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1. Full Text IL28B inhibits hepatitis C virus replication through the JAK–STAT pathway
by Zhang, Leiliang and Jilg, Nikolaus and Shao, Run-Xuan and Lin, Wenyu and Fusco, Dahlene N and Zhao, Hong and Goto, Kaku and Peng, Lee F and Chen, Wen-Chi and Chung, Raymond T
Journal of Hepatology, ISSN 0168-8278, 2010, Volume 55, Issue 2, pp. 289 - 298
Background & Aims The combination of pegylated interferon (IFN) α and ribavirin (RBV) is the standard therapy for patients with chronic HCV infection. However,... 
Gastroenterology and Hepatology | IL28B | STAT | HCV | JAK | IFNS | CLEARANCE | INNATE | RIBAVIRIN | GENOME | LAMBDA | GENETIC-VARIATION | ANTIVIRAL RESPONSES | INTERFERON-ALPHA | CELL-CULTURE | GASTROENTEROLOGY & HEPATOLOGY | RNA, Small Interfering - genetics | Humans | STAT Transcription Factors - metabolism | TYK2 Kinase - metabolism | DNA Primers - genetics | Janus Kinases - metabolism | Interleukins - genetics | Janus Kinase 1 - metabolism | STAT1 Transcription Factor - metabolism | Base Sequence | Hepacivirus - physiology | TYK2 Kinase - antagonists & inhibitors | Interleukins - pharmacology | Phosphorylation - drug effects | Hepacivirus - drug effects | Cell Line | Virus Replication - drug effects | Antiviral Agents - pharmacology | Recombinant Proteins - pharmacology | Interferon-Stimulated Gene Factor 3, gamma Subunit - genetics | Janus Kinase 1 - antagonists & inhibitors | STAT1 Transcription Factor - genetics | Interferon-Stimulated Gene Factor 3, gamma Subunit - metabolism | STAT1 Transcription Factor - antagonists & inhibitors | Receptors, Interferon - metabolism | Janus Kinases - antagonists & inhibitors | STAT2 Transcription Factor - antagonists & inhibitors | Signal Transduction - drug effects | STAT2 Transcription Factor - genetics | Polymorphism, Single Nucleotide | STAT Transcription Factors - antagonists & inhibitors | STAT2 Transcription Factor - metabolism | Virus Replication - physiology | Interferon-Stimulated Gene Factor 3, gamma Subunit - antagonists & inhibitors | Virus diseases | Genetic research | Precipitation (Meteorology) | Biological response modifiers | Hepatitis C virus | Health aspects
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2. Full Text Dual regulation of Stat1 and Stat3 by the tumor suppressor protein PML contributes to interferon α-mediated inhibition of angiogenesis
by Hsu, Kuo-Sheng and Zhao, Xuan and Cheng, Xiwen and Guan, Dongyin and Mahabeleshwar, Ganapati H and Liu, Yu and Borden, Ernest and Jain, Mukesh K and Kao, Hung-Ying
Journal of Biological Chemistry, ISSN 0021-9258, 06/2017, Volume 292, Issue 24, pp. 10048 - 10060
IFNs are effective in inhibiting angiogenesis in preclinical models and in treating several angioproliferative disorders. However, the detailed mechanisms of... 
