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science & technology (509) 509
humans (468) 468
life sciences & biomedicine (452) 452
animals (297) 297
stat2 transcription factor - metabolism (273) 273
stat2 transcription factor (242) 242
signal transduction (234) 234
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phosphorylation (175) 175
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biological response modifiers (66) 66
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cell line, tumor (61) 61
fundamental and applied biological sciences. psychology (61) 61
trans-activators - genetics (61) 61
interferon-stimulated gene factor 3 (59) 59
stat2 (59) 59
stat3 transcription factor (59) 59
research article (58) 58
interferon type i - metabolism (57) 57
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janus kinase 1 (56) 56
multidisciplinary sciences (56) 56
science & technology - other topics (56) 56
signal transduction - physiology (56) 56
amino acid sequence (55) 55
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health aspects (54) 54
interferon-stimulated gene factor 3, gamma subunit (53) 53
hela cells (51) 51
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promoter regions, genetic (34) 34
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tyrosine - metabolism (33) 33
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dna-binding proteins - physiology (31) 31
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1. Full Text Type I interferon‐enhanced IL‐10 expression in human CD4 T cells is regulated by STAT3, STAT2, and BATF transcription factors
by Govender, Umeshree and Corre, Béatrice and Bourdache, Yasmine and Pellegrini, Sandra and Michel, Frédérique
Journal of leukocyte biology, ISSN 0741-5400, 05/2017, Volume 101, Issue 5, pp. 1181 - 1190
gene regulation | Tr1 cells | signaling | Signaling | Gene regulation | Immunology | Life Sciences & Biomedicine | Hematology | Science & Technology | Cell Biology | Interferon-alpha - pharmacology | RNA, Small Interfering - genetics | STAT2 Transcription Factor - immunology | Humans | Basic-Leucine Zipper Transcription Factors - immunology | CD4-Positive T-Lymphocytes - immunology | CD3 Complex - genetics | CD28 Antigens - genetics | CD28 Antigens - antagonists & inhibitors | Basic-Leucine Zipper Transcription Factors - antagonists & inhibitors | Phosphorylation - drug effects | Binding Sites | STAT3 Transcription Factor - genetics | Signal Transduction | CD4-Positive T-Lymphocytes - cytology | Gene Expression Regulation | CD28 Antigens - immunology | Basic-Leucine Zipper Transcription Factors - genetics | STAT1 Transcription Factor - genetics | STAT1 Transcription Factor - antagonists & inhibitors | Cycloheximide - pharmacology | STAT1 Transcription Factor - immunology | Interferon-alpha - immunology | Antibodies - pharmacology | Enhancer Elements, Genetic | STAT2 Transcription Factor - antagonists & inhibitors | Interleukin-10 - genetics | CD3 Complex - immunology | STAT2 Transcription Factor - genetics | Protein Binding | Primary Cell Culture | STAT3 Transcription Factor - immunology | Protein Isoforms - immunology | STAT3 Transcription Factor - antagonists & inhibitors | Interleukin-10 - immunology | CD4-Positive T-Lymphocytes - drug effects | Protein Isoforms - genetics | RNA, Small Interfering - metabolism | Phosphorylation | Transcription factors | CD45RA antigen | Central nervous system | Lymphocytes T | Leucine | Recruitment | T-cell receptor | Signal transduction | Conserved sequence | Pathways | Lymphocytes | Stat1 protein | Immune system | Stat2 protein | RNA-mediated interference | Stat3 protein | Inflammation | T cell receptors | Gene expression | Ribonucleic acid--RNA | CD4 antigen | Leucine zipper proteins | Interleukin 10 | Interferon | Index Medicus | STAT2 Transcription Factor | Interleukin-10 | Interferon-alpha | CD3 Complex | Antibodies | Life Sciences | Cycloheximide | Protein Isoforms | CD28 Antigens | STAT1 Transcription Factor | RNA, Small Interfering | STAT3 Transcription Factor | CD4-Positive T-Lymphocytes | Basic-Leucine Zipper Transcription Factors
