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1. Full Text Programmed death-1-induced interleukin-10 production by monocytes impairs CD4 + T cell activation during HIV infection
by Said, Elias A and Dupuy, Franck P and Trautmann, Lydie and Zhang, Yuwei and Shi, Yu and El-Far, Mohamed and Hill, Brenna J and Noto, Alessandra and Ancuta, Petronela and Peretz, Yoav and Fonseca, Simone G and Van Grevenynghe, Julien and Boulassel, Mohamed R and Bruneau, Julie and Shoukry, Naglaa H and Routy, Jean-Pierre and Douek, Daniel C and Haddad, Elias K and Sekaly, Rafick-Pierre
Nature medicine, ISSN 1078-8956, 2010, Volume 16, Issue 4, pp. 452 - 459
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Phosphorylation | Antigens, CD - biosynthesis | Humans | Monocytes - immunology | CD4-Positive T-Lymphocytes - physiology | Interleukin-10 - physiology | Lymphocyte Subsets - immunology | CD4-Positive T-Lymphocytes - immunology | HIV Infections - immunology | Lymphocyte Activation - immunology | STAT3 Transcription Factor - physiology | Up-Regulation - physiology | CD4-Positive T-Lymphocytes - virology | Monocytes - physiology | Lymphocyte Subsets - physiology | Receptors, IgG - physiology | Viremia - physiopathology | B7-H1 Antigen | Receptors, IgG - immunology | Antigens, CD - physiology | Interleukin-10 - biosynthesis | Viremia - immunology | HIV Infections - physiopathology | STAT3 Transcription Factor - immunology | Lymphocyte Activation - physiology | Immunology | Cellular biology | Cytokines | Microbiology | Human immunodeficiency virus--HIV | Index Medicus
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2. Full Text The activating effect of IFN-γ on monocytes/macrophages is regulated by the LIF-trophoblast-IL-10 axis via Stat1 inhibition and Stat3 activation
by Dallagi, Angham and Girouard, Julie and Hamelin-Morrissette, Jovane and Dadzie, Rachel and Laurent, Laetitia and Vaillancourt, Cathy and Lafond, Julie and Carrier, Christian and Reyes-Moreno, Carlos
Cellular & molecular immunology, ISSN 1672-7681, 05/2015, Volume 12, Issue 3, pp. 326 - 341
macrophage deactivation | embryonic cell | gestational factors | cell migration/motility | inflammatory cell | Life Sciences & Biomedicine | Immunology | Science & Technology | Humans | Gene Expression Regulation | Chorionic Villi - immunology | Monocytes - immunology | STAT1 Transcription Factor - genetics | Leukemia Inhibitory Factor - metabolism | Macrophage Activation | Pregnancy - immunology | Trophoblasts - immunology | STAT1 Transcription Factor - metabolism | Interferon-gamma - immunology | Cell Line, Tumor | Interleukin-10 - metabolism | Female | Interleukin-10 - immunology | Macrophages - immunology | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Index Medicus | Interferon-gamma | Interleukin-10 | Macrophages | Pregnancy | Life Sciences | Monocytes | Trophoblasts | STAT1 Transcription Factor | STAT3 Transcription Factor | Chorionic Villi | Leukemia Inhibitory Factor | cell migration | motility
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3. Full Text Activation of STAT3 integrates common profibrotic pathways to promote fibroblast activation and tissue fibrosis
by Chakraborty, Debomita and Šumová, Barbora and Mallano, Tatjana and Chen, Chih-Wei and Distler, Alfiya and Bergmann, Christina and Ludolph, Ingo and Horch, Raymund E and Gelse, Kolja and Ramming, Andreas and Distler, Oliver and Schett, Georg and Šenolt, Ladislav and Distler, Jörg H. W
Nature communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 1130 - 16
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Phosphorylation | Skin - metabolism | Humans | Middle Aged | Collagen - chemistry | Male | Fibrosis - metabolism | Young Adult | Aminosalicylic Acids - chemistry | Adult | Female | STAT3 Transcription Factor - metabolism | Fibroblasts - metabolism | Scleroderma, Systemic - metabolism | Inflammation | Microscopy, Confocal | Animals | Benzenesulfonates - chemistry | Bleomycin - chemistry | Receptors, Transforming Growth Factor beta - metabolism | Biopsy | Adolescent | Signal Transduction - physiology | Aged | Mice | Enzyme Activation | Transforming Growth Factor beta - metabolism | Medical research | Animal models | Stat3 protein | Clinical trials | JNK protein | Activation | Pharmacology | Kinases | Ablation | Convergence | Signal transduction | Signaling | Pathways | Systemic sclerosis | Rodents | Fibrosis | Fibroblasts | Skin | In vivo methods and tests | Inhibition | Aberration | Index Medicus
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4. Full Text Activation of the protective Survivor Activating Factor Enhancement (SAFE) pathway against reperfusion injury: Does it go beyond the RISK pathway?
