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inhibition (34) 34
kinases (34) 34
protein-tyrosine kinases - metabolism (34) 34
tyrosine phosphorylation (34) 34
abridged index medicus (33) 33
amino acid sequence (33) 33
cell line, tumor (33) 33
transcription factor (33) 33
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dna-binding proteins - genetics (31) 31
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trans-activators - genetics (27) 27
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Journal of Leukocyte Biology, ISSN 0741-5400, 05/2017, Volume 101, Issue 5, pp. 1181 - 1190
The interferon and T cell receptor signaling pathways mediate specific and combined contributions to IL‐10 regulation via STAT and BATF transcription factors.... 
gene regulation | Tr1 cells | signaling | Signaling | Gene regulation | ACTIVATION | Tr 1 cells | PERSISTENT LCMV INFECTION | RECEPTOR | IMMUNOLOGY | CUTTING EDGE | BETA | CELL BIOLOGY | IRF INTERACTIONS | RESPONSES | GENE | DIFFERENTIATION | HEMATOLOGY | IFN-ALPHA | Interferon-alpha - pharmacology | RNA, Small Interfering - genetics | STAT2 Transcription Factor - immunology | Humans | Basic-Leucine Zipper Transcription Factors - immunology | CD4-Positive T-Lymphocytes - immunology | CD3 Complex - genetics | CD28 Antigens - genetics | CD28 Antigens - antagonists & inhibitors | Basic-Leucine Zipper Transcription Factors - antagonists & inhibitors | Phosphorylation - drug effects | Binding Sites | STAT3 Transcription Factor - genetics | Signal Transduction | CD4-Positive T-Lymphocytes - cytology | Gene Expression Regulation | CD28 Antigens - immunology | Basic-Leucine Zipper Transcription Factors - genetics | STAT1 Transcription Factor - genetics | STAT1 Transcription Factor - antagonists & inhibitors | Cycloheximide - pharmacology | STAT1 Transcription Factor - immunology | Interferon-alpha - immunology | Antibodies - pharmacology | Enhancer Elements, Genetic | STAT2 Transcription Factor - antagonists & inhibitors | Interleukin-10 - genetics | CD3 Complex - immunology | STAT2 Transcription Factor - genetics | Protein Binding | Primary Cell Culture | STAT3 Transcription Factor - immunology | Protein Isoforms - immunology | STAT3 Transcription Factor - antagonists & inhibitors | Interleukin-10 - immunology | CD4-Positive T-Lymphocytes - drug effects | Protein Isoforms - genetics | RNA, Small Interfering - metabolism | Phosphorylation | Transcription factors | CD45RA antigen | Central nervous system | Lymphocytes T | Leucine | Recruitment | T-cell receptor | Signal transduction | Conserved sequence | Pathways | Lymphocytes | Stat1 protein | Immune system | Stat2 protein | RNA-mediated interference | Stat3 protein | Inflammation | T cell receptors | Gene expression | Ribonucleic acid--RNA | CD4 antigen | Leucine zipper proteins | Interleukin 10 | Interferon | Index Medicus | STAT2 Transcription Factor | Interleukin-10 | Interferon-alpha | CD3 Complex | Antibodies | Life Sciences | Cycloheximide | Immunology | Protein Isoforms | CD28 Antigens | STAT1 Transcription Factor | RNA, Small Interfering | STAT3 Transcription Factor | CD4-Positive T-Lymphocytes | Basic-Leucine Zipper Transcription Factors
Journal Article
Journal of Immunology, ISSN 0022-1767, 07/2015, Volume 195, Issue 1, pp. 210 - 216
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2017, Volume 127, Issue 12, pp. 4415 - 4420
Primary immunodeficiencies are often monogenic disorders characterized by vulnerability to specific infectious pathogens. Here, we performed whole-exome... 