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2. Full Text Dual regulation of Stat1 and Stat3 by the tumor suppressor protein PML contributes to interferon α-mediated inhibition of angiogenesis
by Hsu, Kuo-Sheng and Zhao, Xuan and Cheng, Xiwen and Guan, Dongyin and Mahabeleshwar, Ganapati H and Liu, Yu and Borden, Ernest and Jain, Mukesh K and Kao, Hung-Ying
The Journal of biological chemistry, ISSN 0021-9258, 06/2017, Volume 292, Issue 24, pp. 10048 - 10060
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Endothelium, Vascular - cytology | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Neovascularization, Pathologic - pathology | Endopeptidases - chemistry | STAT1 Transcription Factor - metabolism | Interferon-alpha - genetics | RNA Interference | Human Umbilical Vein Endothelial Cells - cytology | STAT1 Transcription Factor - agonists | Promyelocytic Leukemia Protein - metabolism | Ubiquitin Thiolesterase - metabolism | Neovascularization, Pathologic - prevention & control | Ubiquitin Thiolesterase - antagonists & inhibitors | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Promyelocytic Leukemia Protein - genetics | Recombinant Proteins - metabolism | Cell Line | Endopeptidases - metabolism | Interferon-alpha - metabolism | Cells, Cultured | STAT1 Transcription Factor - genetics | Ubiquitin Thiolesterase - genetics | Mice, Knockout | STAT2 Transcription Factor - agonists | Animals | Endopeptidases - genetics | Endothelium, Vascular - metabolism | STAT2 Transcription Factor - genetics | Promyelocytic Leukemia Protein - antagonists & inhibitors | Endothelium, Vascular - pathology | Neovascularization, Pathologic - metabolism | Protein Processing, Post-Translational | STAT2 Transcription Factor - metabolism | STAT3 Transcription Factor - antagonists & inhibitors | Neovascularization, Physiologic | Index Medicus | signal transducers and activators of transcription 1 (STAT1) | STAT transcription factor | Signal Transduction | interferon | angiogenesis | STAT3
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3. Full Text IL28B inhibits hepatitis C virus replication through the JAK–STAT pathway
by Zhang, Leiliang and Jilg, Nikolaus and Shao, Run-Xuan and Lin, Wenyu and Fusco, Dahlene N and Zhao, Hong and Goto, Kaku and Peng, Lee F and Chen, Wen-Chi and Chung, Raymond T
Journal of hepatology, ISSN 0168-8278, 2010, Volume 55, Issue 2, pp. 289 - 298
Gastroenterology and Hepatology | IL28B | STAT | HCV | JAK | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Gastroenterology. Liver. Pancreas. Abdomen | Biological and medical sciences | Medical sciences | RNA, Small Interfering - genetics | Humans | STAT Transcription Factors - metabolism | TYK2 Kinase - metabolism | DNA Primers - genetics | Janus Kinases - metabolism | Interleukins - genetics | Janus Kinase 1 - metabolism | STAT1 Transcription Factor - metabolism | Base Sequence | Hepacivirus - physiology | TYK2 Kinase - antagonists & inhibitors | Interleukins - pharmacology | Phosphorylation - drug effects | Hepacivirus - drug effects | Cell Line | Virus Replication - drug effects | Antiviral Agents - pharmacology | Recombinant Proteins - pharmacology | Interferon-Stimulated Gene Factor 3, gamma Subunit - genetics | Janus Kinase 1 - antagonists & inhibitors | STAT1 Transcription Factor - genetics | Interferon-Stimulated Gene Factor 3, gamma Subunit - metabolism | STAT1 Transcription Factor - antagonists & inhibitors | Receptors, Interferon - metabolism | Janus Kinases - antagonists & inhibitors | STAT2 Transcription Factor - antagonists & inhibitors | Signal Transduction - drug effects | STAT2 Transcription Factor - genetics | Polymorphism, Single Nucleotide | STAT Transcription Factors - antagonists & inhibitors | STAT2 Transcription Factor - metabolism | Virus Replication - physiology | Interferon-Stimulated Gene Factor 3, gamma Subunit - antagonists & inhibitors | Virus diseases | Genetic research | Precipitation (Meteorology) | Biological response modifiers | Hepatitis C virus | Health aspects | Index Medicus
