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Journal of the American College of Cardiology, ISSN 0735-1097, 07/2015, Volume 66, Issue 3, pp. 261 - 272
Journal Article
JOURNAL OF GENERAL PHYSIOLOGY, ISSN 0022-1295, 05/2019, Volume 151, Issue 5, pp. 635 - 644
Shifts in myosin heavy chain (MHC) isoforms in cardiac myocytes have been shown to alter cardiac muscle function not only in healthy developing hearts but also... 
ACTOMYOSIN ATPASE ACTIVITY | PHYSIOLOGY | ALPHA-MYOSIN | CARDIAC TROPONIN-T | POWER OUTPUT | GENE-EXPRESSION | FRANK-STARLING MECHANISM | STRETCH ACTIVATION | CA2+ ACTIVATION | ISOFORM EXPRESSION | MYOSIN HEAVY-CHAIN | Calcium | Cardiac muscle | Contractility | Cardiomyocytes | Myocytes | Muscle contraction | Coronary artery disease | Filaments | Myosin | Isoforms | Myocardium | Skin | Heart diseases | Age | 501
Journal Article
Acta Physiologica, ISSN 1748-1708, 11/2019, Volume 227, Issue 3, pp. e13336 - n/a
Aim mTORC1 is regarded as an important key regulator of protein synthesis and hypertrophy following mechanical stimuli in skeletal muscle. However, as... 
EC‐coupling | stretch | tension | eccentric | mTORC1 | mechanotransduction | TARGET | PHYSIOLOGY | TITIN | STIMULI | PROTEIN-SYNTHESIS | HYPERTROPHY | RESISTANCE EXERCISE | PATHWAY | INCREASE | MECHANICAL ACTIVATION | EC-coupling | Musculoskeletal system | Signal transduction | Myosin | Protein biosynthesis | Mechanical stimuli | Muscle contraction | Skeletal muscle | Hypertrophy
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 05/2012, Volume 302, Issue 9, pp. L846 - L856
Ghosh MC, Makena PS, Gorantla V, Sinclair SE, Waters CM. CXCR4 regulates migration of lung alveolar epithelial cells through activation of Rac1 and matrix... 
Acute respiratory distress syndrome | Acute lung injury | Epithelial repair | PHYSIOLOGY | STRETCH | CHEMOKINE RECEPTOR CXCR4 | INJURY | epithelial repair | acute lung injury | IN-VITRO | KERATINOCYTE GROWTH-FACTOR | RESPIRATORY SYSTEM | acute respiratory distress syndrome | II CELLS | TISSUE INHIBITOR | EXTRACELLULAR-MATRIX | PRO-MMP-2 ACTIVATION | PULMONARY-FIBROSIS | Chemokine CXCL12 - physiology | Up-Regulation | Epithelial Cells - metabolism | Heterocyclic Compounds - pharmacology | Male | Chemokine CXCL12 - secretion | Gene Knockdown Techniques | Epithelial Cells - physiology | Tissue Inhibitor of Metalloproteinase-3 - metabolism | Receptors, CXCR4 - genetics | Acute Lung Injury - metabolism | Tissue Inhibitor of Metalloproteinase-3 - genetics | Actin Cytoskeleton - metabolism | Pulmonary Alveoli - pathology | Matrix Metalloproteinase 2 - metabolism | Receptors, CXCR4 - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Matrix Metalloproteinase 14 - metabolism | Acute Lung Injury - pathology | Rats, Sprague-Dawley | Receptors, CXCR4 - metabolism | Receptors, CXCR4 - physiology | Animals | Matrix Metalloproteinase 14 - genetics | rac1 GTP-Binding Protein - antagonists & inhibitors | Mice | Enzyme Activation | Primary Cell Culture | rac1 GTP-Binding Protein - metabolism | Cell Movement | Physiological aspects | Lungs | Epithelial cells | Health aspects | Metalloproteins
Journal Article
Critical Care Medicine, ISSN 0090-3493, 11/2019, Volume 47, Issue 11, pp. e911 - e918
OBJECTIVES:Mechanical ventilation can cause ventilator-induced brain injury via afferent vagal signaling and hippocampal neurotransmitter imbalances. The... 
ventilator-induced | purinergic receptors | mechanical ventilation | RECEPTOR | NEURODEGENERATION | lidocaine | INFLUX | EPITHELIAL-CELLS | TRPV4 | INFLAMMATION | PANNEXIN CHANNELS | ATP RELEASE | MECHANICAL STRETCH | VAGAL | CRITICAL CARE MEDICINE | Brain | Medical research | Usage | Medicine, Experimental | Ventilation | Injuries | Risk factors
Journal Article