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Blood, ISSN 0006-4971, 01/2015, Volume 125, Issue 4, pp. 591 - 599
Journal Article
by Tsoi, L.C and Spain, S.L and Knight, J and Ellinghaus, E and Stuart, P.E and Capon, F and Ding, J and Li, J and Tejasvi, T and Gudjonsson, J.E and Kang, H and Allen, M.H and McManus, R and Novelli, G and Samuelsson, L and Schalkwijk, J and Stahle, M and Burden, A.D and Smith, C.H and Cork, M.J and Estivill, X and Bowcock, A and Krueger, G.G and Weger, W and Worthington, J and Tazi-Ahnini, R and Nestle, F.O and Hayday, A and Hoffmann, P and Winkelmann, J and Wijmenga, C and Langford, C and Edkins, S and Anews, R and Blackburn, H and Strange, A and Band, G and Pearson, R and Vukcevic, D and Spencer, C.C and Deloukas, P and Mrowietz, U and Schreiber, S and Weidinger, S and Koks, S and Kingo, K and Esko, T and Metspalu, A and Lim, H.W and Voorhees, J.J and Weichenthal, M and Wichmann, H.E and Chanan, V and Rosen, C.F and Rahman, P and Gladman, D.D and Griffiths, C.E and Reis, A and Kere, J and Duffin, K.C and Helms, C and Goldgar, D and Li, Y and Paschall, J and Malloy, M.J and Pullinger, C.R and Kane, J.P and Gardner, J and Perlmutter, A and Miner, A and Feng, B.J and Hiremagalore, R and Ike, R.W and Christophers, E and Henseler, T and Ruether, A and Schrodi, S.J and Prahalad, S and Guthery, S.L and Hiregalore, R and Donnelly, P and Collaborative Assoc Study Psoriasi and Psoriasis Assoc Genetics Extension and Genetic Anal Psoriasis Consortium and Wellcome Trust Case Control Consor and Psoriasis Association Genetics Extension and Genetic Analysis of Psoriasis Consortium and Collaborative Association Study of Psoriasis (CASP) and Wellcome Trust Case Control Consortium 2 and Sahlgrenska akademin and Institutionen för biomedicin, avdelningen för medicinsk genetik och klinisk genetik and Göteborgs universitet and Gothenburg University and Institute of Biomedicine, Department of Medical and Clinical Genetics and Sahlgrenska Academy
Nature Genetics, ISSN 1061-4036, 2012, Volume 44, Issue 12, pp. 1341 - 1348
To gain further insight into the genetic architecture of psoriasis, we conducted a meta-analysis of 3 genome-wide association studies (GWAS) and 2 independent... 
COMMON VARIANTS | SIGNALING PATHWAYS | MULTIPLE COMMON | INTERFERON-GAMMA | TH17 DIFFERENTIATION | GENETICS & HEREDITY | GENE-EXPRESSION | T-CELL DEVELOPMENT | GENOME-WIDE ASSOCIATION | CELIAC-DISEASE | NEGATIVE REGULATOR | Oligonucleotide Array Sequence Analysis | Humans | Immunity, Innate - genetics | Genetic Loci | CARD Signaling Adaptor Proteins - genetics | Psoriasis - genetics | Core Binding Factor Alpha 3 Subunit - immunology | STAT3 Transcription Factor - genetics | Skin - immunology | Psoriasis - immunology | CARD Signaling Adaptor Proteins - immunology | DEAD Box Protein 58 | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Guanylate Cyclase - immunology | Membrane Proteins - genetics | Membrane Proteins - immunology | GTPase-Activating Proteins - immunology | DEAD-box RNA Helicases - genetics | Core Binding Factor Alpha 3 Subunit - genetics | DEAD-box RNA Helicases - immunology | Guanylate Cyclase - genetics | T-Lymphocytes - immunology | Polymorphism, Single Nucleotide | GTPase-Activating Proteins - genetics | STAT3 Transcription Factor - immunology | Psoriasis | Physiological aspects | Disease susceptibility | Genetic aspects | Research | Autoimmune diseases | Gene expression | Chromosomes | Risk factors | Index Medicus | genetic analysis | innate immunity | Dermatologi och venereologi | Dermatology and Venereal Diseases | psoriasis
Journal Article
Cancer Cell, ISSN 1535-6108, 04/2015, Volume 27, Issue 4, pp. 516 - 532
Journal Article
Nature, ISSN 0028-0836, 01/2010, Volume 463, Issue 7279, pp. 318 - 325
The inference of transcriptional networks that regulate transitions into physiological or pathological cellular states remains a central challenge in systems... 
