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1. Full Text Slc26a4 -insufficiency causes fluctuating hearing loss and stria vascularis dysfunction
by Ito, Taku and Li, Xiangming and Kurima, Kiyoto and Choi, Byung Yoon and Wangemann, Philine and Griffith, Andrew J
Neurobiology of disease, ISSN 0969-9961, 2014, Volume 66, pp. 53 - 65
Neurology | Inducible | Fluctuation | Doxycycline | Transgenic | Stria vascularis | Conditional | Endocochlear potential | SLC26A4 | Vestibular aqueduct | Hypomorph | Hearing loss | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Immunohistochemistry | Macrophages - physiology | Stria Vascularis - pathology | Gene Expression | Hair Cells, Auditory - pathology | Hearing Loss - physiopathology | Macrophages - pathology | Cochlea - physiopathology | Mice, Transgenic | Hearing Loss - pathology | Anion Transport Proteins - metabolism | Auditory Threshold | Stria Vascularis - physiology | Animals | Evoked Potentials, Auditory, Brain Stem | Hair Cells, Auditory - physiology | Membrane Potentials | Cochlea - pathology | Mice | Otoacoustic Emissions, Spontaneous | Real-Time Polymerase Chain Reaction | Anion Transport Proteins - genetics | Analysis | Genetic engineering | Index Medicus | endocochlear potential | inducible | vestibular aqueduct | transgenic | hearing loss | hypomorph | doxycycline | fluctuation | stria vascularis
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2. Full Text Deafness and Stria Vascularis Defects in S1P2 Receptor-null Mice
by Mari Kono and Inna A. Belyantseva and Athanasia Skoura and Gregory I. Frolenkov and Matthew F. Starost and Jennifer L. Dreier and Darcy Lidington and Steffen-Sebastian Bolz and Thomas B. Friedman and Timothy Hla and Richard L. Proia
The Journal of biological chemistry, ISSN 0021-9258, 04/2007, Volume 282, Issue 14, pp. 10690 - 10696
Epithelium - blood supply | Lysophospholipids - metabolism | Stria Vascularis - pathology | Epithelium - pathology | Deafness - genetics | Epithelial Cells - metabolism | Stria Vascularis - metabolism | Receptors, Lysosphingolipid - deficiency | Deafness - physiopathology | Epithelial Cells - pathology | Deafness - pathology | Sphingosine - analogs & derivatives | Animals | Time Factors | Deafness - metabolism | Mice, Mutant Strains | Evoked Potentials, Auditory, Brain Stem - genetics | Mice | Sphingosine - metabolism | Receptors, Lysosphingolipid - metabolism | Index Medicus
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3. Full Text Development of the stria vascularis and potassium regulation in the human fetal cochlea: Insights into hereditary sensorineural hearing loss
by Locher, Heiko and de Groot, John C.M.J and van Iperen, Liesbeth and Huisman, Margriet A and Frijns, Johan H.M and Chuva de Sousa Lopes, Susana M
Developmental neurobiology (Hoboken, N.J.), ISSN 1932-8451, 11/2015, Volume 75, Issue 11, pp. 1219 - 1240
sensorineural hearing loss | development | melanocytes | human | stria vascularis | Human | Development | Melanocytes | Stria vascularis | Sensorineural hearing loss | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Immunohistochemistry | Potassium - metabolism | Cochlea - embryology | Hearing Loss, Sensorineural - metabolism | Microphthalmia-Associated Transcription Factor - metabolism | Connexin 43 - metabolism | SOXE Transcription Factors - metabolism | Humans | KCNQ1 Potassium Channel - metabolism | Connexin 30 | Cochlea - cytology | Cochlea - physiology | Melanocytes - physiology | Proto-Oncogene Proteins c-kit - metabolism | Connexins - metabolism | Connexin 26 | Microscopy, Confocal | Sodium-Potassium-Exchanging ATPase - metabolism | Stria Vascularis - physiology | Stria Vascularis - cytology | Image Processing, Computer-Assisted | Melanocytes - cytology | Potassium Channels, Inwardly Rectifying - metabolism | Cell Movement | Proteins | Embryonic development | Genetic disorders | Hearing loss | Index Medicus
