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1. Full Text Expression and regulation of intergenic long noncoding RNAs during T cell development and differentiation
by Hu, Gangqing and Tang, Qingsong and Sharma, Suveena and Yu, Fang and Escobar, Thelma M and Muljo, Stefan A and Zhu, Jinfang and Zhao, Keji
Nature immunology, ISSN 1529-2908, 2013, Volume 14, Issue 11, pp. 1190 - 1198
Life Sciences & Biomedicine | Immunology | Science & Technology | GATA3 Transcription Factor - genetics | GATA3 Transcription Factor - immunology | STAT4 Transcription Factor - metabolism | STAT6 Transcription Factor - genetics | T-Box Domain Proteins - immunology | Th2 Cells - immunology | Transcriptome - immunology | Genetic Loci | Th1 Cells - immunology | RNA, Long Noncoding - immunology | Th1 Cells - metabolism | Th17 Cells - metabolism | Precursor Cells, T-Lymphoid - metabolism | Precursor Cells, T-Lymphoid - cytology | Th17 Cells - cytology | Cell Differentiation | GATA3 Transcription Factor - metabolism | STAT4 Transcription Factor - immunology | Signal Transduction | Gene Expression Regulation - immunology | Mice, Inbred C57BL | Molecular Sequence Annotation | Th2 Cells - cytology | RNA, Long Noncoding - genetics | STAT6 Transcription Factor - metabolism | STAT6 Transcription Factor - immunology | T-Box Domain Proteins - genetics | Th2 Cells - metabolism | T-Box Domain Proteins - metabolism | Animals | Protein Binding | Th17 Cells - immunology | STAT4 Transcription Factor - genetics | Mice | Th1 Cells - cytology | Precursor Cells, T-Lymphoid - immunology | Cell Movement | Physiological aspects | Genetic aspects | Research | Cell differentiation | Gene expression | T cells | Index Medicus | Cell migration
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2. Full Text Nuclear Factor of Activated T Cells Regulates the Expression of Interleukin-4 in Th2 Cells in an All-or-none Fashion
by Köck, Juliana and Kreher, Stephan and Lehmann, Katrin and Riedel, René and Bardua, Markus and Lischke, Timo and Jargosch, Manja and Haftmann, Claudia and Bendfeldt, Hanna and Hatam, Farahnaz and Mashreghi, Mir-Farzin and Baumgrass, Ria and Radbruch, Andreas and Chang, Hyun-Dong
The Journal of biological chemistry, ISSN 0021-9258, 09/2014, Volume 289, Issue 39, pp. 26752 - 26761
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Cell Nucleus - immunology | GATA3 Transcription Factor - genetics | Phosphorylation - physiology | Active Transport, Cell Nucleus - physiology | E1A-Associated p300 Protein - immunology | GATA3 Transcription Factor - immunology | E1A-Associated p300 Protein - genetics | E1A-Associated p300 Protein - metabolism | STAT6 Transcription Factor - genetics | Th2 Cells - immunology | DNA Helicases - immunology | Transcription Factor RelA - genetics | Interleukin-4 - biosynthesis | Cell Nucleus - metabolism | NFATC Transcription Factors - immunology | Adaptor Proteins, Signal Transducing - immunology | Transcription Factor RelA - immunology | Receptors, Antigen, T-Cell - immunology | Interleukin-4 - genetics | Nuclear Proteins - genetics | Response Elements - physiology | Transcription Factors - immunology | DNA Helicases - genetics | GATA3 Transcription Factor - metabolism | Receptors, Antigen, T-Cell - metabolism | NFATC Transcription Factors - metabolism | Gene Expression Regulation - physiology | Mice, Transgenic | Nuclear Proteins - metabolism | Th2 Cells - cytology | STAT6 Transcription Factor - metabolism | Transcription Factors - genetics | Nuclear Proteins - immunology | STAT6 Transcription Factor - immunology | Th2 Cells - metabolism | Transcription Factors - metabolism | DNA Helicases - metabolism | Interleukin-4 - immunology | Animals | Cell Nucleus - genetics | Transcription Factor RelA - metabolism | Adaptor Proteins, Signal Transducing - genetics | Mice | Mice, Inbred BALB C | Receptors, Antigen, T-Cell - genetics | Adaptor Proteins, Signal Transducing - metabolism | NFATC Transcription Factors - genetics | Index Medicus | NFAT Transcription Factor | Immunology | T-cell | T-cell Receptor (TCR) | Calcineurin | Cytokine
