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Nature Medicine, ISSN 1078-8956, 04/2010, Volume 16, Issue 4, pp. 452 - 459
Journal Article
Cellular and Molecular Immunology, ISSN 1672-7681, 05/2015, Volume 12, Issue 3, pp. 326 - 341
Journal Article
Immunity, ISSN 1074-7613, 05/2014, Volume 40, Issue 5, pp. 772 - 784
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 147, Issue 4, pp. 847 - 859.e11
Background & Aims Constitutive activation of the transcription factors nuclear factor κB (NF-κB) and STAT3 is involved in the development and progression of... 
Gastroenterology and Hepatology | microRNA 222 | microRNA 221 | Carcinogenesis | Colon Cancer | microRNA | 222 Colon | Cancer | COLITIS-ASSOCIATED CANCER | IKK-ALPHA | TRANSLATION | COLON-CANCER | PATHWAY | INFLAMMATION | DIFFERENTIATION | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | TUMORIGENESIS | PROGRESSION | Cell Proliferation | Colitis - genetics | Colorectal Neoplasms - genetics | Humans | Open Reading Frames | Gene Expression Regulation, Neoplastic | Colitis - pathology | Male | MicroRNAs - metabolism | NF-kappa B - metabolism | LIM Domain Proteins - metabolism | RNA, Messenger - metabolism | Transfection | Colitis - therapy | RNA Interference | Time Factors | Microfilament Proteins - metabolism | 3' Untranslated Regions | Binding Sites | Microfilament Proteins - genetics | Colorectal Neoplasms - metabolism | Genes, Reporter | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Signal Transduction | Cell Survival | HCT116 Cells | Mice, Inbred C57BL | Colorectal Neoplasms - prevention & control | Tumor Burden | HT29 Cells | Xenograft Model Antitumor Assays | Feedback, Physiological | Animals | NF-kappa B - genetics | Transcription Factor RelA - metabolism | Mice, Nude | Colitis - metabolism | LIM Domain Proteins - genetics | Mice | Mice, Inbred BALB C | Colorectal Neoplasms - pathology | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2011, Volume 56, Issue 1, pp. 184 - 191
Background & Aims Mosaic G-protein alpha-subunit ( GNAS )-activating mutations are responsible for the McCune–Albright (MCA) syndrome. This oncogene that... 
Gastroenterology and Hepatology | CRP | interleukine 6 signal transducer | GTP | C-reactive protein | jun N-terminal kinase | serum amyloid A | JNK | macroregenerative nodule | complementary deoxyribonucleic acid | MRN | mitogen activated proteins kinase | cAMP | glycoprotein 130 | neuroendocrine secretory protein 55 | hepatocyte nuclear factor 1A | G-protein alpha stimulatory subunit | cDNA | gp130 | protein kinase A | hepatocellular carcinoma | β catenin | IHCA | McCune-Albright syndrome | HCA | PKA | HCC | MAP kinase | MCA | HNF1A | inflammatory hepatocellular adenoma | SAA | cyclic adenosine monophosphate | hepatocellular adenoma | GNAS | CTNNB1 | NESP55 | guanosine-5′-triphosphate | IL6ST | INACTIVATION | PATHWAY | HEPATOCELLULAR ADENOMA | GS-ALPHA | GROWTH | CLASSIFICATION | HUMAN CANCERS | MCCUNE-ALBRIGHT-SYNDROME | GASTROENTEROLOGY & HEPATOLOGY | G-PROTEIN | BINDING | Liver Neoplasms - classification | Humans | Middle Aged | Adenoma, Liver Cell - pathology | Male | Fibrous Dysplasia, Polyostotic - genetics | GTP-Binding Protein alpha Subunits, Gs - genetics | Chromogranins | DNA Mutational Analysis | Base Sequence | Carcinoma, Hepatocellular - genetics | Adult | Female | Liver Neoplasms - pathology | Adenoma, Liver Cell - genetics | STAT3 Transcription Factor - metabolism | Liver Neoplasms - genetics | Signal Transduction | Adenoma, Liver Cell - metabolism | Models, Biological | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Aged | DNA, Neoplasm - genetics | Mutation | Carcinoma, Hepatocellular - metabolism | Liver cancer | Genetic aspects | Nucleic acids | Adenylic acid | Mitogens | Protein kinases | Index Medicus | GTP-Binding Protein alpha Subunits, Gs | Fibrous Dysplasia, Polyostotic | Liver Neoplasms | Life Sciences | Adenoma, Liver Cell | DNA, Neoplasm | STAT3 Transcription Factor | Carcinoma, Hepatocellular | Cancer
Journal Article
Biochemical Pharmacology, ISSN 0006-2952, 10/2017, Volume 142, pp. 46 - 57
Journal Article
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 1130 - 16
Journal Article
Molecular and Cellular Biochemistry, ISSN 0300-8177, 9/2014, Volume 394, Issue 1, pp. 77 - 90
Human papilloma virus is associated with cervical and other tumors, and several cellular conditions also play an important role in carcinogenesis. Human... 