SURVIVAL | GROWTH-FACTOR RECEPTOR | NEURAL STEM-CELLS | GLIOBLASTOMA | MULTIDISCIPLINARY SCIENCES | DISEASE | C-MYC | MECHANISMS | DIFFERENTIATION | EXPRESSION | BINDING | Neurons - pathology | Prognosis | Glioma - diagnosis | Humans | Brain Neoplasms - pathology | Gene Expression Regulation, Neoplastic | Gene Regulatory Networks | Glioma - genetics | Cell Differentiation - genetics | Cell Transformation, Neoplastic - genetics | Neoplasm Invasiveness - pathology | Glioma - pathology | Transcription, Genetic | Neurons - metabolism | Cellular Reprogramming - genetics | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Reproducibility of Results | Brain Neoplasms - diagnosis | CCAAT-Enhancer-Binding Protein-beta - genetics | Computational Biology | Brain Neoplasms - genetics | Mesenchymal Stromal Cells - metabolism | Mice, SCID | Cell Transformation, Neoplastic - metabolism | CCAAT-Enhancer-Binding Protein-beta - metabolism | Animals | Cell Line, Tumor | Mice, Inbred NOD | Mesoderm - metabolism | Mice | Mesenchymal Stromal Cells - pathology | Cell Transformation, Neoplastic - pathology | Neoplasm Invasiveness - genetics | Mesoderm - pathology | Transcription factors | Gliomas | Physiological aspects | Genetic aspects | Research | Genetic transcription | Risk factors | Proteins | Genotype & phenotype | Brain | Reverse engineering | Brain cancer | Regression analysis | Gene expression | Index Medicus
Journal Article
Immunity, ISSN 1074-7613, 03/2012, Volume 36, Issue 3, pp. 362 - 373
Although Th17 cells are known to promote tissue inflammation and autoimmunity, their role during cancer progression remains elusive. Here, we showed that... 
GROWTH-FACTOR-BETA | EFFECTOR T-CELLS | TGF-BETA | T(H)17 CELLS | ROR-GAMMA-T | SIGNALING PATHWAY | IL-17 | ADENOSINE | GENERATION | IMMUNOLOGY | CANCER | 5'-Nucleotidase - genetics | RNA, Small Interfering - genetics | Antigens, CD - genetics | DNA-Binding Proteins - metabolism | Th17 Cells - drug effects | T-Lymphocytes - metabolism | Th17 Cells - metabolism | Lymphocytes, Tumor-Infiltrating - metabolism | Transcription Factors - immunology | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Promoter Regions, Genetic | DNA-Binding Proteins - immunology | Mice, Inbred C57BL | Gene Expression Regulation | Mice, Transgenic | Binding Sites - genetics | Mice, Knockout | Transcription Factors - metabolism | Transforming Growth Factor beta - pharmacology | Animals | Interleukin-6 - pharmacology | Apyrase - genetics | Th17 Cells - immunology | T-Lymphocytes - immunology | Mice | STAT3 Transcription Factor - immunology | STAT3 Transcription Factor - antagonists & inhibitors | Lymphocytes, Tumor-Infiltrating - immunology | Autoimmunity | DNA binding proteins | T cells | Transforming growth factors | Proteins | Transcription factors | Statistical analysis | Plasmids | Rodents | Cytotoxicity | Deoxyribonucleic acid--DNA | Immune system | Cancer | Adenosine | CD8 antigen | Helper cells | Stat3 protein | Interleukin 23 | Inflammation | Lymphocytes T | Transforming growth factor- beta | Adoptive transfer | Immunosuppressive agents | Promoters | CD4 antigen | Interleukin 6 | Interleukin 1 | CD73 antigen | Ectonucleotidase | Differentiation | Tumors | Index Medicus | 5'-Nucleotidase | T-Lymphocytes | Interleukin-6 | Life Sciences | Lymphocytes, Tumor-Infiltrating | Immunology | Apyrase | RNA, Small Interfering | STAT3 Transcription Factor | Th17 Cells | Transcription Factors | Binding Sites | Transforming Growth Factor beta | Antigens, CD | DNA-Binding Proteins
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2007, Volume 117, Issue 12, pp. 3846 - 3856
Persistently activated or tyrosine-phosphorylated STAT3 (pSTAT3) is found in 50% of lung adenocarcinomas. pSTAT3 is found in primary adenocarcinomas and cell... 