MEDICINE, RESEARCH & EXPERIMENTAL | IMMUNITY | ACTIVATOR | INTERFERON-GAMMA | MYCOBACTERIAL DISEASE | ALPHA/BETA | MENDELIAN SUSCEPTIBILITY | CLINICAL-FEATURES | DEFICIENCY | SIGNAL TRANSDUCER | INBORN-ERRORS | Bacteremia - immunology | Genetic Diseases, Inborn - microbiology | STAT2 Transcription Factor - immunology | Humans | Genetic Diseases, Inborn - genetics | Genetic Diseases, Inborn - virology | Male | Cytomegalovirus Infections - immunology | Cytomegalovirus Infections - genetics | Bacteremia - microbiology | Streptococcal Infections - genetics | Phosphorylation - genetics | Female | Phosphorylation - immunology | Receptor, Interferon alpha-beta - genetics | Fibroblasts - microbiology | Mycobacterium Infections, Nontuberculous - immunology | Immunologic Deficiency Syndromes - immunology | Receptors, Interferon - genetics | Fibroblasts - virology | Genetic Diseases, Inborn - immunology | Viremia - genetics | Viridans Streptococci - immunology | Bacteremia - genetics | Immunologic Deficiency Syndromes - virology | STAT1 Transcription Factor - genetics | Immunologic Deficiency Syndromes - microbiology | STAT1 Transcription Factor - immunology | Cytomegalovirus - immunology | Mycobacterium abscessus - immunology | Receptor, Interferon alpha-beta - immunology | Receptors, Interferon - immunology | Viremia - virology | STAT2 Transcription Factor - genetics | Immunologic Deficiency Syndromes - genetics | Fibroblasts - immunology | Viremia - immunology | Streptococcal Infections - immunology | Mutation | Mycobacterium Infections, Nontuberculous - genetics | Virus diseases | Usage | Immunotherapy | Genetic aspects | Disease susceptibility | Research | Mutation (Biology) | Stat2 protein | Cytomegalovirus | Phosphorylation | Viremia | Immunodeficiency | Stop codon | C-Terminus | Gene deletion | Bacteremia | Nuclear transport | α-Interferon | Clonal deletion | Fibroblasts | Interferon | Codons | Stat1 protein | Viral infections | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2012, Volume 287, Issue 23, pp. 19216 - 19228
Plasmacytoid dendritic cells (pDCs) are characterized by their ability to produce high levels of type 1 interferons in response to ligands that activate TLR7... 
TRANSCRIPTION FACTORS | KAPPA-B KINASE | SYSTEMIC-LUPUS-ERYTHEMATOSUS | AUTOIMMUNE-DISEASES | IMMUNE-RESPONSES | GENE INDUCTION | BIOCHEMISTRY & MOLECULAR BIOLOGY | TOLL-LIKE RECEPTORS | I INTERFERON | ALPHA INDUCTION | NUCLEIC-ACIDS | Thymidine Kinase - immunology | Interferon-beta - immunology | Membrane Glycoproteins - metabolism | STAT2 Transcription Factor - immunology | Dendritic Cells - immunology | Humans | NF-kappa B - immunology | Toll-Like Receptor 9 - genetics | NF-kappa B - metabolism | Janus Kinases - metabolism | Toll-Like Receptor 9 - agonists | Toll-Like Receptor 9 - immunology | STAT1 Transcription Factor - metabolism | I-kappa B Kinase - metabolism | Interferon-alpha - genetics | Toll-Like Receptor 7 - immunology | HEK293 Cells | Interferon-beta - genetics | Janus Kinases - immunology | Plasma Cells - metabolism | Membrane Glycoproteins - immunology | Interleukin-6 - metabolism | Dendritic Cells - metabolism | Toll-Like Receptor 7 - agonists | Toll-Like Receptor 7 - genetics | Interferon-alpha - metabolism | Toll-Like Receptor 7 - metabolism | Interleukin-6 - genetics | Janus Kinases - genetics | Thymidine Kinase - genetics | Membrane Glycoproteins - agonists | I-kappa B Kinase - genetics | STAT1 Transcription Factor - genetics | STAT1 Transcription Factor - immunology | Interferon-alpha - immunology | Membrane Glycoproteins - genetics | Animals | Interferon-beta - metabolism | NF-kappa B - genetics | Interleukin-6 - immunology | STAT2 Transcription Factor - genetics | Thymidine Kinase - metabolism | I-kappa B Kinase - immunology | Mice | STAT2 Transcription Factor - metabolism | Toll-Like Receptor 9 - metabolism | Plasma Cells - immunology | Index Medicus | Toll-like Receptors (TLR) | Immunology | Plasmacytoid Dendritic Cells | TAK1 | Innate Immunity | Interferon | Protein Phosphorylation | IKKβ | Kinase Inhibitor
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 11/2015, Volume 98, Issue 5, pp. 689 - 702
In vivo and in vitro inhibition of IL‐12, IL‐23, and IL‐10 stimulation by IFNβ. MS is an autoimmune disease characterized by immune cell infiltration in the... 