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3. Full Text STAT6 Transcription Factor Is a Facilitator of the Nuclear Receptor PPARγ-Regulated Gene Expression in Macrophages and Dendritic Cells
by Szanto, Attila and Balint, Balint L and Nagy, Zsuzsanna S and Barta, Endre and Dezso, Balazs and Pap, Attila and Szeles, Lajos and Poliska, Szilard and Oros, Melinda and Evans, Ronald M and Barak, Yaacov and Schwabe, John and Nagy, Laszlo
Immunity (Cambridge, Mass.), ISSN 1074-7613, 2010, Volume 33, Issue 5, pp. 699 - 712
Life Sciences & Biomedicine | Immunology | Science & Technology | Promoter Regions, Genetic | Macrophages - metabolism | Animals | Humans | Interleukin-4 - metabolism | Gene Expression Regulation | Inflammation Mediators - metabolism | Mice | Fatty Acid-Binding Proteins - metabolism | STAT6 Transcription Factor - metabolism | PPAR gamma - metabolism | Dendritic Cells - metabolism | Index Medicus | DNA
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4. Full Text Role of C/EBP homologous protein and endoplasmic reticulum stress in asthma exacerbation by regulating the IL-4/signal transducer and activator of transcription 6/transcription factor EC/IL-4 receptor α positive feedback loop in M2 macrophages
by Wang, Yi, PhD and Zhu, Jianghui, MD and Zhang, Lei, MD and Zhang, Zhijun, PhD and He, Long, PhD and Mou, Yong, MD and Deng, Yanhan, MD and Cao, Yong, MD, PhD and Yang, Ping, MD and Su, Yunchao, PhD and Zhao, Jianping, MD, PhD and Zhang, Shu, PhD and Yu, Qilin, MD, PhD and Hu, Jifa, PhD and Chen, Zhishui, MD, PhD and Ning, Qin, MD, PhD and Xiang, Xudong, MD, PhD and Xu, Yongjian, MD, PhD and Wang, Cong-Yi, MD, PhD and Xiong, Weining, MD, PhD
Journal of allergy and clinical immunology, ISSN 0091-6749, 2017, Volume 140, Issue 6, pp. 1550 - 1561.e8
Allergy and Immunology | macrophage | asthma | C/EBP homologous protein | endoplasmic reticulum stress | Allergy | Immunology | Life Sciences & Biomedicine | Science & Technology | CCAAT-Enhancer-Binding Proteins - metabolism | Transcription Factor CHOP - genetics | Humans | Interleukin-4 - metabolism | Middle Aged | Cells, Cultured | Receptors, Cell Surface - metabolism | Male | STAT6 Transcription Factor - metabolism | Disease Progression | Endoplasmic Reticulum Stress - immunology | Mice, Knockout | Feedback, Physiological | Animals | Asthma - immunology | Adult | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Female | Cell Differentiation | Mice | Transcription Factor CHOP - metabolism | Macrophages - immunology | Phosphorylation | Transcription factors | Pathogenesis | Programming | Homology | Feedback loops | CCAAT/enhancer-binding protein | Macrophages | Positive feedback | Respiratory tract | Proteins | Airway management | Interleukin 4 | Feedback | Rodents | Respiratory tract diseases | Stresses | Ovalbumin | Inflammation | Patients | Allergies | Stress | Asthma | Signaling | Lungs | Software | Aberration | Endoplasmic reticulum | Index Medicus | Abridged Index Medicus
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5. Full Text IL-4 inhibits TGF-β-induced Foxp3 + T cells and, together with TGF-β, generates IL-9 + IL-10 + Foxp3 − effector T cells