RECEPTOR GENE-MUTATIONS | BREAST-CANCER | MEDICINE, RESEARCH & EXPERIMENTAL | SIGNALING PATHWAYS | CONSTITUTIVE ACTIVATION | EPIDERMAL-GROWTH-FACTOR | PROSTATE-CANCER CELLS | SQUAMOUS-CELL CARCINOMA | TYROSINE KINASES | NF-KAPPA-B | MAMMARY EPITHELIAL-CELLS | Janus Kinases | Neoplasm Transplantation | Receptor, Epidermal Growth Factor - genetics | Transcription, Genetic - drug effects | Adenocarcinoma - pathology | Interleukin-6 - antagonists & inhibitors | Cytokine Receptor gp130 - genetics | Humans | Lung Neoplasms - metabolism | Lung Neoplasms - pathology | Receptor, Epidermal Growth Factor - metabolism | Adenocarcinoma - metabolism | Female | Adenocarcinoma - genetics | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Gene Expression Regulation, Neoplastic - genetics | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Lung Neoplasms - genetics | Cytokine Receptor gp130 - metabolism | Interleukin-6 - genetics | RNA, Small Interfering - pharmacology | Enzyme Inhibitors - pharmacology | Lung Neoplasms - therapy | Signal Transduction - genetics | Animals | Adenocarcinoma - therapy | Signal Transduction - drug effects | Mice, Nude | Cell Line, Tumor | Interleukin-6 - biosynthesis | Mice | Mutation | Transcription, Genetic - genetics | Research | Gene mutations | Lung cancer | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Cancer Cell, ISSN 1535-6108, 08/2017, Volume 32, Issue 2, pp. 169 - 184.e7
Journal Article
Molecular Cell, ISSN 1097-2765, 03/2012, Volume 45, Issue 6, pp. 777 - 789
Abnormal inflammatory signaling activation occurs commonly in cancer cells. However, how it is initiated and maintained and its roles in early stages of... 
BREAST-CANCER | PATHWAYS | CELLS | PHOSPHORYLATION | JNK | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROSTATE-CANCER | IN-VIVO | ALPHA | CYTOKINE | NF-KAPPA-B | CELL BIOLOGY | Mitogen-Activated Protein Kinase Kinases - genetics | Epithelial Cells - metabolism | Humans | JNK Mitogen-Activated Protein Kinases - metabolism | MicroRNAs - metabolism | NF-kappa B - metabolism | Mitogen-Activated Protein Kinase 9 - genetics | Transcription Factor RelA - genetics | Heat Shock Transcription Factors | DNA-Binding Proteins - metabolism | Mitogen-Activated Protein Kinase Kinases - metabolism | Inflammation - metabolism | Cell Transformation, Neoplastic - genetics | Estrogen Receptor alpha - metabolism | Female | Chemokine CCL2 - metabolism | JNK Mitogen-Activated Protein Kinases - genetics | Interleukin-6 - metabolism | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Promoter Regions, Genetic | Interleukin-6 - genetics | Breast - cytology | Mitogen-Activated Protein Kinase 9 - metabolism | Chemokine CCL2 - genetics | Epithelial Cells - pathology | Mice, Transgenic | Signal Transduction - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Transcription Factors - metabolism | Animals | NF-kappa B - genetics | Transcription Factor RelA - metabolism | Inflammation - genetics | Mice | Analysis | Tumors | Index Medicus
Journal Article