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Oncogene, ISSN 0950-9232, 04/2009, Volume 28, Issue 14, pp. 1653 - 1668
The mammalian SWI/SNF complexes mediate ATP-dependent chromatin remodeling processes that are critical for differentiation and proliferation. Not surprisingly,... 
BAF47 | BRM | BRG1 | Tumor suppressor | Chromatin remodeling | STRAND BREAK REPAIR | TRANSCRIPTION IN-VIVO | BIOCHEMISTRY & MOLECULAR BIOLOGY | HUMAN LUNG-CANCER | chromatin remodeling | TUMOR-SUPPRESSOR GENE | GLUCOCORTICOID-INDUCED APOPTOSIS | tumor suppressor | CELL-CYCLE ARREST | SACCHAROMYCES-CEREVISIAE | CELL BIOLOGY | CHROMATIN-REMODELING COMPLEX | ONCOLOGY | GENETICS & HEREDITY | RETINOIC ACID RECEPTOR | SWI-SNF COMPLEX | Immune System - physiology | Transcription Factors - chemistry | Humans | Loss of Heterozygosity | SMARCB1 Protein | Neoplasms - genetics | Cell Differentiation | Chromosomal Proteins, Non-Histone - physiology | Adenosine Triphosphate - physiology | Nuclear Proteins - genetics | DNA Helicases - genetics | DNA Helicases - chemistry | DNA-Binding Proteins - physiology | Transcription Factors - physiology | Gene Expression Regulation | Gene Silencing | Chromatin Assembly and Disassembly | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Nuclear Proteins - chemistry | Cell Adhesion | Chromosomal Proteins, Non-Histone - genetics | Tumor Suppressor Proteins - physiology | Animals | DNA Repair | Nuclear Proteins - physiology | DNA Helicases - physiology | Physiological aspects | Nucleosomes | Tumor suppressor genes | Genetic aspects | Research | Structure | DNA repair | Cancer | Chromatin | Genes
Journal Article
Nature Genetics, ISSN 1061-4036, 11/2013, Volume 45, Issue 11, pp. 1392 - 1403
Journal Article
Nature Genetics, ISSN 1061-4036, 11/2017, Volume 49, Issue 11, pp. 1613 - 1623
Perturbations to mammalian SWI/SNF (mSWI/SNF or BAF) complexes contribute to more than 20% of human cancers, with driving roles first identified in malignant... 
CHROMATIN REMODELING COMPLEX | DISRUPTION | SUPER-ENHANCERS | SUPPRESSOR GENE | SWI/SNF COMPLEXES | CELL IDENTITY | GENETICS & HEREDITY | MUTATIONS | POLYCOMB | GENOME | MALIGNANT RHABDOID TUMORS | Chromatin - metabolism | Humans | Gene Expression Regulation, Neoplastic | SMARCB1 Protein - genetics | Polycomb-Group Proteins - metabolism | Rhabdoid Tumor - pathology | Carcinogenesis - metabolism | Genetic Complementation Test | Rhabdoid Tumor - metabolism | SMARCB1 Protein - deficiency | Sarcoma - genetics | Chromatin - chemistry | Promoter Regions, Genetic | Chromosomal Proteins, Non-Histone - metabolism | Carcinogenesis - genetics | Chromatin Assembly and Disassembly | Transcription Factors - genetics | Sarcoma - pathology | Chromosomal Proteins, Non-Histone - genetics | Carcinogenesis - pathology | Sarcoma - metabolism | Transcription Factors - metabolism | Rhabdoid Tumor - genetics | Enhancer Elements, Genetic | Polycomb-Group Proteins - genetics | Cell Line, Tumor | Cellular proteins | Genetic aspects | Gene expression | Health aspects | Carcinogenesis | Malignant rhabdoid tumor | Pediatrics | Chromatin | Deactivation | Sarcoma | Ontology | Activation | Genomes | Tumor cell lines | Mammals | Inactivation | Promoters | Proteins | Consortia | Polycomb group proteins | Enhancers | Collaboration | Cell lines | Stem cells | Epigenetics | Tumorigenesis | Bioinformatics | Cancer
Journal Article
Nature Cell Biology, ISSN 1465-7392, 12/2018, Volume 20, Issue 12, pp. 1410 - 1420
Journal Article