by Oukka, Mohamed and Mitsdoerffer, Meike and Strom, Terry B and Kuchroo, Vijay K and Elyaman, Wassim and Dardalhon, Valérie and Galileos, George and Awasthi, Amit and Sobel, Raymond A and Ho, I-Cheng and Kwon, Hyoung and Gao, Wenda and Khoury, Samia
Nature immunology, ISSN 1529-2908, 12/2008, Volume 9, Issue 12, pp. 1347 - 1355
Life Sciences & Biomedicine | Immunology | Science & Technology | Forkhead Transcription Factors - immunology | T-Lymphocyte Subsets - immunology | Transforming Growth Factor beta - immunology | T-Lymphocyte Subsets - cytology | GATA3 Transcription Factor - immunology | Interleukin-4 - metabolism | Mice, Transgenic | Adoptive Transfer | STAT6 Transcription Factor - metabolism | STAT6 Transcription Factor - immunology | T-Lymphocytes, Regulatory - immunology | Cell Differentiation - immunology | Interleukin-4 - immunology | Lymphocyte Activation - immunology | Animals | Forkhead Transcription Factors - metabolism | T-Lymphocytes, Regulatory - cytology | Interleukin-10 - metabolism | Interleukin-9 - metabolism | Mice | GATA3 Transcription Factor - metabolism | Interleukin-10 - immunology | Interleukin-9 - immunology | Transforming Growth Factor beta - metabolism | Index Medicus
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6. Full Text Transcription factors STAT6 and KLF4 implement macrophage polarization via the dual catalytic powers of MCPIP
by Kapoor, Nidhi and Niu, Jianli and Saad, Yasser and Kumar, Sanjay and Sirakova, Tatiana and Becerra, Edilu and Li, Xiaoman and Kolattukudy, Pappachan E
The Journal of immunology (1950), ISSN 0022-1767, 06/2015, Volume 194, Issue 12, pp. 6011 - 6023
Life Sciences & Biomedicine | Immunology | Science & Technology | Ribonucleases - genetics | Macrophage Activation - genetics | Reactive Oxygen Species - metabolism | Humans | NF-kappa B - metabolism | PPAR gamma - metabolism | Ribonucleases - metabolism | RNA Interference | Kruppel-Like Transcription Factors - metabolism | Autophagy - genetics | Catalysis | Macrophages - immunology | Gene Expression | Macrophage Activation - immunology | Interleukin-4 - metabolism | Mice, Transgenic | STAT6 Transcription Factor - metabolism | Gene Knockout Techniques | CCAAT-Enhancer-Binding Protein-beta - metabolism | Macrophages - metabolism | Animals | Models, Biological | Endoplasmic Reticulum Stress | Mice | Mutation | Index Medicus | Abridged Index Medicus | KLF4 induce MCPIP | STAT6 | Macrophage polarization | MCPIP | M2 polarization by IL-4 mediated via MCPIP | induction of polarization using both catalytic activities of MCPIP
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7. Full Text Deregulation in STAT signaling is important for cutaneous T-cell lymphoma (CTCL) pathogenesis and cancer progression
by Netchiporouk, Elena and Litvinov, Ivan V and Moreau, Linda and Gilbert, Martin and Sasseville, Denis and Duvic, Madeleine
Cell cycle (Georgetown, Tex.), ISSN 1538-4101, 11/2014, Volume 13, Issue 21, pp. 3331 - 3335
Cutaneous T-Cell Lymphoma (CTCL) | Sézary Syndrome | STAT3 | STAT4 | STAT5 | Mycosis Fungoides | and STAT6 | Mycosis fungoides | Cutaneous T-cell lymphoma (CTCL) | Sézary syndrome | And STAT6 | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Interleukin-2 - metabolism | Signal Transduction | Humans | STAT Transcription Factors - metabolism | STAT4 Transcription Factor - metabolism | MicroRNAs - metabolism | Th2 Cells - cytology | Th2 Cells - immunology | STAT6 Transcription Factor - metabolism | RNA, Messenger - metabolism | Th1 Cells - immunology | Th2 Cells - metabolism | Disease Progression | Lymphoma, T-Cell, Cutaneous - metabolism | Lymphoma, T-Cell, Cutaneous - pathology | Th1 Cells - metabolism | Janus Kinase 1 - metabolism | STAT5 Transcription Factor - metabolism | Interleukin-15 - metabolism | Janus Kinase 2 - metabolism | STAT4 Transcription Factor - genetics | Th1 Cells - cytology | Interleukin-7 - metabolism | STAT3 Transcription Factor - metabolism | Index Medicus
