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PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 9, p. e0162460
Background Quantitative susceptibility mapping (QSM) and R2* relaxation rate mapping have demonstrated increased iron deposition in the substantia nigra of... 
DIAGNOSIS | DEPOSITION | NUCLEUS | MULTIDISCIPLINARY SCIENCES | MOVEMENT | VALIDATION | QSM | RATING-SCALE | SUBSTANTIA-NIGRA | PROGRESSION | BRAIN IRON | Red Nucleus - diagnostic imaging | Thalamus - metabolism | Substantia Nigra - pathology | Humans | Middle Aged | Image Interpretation, Computer-Assisted | Male | Parkinson Disease - drug therapy | Thalamus - pathology | Substantia Nigra - metabolism | Case-Control Studies | Globus Pallidus - diagnostic imaging | Antiparkinson Agents - therapeutic use | Globus Pallidus - metabolism | Female | Parkinson Disease - metabolism | Globus Pallidus - pathology | Severity of Illness Index | Parkinson Disease - pathology | Parkinson Disease - diagnostic imaging | Levodopa - therapeutic use | Thalamus - diagnostic imaging | Brain Mapping - methods | Iron - metabolism | Thalamus - drug effects | Globus Pallidus - drug effects | Magnetic Resonance Imaging | Red Nucleus - metabolism | Substantia Nigra - drug effects | Red Nucleus - drug effects | Substantia Nigra - diagnostic imaging | Aged | Red Nucleus - pathology | Care and treatment | Usage | Diagnostic imaging | Parkinson's disease | Multiple sclerosis | Substantia nigra | Parkinsons disease | Iron | Mapping | Health physics | Substantia grisea | Nuclei | Magnetic susceptibility | Ethics | Maps | Thalamus | Levodopa | Age | Movement disorders | Dopamine receptors | Dopamine | Neurodegenerative diseases | Globus pallidus | Histology | Disease control | Patients | Neurology | Sensitivity | Brain research | Magnetic resonance imaging | Echo surveys | Magnetic permeability | Informed consent | Gene mapping
Journal Article
Glia, ISSN 1098-1136, 2007, Volume 55, Issue 5, pp. 453 - 462
Inflammation is implicated in the progressive nature of neurodegenerative diseases, such as Parkinson's disease, but the mechanisms are poorly understood. A... 
neurodegeneration | TNFα | substantia nigra | neuroinflammation | microglia | LPS | Neuroinflammation | Neurodegeneration | Substantia nigra | Microglia | TNF alpha | MICROGLIAL ACTIVATION | TNF-ALPHA TRANSPORT | TRIGGERS | BACTERIAL-ENDOTOXIN EXPOSURE | DOPAMINE NEURON LOSS | SUBSTANTIA-NIGRA | NEUROSCIENCES | TUMOR-NECROSIS-FACTOR | LIPOPOLYSACCHARIDE-INDUCED NEUROTOXICITY | RAT-BRAIN | INNATE IMMUNITY | Inflammation - chemically induced | Tumor Necrosis Factor-alpha - metabolism | Neurodegenerative Diseases - etiology | Lipopolysaccharides - administration & dosage | Tumor Necrosis Factor-alpha - genetics | Cell Death - immunology | RNA, Messenger - analysis | Substantia Nigra - immunology | Male | Neurons - cytology | Substantia Nigra - metabolism | Neurodegenerative Diseases - immunology | Lipopolysaccharides - immunology | Substantia Nigra - cytology | Brain - metabolism | Inflammation - complications | Microglia - immunology | Time Factors | Inflammation Mediators - metabolism | Neurons - metabolism | Parkinson Disease - metabolism | Dopamine - metabolism | Disease Models, Animal | Inflammation Mediators - immunology | Brain - cytology | Parkinson Disease - immunology | Liver - metabolism | Mice, Inbred C57BL | Injections, Intraperitoneal | Neurodegenerative Diseases - metabolism | Mice, Inbred Strains | Mice, Knockout | Animals | Mice | Brain - immunology
Journal Article
Science (American Association for the Advancement of Science), ISSN 1095-9203, 2017, Volume 357, Issue 6357, pp. 1255 - 1261
Mitochondrial and lysosomal dysfunction have been implicated in substantia nigra dopaminergic neurodegeneration in Parkinson's disease (PD), but how these... 