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4. Full Text Estrogen-Related Receptor β/NR3B2 Controls Epithelial Cell Fate and Endolymph Production by the Stria Vascularis
by Chen, Jichao and Nathans, Jeremy
Developmental cell, ISSN 1534-5807, 2007, Volume 13, Issue 3, pp. 325 - 337
DEVBIO | Fundamental and applied biological sciences. Psychology | Biological and medical sciences | Cell physiology | Molecular and cellular biology | Cell differentiation, maturation, development, hematopoiesis | Immunohistochemistry | Gene Expression | Epithelial Cells - metabolism | Stria Vascularis - metabolism | Ear, Inner - cytology | Deafness - etiology | Mice, Knockout | Deafness - pathology | Estrogen Receptor beta - metabolism | Epithelial Cells - physiology | Ear, Inner - metabolism | Endolymph - metabolism | Animals | In Situ Hybridization | Stria Vascularis - cytology | Models, Biological | Alleles | Mosaicism | Cell Differentiation | Mice | Index Medicus
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5. Full Text Differential localizations of the myo-inositol transporters HMIT and SMIT1 in the cochlear stria vascularis
by Edamatsu, Midori and Kondo, Yasuhiro and Ando, Motonori
Neuroscience letters, ISSN 0304-3940, 05/2018, Volume 674, pp. 88 - 93
Osmoregulation | Endolymph | Rat (Brown Norway) | Inner ear | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Glucose Transport Proteins, Facilitative - metabolism | Animals | Stria Vascularis - cytology | Stria Vascularis - metabolism | Rats | Symporters - metabolism | Protein Transport | Immunohistochemistry | Inositol | Index Medicus
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6. Full Text TBX1 is required for normal stria vascularis and semicircular canal development
by Tian, Cong and Johnson, Kenneth R
Developmental biology, ISSN 0012-1606, 01/2020, Volume 457, Issue 1, pp. 91 - 103
TBX1 | Inner ear development | Stria vascularis | Semicircular canal | Mouse mutation | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Index Medicus
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7. Full Text Progressive irreversible hearing loss is caused by stria vascularis degeneration in an Slc26a4 -insufficient mouse model of large vestibular aqueduct syndrome
by Ito, T and Nishio, A and Wangemann, P and Griffith, A.J
Neuroscience, ISSN 0306-4522, 2015, Volume 310, pp. 188 - 197
Neurology | deafness | doxycycline | hypomorphic | SLC26A4 | fluctuation | stria vascularis | Deafness | Stria vascularis | Hypomorphic | Fluctuation | Doxycycline | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Stria Vascularis - pathology | Hair Cells, Auditory - pathology | KCNQ1 Potassium Channel - genetics | Vestibular Diseases - complications | Hearing Loss - etiology | KCNQ1 Potassium Channel - metabolism | Sulfate Transporters | Cell Death - genetics | Anion Transport Proteins - deficiency | Disease Models, Animal | Hearing Loss - physiopathology | Acoustic Stimulation | Gene Expression Regulation - genetics | Genotype | Mice, Transgenic | Potassium Channels, Inwardly Rectifying - genetics | Mutation - genetics | Hearing Loss - genetics | Animals | Vestibular Aqueduct - pathology | Evoked Potentials, Auditory, Brain Stem - genetics | Auditory Threshold - physiology | Mice | Vestibular Diseases - genetics | Potassium Channels, Inwardly Rectifying - metabolism | Anion Transport Proteins - genetics | Evoked Potentials, Auditory, Brain Stem - physiology | Physiological aspects | Hearing loss | Index Medicus | DFNB4 | presbycusis | pendrin
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8. Full Text Isolation, cultivation, and characterization of primary bovine cochlear pericytes: A new in vitro model of stria vascularis
by Giurdanella, Giovanni and Montalbano, Giuseppe and Gennuso, Florinda and Brancati, Serena and Lo Furno, Debora and Augello, Antonio and Bucolo, Claudio and Drago, Filippo and Salomone, Salvatore
