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1. Full Text Cistrome-GO: a web server for functional enrichment analysis of transcription factor ChIP-seq peaks
by Li, SJ and Wan, CX and Zheng, RB and Fan, JY and Dong, X and Meyer, CA and Liu, XS
NUCLEIC ACIDS RESEARCH, ISSN 0305-1048, 07/2019, Volume 47, Issue W1, pp. W206 - W211
Characterizing the ontologies of genes directly regulated by a transcription factor (TF), can help to elucidate the TF's biological role. Previously, we... 
CANCER | BIOCHEMISTRY & MOLECULAR BIOLOGY | STAT3 | Web Server Issue
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2. Full Text Reconstruction and analysis of transcription factor-miRNA co-regulatory feed-forward loops in human cancers using filter-wrapper feature selection
by Peng, Chen and Wang, Minghui and Shen, Yi and Feng, Huanqing and Li, Ao
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, pp. e78197 - e78197
Background: As one of the most common types of co-regulatory motifs, feed-forward loops (FFLs) control many cell functions and play an important role in human... 
LUNG-CANCER | NETWORK | DATABASE | GENE | MICROARRAY | MULTIDISCIPLINARY SCIENCES | CELL PANEL | TARGETS | EXPRESSION PROFILES | MICRORNA | Gene Regulatory Networks - genetics | Neoplasms - genetics | Humans | Carcinogenesis - genetics | MicroRNAs - genetics | Models, Genetic | Signal Transduction - genetics | Transcription Factors - genetics | Gene Expression Regulation, Neoplastic - genetics | STAT3 Transcription Factor - genetics | MicroRNA | Analysis | Genes | Cancer | Regulations | Regulators | Transcription factors | Gene regulation | Science | Genomes | Carcinogenesis | Information science | Carcinogens | Cell cycle | Bioinformatics | Biomedical engineering | Medical research | MiRNA | Stat3 protein | Gene expression | Ribonucleic acid--RNA | Studies | Signaling | MicroRNAs | Data sets | Computer applications | Predictions | Regulatory mechanisms (biology) | Prostate cancer | Prostate | Binding sites | Index Medicus | RNA | Ribonucleic acid
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3. Full Text mRNA chip-based analysis on transcription factor regulatory network central nodes of protection targets of Deproteinized Extract of Calf Blood on acute liver injury in mice
by Xu, Guangyu and Xu, Jinhe and Han, Xiao and Li, Hongyu and Yuan, Guangxin and An, Liping and Du, Peige
International Immunopharmacology, ISSN 1567-5769, 03/2018, Volume 56, pp. 212 - 216
Our previous study found that Deproteinized Extract of Calf Blood (DECB) could protect the acute liver injury induced by carbon tetrachloride in mice, but the... 
Deproteinated Extract of Calf Blood | Transcription factors | Regulatory networks | Network nodes | ACTIVATION | T-CELL RESPONSE | GENES | RECEPTOR | PHARMACOLOGY & PHARMACY | IMMUNOLOGY | EXPRESSION | Messenger RNA | DNA binding proteins | Cattle | Analysis | Liver | Carbon tetrachloride | Liver diseases | Cytokines | Genes | Stat3 protein | Inflammation | Gene expression | Blood | Interleukin 6 | Proteins | Signal transduction | Signaling | DNA microarrays | Hepatocytes | Tumor necrosis factor | Interleukin 1 | Mice | Apoptosis | Index Medicus
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4. Full Text IL-27 increases the proliferation and effector functions of human naïve CD8+ T lymphocytes and promotes their development into Tc1 cells
by Schneider, Raphael and Yaneva, Teodora and Beauseigle, Diane and El-Khoury, Lama and Arbour, Nathalie
European Journal of Immunology, ISSN 0014-2980, 2011, Volume 41, Issue 1, pp. 47 - 59
IL-27 has been shown to exhibit both pro-and anti-inflammatory properties; it favors mouse naive CD4(+) T-cell differentiation into Th1 cells to the detriment... 