APOPTOSIS | ACTIVATION | NETWORK | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOTHELIAL-CELLS | TNF-ALPHA | INDUCTION | RAR-ALPHA | TRANSCRIPTION FACTOR | EXPRESSION | Endothelium, Vascular - cytology | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Neovascularization, Pathologic - pathology | Endopeptidases - chemistry | STAT1 Transcription Factor - metabolism | Interferon-alpha - genetics | RNA Interference | Human Umbilical Vein Endothelial Cells - cytology | STAT1 Transcription Factor - agonists | Promyelocytic Leukemia Protein - metabolism | Ubiquitin Thiolesterase - metabolism | Neovascularization, Pathologic - prevention & control | Ubiquitin Thiolesterase - antagonists & inhibitors | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Promyelocytic Leukemia Protein - genetics | Recombinant Proteins - metabolism | Cell Line | Endopeptidases - metabolism | Interferon-alpha - metabolism | Cells, Cultured | STAT1 Transcription Factor - genetics | Ubiquitin Thiolesterase - genetics | Mice, Knockout | STAT2 Transcription Factor - agonists | Animals | Endopeptidases - genetics | Endothelium, Vascular - metabolism | STAT2 Transcription Factor - genetics | Promyelocytic Leukemia Protein - antagonists & inhibitors | Endothelium, Vascular - pathology | Neovascularization, Pathologic - metabolism | Protein Processing, Post-Translational | STAT2 Transcription Factor - metabolism | STAT3 Transcription Factor - antagonists & inhibitors | Neovascularization, Physiologic | signal transducers and activators of transcription 1 (STAT1) | STAT transcription factor | Signal Transduction | interferon | angiogenesis | STAT3
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3. Full Text Zika Virus Targets Human STAT2 to Inhibit Type I Interferon Signaling
by Grant, Alesha and Ponia, Sanket S and Tripathi, Shashank and Balasubramaniam, Vinod and Miorin, Lisa and Sourisseau, Marion and Schwarz, Megan C and Sánchez-Seco, Mari Paz and Evans, Matthew J and Best, Sonja M and García-Sastre, Adolfo
Cell Host & Microbe, ISSN 1931-3128, 06/2016, Volume 19, Issue 6, pp. 882 - 890
The ongoing epidemic of Zika virus (ZIKV) illustrates the importance of flaviviruses as emerging human pathogens. All vector-borne flaviviruses studied thus... 
ACTIVATION | ANTAGONIST | WEST-NILE-VIRUS | MICROBIOLOGY | MECHANISMS | IDENTIFICATION | EMERGENCE | EVOLUTION | VIROLOGY | FLAVIVIRUS | INFECTION | NS5 PROTEIN | PARASITOLOGY | Signal Transduction | Humans | Ubiquitin-Protein Ligases - metabolism | Cercopithecus aethiops | Viral Nonstructural Proteins - genetics | Calmodulin-Binding Proteins - metabolism | Phylogeny | Zika Virus Infection - metabolism | HEK293 Cells - drug effects | Zika Virus Infection - virology | Animals | Base Sequence | Interferon Type I - metabolism | Cytoskeletal Proteins - metabolism | Viral Nonstructural Proteins - metabolism | Mice | STAT2 Transcription Factor - metabolism | Vero Cells | Zika Virus - metabolism | Disease Models, Animal | Interferon Type I - antagonists & inhibitors | Virus diseases | Ubiquitin | Medical colleges | Microbiology | Ligases | Dengue viruses | Interferon | Biological response modifiers
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4. Full Text A variant upstream of IFNL3 (IL28B) creating a new interferon gene IFNL4 is associated with impaired clearance of hepatitis C virus
by Prokunina-Olsson, Ludmila and Muchmore, Brian and Tang, Wei and Pfeiffer, Ruth M and Park, Heiyoung and Dickensheets, Harold and Hergott, Dianna and Porter-Gill, Patricia and Mumy, Adam and Kohaar, Indu and Chen, Sabrina and Brand, Nathan and Tarway, McAnthony and Liu, Luyang and Sheikh, Faruk and Astemborski, Jacquie and Bonkovsky, Herbert L and Edlin, Brian R and Howell, Charles D and Morgan, Timothy R and Thomas, David L and Rehermann, Barbara and Donnelly, Raymond P and O'Brien, Thomas R
Nature Genetics, ISSN 1061-4036, 02/2013, Volume 45, Issue 2, pp. 164 - 171
Chronic infection with hepatitis C virus (HCV) is a common cause of liver cirrhosis and cancer. We performed RNA sequencing in primary human hepatocytes... 
