | Interferon Regulatory Factor-7 - physiology | Repressor Proteins - physiology | STAT3 Transcription Factor - physiology | Phosphorylation - drug effects | Interferon Regulatory Factor-7 - analysis | Intracellular Signaling Peptides and Proteins - genetics | STAT3 Transcription Factor - genetics | STAT1 Transcription Factor - physiology | Chemokine CCL4 | Interferon Regulatory Factor-3 - analysis | Poly I-C - pharmacology | Chemokine CCL3 | Immunity, Innate - drug effects | Macrophage Inflammatory Proteins - metabolism | Cells, Cultured | Intracellular Signaling Peptides and Proteins - analysis | Repressor Proteins - genetics | Suppressor of Cytokine Signaling Proteins - genetics | STAT3 Transcription Factor - analysis | STAT1 Transcription Factor - genetics | Genetic Vectors - genetics | Keratinocytes - immunology | Interferon Regulatory Factor-3 - physiology | Gene Expression Regulation - drug effects | Immunity, Innate - immunology | Toll-Like Receptor 3 - physiology | Signal Transduction - drug effects | Homeostasis - physiology | STAT3 Transcription Factor - drug effects | NF-kappa B - physiology | RNA, Double-Stranded - pharmacology | Signal Transduction - physiology | Suppressor of Cytokine Signaling Proteins - physiology | Adenoviruses, Human - genetics | Feedback, Physiological - physiology | Intracellular Signaling Peptides and Proteins - physiology | Suppressor of Cytokine Signaling Proteins - analysis | Keratinocytes - physiology | Index Medicus | Phosphorylation | Immune response | Transcription | macrophage inflammatory protein 1 | Dermatology | Double-stranded RNA | Stat3 protein | Keratinocytes | Intracellular signalling | Poly (I:C) | Nuclear transport | TLR3 protein | Infection | Interferon regulatory factor 7 | Signal transduction | NF- Kappa B protein | Feedback | Toll-like receptors | Interferon | Internet | Stat1 protein | Macrophage inflammatory protein | Expression vectors
Journal Article