Human | STAT | SOCS | Cytokines | Cytotoxic T cells | ACTIVATION | STAT1 | RECEPTOR | IMMUNOLOGY | HUMAN MONOCYTES | AUTOIMMUNE ENCEPHALOMYELITIS | TH1 DIFFERENTIATION | RESPONSES | INHIBITION | INTERLEUKIN-27 | EXPRESSION | Cell Proliferation | Granzymes - metabolism | Humans | STAT1 Transcription Factor - analysis | Interleukin-17 - immunology | STAT5 Transcription Factor - immunology | T-Box Domain Proteins - immunology | Interferon-gamma - metabolism | Th1 Cells - immunology | Th1 Cells - metabolism | STAT5 Transcription Factor - metabolism | Lymphocyte Activation - immunology | STAT1 Transcription Factor - metabolism | Antigens, Neoplasm - metabolism | CD8-Positive T-Lymphocytes - metabolism | T-Box Domain Proteins - analysis | Granzymes - immunology | Antigens, Neoplasm - analysis | Carrier Proteins - analysis | Carrier Proteins - immunology | STAT3 Transcription Factor - metabolism | T-Lymphocytes, Cytotoxic - immunology | Granzymes - analysis | Cytokine Receptor gp130 - metabolism | Antigens, Neoplasm - immunology | Cells, Cultured | Cytokine Receptor gp130 - immunology | STAT3 Transcription Factor - analysis | Cytokine Receptor gp130 - analysis | STAT1 Transcription Factor - immunology | T-Box Domain Proteins - metabolism | Interleukin-17 - metabolism | Interferon-gamma - analysis | Carrier Proteins - metabolism | T-Lymphocytes, Cytotoxic - metabolism | Interferon-gamma - immunology | CD3 Complex - immunology | STAT3 Transcription Factor - immunology | CD8-Positive T-Lymphocytes - immunology | Index Medicus
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5. Full Text Leptin Activates Human B Cells to Secrete TNF-α, IL-6, and IL-10 via JAK2/STAT3 and p38MAPK/ERK1/2 Signaling Pathway
by Agrawal, Sudhanshu and Gollapudi, Sastry and Su, Houfen and Gupta, Sudhir
Journal of Clinical Immunology, ISSN 0271-9142, 6/2011, Volume 31, Issue 3, pp. 472 - 478
Leptin, one of the adipokines, functions as a hormone and a cytokine. In this investigation, we show for the first time that leptin, in a... 
Medical Microbiology | Biomedicine | Immunology | Internal Medicine | Infectious Diseases | activation markers | cytokines | B cells | PROTEIN-KINASE | TRANSDUCER | RECEPTOR | PROLIFERATION | IMMUNOLOGY | BLOOD MONONUCLEAR-CELLS | DEFICIENCY | PI3K/AKT | IMMUNE-RESPONSES | T-CELLS | Autoimmunity | Interleukin-6 - analysis | Janus Kinase 2 - biosynthesis | Mitogen-Activated Protein Kinase 3 - analysis | Humans | p38 Mitogen-Activated Protein Kinases - biosynthesis | p38 Mitogen-Activated Protein Kinases - analysis | Dose-Response Relationship, Drug | Signal Transduction - immunology | Inflammation - metabolism | Mitogen-Activated Protein Kinases - analysis | Mitogen-Activated Protein Kinase 3 - biosynthesis | Mitogen-Activated Protein Kinase 6 - analysis | Leptin - pharmacology | Phosphorylation - drug effects | Interleukin-10 - analysis | B-Lymphocytes - metabolism | Janus Kinase 2 - analysis | Enzyme-Linked Immunosorbent Assay | Lymphocyte Activation | Cells, Cultured | Mitogen-Activated Protein Kinases - biosynthesis | Receptors, Leptin - immunology | STAT3 Transcription Factor - analysis | Inflammation - immunology | Tumor Necrosis Factor-alpha - analysis | STAT3 Transcription Factor - biosynthesis | B-Lymphocytes - drug effects | B-Lymphocytes - immunology | Mitogen-Activated Protein Kinase 6 - biosynthesis | Interleukin-6 - biosynthesis | Interleukin-10 - biosynthesis | Receptors, Leptin - metabolism | Tumor Necrosis Factor-alpha - biosynthesis | Index Medicus
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6. Full Text Nonreceptor Tyrosine Kinase BMX Maintains Self-Renewal and Tumorigenic Potential of Glioblastoma Stem Cells by Activating STAT3
by Guryanova, Olga A and Wu, Qiulian and Cheng, Lin and Lathia, Justin D and Huang, Zhi and Yang, Jinbo and MacSwords, Jennifer and Eyler, Christine E and McLendon, Roger E and Heddleston, John M and Shou, Weinian and Hambardzumyan, Dolores and Lee, Jeongwu and Hjelmeland, Anita B and Sloan, Andrew E and Bredel, Markus and Stark, George R and Rich, Jeremy N and Bao, Shideng
Cancer Cell, ISSN 1535-6108, 2011, Volume 19, Issue 4, pp. 498 - 511
Glioblastomas display cellular hierarchies containing tumor-propagating glioblastoma stem cells (GSCs). STAT3 is a critical signaling node in GSC maintenance... 
ANGIOGENESIS | ONCOLOGY | GLIOMA | ETK/BMX | PROSTATE-CANCER | BIOLOGY | FACTOR RECEPTOR | EXPRESSION | CELLULAR-TRANSFORMATION | ENDOTHELIAL GROWTH-FACTOR | PH-DOMAIN | CELL BIOLOGY | Cell Proliferation | Humans | Mice, Inbred C57BL | Brain Neoplasms - pathology | Nerve Tissue Proteins - analysis | Basic Helix-Loop-Helix Transcription Factors - analysis | Oligodendrocyte Transcription Factor 2 | Protein-Tyrosine Kinases - physiology | Receptors, Interleukin-6 - physiology | Animals | STAT3 Transcription Factor - physiology | Glioblastoma - pathology | Cell Line, Tumor | Neoplastic Stem Cells - pathology | Mice | Vascular Endothelial Growth Factor A - physiology | Tyrosine | Medical colleges | Stem cell research | Molecular genetics | Oncology, Experimental | Stem cells | Phenols | Research | DNA binding proteins | Glioblastoma multiforme | Cancer | Index Medicus
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7. Full Text IL-17 induces AKT-dependent IL-6/JAK2/STAT3 activation and tumor progression in hepatocellular carcinoma
by Gu, Fang-Ming and Li, Quan-Lin and Gao, Qiang and Jiang, Jia-Hao and Zhu, Kai and Huang, Xiao-Yong and Pan, Jin-Feng and Yan, Jun and Hu, Jin-Hui and Wang, Zheng and Dai, Zhi and Fan, Jia and Zhou, Jian
Molecular Cancer, ISSN 1476-4598, 12/2011, Volume 10, Issue 1, pp. 150 - 150
Background: The Th17 subset and IL-17 have been found in increased frequencies within certain tumors. However, their relevance in cancer biology remains... 