GENOTYPE 1 | EARLY VIRAL KINETICS | THERAPY | INTERLEUKIN 28B | STIMULATED GENES | MULTIETHNIC COHORT | GENETICS & HEREDITY | EXPRESSION | PEGINTERFERON | GENOME-WIDE ASSOCIATION | LAMBDA | Phosphorylation | Species Specificity | Humans | Gene Expression Profiling | Antibodies, Monoclonal | Interleukins - metabolism | Hep G2 Cells | Linkage Disequilibrium | Chromosomes, Human, Pair 19 - genetics | Hepatitis C - immunology | Interleukins - genetics | Microscopy, Confocal | STAT1 Transcription Factor - metabolism | Sequence Analysis, RNA | Models, Biological | Genetic Markers - genetics | Interleukins - immunology | Statistics, Nonparametric | Polymorphism, Genetic - genetics | STAT2 Transcription Factor - metabolism | Hepatitis C - prevention & control | Causes of | Interferon | Genetic aspects | Research | Hepatitis C virus | Health aspects | Liver cirrhosis | Proteins | Genealogy | Genomes | Diabetes | Experiments | Epidemiology | Cancer
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5. Full Text IFNβ‐dependent increases in STAT1, STAT2, and IRF9 mediate resistance to viruses and DNA damage
by Cheon, HyeonJoo and Holvey‐Bates, Elise G and Schoggins, John W and Forster, Samuel and Hertzog, Paul and Imanaka, Naoko and Rice, Charles M and Jackson, Mark W and Junk, Damian J and Stark, George R
The EMBO Journal, ISSN 0261-4189, 10/2013, Volume 32, Issue 20, pp. 2751 - 2763
A single high dose of interferon‐β (IFNβ) activates powerful cellular responses, in which many anti‐viral, pro‐apoptotic, and anti‐proliferative proteins are... 
DNA damage resistance | interferon‐β | unphosphorylated interferon‐stimulated gene factor 3 (U‐ISGF3) | anti‐viral genes | interferon-β | anti-viral genes | unphosphorylated interferon-stimulated gene factor 3 (U-ISGF3) | PROTEIN | CANCER CELLS | IKK-EPSILON | BIOCHEMISTRY & MOLECULAR BIOLOGY | IDENTIFICATION | RADIATION | CELL BIOLOGY | BREAST-CANCER | interferon-beta | ALPHA/BETA-INTERFERON-PRODUCTION | GENE-EXPRESSION | ANTIVIRAL ACTIVITY | UP-REGULATION | Tumor Escape - drug effects | Viruses - pathogenicity | Tumor Escape - genetics | Humans | Cercopithecus aethiops | STAT1 Transcription Factor - metabolism | DNA Damage - genetics | Virus Diseases - genetics | Disease Resistance - genetics | Vero Cells | DNA Damage - drug effects | Antiviral Agents - pharmacology | Virus Diseases - immunology | Cells, Cultured | Disease Resistance - drug effects | Gene Expression Regulation - physiology | Viruses - drug effects | Viruses - immunology | Interferon-Stimulated Gene Factor 3, gamma Subunit - genetics | STAT1 Transcription Factor - genetics | Interferon-Stimulated Gene Factor 3, gamma Subunit - metabolism | Gene Expression Regulation - drug effects | Animals | Interferon-beta - pharmacology | STAT2 Transcription Factor - genetics | STAT2 Transcription Factor - metabolism | Virus Diseases - prevention & control
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6. Full Text High basal STAT4 balanced by STAT1 induction to control type 1 interferon effects in natural killer cells
by Miyagi, Takuya and Gil, M. Pilar and Wang, Xin and Louten, Jennifer and Chu, Wen-Ming and Biron, Christine A
Journal of Experimental Medicine, ISSN 0022-1007, 10/2007, Volume 204, Issue 10, pp. 2383 - 2396
The best- characterized type 1 interferon ( IFN) signaling pathway depends on signal transducer and activator of transcription 1 ( STAT1) and STAT2. The... 