cytokines | Type I interferons | STAT1/STAT2 | IRF1/IRF7 | PI3K/GSK3 | Cytokines | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | ACTIVATION | GLYCOGEN-SYNTHASE KINASE-3 | INTERLEUKIN-23 | INTERFERON-BETA | IMMUNOLOGY | MYELOID CELLS | CELL BIOLOGY | MULTIPLE-SCLEROSIS | IN-VIVO | GENE-EXPRESSION | CENTRAL-NERVOUS-SYSTEM | HEMATOLOGY | Interleukin-12 Subunit p40 - genetics | Interferon-beta - immunology | Myelin-Oligodendrocyte Glycoprotein - pharmacology | Spleen - immunology | Dendritic Cells - immunology | Peptide Fragments - pharmacology | Interferon Regulatory Factor-7 - genetics | Phosphatidylinositol 3-Kinases - immunology | Interleukin-23 - genetics | Signal Transduction - immunology | Interferon-beta - genetics | Th17 Cells - cytology | Interleukin-12 Subunit p40 - immunology | STAT3 Transcription Factor - genetics | Gene Expression Regulation - immunology | Interferon Regulatory Factor-1 - immunology | Toll-Like Receptors - immunology | Interferon Regulatory Factor-7 - immunology | Interleukin-12 Subunit p35 - genetics | Signal Transduction - genetics | Spleen - cytology | STAT1 Transcription Factor - genetics | Interleukin-23 - immunology | STAT1 Transcription Factor - immunology | Interferon Regulatory Factor-1 - genetics | Mice, Knockout | Gene Expression Regulation - drug effects | Phosphatidylinositol 3-Kinases - genetics | Animals | Interleukin-12 Subunit p35 - immunology | Signal Transduction - drug effects | Interleukin-10 - genetics | Toll-Like Receptors - genetics | Th17 Cells - immunology | Dendritic Cells - cytology | Mice | STAT3 Transcription Factor - immunology | Interleukin-10 - immunology | Index Medicus | Spotlight on Leading Edge Research | PI3K | STAT1 | GSK3 | STAT2 | IRF1 | IRF7
Journal Article
Blood, ISSN 0006-4971, 03/2005, Volume 105, Issue 6, pp. 2458 - 2464
There is now considerable in vitro evidence that tumor necrosis factor (TNF)-related apoptosis-inducing ligand (TRAIL) is involved in HIV-1 pathogenesis by... 