IMMUNITY | INTERLEUKIN-17 | ONCOLOGY | INFLAMMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | RECEPTOR | CYTOKINE | CANCER | EXPRESSION | T-CELLS | TH17 CELLS | Interleukin-6 - analysis | Humans | Male | Transplantation, Heterologous | Vascular Endothelial Growth Factor A - metabolism | Interleukin-17 - pharmacology | Liver Neoplasms - chemistry | Proto-Oncogene Proteins c-akt - genetics | Janus Kinase 2 - metabolism | Liver Neoplasms - pathology | Carcinoma, Hepatocellular - chemistry | Proto-Oncogene Proteins c-akt - metabolism | Interleukin-6 - metabolism | Janus Kinase 2 - analysis | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Neoplasm Invasiveness | Kaplan-Meier Estimate | STAT3 Transcription Factor - analysis | Chi-Square Distribution | Disease Progression | Up-Regulation - drug effects | Cell Movement - drug effects | Interleukin-17 - metabolism | Animals | Mice, Nude | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Line, Tumor | Mice | Mice, Inbred BALB C | Carcinoma, Hepatocellular - metabolism | Immunohistochemistry | Medical research | Carcinoma | Endothelial growth factors | Oncology, Experimental | Research | Hepatoma | Liver cancer | Medicine, Experimental | Vascular endothelial growth factor | Enzyme-linked immunosorbent assay | Cancer | Tumors | Studies | Proteins | Index Medicus
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8. Full Text MicroRNA and transcription factor gene regulatory network analysis reveals key regulatory elements associated with prostate cancer progression
by Sadeghi, Mehdi and Ranjbar, Bijan and Ganjalikhany, Mohamad Reza and Khan, Faiz M and Schmitz, Ulf and Wolkenhauer, Olaf and Gupta, Shailendra K
PLoS ONE, ISSN 1932-6203, 12/2016, Volume 11, Issue 12, pp. e0168760 - e0168760
Technological and methodological advances in multi-omics data generation and integration approaches help elucidate genetic features of complex biological... 
BREAST-CANCER | CELLS | OVEREXPRESSION | TARGET GENES | CARCINOGENESIS | BIOLOGICAL NETWORKS | MULTIDISCIPLINARY SCIENCES | STAT3 | TUMOR-SUPPRESSOR | PROGESTERONE-RECEPTOR EXPRESSION | AP-1 | Prostatic Neoplasms - pathology | Computational Biology - methods | Prostatic Neoplasms - genetics | Humans | Gene Expression Regulation, Neoplastic | Male | Gene Expression Profiling | MicroRNAs - genetics | Transcriptome - genetics | Transcription Factors - genetics | Gene Regulatory Networks | Disease Progression | Transcription factors | MicroRNA | Analysis | Development and progression | Genetic aspects | Research | Genetic regulation | Prostate cancer | MiRNA | Stat3 protein | Biology | Regulatory sequences | Metastasis | Biophysics | Gene expression | Metastases | JunB protein | Studies | Survival analysis | Ribonucleic acids | MicroRNAs | Network analysis | Bioinformatics | Prostate | Tumors | Cancer | Apoptosis | Index Medicus
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9. Full Text EGFR, HER2, survivin, and loss of pSTAT3 characterize high-grade malignancy in salivary gland cancer with impact on prognosis
by Ettl, Tobias, MD, DMD and Stiegler, Clemens, MD and Zeitler, Katharina, MD and Agaimy, Abbas, MD, PhD and Zenk, Johannes, MD, PhD and Reichert, Torsten E., MD, DMD, PhD and Gosau, Martin, MD, DMD, PhD and Kühnel, Thomas, MD, PhD and Brockhoff, Gero, PhD and Schwarz, Stephan, MD, PhD
Human Pathology, ISSN 0046-8177, 2012, Volume 43, Issue 6, pp. 921 - 931
Summary Increased gene copy number (high polysomy or amplification) of EGFR and HER2 has evolved as a predictor for response to targeted therapy. STAT3 and the... 