MEDICINE, RESEARCH & EXPERIMENTAL | IMMUNE-RESPONSE | TARGETED DISRUPTION | MURINE CYTOMEGALOVIRUS-INFECTION | ANTIVIRAL DEFENSE | GENERALIZED SHWARTZMAN REACTION | IFN-GAMMA | IN-VIVO | CHORIOMENINGITIS VIRUS-INFECTION | NECROSIS-FACTOR-ALPHA | IMMUNOLOGY | VIRAL-INFECTION | Lymphocytic choriomeningitis virus - immunology | STAT1 Transcription Factor - deficiency | Lymphocyte Activation | Gene Expression Regulation | STAT4 Transcription Factor - metabolism | Mice, Transgenic | Interferon-gamma - metabolism | Lymphocytic Choriomeningitis - immunology | STAT1 Transcription Factor - genetics | STAT2 Transcription Factor - deficiency | Lymphocytic Choriomeningitis - metabolism | STAT1 Transcription Factor - metabolism | Animals | STAT2 Transcription Factor - genetics | Interferon Type I - genetics | Interferon Type I - metabolism | Killer Cells, Natural - immunology | STAT4 Transcription Factor - genetics | Mice | STAT2 Transcription Factor - metabolism | Killer Cells, Natural - metabolism | STAT4 Transcription Factor - deficiency | Receptor, Interferon alpha-beta - metabolism
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7. Full Text The Interferon Signaling Antagonist Function of Yellow Fever Virus NS5 Protein Is Activated by Type I Interferon
by Laurent-Rolle, Maudry and Morrison, Juliet and Rajsbaum, Ricardo and Macleod, Jesica M. Levingston and Pisanelli, Giuseppe and Pham, Alissa and Ayllon, Juan and Miorin, Lisa and Martínez-Romero, Carles and tenOever, Benjamin R and García-Sastre, Adolfo
Cell Host & Microbe, ISSN 1931-3128, 09/2014, Volume 16, Issue 3, pp. 314 - 327
To successfully establish infection, flaviviruses have to overcome the antiviral state induced by type I interferon (IFN-I). The nonstructural NS5 proteins of... 
PATHWAYS | HUMAN-CELLS | MICROBIOLOGY | IDENTIFICATION | ALPHA-INTERFERON | ANTIVIRAL RESPONSE | STAT | REPLICATION | VIROLOGY | FLAVIVIRUS | WEST NILE VIRUS | NUCLEOTIDE-SEQUENCE | PARASITOLOGY | Phosphorylation | Humans | GTP-Binding Proteins - genetics | STAT1 Transcription Factor - metabolism | Viral Nonstructural Proteins - chemistry | Yellow Fever - metabolism | Yellow Fever - virology | STAT1 Transcription Factor - chemistry | Yellow fever virus - metabolism | Cell Line | Signal Transduction | Viral Nonstructural Proteins - genetics | STAT1 Transcription Factor - genetics | Amino Acid Motifs | Host-Pathogen Interactions | Animals | Interferon-beta - metabolism | STAT2 Transcription Factor - genetics | Protein Binding | Yellow Fever - genetics | Viral Nonstructural Proteins - metabolism | STAT2 Transcription Factor - metabolism | Viral Nonstructural Proteins - antagonists & inhibitors | GTP-Binding Proteins - metabolism | Yellow fever virus - genetics | Interferon | Biological response modifiers | Ligases | Health aspects | Yellow fever
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8. Full Text Positive Role of Promyelocytic Leukemia Protein in Type I Interferon Response and Its Regulation by Human Cytomegalovirus
by Kim, Young-Eui and Ahn, Jin-Hyun
PLoS Pathogens, ISSN 1553-7366, 03/2015, Volume 11, Issue 3, pp. 1 - 24
Promyelocytic leukemia protein (PML), a major component of PML nuclear bodies (also known as nuclear domain 10), is involved in diverse cellular processes such... 