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4. Full Text High basal STAT4 balanced by STAT1 induction to control type 1 interferon effects in natural killer cells
by Miyagi, Takuya and Gil, M. Pilar and Wang, Xin and Louten, Jennifer and Chu, Wen-Ming and Biron, Christine A
The Journal of experimental medicine, ISSN 0022-1007, 10/2007, Volume 204, Issue 10, pp. 2383 - 2396
Immunology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Lymphocytic choriomeningitis virus - immunology | STAT1 Transcription Factor - deficiency | Lymphocyte Activation | Gene Expression Regulation | STAT4 Transcription Factor - metabolism | Mice, Transgenic | Interferon-gamma - metabolism | Lymphocytic Choriomeningitis - immunology | STAT1 Transcription Factor - genetics | STAT2 Transcription Factor - deficiency | Lymphocytic Choriomeningitis - metabolism | STAT1 Transcription Factor - metabolism | Animals | STAT2 Transcription Factor - genetics | Interferon Type I - genetics | Interferon Type I - metabolism | Killer Cells, Natural - immunology | STAT4 Transcription Factor - genetics | Mice | STAT2 Transcription Factor - metabolism | Killer Cells, Natural - metabolism | STAT4 Transcription Factor - deficiency | Receptor, Interferon alpha-beta - metabolism | Index Medicus
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5. Full Text A molecular switch from STAT2-IRF9 to ISGF3 underlies interferon-induced gene transcription
by Platanitis, Ekaterini and Demiroz, Duygu and Schneller, Anja and Fischer, Katrin and Capelle, Christophe and Hartl, Markus and Gossenreiter, Thomas and Mueller, Mathias and Novatchkova, Maria and Decker, Thomas
Nature communications, ISSN 2041-1723, 07/2019, Volume 10, Issue 1, pp. 2921 - 17
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Promoter Regions, Genetic | Humans | Mice, Inbred C57BL | Gene Expression Regulation | Male | Interferon-Stimulated Gene Factor 3, gamma Subunit - genetics | STAT1 Transcription Factor - genetics | Interferon-Stimulated Gene Factor 3, gamma Subunit - metabolism | Mice, Knockout | Macrophages - metabolism | STAT1 Transcription Factor - metabolism | Animals | Interferon-Stimulated Gene Factor 3 - genetics | Interferon-Stimulated Gene Factor 3 - metabolism | STAT2 Transcription Factor - genetics | Interferons - metabolism | Female | RAW 264.7 Cells | Transcription, Genetic | Mice | STAT2 Transcription Factor - metabolism | Binding | Molecular machines | Stat2 protein | Homeostasis | Labelling | Intracellular signalling | Gene expression | Macrophages | Cascades | γ-Interferon | Interferon | Stat1 protein | Deoxyribonucleic acid--DNA | Cytoplasm | Index Medicus
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6. Full Text The Interferon Signaling Antagonist Function of Yellow Fever Virus NS5 Protein Is Activated by Type I Interferon
by Laurent-Rolle, Maudry and Morrison, Juliet and Rajsbaum, Ricardo and Macleod, Jesica M. Levingston and Pisanelli, Giuseppe and Pham, Alissa and Ayllon, Juan and Miorin, Lisa and Martínez-Romero, Carles and tenOever, Benjamin R and García-Sastre, Adolfo
Cell host & microbe, ISSN 1931-3128, 09/2014, Volume 16, Issue 3, pp. 314 - 327