CANCER-CELLS | TUMOR-NECROSIS-FACTOR | HUMAN LYMPHOID-TISSUE | HIV-INFECTION | DENDRITIC CELLS | SIGNALING PATHWAY | IMMUNODEFICIENCY-VIRUS TYPE-1 | MONOCYTES | CD4(+) T-CELLS | INDUCTION | HEMATOLOGY | STAT2 Transcription Factor - immunology | Dendritic Cells - immunology | Humans | Membrane Glycoproteins - biosynthesis | Cell Death - immunology | Dendritic Cells - pathology | Male | Monocytes - metabolism | Monocytes - immunology | CD4-Positive T-Lymphocytes - pathology | Interferon Type I - immunology | Viral Load | CD4-Positive T-Lymphocytes - immunology | Acquired Immunodeficiency Syndrome - immunology | Signal Transduction - immunology | STAT1 Transcription Factor - metabolism | TNF-Related Apoptosis-Inducing Ligand | Monocytes - pathology | Tumor Necrosis Factor-alpha - immunology | Female | Membrane Glycoproteins - immunology | CD4-Positive T-Lymphocytes - virology | Dendritic Cells - virology | Dendritic Cells - metabolism | Apoptosis Regulatory Proteins - biosynthesis | CD4 Antigens - immunology | Acquired Immunodeficiency Syndrome - metabolism | Apoptosis Regulatory Proteins - immunology | Gene Expression Regulation - immunology | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Acquired Immunodeficiency Syndrome - drug therapy | STAT1 Transcription Factor - immunology | Gene Expression Regulation - drug effects | Monocytes - virology | HIV-1 - immunology | Acquired Immunodeficiency Syndrome - pathology | STAT2 Transcription Factor - metabolism | CD4 Antigens - pharmacology | Tumor Necrosis Factor-alpha - biosynthesis | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Journal Article
Journal of Virology, ISSN 0022-538X, 06/2012, Volume 86, Issue 12, pp. 6932 - 6946
Journal Article
Cellular Signalling, ISSN 0898-6568, 03/2014, Volume 26, Issue 3, pp. 619 - 628
New negative regulators of interferon (IFN) signaling, preferably with tissue specificity, are needed to develop therapeutic means to enhance the efficacy of... 
Interferon | SHP-2 | Luteolin | PKA | Phosphodiesterase | INDUCED APOPTOSIS | STAT1 | MOTIF-CONTAINING PROTEIN | RECEPTOR | CELL BIOLOGY | JAK/STAT PATHWAY | FLAVONOID LUTEOLIN | DEGRADATION | EXPRESSION | CAMP | IFN-ALPHA | Interferon-alpha - pharmacology | Adjuvants, Immunologic - pharmacology | STAT2 Transcription Factor - immunology | Humans | Cyclic AMP-Dependent Protein Kinases - chemistry | TYK2 Kinase - metabolism | Receptors, Cell Surface | TYK2 Kinase - immunology | Janus Kinase 1 - metabolism | STAT1 Transcription Factor - metabolism | Neoplasm Proteins | HEK293 Cells | Receptors for Activated C Kinase | Antibodies - immunology | Phosphorylation - drug effects | Receptor, Interferon alpha-beta - chemistry | Cyclic AMP - metabolism | Receptor, Interferon alpha-beta - metabolism | Phosphoric Diester Hydrolases - metabolism | Cell Survival | Colforsin - pharmacology | Protein Tyrosine Phosphatase, Non-Receptor Type 11 - antagonists & inhibitors | Phosphodiesterase Inhibitors - pharmacology | Janus Kinase 1 - antagonists & inhibitors | Luteolin - pharmacology | STAT1 Transcription Factor - antagonists & inhibitors | Janus Kinase 1 - immunology | STAT1 Transcription Factor - immunology | Hep G2 Cells | Interferon-beta - pharmacology | STAT2 Transcription Factor - antagonists & inhibitors | Signal Transduction - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | STAT2 Transcription Factor - metabolism | GTP-Binding Proteins | HeLa Cells | Immunologic Factors - pharmacology | Index Medicus | Proteins | Transducers | Pathways | Partial differential equations | Activation | Inhibition | Kinases
Journal Article
Clinical Immunology, ISSN 1521-6616, 2015, Volume 161, Issue 2, pp. 260 - 269
Journal Article