by Lecour, Sandrine
Journal of molecular and cellular cardiology, ISSN 0022-2828, 2009, Volume 47, Issue 1, pp. 32 - 40
Cardiovascular | Signal transducer and activator of transcription | Tumor necrosis factor alpha | Ischemia | Protective signaling pathways | Reperfusion injury | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cell Biology | Tumor Necrosis Factor-alpha - metabolism | Myocardial Reperfusion Injury - metabolism | Animals | Models, Biological | Humans | Tumor Necrosis Factor-alpha - physiology | Signal Transduction - physiology | STAT3 Transcription Factor - metabolism | Index Medicus
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5. Full Text IL-22+CD4+ T Cells Promote Colorectal Cancer Stemness via STAT3 Transcription Factor Activation and Induction of the Methyltransferase DOT1L
by Kryczek, Ilona and Lin, Yanwei and Nagarsheth, Nisha and Peng, Dongjun and Zhao, Lili and Zhao, Ende and Vatan, Linda and Szeliga, Wojciech and Dou, Yali and Owens, Scott and Zgodzinski, Witold and Majewski, Marek and Wallner, Grzegorz and Fang, Jingyuan and Huang, Emina and Zou, Weiping
Immunity (Cambridge, Mass.), ISSN 1074-7613, 05/2014, Volume 40, Issue 5, pp. 772 - 784
Life Sciences & Biomedicine | Immunology | Science & Technology | Receptors, CCR6 - immunology | Neoplasm Transplantation | Chemokine CCL20 - metabolism | Cell Proliferation | Homeodomain Proteins - metabolism | Humans | Methyltransferases - metabolism | Neoplastic Stem Cells - immunology | Homeodomain Proteins - immunology | Methyltransferases - immunology | Enzyme Activation - immunology | SOXB1 Transcription Factors - metabolism | CD4-Positive T-Lymphocytes - immunology | Chemokine CCL20 - immunology | SOXB1 Transcription Factors - immunology | Interleukins - immunology | Receptors, CCR6 - metabolism | Neoplastic Stem Cells - pathology | Octamer Transcription Factor-3 - immunology | STAT3 Transcription Factor - metabolism | Colorectal Neoplasms - mortality | Nanog Homeobox Protein | HT29 Cells | Animals | Colorectal Neoplasms - immunology | Octamer Transcription Factor-3 - metabolism | Cell Line, Tumor | Mice | STAT3 Transcription Factor - immunology | Colorectal Neoplasms - pathology | Medical research | Colon | Rodents | Colorectal cancer | Stem cells | Index Medicus | Th22 | colon cancer | DOT1L | STAT3 | ILC22 | Cancer stem cell | CCR6 | H3K79 | IL-22 | epigenetics
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6. Full Text A microRNA 221– and 222–Mediated Feedback Loop Maintains Constitutive Activation of NFκB and STAT3 in Colorectal Cancer Cells
by Liu, Sanhong and Sun, Xiaohua and Wang, Mingliang and Hou, Yingyong and Zhan, Yu and Jiang, Yuhang and Liu, Zhanjie and Cao, Xinwei and Chen, Pengfei and Liu, Zhi and Chen, Xi and Tao, Yu and Xu, Chen and Mao, Jie and Cheng, Chunyan and Li, Cuifeng and Hu, Yiming and Wang, Lunshan and Chin, Y. Eugene and Shi, Yufang and Siebenlist, Ulrich and Zhang, Xiaoren
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2014, Volume 147, Issue 4, pp. 847 - 859.e11
Gastroenterology and Hepatology | microRNA 222 | microRNA 221 | Carcinogenesis | Colon Cancer | microRNA | 222 Colon | Cancer | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Cell Proliferation | Colitis - genetics | Colorectal Neoplasms - genetics | Humans | Open Reading Frames | Gene Expression Regulation, Neoplastic | Colitis - pathology | Male | MicroRNAs - metabolism | NF-kappa B - metabolism | LIM Domain Proteins - metabolism | RNA, Messenger - metabolism | Transfection | Colitis - therapy | RNA Interference | Time Factors | Microfilament Proteins - metabolism | 3' Untranslated Regions | Binding Sites | Microfilament Proteins - genetics | Colorectal Neoplasms - metabolism | Genes, Reporter | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Signal Transduction | Cell Survival | HCT116 Cells | Mice, Inbred C57BL | Colorectal Neoplasms - prevention & control | Tumor Burden | HT29 Cells | Xenograft Model Antitumor Assays | Feedback, Physiological | Animals | NF-kappa B - genetics | Transcription Factor RelA - metabolism | Mice, Nude | Colitis - metabolism | LIM Domain Proteins - genetics | Mice | Mice, Inbred BALB C | Colorectal Neoplasms - pathology | Index Medicus | Abridged Index Medicus
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7. Full Text Constitutive activation of STAT3 in breast cancer cells: A review
by Banerjee, Kasturi and Resat, Haluk
International journal of cancer, ISSN 0020-7136, 06/2016, Volume 138, Issue 11, pp. 2570 - 2578
transcription factor | breast cancer | tumor growth | regulation | target genes | STAT3 | Life Sciences & Biomedicine | Oncology | Science & Technology | Receptor, Epidermal Growth Factor - genetics | Transcriptional Activation - genetics | Interleukin-6 - genetics | Janus Kinases - genetics | Signal Transduction | Humans | Gene Expression Regulation, Neoplastic | STAT3 Transcription Factor - biosynthesis | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Female | STAT3 Transcription Factor - genetics | Vascular Endothelial Growth Factor Receptor-1 - genetics | Development and progression | Vascular endothelial growth factor | Breast cancer | Signal transduction | Transcription factors | Index Medicus
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