Journal of cellular physiology, ISSN 0021-9541, 03/2019, Volume 234, Issue 3, pp. 1978 - 1986
gentamicin | cisplatin | ototoxicity | primary culture | stria vascularis | pericytes | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Pericytes - drug effects | Stria Vascularis - drug effects | Pericytes - cytology | Actins - metabolism | Gentamicins - toxicity | Cell Culture Techniques - methods | Cisplatin - toxicity | Stria Vascularis - cytology | Cattle | Cochlea - metabolism | Culture Media | Receptor, Platelet-Derived Growth Factor beta - metabolism | Biomarkers - metabolism | Drug Evaluation, Preclinical - methods | Stria Vascularis - metabolism | Cell Survival | Pericytes - metabolism | Proteoglycans - metabolism | Ototoxicity - pathology | Antigens - metabolism | Cochlea - cytology | Ototoxicity - metabolism | Animals | Cochlea - drug effects | Models, Biological | In Vitro Techniques | Ototoxicity - etiology | Physiological aspects | Aminoglycosides | Platelet-derived growth factor | Analysis | Von Willebrand factor | Sensitivity analysis | Cultivation | Gentamicin | Muscles | Retina | Smooth muscle | Drug development | Neuronal-glial interactions | Cisplatin | Endothelial cells | Lipopolysaccharides | Biomedical materials | Actin | Cochlea | Pericytes | Stria vascularis | Biocompatibility | In vivo methods and tests | Growth factors | Ototoxicity | Index Medicus
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9. Full Text Noncoding microdeletion in mouse HGF disrupts neural crest migration into the stria vascularis, reduces the endocochlear potential, and suggests the neuropathology for human nonsyndromic deafness DFNB39
by Morell, Robert J and Olszewski, Rafal and Tona, Risa and Leitess, Samuel and Wafa, Talah T and Taukulis, Ian and Schultz, Julie M and Thomason, Elizabeth J and Richards, Keri and Whitley, Brittany N and Hill, Connor and Saunders, Thomas and Starost, Matthew F and Fitzgerald, Tracy and Wilson, Elizabeth and Ohyama, Takahiro and Friedman, Thomas B and Hoa, Michael
The Journal of neuroscience, ISSN 0270-6474, 04/2020, Volume 40, Issue 15, pp. 2976 - 2992
Deafness | Stria vascularis | DFNB39 | Hepatocyte growth factor | HGF | Neural crest cells | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Cell proliferation | Immunohistochemistry | Leukocyte migration | Brain stem | Homeostasis | Inner ear | c-Met protein | Rodents | Growth factors | Cell survival | Wound healing | Abnormalities | Neural crest | Hearing loss | Homozygotes | Ear | Phenotyping | Auditory system | Cochlea | Mutation | Auditory defects | Cell migration | Index Medicus | neural crest cells | s | hepatocyte growth factor | deafness | stria vascularis
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10. Full Text Molecular architecture of the stria vascularis membrane transport system, which is essential for physiological functions of the mammalian cochlea
by Uetsuka, Satoru and Ogata, Genki and Nagamori, Shushi and Isozumi, Noriyoshi and Nin, Fumiaki and Yoshida, Takamasa and Komune, Shizuo and Kitahara, Tadashi and Kikkawa, Yoshiaki and Inohara, Hidenori and Kanai, Yoshikatsu and Hibino, Hiroshi
The European journal of neuroscience, ISSN 0953-816X, 08/2015, Volume 42, Issue 3, pp. 1984 - 2002
mass spectrometry | proteomics | rat | ion channel | transporter | Rat | Ion channel | Mass spectrometry | Transporter | Proteomics | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Deafness - genetics | Stria Vascularis - metabolism | Humans | Databases, Genetic | Molecular Sequence Data | Rats | Deafness - physiopathology | Male | Animals | Membrane Transport Proteins - genetics | Membrane Transport Proteins - metabolism | Mice | Physiological aspects | Deafness | Liquid chromatography | Analysis | Membrane proteins | Index Medicus
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