VIRAL GENE-EXPRESSION | MICROBIOLOGY | INTRINSIC IMMUNITY | STIMULATED TRANSCRIPTION | DNA-REPLICATION | NUCLEAR-BODIES | ANTIVIRAL DEFENSE | VIROLOGY | PML PROTEIN | IE1 PROTEIN | POLYMERASE-II | HISTONE DEACETYLASE | PARASITOLOGY | Humans | Immediate-Early Proteins - metabolism | STAT1 Transcription Factor - metabolism | Tumor Suppressor Proteins - genetics | HEK293 Cells | Interferon Type I - metabolism | Nuclear Proteins - genetics | Histone Deacetylase 1 - genetics | Cytomegalovirus - genetics | Histone Deacetylase 2 - genetics | Tumor Suppressor Proteins - metabolism | Cytomegalovirus - metabolism | Nuclear Proteins - metabolism | Transcription Factors - genetics | Interferon-Stimulated Gene Factor 3, gamma Subunit - genetics | STAT1 Transcription Factor - genetics | Interferon-Stimulated Gene Factor 3, gamma Subunit - metabolism | Transcription Factors - metabolism | Immediate-Early Proteins - genetics | STAT2 Transcription Factor - genetics | Interferon Type I - genetics | STAT2 Transcription Factor - metabolism | Histone Deacetylase 2 - metabolism | Promyelocytic Leukemia Protein | Histone Deacetylase 1 - metabolism | Interferon | Genetic aspects | Host-virus relationships | Viral proteins | Cytomegaloviruses | Health aspects | Cytomegalovirus | Viruses | Infections | Genomes | Kinases | Gene expression | Proteins | Plasmids | Deoxyribonucleic acid | DNA | Viral infections | Immune system | Apoptosis
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9. Full Text NS5 of Dengue Virus Mediates STAT2 Binding and Degradation
by Joseph Ashour and Maudry Laurent-Rolle and Pei-Yong Shi and Adolfo García-Sastre
Journal of Virology, ISSN 0022-538X, 06/2009, Volume 83, Issue 11, pp. 5408 - 5418
Article Usage Stats Services JVI Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
ACTIVATION | ANTIVIRAL STATE | VIROLOGY | TYROSINE PHOSPHORYLATION | I INTERFERON RECEPTOR | WEST-NILE-VIRUS | C-PROTEIN | STIMULATED-GENE FACTOR-3 | ALPHA-INTERFERON | V-PROTEIN | SIGNAL TRANSDUCER | Cell Line | Humans | Gene Expression Regulation | Ubiquitin - metabolism | Cercopithecus aethiops | Viral Nonstructural Proteins - genetics | Amino Acids - genetics | Amino Acids - metabolism | Dengue Virus - genetics | Animals | STAT2 Transcription Factor - genetics | Protein Binding | Viral Nonstructural Proteins - metabolism | STAT2 Transcription Factor - metabolism | Dengue Virus - metabolism | Proteasome Endopeptidase Complex - metabolism | Cellular Response to Infection
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10. Full Text The chemopreventive phytochemical moringin isolated from Moringa oleifera seeds inhibits JAK/STAT signaling
by Michl, Carina and Vivarelli, Fabio and Weigl, Julia and De Nicola, Gina Rosalinda and Canistro, Donatella and Paolini, Moreno and Iori, Renato and Rascle, Anne
PLoS ONE, ISSN 1932-6203, 06/2016, Volume 11, Issue 6, pp. e0157430 - e0157430
Sulforaphane (SFN) and moringin (GMG-ITC) are edible isothiocyanates present as glucosinolate precursors in cruciferous vegetables and in the plant Moringa... 
CANCER-CELLS | GLUCOMORINGIN ISOTHIOCYANATE | JAK-STAT PATHWAY | CHEMICAL BIOLOGY | TRANSCRIPTION 3 | MULTIDISCIPLINARY SCIENCES | ENDOTHELIAL-CELLS | IN-VIVO | DEACETYLASE INHIBITORS | MONOCYTE ADHESION | NF-KAPPA-B | Interferon-alpha - pharmacology | Gene Expression - drug effects | Humans | Precursor Cells, B-Lymphoid - metabolism | STAT Transcription Factors - metabolism | Cell Survival - genetics | Janus Kinases - metabolism | Precursor Cells, B-Lymphoid - drug effects | Interleukin-3 - pharmacology | Dose-Response Relationship, Drug | Moringa oleifera - chemistry | STAT5 Transcription Factor - genetics | STAT5 Transcription Factor - metabolism | STAT1 Transcription Factor - metabolism | Isothiocyanates - chemistry | Molecular Structure | Seeds - chemistry | Isothiocyanates - isolation & purification | Phosphorylation - drug effects | Isothiocyanates - pharmacology | Cell Line | Cell Survival - drug effects | Janus Kinases - genetics | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | STAT1 Transcription Factor - genetics | Blotting, Western | STAT Transcription Factors - genetics | Animals | Signal Transduction - drug effects | STAT2 Transcription Factor - genetics | Mice | STAT2 Transcription Factor - metabolism | HeLa Cells | Biotechnology | Phosphorylation | Transformation | Vegetables | Seeds | Disorders | Kinases | Inflammatory diseases | Mimicry | Signal transduction | Toxicology | Cell growth | Immunology | Illnesses | Pathways | Interleukin 3 | Rodents | Stat1 protein | Sulforafan | Medicinal plants | Stat2 protein | NF-κB protein | Cell survival | Stat5 protein | Gene expression | Disease prevention | Studies | Signaling | Diet | Tumor necrosis factor | Cell death | Pharmacy | Prostate cancer | Binding sites | Cancer | Index Medicus
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11. Full Text Acetylation-Dependent Signal Transduction for Type I Interferon Receptor
by Tang, Xiaoli and Gao, Jin-Song and Guan, Ying-jie and McLane, Katya E and Yuan, Zheng-Long and Ramratnam, Bharat and Chin, Y. Eugene
Cell, ISSN 0092-8674, 2007, Volume 131, Issue 1, pp. 93 - 105
Cytokine-activated receptors initiate intracellular signaling by recruiting protein kinases that phosphorylate the receptors on tyrosine residues, thus... 