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8. Full Text Early B-cell factor 1 (EBF1) is critical for transcriptional control of SLAMF1 gene in human B cells
by Schwartz, Anton M and Putlyaeva, Lidia V and Covich, Milica and Klepikova, Anna V and Akulich, Kseniya A and Vorontsov, Ilya E and Korneev, Kirill V and Dmitriev, Sergey E and Polanovsky, Oleg L and Sidorenko, Svetlana P and Kulakovskiy, Ivan V and Kuprash, Dmitry V
Biochimica et biophysica acta. Gene regulatory mechanisms, ISSN 1874-9399, 10/2016, Volume 1859, Issue 10, pp. 1259 - 1268
EBF1 | B cell | RNA isoform | Trascription factor | SLAMF | Transcriptional regulation | Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | Humans | Interferon Regulatory Factors - metabolism | NF-kappa B - metabolism | Sp1 Transcription Factor - metabolism | STAT6 Transcription Factor - genetics | Luciferases - genetics | Signaling Lymphocytic Activation Molecule Family Member 1 - genetics | Signaling Lymphocytic Activation Molecule Family Member 1 - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | HEK293 Cells | Trans-Activators - genetics | Transcription, Genetic | Nuclear Proteins - genetics | Binding Sites | B-Lymphocytes - metabolism | Genes, Reporter | Proto-Oncogene Proteins - metabolism | Promoter Regions, Genetic | B-Lymphocytes - cytology | Basic Helix-Loop-Helix Transcription Factors - genetics | Signal Transduction | Gene Expression Regulation | Interferon Regulatory Factors - genetics | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | STAT6 Transcription Factor - metabolism | Transcription Factors - genetics | Transcription Factors - metabolism | Enhancer Elements, Genetic | NF-kappa B - genetics | Sp1 Transcription Factor - genetics | Cell Line, Tumor | Protein Binding | Trans-Activators - metabolism | Mutation | Primary Cell Culture | Autoimmunity | Promoters (Genetics) | Leukemia | Genes | Genetic aspects | Genetic transcription | B cells | DNA binding proteins | Cell differentiation
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9. Full Text Helper T cell IL-2 production is limited by negative feedback and STAT-dependent cytokine signals
by Villarino, Alejandro V and Tato, Cristina M and Stumhofer, Jason S and Yao, Zhengju and Cui, Yongzhi K and Hennighausen, Lothar and O'Shea, John J and Hunter, Christopher A
The Journal of experimental medicine, ISSN 0022-1007, 01/2007, Volume 204, Issue 1, pp. 65 - 71
Immunology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Ovalbumin - immunology | STAT Transcription Factors - metabolism | STAT4 Transcription Factor - metabolism | STAT6 Transcription Factor - genetics | Th2 Cells - immunology | Th1 Cells - immunology | STAT Transcription Factors - deficiency | STAT5 Transcription Factor - genetics | STAT5 Transcription Factor - metabolism | Feedback | T-Lymphocytes, Helper-Inducer - immunology | STAT5 Transcription Factor - deficiency | Immunization | Cytokines - metabolism | Signal Transduction | Mice, Inbred C57BL | Mice, Transgenic | STAT6 Transcription Factor - metabolism | Receptors, Antigen, T-Cell, alpha-beta - genetics | Mice, Knockout | STAT Transcription Factors - genetics | Animals | STAT6 Transcription Factor - deficiency | Interleukin-2 - pharmacology | STAT4 Transcription Factor - genetics | Mice | Receptors, Antigen, T-Cell, alpha-beta - metabolism | Interleukin-2 - biosynthesis | STAT4 Transcription Factor - deficiency | Index Medicus | Brief Definitive Reports
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