Life Sciences | Biochemistry, general | NF-κB | TNF-α | STAT3 | Medical Biochemistry | Oncology | HPV16 E2 | Cardiology | IL-6 | RelA | HUMAN-PAPILLOMAVIRUS TYPE-16 | TNF-alpha | NECROSIS-FACTOR-ALPHA | CERVICAL INTRAEPITHELIAL NEOPLASIA | UNPHOSPHORYLATED STAT3 | CELL BIOLOGY | UTERINE CERVIX | NF-kappa B | CANCER DEVELOPMENT | HELA-CELLS | HIGH-RISK | SQUAMOUS-CELL CARCINOMA | E2 PROTEIN | Epithelial Cells - metabolism | Apoptosis - drug effects | Epithelial Cells - drug effects | Humans | Human papillomavirus 16 - immunology | Transcription Factor RelA - genetics | DNA-Binding Proteins - metabolism | Dose-Response Relationship, Drug | Papillomavirus Infections - pathology | Transfection | MCF-7 Cells | RNA Interference | HEK293 Cells | Papillomavirus Infections - immunology | Oncogene Proteins, Viral - genetics | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Oncogene Proteins, Viral - metabolism | Cell Survival - drug effects | Human papillomavirus 16 - pathogenicity | DNA-Binding Proteins - genetics | Human papillomavirus 16 - metabolism | Apoptosis Regulatory Proteins - metabolism | Papillomavirus Infections - genetics | Tumor Necrosis Factor-alpha - pharmacology | Signal Transduction - drug effects | Transcription Factor RelA - metabolism | Papillomavirus Infections - metabolism | Epithelial Cells - immunology | Epithelial Cells - virology | HeLa Cells | Human papillomavirus 16 - genetics | Index Medicus
Journal Article
Nature immunology, ISSN 1529-2908, 2005, Volume 6, Issue 8, pp. 844 - 851
Journal Article
by Yu, JP and Wang, Y and Yan, F and Zhang, P and Li, H and Zhao, H and Yan, CH and Ren, XB
JOURNAL OF IMMUNOLOGY, ISSN 0022-1767, 09/2014, Volume 193, Issue 5, pp. 2574 - 2586
Immunotherapy for cancer treatment is achieved through the activation of competent immune effector cells and the inhibition of immunosuppressive cells, such as... 
LUNG-CANCER | STAT3 ACTIVATION | SIGNALING PATHWAYS | DENDRITIC CELLS | SYNERGISTIC INDUCTION | IFN-GAMMA | INDOLEAMINE 2,3-DIOXYGENASE | IKK-ALPHA | TNF-ALPHA | IMMUNOLOGY | BLOOD MONONUCLEAR-CELLS | Cell Nucleus - immunology | Indoleamine-Pyrrole 2,3,-Dioxygenase - genetics | Triterpenes - pharmacology | Breast Neoplasms - immunology | Humans | Transcription Factor RelB - immunology | Neoplasm Proteins - immunology | Indoleamine-Pyrrole 2,3,-Dioxygenase - immunology | Cell Nucleus - pathology | Myeloid Cells - immunology | Female | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | Active Transport, Cell Nucleus - genetics | STAT3 Transcription Factor - genetics | Transcription Factor RelB - genetics | Interleukin-6 - genetics | Protein-Serine-Threonine Kinases - genetics | Gene Expression Regulation, Neoplastic - immunology | Up-Regulation - drug effects | Breast Neoplasms - genetics | Cell Nucleus - genetics | Up-Regulation - immunology | Breast Neoplasms - pathology | Active Transport, Cell Nucleus - drug effects | Interleukin-6 - immunology | Protein-Serine-Threonine Kinases - immunology | Active Transport, Cell Nucleus - immunology | Myeloid Cells - pathology | STAT3 Transcription Factor - immunology | Index Medicus | Abridged Index Medicus | MDSCs | Breast cancer | IDO | STAT3 | Noncanonical NF-κB
Journal Article