Pathology | pSTAT3 | Prognosis | Survivin | HER2 | Salivary gland cancer | EGFR | IN-SITU HYBRIDIZATION | CYTOPLASMIC EXPRESSION | PATHOLOGY | BREAST-CANCER | POOR-PROGNOSIS | COLORECTAL-CANCER | EPIDERMAL-GROWTH-FACTOR | ADENOID CYSTIC CARCINOMA | SQUAMOUS-CELL CARCINOMAS | GENE COPY NUMBER | FACTOR RECEPTOR GENE | Receptor, Epidermal Growth Factor - genetics | Receptor, ErbB-2 - genetics | Inhibitor of Apoptosis Proteins - genetics | Salivary Gland Neoplasms - genetics | Tissue Array Analysis | Humans | Male | Salivary Gland Neoplasms - metabolism | Female | Carcinoma - pathology | Inhibitor of Apoptosis Proteins - analysis | STAT3 Transcription Factor - genetics | Inhibitor of Apoptosis Proteins - biosynthesis | Receptor, Epidermal Growth Factor - biosynthesis | Receptor, ErbB-2 - biosynthesis | Biomarkers, Tumor - analysis | Kaplan-Meier Estimate | Proportional Hazards Models | In Situ Hybridization, Fluorescence | STAT3 Transcription Factor - analysis | Gene Dosage | Receptor, Epidermal Growth Factor - analysis | STAT3 Transcription Factor - biosynthesis | Genes, erbB-1 | Genes, erbB-2 | Carcinoma - genetics | Biomarkers, Tumor - genetics | Carcinoma - metabolism | Receptor, ErbB-2 - analysis | Neoplasm Staging | Salivary Gland Neoplasms - pathology | Immunohistochemistry | Genes | Tumors | Cancer | Confidence intervals | Gene amplification | Lung cancer | Cell cycle | Breast cancer | Histology | Kinases | Multivariate analysis | Microwave heating | Apoptosis | Index Medicus
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10. Full Text Stat3-dependent acute Rantes production in vascular smooth muscle cells modulates inflammation following arterial injury in mice
by Kovacic, Jason C and Gupta, Rohit and Lee, Angela C and Ma, Mingchao and Fang, Fang and Tolbert, Claire N and Walts, Avram D and Beltran, Leilani E and San, Hong and Chen, Guibin and St Hilaire, Cynthia and Boehm, Manfred
Journal of Clinical Investigation, ISSN 0021-9738, 01/2010, Volume 120, Issue 1, pp. 303 - 314
Inflammation is a key component of arterial injury, with VSMC proliferation and neointimal formation serving as the final outcomes of this process. However,... 
SIGNAL-TRANSDUCTION | MEDICINE, RESEARCH & EXPERIMENTAL | MONOCYTE RECRUITMENT | P-SELECTIN | ATHEROSCLEROSIS | NECROSIS-FACTOR-ALPHA | INTERFERON REGULATORY FACTOR-1 | NEOINTIMA FORMATION | DEPENDENT KINASE INHIBITOR | NF-KAPPA-B | TRANSCRIPTION FACTOR | T-Lymphocytes - physiology | Muscle, Smooth, Vascular - metabolism | Vasculitis - etiology | Male | Chemokine CCL5 - analysis | Arteries - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | STAT3 Transcription Factor - physiology | Female | Cyclin-Dependent Kinase Inhibitor p21 - physiology | Myocytes, Smooth Muscle - metabolism | STAT3 Transcription Factor - genetics | Macrophages - physiology | Mice, Inbred C57BL | Cells, Cultured | STAT3 Transcription Factor - analysis | Tumor Necrosis Factor-alpha - pharmacology | Cyclin-Dependent Kinase Inhibitor p21 - analysis | Animals | Chemokine CCL5 - biosynthesis | Interleukin-6 - pharmacology | Chemokine CCL5 - genetics | NF-kappa B - physiology | Chemokine CCL2 - biosynthesis | Mice | Care and treatment | Development and progression | Inflammation | Cell physiology | Vascular smooth muscle | Research | Injuries | Arteries | Index Medicus | Abridged Index Medicus
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11. Full Text Dual JAK1 and STAT3 mutations in a breast implant-associated anaplastic large cell lymphoma
by Letourneau, Audrey and Maerevoet, Marie and Milowich, Dina and Dewind, Roland and Bisig, Bettina and Missiaglia, Edoardo and de Leval, Laurence
Virchows Archiv, ISSN 0945-6317, 10/2018, Volume 473, Issue 4, pp. 505 - 511