PROTEINS | MOLIMMUNO | SIGNALING | APOPTOSIS | AUTOINHIBITION | COMPLEX-FORMATION | DNA-BINDING | DOMAIN | PHOSPHORYLATION | CRYSTAL-STRUCTURE | STAT2 | BIOCHEMISTRY & MOLECULAR BIOLOGY | ALPHA-STIMULATED TRANSCRIPTION | PREASSOCIATION | CELL BIOLOGY | RNA, Small Interfering - genetics | Humans | Molecular Sequence Data | Recombinant Fusion Proteins - metabolism | CREB-Binding Protein - genetics | CREB-Binding Protein - metabolism | STAT1 Transcription Factor - metabolism | Lysine - metabolism | Receptor, Interferon alpha-beta - genetics | Acetylation | Binding Sites | Receptors, Interferon - genetics | Receptor, Interferon alpha-beta - metabolism | Amino Acid Sequence | Interferon-alpha - metabolism | Interferon-Stimulated Gene Factor 3, gamma Subunit - genetics | STAT1 Transcription Factor - genetics | Interferon-Stimulated Gene Factor 3, gamma Subunit - metabolism | Serine - metabolism | Receptors, Interferon - metabolism | Sequence Alignment | Animals | STAT2 Transcription Factor - genetics | Protein Binding | Recombinant Fusion Proteins - genetics | Signal Transduction - physiology | STAT2 Transcription Factor - metabolism | HeLa Cells | RNA, Small Interfering - metabolism | Immunological adjuvants | Recombinant proteins | Interferon
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12. Full Text Porcine Reproductive and Respiratory Syndrome Virus Nsp1β Inhibits Interferon-Activated JAK/STAT Signal Transduction by Inducing Karyopherin-α1 Degradation
by Rong Wang and Yuchen Nan and Ying Yu and Yan-Jin Zhang
Journal of Virology, ISSN 0022-538X, 05/2013, Volume 87, Issue 9, pp. 5219 - 5228
Article Usage Stats Services JVI Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
alpha Karyopherins - metabolism | Cell Line | Interferon-alpha - metabolism | Janus Kinases - genetics | Signal Transduction | Down-Regulation | Porcine Reproductive and Respiratory Syndrome - metabolism | Viral Nonstructural Proteins - genetics | Janus Kinases - metabolism | STAT1 Transcription Factor - genetics | Porcine respiratory and reproductive syndrome virus - genetics | Porcine Reproductive and Respiratory Syndrome - virology | Porcine respiratory and reproductive syndrome virus - metabolism | STAT1 Transcription Factor - metabolism | Animals | Interferon-alpha - genetics | Porcine Reproductive and Respiratory Syndrome - genetics | Proteolysis | Swine | STAT2 Transcription Factor - genetics | alpha Karyopherins - genetics | Viral Nonstructural Proteins - metabolism | STAT2 Transcription Factor - metabolism | Virus-Cell Interactions
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