GLUCOCEREBROSIDASE | PACEMAKING | DJ-1 | ALPHA-SYNUCLEIN | SUBSTANTIA-NIGRA | MULTIDISCIPLINARY SCIENCES | Mitochondria - enzymology | Mesencephalon - metabolism | Humans | Protein Deglycase DJ-1 - genetics | Substantia Nigra - metabolism | Melanins - metabolism | Glucosylceramidase - deficiency | Lysosomes - metabolism | Dopaminergic Neurons - metabolism | Tacrolimus - pharmacology | Parkinson Disease - metabolism | Dopamine - metabolism | Disease Models, Animal | Substantia Nigra - enzymology | Cell Line | Calcineurin Inhibitors - pharmacology | Oxidation-Reduction | Mesencephalon - enzymology | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - drug effects | Parkinson Disease - genetics | Mice, Knockout | Animals | Parkinson Disease - enzymology | Mice | Oxidative Stress - drug effects | alpha-Synuclein - metabolism | Oxidation-reduction reaction | Development and progression | Mitochondria | Dopamine | Parkinson's disease | Health aspects | Brain | Energy metabolism | Animal models | Target recognition | Mesencephalon | Pathogenesis | Substantia nigra | Parkinsons disease | Lysosomes | Synuclein | Accumulation | Pathways | Enzymatic activity | Neurodegeneration | Rodents | Oxidation | Degeneration | Species | Movement disorders | Dopamine receptors | Neurodegenerative diseases | Neurons | Medical treatment | Metabolism | Patients | Glucosylceramidase | Index Medicus
Journal Article
Brain (London, England : 1878), ISSN 1460-2156, 2013, Volume 136, Issue 11, pp. 3242 - 3251
Journal Article
Neuroscience, ISSN 0306-4522, 2008, Volume 152, Issue 4, pp. 1024 - 1031
Abstract Midbrain dopamine neurons in the ventral tegmental area, substantia nigra and retrorubral field play key roles in reward processing, learning and... 
Neurology | reward | midbrain | basal ganglia | GAD | VGluT2 | TYROSINE-HYDROXYLASE | NEUROSCIENCES | IN-VITRO | INHIBITION | GABA-CONTAINING NEURONS | INTERFASCICULAR NUCLEUS | SYNAPSES | BRAIN | Tyrosine 3-Monooxygenase - metabolism | gamma-Aminobutyric Acid - metabolism | Vesicular Glutamate Transport Protein 2 - metabolism | Rats | Glutamate Decarboxylase - genetics | Male | Ventral Tegmental Area - cytology | RNA, Messenger - metabolism | Rats, Sprague-Dawley | Substantia Nigra - cytology | Stereotaxic Techniques | Tyrosine 3-Monooxygenase - genetics | Mesencephalon - anatomy & histology | Animals | Cell Count - methods | Glutamate Decarboxylase - metabolism | Glutamic Acid - metabolism | Neurons - metabolism | Dopamine - metabolism | Tyrosine | GABA | Neurosciences | Glutamate decarboxylase | Glutamate | Neurons | SN, substantia nigra | lSN, lateral substantia nigra | VTA, ventral tegmental area | Neuroanatomy | PBS-T, phosphate-buffered saline containing 0.1% Tween 20 | VGluT, vesicular glutamate transporter | HB, hybridization buffer | PN, paranigral nucleus | SNpc, substantia nigra pars compacta | PBS, phosphate-buffered saline | PBP, parabrachial nucleus | IFN, interfascicular subnucleus of the ventral tegmental area | TH, tyrosine hydroxylase | GAD, glutamic acid decarboxylase | vSN, ventral substantia nigra | RLi, rostral linear nucleus | RRF, retrorubral field | RT-PCR, reverse transcriptase–polymerase chain reaction | IP, interpeduncular nucleus
Journal Article
Journal Article
Neurobiology of disease, ISSN 0969-9961, 2015, Volume 82, pp. 185 - 199
Journal Article
Journal Article