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1007/s00428-018-2352-y 
Medicine & Public Health | Pathology | LEUKEMIA | PATHOLOGY | Immunohistochemistry | Lymphoma, Large-Cell, Anaplastic - therapy | Phosphorylation | Biomarkers, Tumor - analysis | Breast Implantation - instrumentation | Breast Implants - adverse effects | Humans | In Situ Hybridization, Fluorescence | STAT3 Transcription Factor - analysis | Lymphoma, Large-Cell, Anaplastic - enzymology | Positron Emission Tomography Computed Tomography | Breast Implantation - adverse effects | Janus Kinase 1 - genetics | Female | Aged | Biomarkers, Tumor - genetics | Lymphoma, Large-Cell, Anaplastic - pathology | Mutation | STAT3 Transcription Factor - genetics | Lymphoma, Large-Cell, Anaplastic - genetics | Lymphomas | Genetic aspects | Implants, Artificial | Prosthesis | Janus kinase | Anaplastic large-cell lymphoma | Lymphoma | Stat3 protein | Index Medicus
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12. Full Text Suppression of leptin-induced hypothalamic JAK/STAT signalling and feeding response during pregnancy in the mouse
by Ladyman, S.R and Fieldwick, D.M and Grattan, D.R
Reproduction, ISSN 1470-1626, 07/2012, Volume 144, Issue 1, pp. 83 - 90
Hyperphagia during pregnancy, despite rising concentrations of the satiety hormone leptin, suggests that a state of leptin resistance develops. This study... 
ENERGY-BALANCE | REPRODUCTIVE BIOLOGY | GROWTH-HORMONE | FOOD-INTAKE | OBESE GENE | OB/OB MICE | LONG FORM | RATS IN-VIVO | HORMONE-SECRETION | RESISTANCE | RECEPTOR | DEVELOPMENTAL BIOLOGY | Ventromedial Hypothalamic Nucleus - drug effects | Satiation - drug effects | STAT5 Transcription Factor - metabolism | Arcuate Nucleus of Hypothalamus - drug effects | Female | Leptin - pharmacology | Hypothalamus - drug effects | Phosphorylation - drug effects | Hypothalamus - chemistry | Drug Resistance | STAT3 Transcription Factor - metabolism | Arcuate Nucleus of Hypothalamus - chemistry | Hypothalamus - physiology | Pregnancy, Animal - physiology | Mice, Inbred C57BL | Ventromedial Hypothalamic Nucleus - chemistry | STAT5 Transcription Factor - drug effects | STAT3 Transcription Factor - analysis | Pregnancy | Eating - drug effects | Animals | STAT5 Transcription Factor - analysis | Signal Transduction - drug effects | STAT3 Transcription Factor - drug effects | Mice | Index Medicus
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13. Full Text Receptor Trafficking Controls Weak Signal Delivery: A Strategy Used by c-Met for STAT3 Nuclear Accumulation
by Stéphanie Kermorgant and Peter J. Parker
The Journal of Cell Biology, ISSN 0021-9525, 9/2008, Volume 182, Issue 5, pp. 855 - 863
C-Met, the receptor of hepatocyte growth factor (HGF), through overexpression or mutation, is a major protooncogene that provides an attractive molecular... 
Receptors | Phosphorylation | Endocytosis | Report | Vanadates | Cell nucleus | Biological transport | HeLa cells | Antibodies | Cell membranes | Endosomes | COMPLEX | BEHAVIOR | GROWTH | TIME | ENDOCYTOSIS | CELL | CANCER | CELL BIOLOGY | Proto-Oncogene Proteins c-met - metabolism | Signal Transduction | Humans | STAT3 Transcription Factor - analysis | Endosomes - metabolism | Hepatocyte Growth Factor - metabolism | Protein Transport | Microtubules - metabolism | Animals | Cell Nucleus - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Proto-Oncogene Proteins c-met - analysis | Mice | STAT3 Transcription Factor - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Physiological aspects | Cellular signal transduction | Research | Growth factor receptors | Properties | Carcinogenesis | Oncogenes | Signal transduction | Biochemistry | Mutation | Cytokines | Cancer | Tumors | Index Medicus
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