Parasitology | Life Sciences & Biomedicine | Microbiology | Science & Technology | Virology | Phosphorylation | Humans | GTP-Binding Proteins - genetics | STAT1 Transcription Factor - metabolism | Viral Nonstructural Proteins - chemistry | Yellow Fever - metabolism | Yellow Fever - virology | STAT1 Transcription Factor - chemistry | Yellow fever virus - metabolism | Cell Line | Signal Transduction | Viral Nonstructural Proteins - genetics | STAT1 Transcription Factor - genetics | Amino Acid Motifs | Host-Pathogen Interactions | Animals | Interferon-beta - metabolism | STAT2 Transcription Factor - genetics | Protein Binding | Yellow Fever - genetics | Viral Nonstructural Proteins - metabolism | STAT2 Transcription Factor - metabolism | Viral Nonstructural Proteins - antagonists & inhibitors | GTP-Binding Proteins - metabolism | Yellow fever virus - genetics | Interferon | Biological response modifiers | Ligases | Health aspects | Yellow fever | Index Medicus
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7. Full Text A positive feedback amplifier circuit that regulates interferon (IFN)-stimulated gene expression and controls type I and type II IFN responses
by Michalska, Agata and Blaszczyk, Katarzyna and Wesoly, Joanna and Bluyssen, Hans A.R
Frontiers in immunology, ISSN 1664-3224, 05/2018, Volume 9, Issue MAY, pp. 1135 - 1135
Interferon-stimulated gene factor 3 | Signal transducer and activator of transcriptions | Antiviral activity | Interferon | Interferon regulatory factor 1 | JAK/signal transducer and activator of transcription signaling pathway | Transcriptional regulation | Life Sciences & Biomedicine | Immunology | Science & Technology | Genome-Wide Association Study | Humans | Gene Expression Regulation | Interferon Regulatory Factors - metabolism | Interferon Regulatory Factors - genetics | Feedback, Physiological | STAT1 Transcription Factor - metabolism | Animals | Interferon-Stimulated Gene Factor 3 - genetics | Interferon-Stimulated Gene Factor 3 - metabolism | STAT2 Transcription Factor - chemistry | STAT1 Transcription Factor - chemistry | Germ-Line Mutation | Interferons - metabolism | Protein Binding | STAT2 Transcription Factor - metabolism | Mutation | Physiological aspects | Genetic aspects | Immune response | Research | Gene expression | interferon-stimulated gene factor 3 | interferon regulatory factor 1 | interferon | transcriptional regulation | signal transducer and activator of transcriptions
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8. Full Text STAT2 is an essential adaptor in USP18-mediated suppression of type i interferon signaling
by Arimoto, Kei-Ichiro and Löchte, Sara and Stoner, Samuel A and Burkart, Christoph and Zhang, Yue and Miyauchi, Sayuri and Wilmes, Stephan and Fan, Jun-Bao and Heinisch, Jürgen J and Li, Zhi and Yan, Ming and Pellegrini, Sandra and Colland, Frédéric and Piehler, Jacob and Zhang, Dong-Er
Nature structural & molecular biology, ISSN 1545-9993, 03/2017, Volume 24, Issue 3, pp. 279 - 289
Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Endopeptidases - metabolism | Signal Transduction | Humans | Immunoblotting | Mutant Proteins - metabolism | Two-Hybrid System Techniques | Feedback, Physiological | Animals | Mutant Proteins - chemistry | STAT2 Transcription Factor - chemistry | Cell Line, Tumor | Interferon Type I - metabolism | Protein Binding | Protein Domains | Mice | STAT2 Transcription Factor - metabolism | Receptor, Interferon alpha-beta - metabolism | Genetic susceptibility | Cellular signal transduction | Genetic aspects | Ubiquitin-proteasome system | Gene expression | Health aspects | STAT proteins | Proteins | Enzymes | Pathogens | Genetic disorders | Cytokines | Index Medicus | STAT2 Transcription Factor | Biochemistry, Molecular Biology | Cellular Biology | Interferon Type I | Receptor, Interferon alpha-beta | Mutant Proteins | Life Sciences | Endopeptidases
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